ライフサイエンスコーパス: intestinal infection

1 x mutual regulation of both cytokines during intestinal infection.

2 al MPs in ILC3-mediated host defence against intestinal infection.

3 s were neutropenia, febrile neutropenia, and intestinal infection.

4 ost frequently identified protozoan cause of intestinal infection.

5 of intranasally infected animals in clearing intestinal infection.

6 efficacy against asymptomatic E. histolytica intestinal infection.

7 e is known about the mechanisms that control intestinal infection.

8 ed in vitro and in the infant mouse model of intestinal infection.

9 for the mucosal influx of neutrophils during intestinal infection.

10 a coli often develop septicemia secondary to intestinal infection.

11 ntified by a screen for genes induced during intestinal infection.

12 promotes health and reduces the incidence of intestinal infections.

13 istance is key to the prevention and cure of intestinal infections.

14 Four MAbs given therapeutically each reduced intestinal infection 34.4 to 42.2% compared to isotype-m

15 E. coli strains causing extra-intestinal infections accumulate hotspot mutations at th

16 t public health concerns by causing an acute intestinal infection afflicting millions of people each

17 1.09; 95% CI: 1.02-1.17), and ED visits for intestinal infection and heat waves defined by average t

18 mportant role for TLR11 in preventing murine intestinal infection and modulating antigen transportati

19 Intestinal infection and the associated diarrhea are sig

20 be elucidated, the likelihood of TV causing intestinal infection and the availability of a tissue cu

21 is essential for understanding and treating intestinal infections and inflammatory diseases.

22 tic toxin interactions during C. perfringens intestinal infections and support a possible role for CP

23 e treatment of inflammatory bowl disease and intestinal infections and to new immunization strategies

24 me an antiparasite effect of IL-22 during an intestinal infection, and they suggest that IL-17A and I

25 RPOSE OF REVIEW: Clostridium difficile is an intestinal infection associated with antibiotic use, com

26 amebiasis can develop in some children after intestinal infection, but protective immunity may be tra

27 MAbs or combinations of two MAbs and reduced intestinal infection by 86 to 93%.

28 CD98 in the innate host defense response to intestinal infection by attaching and effacing (A/E) pat

29 effects of leptin receptor polymorphisms on intestinal infection by E. histolytica.

30 tic uremic syndrome (HUS) is associated with intestinal infection by enterohemorrhagic Escherichia co

31 Primary intestinal infection by T. spiralis induces mastocytosis

32 We found increased susceptibility to intestinal infection by this parasite associated with an

33 Intestinal infections by attaching and effacing (A/E) ba

34 rome (HUS) is the life-threatenig sequela of intestinal infections by Shiga toxin (Stx)-producing Esc

35 Cholera is an acute intestinal infection caused by the bacterium Vibrio chol

36 reased lethality in response to C. difficile intestinal infection despite comparable levels of intest

37 which is sufficient to protect mice against intestinal infections from S. Typhimurium.

38 ns to pathogenesis, we utilized the model of intestinal infection in adult mice sensitive to the acti

39 cile is an important pathogen causing severe intestinal infections in humans and animals.

40 utic tools for the treatment of pathological intestinal infections in infants.

41 host susceptibility to intestinal and extra-intestinal infections, including those caused by viruses

42 strains known to cause intestinal and extra intestinal infections, induce reorganization of the acti

43 However, these intestinal infection-induced plasmablasts lack the CLA h

44 Intra-amniotic C.albicans caused intestinal infection, injury and inflammation.

45 mansoni (and rarely Schistosoma haematobium) intestinal infection is also not very common and is foun

46 Immunity to Entamoeba species intestinal infection is associated with the presence of

47 ortality in Africa, but host defense against intestinal infection is poorly understood and may depend

48 biological significance of this event during intestinal infection is unclear.

49 stressors, including antibiotic therapy and intestinal infections, is associated with multiple healt

50 ium subsp. paratuberculosis causes a chronic intestinal infection leading to a chronic wasting diseas

51 aerophilic bacterium associated with chronic intestinal infection leading to hepatitis and colonic an

52 ntibody-secreting cells in response to acute intestinal infection, likely helping target these cells

53 n inside macrophages, resulting in increased intestinal infection loads in DBA/2J mice.

54 Our data also suggest that intestinal infection might be initiated from the basolat

55 rulent than the wild-type strain in a murine intestinal infection model, suggesting that survival and

56 Using an adult mouse intestinal infection model, this study examines the cont

57 Using an adult mouse intestinal infection model, this study examines the cont

58 dine KDU731 results in a potent reduction in intestinal infection of immunocompromised mice.

59 racellular growth of L. monocytogenes during intestinal infection of mice.

60 In contrast, responses to intestinal infection or flagellin administration were un

61 or without concurrent Entamoeba histolytica intestinal infection or were infection free 1 year after

62 anisms by which they protect the host during intestinal infection remains poorly understood.

63 phocytes were important for the clearance of intestinal infection, since severe combined immunodefici

64 nferring protection against subsequent extra-intestinal infections, such as urinary tract infections.

65 N-beta, and Mx-1 in PPs and caused localized intestinal infection that was cleared in 10 d.

66 y of the antigen-specific T-cell response to intestinal infection, the prominence of microbial mechan

67 We found that resistance to intestinal infection was independent of lymphocyte activ

68 neutrophils, and T lymphocytes during human intestinal infection with Campylobacter.

69 ion from TH17 but not ILCs in the context of intestinal infection with Citrobacter rodentium, resulti

70 IL-22 and resulted in crippled responses to intestinal infection with Citrobacter rodentium.

71 tomycin-fed C57BL/6 mice were susceptible to intestinal infection with El Tor strains, which caused r

72 Establishment of intestinal infection with Entamoeba histolytica depends

73 titers of antibody and is protective against intestinal infection with ETEC.

74 In an independent IgE-inducing model, i.e., intestinal infection with H. polygyrus, we validated the

75 id and 17% triglycerides), as well as distal intestinal infection with Helicobacter hepaticus, influe

76 atf3 deficiency converted a normally chronic intestinal infection with Heligmosomoides polygyrus into

77 (KO) mice following sensitization to OVA or intestinal infection with Heligmosomoides polygyrus Spec

78 the infecting serotype, or who had previous intestinal infection with homologous poliovirus.

79 of increased fluid secretion in response to intestinal infection with invasive bacteria.

80 bolization with eggs of Schistosoma mansoni, intestinal infection with Nippostrongylus brasiliensis,

81 hila model of gut pathogenesis, we show that intestinal infection with Pseudomonas aeruginosa, a huma

82 Most cases are caused by an intestinal infection with Shiga toxin-producing strains

83 ve as a model for renal disease secondary to intestinal infection with STEC.

84 pecific IgG2c failed to protect rats against intestinal infection with T. spiralis.

85 Intestinal infection with the parasitic nematode, Trichi

86 During intestinal infection with the pathogen Salmonella Typhim

87 eless, T-bet(-/-) mice respond vigorously to intestinal infection with Trichinella spiralis, eliminat

88 naerobic Gram-positive bacterium that causes intestinal infections with symptoms ranging from mild di

89 th spread to respiratory tissues, or through intestinal infection, with spread to the liver and pancr

90 r, unlike wild-type mice, which resolved the intestinal infection within 10 days, LTalpha(-/-) mice s