ライフサイエンスコーパス: intestinal lumen

1 esumably caused by excess fatty acids in the intestinal lumen.

2 can participate in antigen sampling from the intestinal lumen.

3 did not affect the expression of pmrH in the intestinal lumen.

4 d signal-induced release to be active in the intestinal lumen.

5 is involved in dietary zinc uptake from the intestinal lumen.

6 led code for the presence of a threat in the intestinal lumen.

7 express and release leptin apically into the intestinal lumen.

8 generate microbicidal concentrations in the intestinal lumen.

9 ating takes place extracellularly within the intestinal lumen.

10 polarized secretion of interleukin-6 to the intestinal lumen.

11 th commensal microbes that reside within the intestinal lumen.

12 (PMN) across the epithelial monolayer to the intestinal lumen.

13 ctivity of neutrophils as they arrive in the intestinal lumen.

14 ely across the epithelial monolayer into the intestinal lumen.

15 ation across the epithelial monolayer to the intestinal lumen.

16 sive inflammation to the normal flora in the intestinal lumen.

17 y infects the host by translocating from the intestinal lumen.

18 usually considered a digestive enzyme in the intestinal lumen.

19 um, while 2570.00 microg IgE/day entered the intestinal lumen.

20 ation of NaCl from interstitial fluid to the intestinal lumen.

21 dically exposed to pancreatic trypsin in the intestinal lumen.

22 ered by the presence of bacterial LPS in the intestinal lumen.

23 ese mice, hREG3A travels via the bile to the intestinal lumen.

24 the commensal microbes during growth in the intestinal lumen.

25 ere translocated into tissues outside of the intestinal lumen.

26 and abrogated mucus secretion into the large intestinal lumen.

27 estine, to be detached and released into the intestinal lumen.

28 he presence of viable tachyzoites within the intestinal lumen.

29 tips of IEC microvilli and accumulate in the intestinal lumen.

30 r reflects the low-oxygen environment of the intestinal lumen.

31 macrophage-derived [(3)H]cholesterol in the intestinal lumen.

32 a lamprey secreted taurocholic acid into its intestinal lumen.

33 tivation and immune cell infiltration in the intestinal lumen.

34 les and thus are secreted to function in the intestinal lumen.

35 helium and their subsequent release into the intestinal lumen.

36 ecause of the lack of cognate antigen in the intestinal lumen.

37 recognize ligands that co-localize along the intestinal lumen.

38 to promote transmucosal fluid effux into the intestinal lumen.

39 ainst parasitic helminthes that dwell in the intestinal lumen.

40 derived resistin-like molecule beta into the intestinal lumen.

41 ytes to deploy catalytic activities into the intestinal lumen.

42 esent at nearly 50 mum concentrations in the intestinal lumen.

43 on, with concomitant water movement into the intestinal lumen.

44 s limited to the apical membranes facing the intestinal lumen.

45 and subsequent accumulation of fluid in the intestinal lumen.

46 trophic feedings and the microecology of the intestinal lumen.

47 the release of BB membrane vesicles into the intestinal lumen.

48 atory responses and fluid secretion into the intestinal lumen and also can enhance the immunogenicity

49 population, reside in close proximity to the intestinal lumen and are conserved throughout vertebrate

50 hat the [(3)H]AS-binding site resides in the intestinal lumen and are consistent with preclinical dat

51 s mislocalize birefringent material into the intestinal lumen and are lacking in acidified intestinal

52 sequestrants (BAS) complex bile acids in the intestinal lumen and decrease intestinal FXR activity.

53 In contrast, they are secreted into the intestinal lumen and degraded during dauer arrest, only

54 to successfully establish replication in the intestinal lumen and does not result from ordered spread

55 s from wheat, rye, and barley persist in the intestinal lumen and elicit an immune response in geneti

56 gA production, which is also abundant in the intestinal lumen and has a crucial role as the first def

57 e gut microbiota is compartmentalized in the intestinal lumen and induces local immune responses, but

58 d toxin A receptor sequesters toxin A in the intestinal lumen and inhibits its effects of ileal mucos

59 gA promoted V. cholerae agglutination in the intestinal lumen and limited the ability of the bacteria

60 ed back from intestinal villi into the small intestinal lumen and reinfected the Peyer's patches.

61 eased from the intestinal epithelia into the intestinal lumen and systemic circulation and bind to th

62 distributing host defense machinery into the intestinal lumen and that microvillus-derived LVs modula

63 l barrier between the high Ag content of the intestinal lumen and the sterile environment beyond the

64 ypically avirulent pathogens remained in the intestinal lumen and were eventually outcompeted by the

65 cidal enteric alpha-defensins into the small intestinal lumen, and cryptdin-4 (Crp4) is the most bact

66 organisms mainly infected the intestines and intestinal lumen, and we only sporadically observed few

67 on; increased leak of serum protein into the intestinal lumen; and altered tight junction structure.

