ライフサイエンスコーパス: intestinal mucosa

1 eron-gamma and interleukin-17A in the murine intestinal mucosa.

2 LFABP; FABP1) is expressed both in liver and intestinal mucosa.

3 3 and T helper 17 signature cytokines in the intestinal mucosa.

4 dence for mosaicism in APC in non-neoplastic intestinal mucosa.

5 rative colitis patients compared with normal intestinal mucosa.

6 e with hallmarks of adaptive immunity in the intestinal mucosa.

7 h are substantially enriched at the inflamed intestinal mucosa.

8 and are critical for barrier function of the intestinal mucosa.

9 -22, in response to bacterial entry into the intestinal mucosa.

10 mediators of innate immune signaling in the intestinal mucosa.

11 inducible regulatory T cells (iTreg) in the intestinal mucosa.

12 rption and buildup of DGAT substrates in the intestinal mucosa.

13 accumulate in the pulmonary compartment and intestinal mucosa.

14 uced LPS-mediated signaling within the fetal intestinal mucosa.

15 hogenic microbes direct host defenses at the intestinal mucosa.

16 (+)CD4(+)cells in lymphoid organs and in the intestinal mucosa.

17 ncountered systemically or delivered via the intestinal mucosa.

18 on of active immunity or inflammation in the intestinal mucosa.

19 re with normal tolerogenic mechanisms in the intestinal mucosa.

20 afficking of Ly6C(+) monocytes from blood to intestinal mucosa.

21 ex regulation of barrier protection with the intestinal mucosa.

22 ic mechanisms and induce inflammation in the intestinal mucosa.

23 ruminant hosts by translocation through the intestinal mucosa.

24 mediate induction of homing potential to the intestinal mucosa.

25 D4 T cell priming in lymphoid tissue and the intestinal mucosa.

26 maintaining the Treg/Th17 balance within the intestinal mucosa.

27 ceptor superfamily, is expressed in inflamed intestinal mucosa.

28 ed inhibition of TLR4 signaling in the small intestinal mucosa.

29 ved in all VCMsh2 strains and limited to the intestinal mucosa.

30 its preferred colonization site at the host intestinal mucosa.

31 cant numbers in their tissues, including the intestinal mucosa.

32 sely mirrored the transcriptome of the small intestinal mucosa.

33 stemic circulation after passing through the intestinal mucosa.

34 ithelial migration of neutrophils across the intestinal mucosa.

35 nt was maintained in the upper crypts of the intestinal mucosa.

36 on of the variety of these structures in the intestinal mucosa.

37 or crypt cell regions of healthy human small intestinal mucosa.

38 causes systemic infection by traversing the intestinal mucosa.

39 infection of cattle takes place through the intestinal mucosa.

40 wn to be associated with inflammation of the intestinal mucosa.

41 loss in lymphoid tissues and necrosis of the intestinal mucosa.

42 . enterica serovar Typhimurium with the host intestinal mucosa.

43 actors participate in antiviral responses in intestinal mucosa.

44 man intestinal Caco-2 cells and rodent small intestinal mucosa.

45 systemic spread of pathogens that infect the intestinal mucosa.

46 s or villous motility may be trophic for the intestinal mucosa.

47 uced numbers of endogenous Th17 cells in the intestinal mucosa.

48 or mediating the ITF healing response of the intestinal mucosa.

49 ncreased phospho-p38 and COX-2 levels in the intestinal mucosa.

50 6e significantly reduced inflammation in the intestinal mucosa.

51 n various tissues, including the gastric and intestinal mucosa.

52 -derived CRC samples and adjacent uninvolved intestinal mucosa.

53 e cellular and molecular interactions in the intestinal mucosa.

54 L-18, TL1A, and DR3 converge in the inflamed intestinal mucosa.

55 sence of gross or microscopic lesions in the intestinal mucosa.

56 on and promotion of epithelial repair in the intestinal mucosa.

57 of cell differentiation and apoptosis in the intestinal mucosa.

58 ial cancers but not in normal tissues except intestinal mucosa.

59 tion of TLR-dependent IL-8 production in the intestinal mucosa.

60 Niemann-Pick C1-like 1 (NPC1L1) in proximal intestinal mucosa.

61 n the migration of CD4+ T lymphocytes to the intestinal mucosa.

