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1 -operated mice in the presence or absence of intestinal flora.
2 ut did not affect Escherichia coli of normal intestinal flora.
3 base, queuine, obtained from the diet or the intestinal flora.
4 titutive TLR signals are driven by commensal intestinal flora.
5 nt host immunity or the emergence of harmful intestinal flora.
6 inflammation requires the presence of viable intestinal flora.
7 art by a dysregulated immune response to the intestinal flora.
8 stinal epithelium serves as a barrier to the intestinal flora.
9 tion) on antibiotic resistance in the normal intestinal flora.
10 a short chain fatty acid produced by normal intestinal flora.
11 duced by nonpathogenic E. coli of the normal intestinal flora.
12 evelopment occurring independently of viable intestinal flora.
13 min K2 and lithocholic acid, a by-product of intestinal flora, activate pregnane X receptor (PXR) and
14 metabolite of cholate produced by the normal intestinal flora, also induced germination of C. diffici
15 low skatole deposition in wild boars such as intestinal flora and anatomy, dietary composition, housi
16 cognized gut-brain axis and an impact of the intestinal flora and meningeal IL-17(+) gammadelta T cel
17 the abundance of a group of bacteria in the intestinal flora and relapse/progression of disease afte
18 Recently, the bacterial diversity of the intestinal flora and the diversity of various environmen
20 reported previously that alterations in the intestinal flora are associated with GVHD, bacteremia, a
21 llo-HSCT, the diversity and stability of the intestinal flora are disrupted, resulting in domination
22 Enterococci are members of the healthy human intestinal flora, but are also leading causes of highly
25 ects, we hypothesized that components of the intestinal flora could be associated with relapse after
26 lude that specific members of the commensal, intestinal flora drive GALT development through a specif
27 ial therapy, which alters the balance of the intestinal flora, enabling C. difficile to proliferate i
28 ted in maintenance of the composition of the intestinal flora, even after a moderate gluten challenge
29 for risk assessment and manipulation of the intestinal flora for prevention and treatment of radiati
30 to be functionally hyporesponsive to normal intestinal flora, human intestinal epithelial cells can
32 here is a difference in response towards the intestinal flora in dectin-1 deficient macrophages, duri
33 rs triggering a loss of tolerance for normal intestinal flora in genetically susceptible individuals.
34 onal significance, since manipulation of the intestinal flora in individuals with congenital biopteri
36 ther, these results support a vital role for intestinal flora in the maturation of intestinal barrier
38 a support the hypothesis that the indigenous intestinal flora inhibit growth of VRE and suggest that
42 teroides fragilis, a component of the normal intestinal flora, is an obligate anaerobe capable of lon
46 erococcus species combined with conventional intestinal flora obtained from the gastrointestinal trac
49 have investigated the influence of maternal intestinal flora on wheezing and eczema in early childho
51 s, treatment with a probiotic (to modify the intestinal flora) or anti-TNF antibodies (to inhibit TNF
53 w that antibiotic-induced alterations in the intestinal flora reduce ischemic brain injury in mice, a
54 been demonstrated to alter the pre-existing intestinal flora so as to provide an advantage to the ho
55 athogenic strains from within the host's own intestinal flora, strain sharing between household membe
56 estion or to certain metabolites produced by intestinal flora that degraded alcohol was investigated.
60 in integrating signals from the diet and the intestinal flora to modulate ongoing inflammation in the
61 sults suggest that the susceptibility of the intestinal flora to the Shiga toxin phage could exert ei
62 , these junctions become loose, enabling the intestinal flora to trigger a constitutive activation of
63 n vivo effect of diosgenin on the density of intestinal flora was examined in a murine model of food
64 ce an aberrant immune response to indigenous intestinal flora, which might favor IBD development.
65 ates the decolonization of oxalate degrading intestinal flora with a higher risk of calcium oxalate s
66 nsplantation (FMT) re-establishes a balanced intestinal flora with resultant cure of recurrent Clostr
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