68 lial MMCs, and levels in the bloodstream and intestinal lumen are maximal at the time of worm expulsi

69 HD5 peptides isolated from the intestinal lumen are proteolytically processed forms--HD

70 es that express hREG3A, which travels to the intestinal lumen, are less sensitive to colitis than con

71 t accumulation of dietary cholesterol in the intestinal lumen at the end of 6-hour lipid infusion and

72 mosomoides polygyrus, which both live in the intestinal lumen, but it does not contribute to immunity

73 is thought to promote uptake of Stx from the intestinal lumen by compromising the epithelial barrier.

74 ay gain a specific survival advantage in the intestinal lumen by decreasing the expression of microbi

75 ins, termed cryptdins, are secreted into the intestinal lumen by mouse Paneth cells in response to mi

76 onmental dsRNAs are imported from the acidic intestinal lumen by SID-2 via endocytosis and are releas

77 e chloride secretion by enterocytes into the intestinal lumen by the cystic fibrosis transmembrane co

78 uggested that histones are released into the intestinal lumen by the high turnover of the intestinal

79 ins in the basolateral membranes, and in the intestinal lumen by weaker interactions with apical memb

80 that commensal microorganisms present in the intestinal lumen can affect the efficiency of serovar Ty

81 in and that movement of bacteria through the intestinal lumen can occur very rapidly in the absence o

82 nced by exogenous factors present within the intestinal lumen, CD1d expression was analyzed in severa

83 himurium bacteria harvested from the chicken intestinal lumen (cecum) was compared with that of a lat

84 ocalization of gut granule contents into the intestinal lumen, consistent with a defect in intracellu

85 ransfer of CX3CR1(+) cells directly into the intestinal lumen, consistent with intraluminal CX3CR1(+)

86 ransfer of CX3CR1(+) cells directly into the intestinal lumen, consistent with intraluminal CX3CR1(+)

87 sting that a reduced ability to colonize the intestinal lumen contributed to its attenuation.

88 icochemical environment of the center of the intestinal lumen differs from that of the epithelial sur

89 iously shown that adenosine is formed in the intestinal lumen during active inflammation from neutrop

90 Adenosine is formed in the intestinal lumen during active inflammation from neutrop

91 We conclude that adenosine released in the intestinal lumen during active inflammation may induce I

92 Shiga toxins expressed in the intestinal lumen during infection with Shiga-toxigenic E

93 We show that IL-4 and IL-13 protect against intestinal lumen-dwelling worms primarily by inducing in

94 dicated that under conditions similar to the intestinal lumen environment, the genes identified are u

95 profile similar to that of bacteria from the intestinal lumen, except that levels of transcription, t

96 up 1B PLA2 digestion of phospholipids in the intestinal lumen facilitates postprandial lysophospholip

97 oteins involved in their absorption from the intestinal lumen fluctuate in a circadian manner.

98 are minimally absorbed and washed out of the intestinal lumen for application as antisecretory agents

99 and that its presence is not required in the intestinal lumen for dietary iron absorption.

100 plant sterols from enterocytes back into the intestinal lumen for excretion.

101 of S. typhimurium persists in the C. elegans intestinal lumen for the rest of the worms' life.

102 is driven by fluid transport, recapitulating intestinal lumen formation.

103 rier limiting micro-organisms present in the intestinal lumen from colonizing enterocytes.

104 intestine forms a barrier that separates the intestinal lumen from the host's internal milieu and is

105 ed the apical compartment that simulates the intestinal lumen, from the BC which represented the bloo

106 ens in microbe-rich environments such as the intestinal lumen; however, the mechanisms are unclear.

107 bulin isotypes that are transported into the intestinal lumen, IgA and IgM, for antigiardial host def

108 d fermentation acids, factors present in the intestinal lumen in patients with active IBDs.

109 y to secrete the bioactive peptides into the intestinal lumen in response to dietary factors; release

110 asolaterally, and is hence found in both the intestinal lumen in the mucosal layer as well as in the

111 ysis and metabolic parameters from the small intestinal lumen indicate onset of ischemia earlier than

112 and that psyllium, through its action in the intestinal lumen, indirectly affected the intravascular

113 Dietary sugars are transported from the intestinal lumen into absorptive enterocytes by the sodi

114 te the absorption of dietary copper from the intestinal lumen into blood as well as utilizing copper

115 Although these mice take up iron from the intestinal lumen into mature epithelial cells normally,

116 nsient transference of microspheres from the intestinal lumen into mesenteric lymph that was not obse

117 ts in the translocation of microbes from the intestinal lumen into the circulation.

118 n is induced when S. Typhi transits from the intestinal lumen into the ileal mucosa.

119 C) expression when bacteria transit from the intestinal lumen into the intestinal mucosa.

120 6 antigen were tested for movement from the intestinal lumen into the subepithelium.