62 r of proinflammatory T-T interactions in the intestinal mucosa.

63 e of Listeria monocytogenes infection in the intestinal mucosa.

64 ithelial cells in renal proximal tubules and intestinal mucosa.

65 which promotes dispersal of EAEC across the intestinal mucosa.

66 nic organisms, focusing in particular on the intestinal mucosa.

67 ponses both systemically and at the oral and intestinal mucosa.

68 ost-environment interfaces, such as skin and intestinal mucosa.

69 ry signals and inflammatory mediators in the intestinal mucosa.

70 role in fluid and water transport across the intestinal mucosa.

71 MT1 was found to be markedly up-regulated in intestinal mucosa.

72 and protection of epithelial barriers in the intestinal mucosa.

73 a transit from the intestinal lumen into the intestinal mucosa.

74 fferent colonization factors that target the intestinal mucosa.

75 omoted the translocation of Stx2a across the intestinal mucosa.

76 specific inflammatory conditions within the intestinal mucosa.

77 ed (reduced and methylated) to 5-MTHF in the intestinal mucosa.

78 tervillous spaces, and increased IELs in the intestinal mucosa.

79 ct of viral infection on miRNA expression in intestinal mucosa.

80 nt, and is integral to the repair of damaged intestinal mucosa(1-3).

81 Among the innate defense mechanisms of intestinal mucosa, a defective tight junction (TJ) barri

82 tion of structural/functional changes in the intestinal mucosa after food challenge.

83 e c, and the cleaved caspase 9 expression in intestinal mucosa after intestinal I/R injury (P<0.05).

84 ed epithelial conditioning that protects the intestinal mucosa against bacterial invasion by inducing

85 st to confer antiapoptotic protection of the intestinal mucosa against inflammatory stress-induced da

86 TI may provide a novel method of stabilizing intestinal mucosa against noxious agents and stimulating

87 g of T cells by dendritic cells (DCs) in the intestinal mucosa and associated lymphoid tissues helps

88 ells whose presence had been reported in the intestinal mucosa and blood.

89 ed these data to RNA-Seq from both the small intestinal mucosa and colonic mucosa of healthy control

90 ients with DE exhibit abnormalities in their intestinal mucosa and CoSCs, which fail to generate in v

91 nd that embryonic precursor cells seeded the intestinal mucosa and demonstrated extensive in situ pro

92 sitions for heparins from porcine and bovine intestinal mucosa and heparan sulfate from bovine kidney

93 hat classical monocytes constantly enter the intestinal mucosa and how the environment dictates their

94 s involved in lymphocyte localization to the intestinal mucosa and how they can be applied to therapy

95 therapeutic antibodies are lost through the intestinal mucosa and how this process affects treatment

96 tion of Toll-like receptors (TLRs) in murine intestinal mucosa and human intestinal epithelial cells

97 nflammatory macrophages and T cells into the intestinal mucosa and increased expression of inflammato

98 rched for T-UCRs that regulate growth of the intestinal mucosa and investigated the mechanism by whic

99 h a novel mechanism that is localized to the intestinal mucosa and is associated with significant cha

100 induction of the murine Hbeta D2 ortholog in intestinal mucosa and is dependent upon both TLR4 and CD

101 ne quantitative polymerase chain reaction of intestinal mucosa and mesenteric lymph nodes of Duoxa(-/

102 epithelium, as well as CD8(+) T cells in the intestinal mucosa and mesenteric lymph nodes, express th

103 c survival and promote interactions with the intestinal mucosa and microbiota.

104 and migrate toward netrins expressed by the intestinal mucosa and pancreas; this attraction is requi

105 posaccharide level and bacteria loads in the intestinal mucosa and peripheral organs were elevated in

106 of tissue-resident memory in organs such as intestinal mucosa and skin.

107 3 noncoding RNA that regulates growth of the intestinal mucosa and stimulates intestinal epithelial r

108 The intestinal mucosa and submucosa contain three types of l

109 with increased aquaporin 4 expression in the intestinal mucosa and submucosa.

