ライフサイエンスコーパス: intestinal microflora

1 mediated in part by an aberrant response to intestinal microflora.

2 timulating growth of selected members of the intestinal microflora.

3 nd ligated the appendix to sequester it from intestinal microflora.

4 s results from interactions between GALT and intestinal microflora.

5 nent component of the normal mouse and human intestinal microflora.

6 lects increased production of ethanol by the intestinal microflora.

7 hese studies demonstrate a transition in the intestinal microflora accompanied by a dynamic change of

8 The intestinal microflora also impacts immunity but its role

9 ary selenium affects both composition of the intestinal microflora and colonization of the gastrointe

10 ble transfer of transgenes from GM plants to intestinal microflora and enterocytes.

11 deficiency contributed colitis-predisposing intestinal microflora and increased intestinal ATP, wher

12 ons between the host genetic susceptibility, intestinal microflora and mucosal immune responses throu

13 facilitate the early onset of CD by altering intestinal microflora and the gut mucosal barrier.

14 o investigate the relationship between GALT, intestinal microflora, and modulation of the antibody re

15 strains of bacteria normally present in the intestinal microflora are able to trigger redistribution

16 In rabbits, the intestinal microflora are also required for developing t

17 Indigenous intestinal microflora are known to afford protection aga

18 nclude that specific, currently unidentified intestinal microflora are required for Ab repertoire div

19 s in PBL were undiversified, indicating that intestinal microflora are required for somatically diver

20 and equol (a daidzein metabolite produced by intestinal microflora) are antioxidants in vitro; equol

21 f ingested soy protein are biotransformed by intestinal microflora, are absorbed, undergo enterohepat

22 e and to evaluate the variation in the small intestinal microflora at repeated sampling.

23 vation of NOD2 regulates innate responses to intestinal microflora by downregulating multiple TLR res

24 The intestinal microflora consists of a heterogeneous popula

25 As this low level of epsps in the intestinal microflora did not increase after consumption

26 Suppression of intestinal microflora in atherosclerosis-prone mice inhi

27 dysregulated mucosal immune response to the intestinal microflora in genetically predisposed hosts.

28 immune responses driven by apparently normal intestinal microflora in genetically susceptible hosts.

29 ince the 1910s have explored the role of the intestinal microflora in human health.

30 immune system and its interactions with the intestinal microflora in the pathogenesis of inflammator

31 ould turn to the role of antibiotics and the intestinal microflora in the rising obesity epidemic.

32 equirement for interactions between GALT and intestinal microflora in the selective expansion of V(H)

33 mune responses to distinct components of the intestinal microflora induce intestinal inflammation, we

34 The human endogenous intestinal microflora is an essential "organ" in providi

35 However, the contribution of the intestinal microflora is beyond simple microbial translo

36 Intestinal microflora keeps C. difficile in the spore st

37 is relies on a balance in the composition of intestinal microflora; long-lived macrofauna have also b

38 eroides thetaiotaomicron, a component of the intestinal microflora of mice and humans, uses a repress

39 Quantitative changes in the intestinal microflora of these animals were assessed fir

40 expression by CD4 T cells is induced by the intestinal microflora, oral delivery of specific Ag, and

41 It is now recognized that innate immunity to intestinal microflora plays a significant role in mediat

42 dynamic interactions between a host and its intestinal microflora that lead to commensalism are uncl

43 The alterations in the intestinal microflora that occur after B16 infection rem

44 ensins modulate the composition of the small intestinal microflora, that development of crypt organoi

45 mechanistic explanation for the capacity of intestinal microflora to control liver inflammation.

46 iated lymphoid tissues (GALTs) interact with intestinal microflora to drive GALT development and dive

47 The intestinal microflora, typically equated with bacteria,

48 The immunostimulatory activity of the intestinal microflora varied among individual mice but w

49 The major stimulatory activity of the intestinal microflora was still intact in NOD1-, NOD2-,

50 To understand how selenium regulates the intestinal microflora, we used high-throughput sequencin