121 translocation of microbial products from the intestinal lumen into the systemic circulation occurs du

122 lear leukocytes (PMNs; neutrophils) into the intestinal lumen is a classical phenomenon associated wi

123 that although phospholipid digestion in the intestinal lumen is a prerequisite for efficient cholest

124 In conclusion, pro-HD-5 in the intestinal lumen is processed by trypsin in a complex in

125 e immunity in the hostile environment of the intestinal lumen, it should be of interest to define bio

126 butyrate-producing Clostridia from the mouse intestinal lumen, leading to decreased butyrate levels,

127 ctors and bound virulent bacteria within the intestinal lumen, leading to their engulfment by neutrop

128 ority of commensal bacteria are found in the intestinal lumen, many species have also adapted to colo

129 t study tests the hypothesis that CEL in the intestinal lumen may influence the type of lipoproteins

130 al bacterial communities residing within the intestinal lumen of mammals have evolved to flourish in

131 step in the translocation of prions from the intestinal lumen of mammals in vivo, which is a precurso

132 peptidoglycan or Pam(3)Cys injected into the intestinal lumen of mice resulted in receptor redistribu

133 o colonize the mucus that accumulates in the intestinal lumen of mice with CF.

134 cherichia coli strains can take place in the intestinal lumen of N2 wild-type worms at a rate of 10(-

135 The half-life of intact IgE in the intestinal lumen of rats 10 days after infection was 3.2

136 esent, they increase water movement into the intestinal lumen over and above the levels observed with

137 cell alpha-defensins secreted into the small intestinal lumen persist as intact and functional forms

138 Long chain triglyceride (>C12) in the intestinal lumen potently inhibits gastric emptying and

139 y, reduced growth, emaciated body, distended intestinal lumen, rectal swelling, and prolific infectio

140 thelia induces neutrophil recruitment to the intestinal lumen, resulting in neutrophil extravasation

141 Notably, neutrophil emigration to the intestinal lumen results in the generation of organized

142 ntestine: first, they are eliminated via the intestinal lumen; second, pro-inflammatory T(H)17 cells

143 hen Salmonella typhimurium is present in the intestinal lumen, several environmental and regulatory c

144 The biocomplexity of the intestinal lumen suggests that interactions between the

145 that regulates the secretion of IgA into the intestinal lumen, the polymeric immunoglobulin receptor,

146 ite displaying wild-type colonization of the intestinal lumen, the straight-rod Deltapgp1 and Deltapg

147 ree fatty acids and monoacylglycerols in the intestinal lumen, the uptake of these products into ente

148 ovilli release unilamellar vesicles into the intestinal lumen; these vesicles retain the right side o

149 on-absorbable disaccharide, which alters the intestinal lumen through multiple mechanisms that lead t

150 Iron uptake from the intestinal lumen through the apical surface of polarized

151 very early after infection, migrate into the intestinal lumen to capture bacteria.

152 During late gestation, villi extend into the intestinal lumen to dramatically increase the surface ar

153 , allow dendritic cells direct access to the intestinal lumen to obtain information about commensal a

154 cid transporter reclaims bile acids from the intestinal lumen to preserve their enterohepatic recircu

155 ctively targets virulent C. rodentium in the intestinal lumen to promote pathogen eradication and hos

156 Glucose amplifies its own transport from the intestinal lumen to the bloodstream by increasing lumina

157 t constitutes a signaling mechanism from the intestinal lumen to the body's defenses.

158 data suggest that a transport nexus from the intestinal lumen to the eggs introduces bottlenecks that

159 lesterol and triglyceride transport from the intestinal lumen to the lymph.

160 ase inhibition on cholesterol transport from intestinal lumen to the lymphatics was evaluated in lymp

161 its information about the composition of the intestinal lumen to the mucosal immune system.

162 w molecular weight soluble antigens from the intestinal lumen to underlying CD103(+) LP-DCs.

163 e transepithelial transport of SIgA from the intestinal lumen to underlying gut-associated organized

164 ce, sodium ions and water also flow into the intestinal lumen via a paracellular route.

165 stages, and that pancreatic AFGPs enter the intestinal lumen via the pancreatic duct to prevent inge

166 ted robust secretion of cholesterol into the intestinal lumen via the sterol-exporting heterodimer ad

167 oximately 3-4% of the dose injected into the intestinal lumen was absorbed, relative to a subcutaneou

168 Recovery of dietary FA in the intestinal lumen was comparable between WT and KO, consi

169 icial roles of nonpathogenic bacteria in the intestinal lumen were described.

170 bed, they were expected to accumulate in the intestinal lumen where a potential inhibition capacity o

171 rbed, they are expected to accumulate in the intestinal lumen where a potential inhibition capacity o

172 ich led to relocation of the pathogen to the intestinal lumen where it was outcompeted by commensals.

173 nteric alpha-defensins are secreted into the intestinal lumen where they have potent microbicidal act

174 porulating C. perfringens cells in the small intestinal lumen, where it then causes epithelial cell d

175 upon inflammation they are secreted into the intestinal lumen, where they promote prostaglandin synth

176 e hormone that induces bile release into the intestinal lumen which in turn aids in fat digestion and

177 V. cholerae caused more clouds of DNA in the intestinal lumen, which appeared to be neutrophil extrac

178 ocated at the critical interface between the intestinal lumen, which is chronically exposed to food a

179 cations in volume, bulk and viscosity in the intestinal lumen, which will alter metabolic pathways of

180 l passage of ions and water to lubricate the intestinal lumen while restricting fluid loss.