110 t on the structure and function of the small intestinal mucosa and suggest signaling through this pat

111 ted robust T cell responses primarily in the intestinal mucosa and that MNV-specific CD8 T cells dyna

112 ted with increased expression of TLR4 in the intestinal mucosa and that physiological stressors assoc

113 ed in the differentiation compartment of the intestinal mucosa and that the expression of JNK1 was si

114 flux transporters in Caco-2 cells and in the intestinal mucosa and the BBB in vivo are most likely du

115 OH) exhibited low permeation across both the intestinal mucosa and the blood-brain barrier (BBB).

116 facilitation of bacterial invasion into the intestinal mucosa and the development of acute colitis.

117 nderstanding of the interactions between the intestinal mucosa and the enteric microbiota.

118 te miRNA expression profile of the mammalian intestinal mucosa and to determine the contribution of m

119 y vitamin folates, are biotransformed in the intestinal mucosa and transferred to the portal vein as

120 cyclase C (GUCY2C), a receptor expressed by intestinal mucosa and universally expressed by metastati

121 feration and stimulation of apoptosis in the intestinal mucosa and was associated with decreased acti

122 ntain a healthy inflammatory tone within the intestinal mucosa and, thus, enhances resistance to infe

123 nergy absorption and in the integrity of the intestinal mucosa, and a GLP-2R agonist, teduglutide, is

124 key regulator for the normal turnover of the intestinal mucosa, and abnormalities associated with thi

125 lycans present in our diets, secreted by our intestinal mucosa, and displayed on the surfaces of othe

126 gens, lymphoid follicular hyperplasia in the intestinal mucosa, and elevated host-defence ability aga

127 ed from animal organs, predominantly porcine intestinal mucosa, and goes through an extensive process

128 nt, colitis-attenuating SodA to the inflamed intestinal mucosa, and host antimicrobials may play a cr

129 ity, reduces subclinical inflammation of the intestinal mucosa, and improves gut barrier function to

130 d with an acute inflammatory response in the intestinal mucosa, and lethal hemorrhagic colitis may oc

131 essential signals to cells in the underlying intestinal mucosa, and that intestinal epithelial cells,

132 highly expressed in both the small and large intestinal mucosa, and there is a 53% overlap in the top

133 etics, enhancement of permeation through the intestinal mucosa, and triggering drug precipitation upo

134 adherin, the natural ligand of KLRG1, in the intestinal mucosa; and have elevated levels of systemic

135 The major fuels for the small intestinal mucosa are amino acids (glutamine, glutamate,

136 zation and homeostasis of glial cells in the intestinal mucosa are regulated by the indigenous gut mi

137 and proinflammatory cytokine response in the intestinal mucosa are significantly higher in AW-recipie

138 ociated with a type 2 immune response in the intestinal mucosa are up-regulated in treatment-naive pe

139 ust reach the circulatory system through the intestinal mucosa as a sufficiently large fragment with

140 rat, a model in which the adaptation of the intestinal mucosa, at least to fasting, is quite differe

141 in mice depleted constitutive Th17 cells in intestinal mucosa, blocked Th17 cell generation in the l

142 (+) and IgA(+) cells were seen in the normal intestinal mucosa, but they were significantly more freq

143 m subsp. paratuberculosis interacts with the intestinal mucosa by crossing both Peyer's patches and n

144 Growth inhibition of the small intestinal mucosa by fasting in mice was associated with

145 Analysis of CD3(+) lymphocytes in the intestinal mucosa by flow cytometry revealed that alphab

146 dies prevents commensal association with the intestinal mucosa by limiting bacterial motility.

147 The invasion of the intestinal mucosa by M. avium subsp. paratuberculosis an

148 n, we investigated expression of DMT1 in the intestinal mucosa by quantitative real-time polymerase c

149 yndrome characterized by damage of the small intestinal mucosa caused by the gluten fraction of wheat

150 cells, E histolytica trophozoites invade the intestinal mucosa, causing amoebic colitis.

151 The intestinal mucosa comprises the inner lining of the inte

152 Normal intestinal mucosa contains abundant immunoglobulin A (Ig

153 Intestinal mucosa contains leptin receptors, and leptin

154 oid-mediated ST2 production was evaluated in intestinal mucosa cultures.

155 al ligation and puncture, Chk2 levels in the intestinal mucosa decreased, associated with an inhibiti

156 e function and structural maintenance of the intestinal mucosa depend on a constant process of prolif

157 late intestinal epithelium to produce IDENs (intestinal mucosa-derived exosome-like nanoparticles) co

158 nce of inflammation characteristic of normal intestinal mucosa despite the close proximity of immunos

159 arget in the study of innate immunity in the intestinal mucosa due to their involvement in the regula

160 ze recent findings related to aspects of the intestinal mucosa during acute GVHD.

161 d IL-17A in response to curli fibrils in the intestinal mucosa during S. Typhimurium infection.

162 plification of inflammatory responses in the intestinal mucosa during serotype Typhimurium infection.

163 ed for in vitro dissolution, mucoadhesion to intestinal mucosa, enhancement of drug absorption in vit

164 The mammalian intestinal mucosa exhibits a spectrum of responses after

165 Mice overexpressing progastrin (PG) in intestinal mucosa (fatty acid-binding protein (Fabp)-PG

166 ve immune responses that are elicited in the intestinal mucosa following ricin exposure and will prov

167 intestinal stem cells maintains a functional intestinal mucosa for a lifetime.

168 (-) cotransporter 1 might be a target in the intestinal mucosa for treatment of secretory diarrheas.

169 The intestinal mucosa forms the first line of defense agains

170 Dysregulated energy homeostasis in the intestinal mucosa frequently is observed in patients wit

171 d cells (ILCs) of the ILC22 type protect the intestinal mucosa from infection by secreting interleuki

172 amounts of mPGES-1 were detected in inflamed intestinal mucosa from patients with inflammatory bowel

173 ceptor and can be produced in the vertebrate intestinal mucosa from the oxidation of thiosulphate (S2

174 miRNA abundance varies dramatically in the intestinal mucosa, from 1 read per million to 250,000.

175 The intestinal mucosa functions is an immunologic organ that

176 key roles for the CX3CR1/CX3CL1 axis in the intestinal mucosa; further clarification of CX3CR1 funct

177 microbial activation and populate the human intestinal mucosa, generating functionally distinct CD10

178 ripheral blood, the cellular response at the intestinal mucosa has never been directly assessed.

179 ut, the physiological roles of MFG-E8 in the intestinal mucosa have not been explored.

180 ts of constitutively activated PI3K upon the intestinal mucosa have not been previously studied in an

181 xamining tolerogenic cell populations of the intestinal mucosa highlight the progress in understandin

182 In the intestinal mucosa, IL-17A was produced by three distinct

183 Most EHEC strains intimately adhere to the intestinal mucosa in a characteristic attaching and effa

184 e production, and consequences for the large intestinal mucosa in humans.A randomized, double-blind,

185 r 5-HT affects growth and maintenance of the intestinal mucosa in mice.

186 e mesenteric lymph nodes (mLNs) and inflamed intestinal mucosa in patients with Crohn's disease (CD).

187 eated T84 cell monolayers and inflamed human intestinal mucosa in vivo.

188 duced endogenous miR-29b levels in the small intestinal mucosa increases cyclin-dependent kinase 2 (C

189 volved histology and immunohistochemistry of intestinal mucosa, indirect calorimetric measurements, w

190 CD8(+) T cells in the intestinal mucosa influence the HIV-associated pathogene

191 play an important role in the maintenance of intestinal mucosa integrity.

192 Morphological development of the small intestinal mucosa involves the stepwise remodeling of a

193 Since immune activation at the level of intestinal mucosa is a hallmark of C. difficile-induced

194 Acute injury to the intestinal mucosa is a major dose-limiting complication

195 The epithelium of the intestinal mucosa is a rapidly self-renewing tissue in t

196 These data suggest that invasion of the intestinal mucosa is an event that requires the particip

197 s reveal that growth inhibition of the small intestinal mucosa is associated with increased expressio

198 into the structure and function of the small intestinal mucosa is becoming increasingly focused on th

199 The fetal intestinal mucosa is characterized by elevated Toll-like

200 Although the small intestinal mucosa is designed to transport large quantit

201 The small intestinal mucosa is highly specialized for terminal dig

202 ever, it is assumed that crossing the bovine intestinal mucosa is important in order for M. avium sub

203 teric glial cells (EGCs) residing within the intestinal mucosa is integrated into the dynamic microen

204 e process by which the toxin transits across intestinal mucosa is not completely understood.

205 ontrast, when pathogenic bacteria invade the intestinal mucosa, it is necessary to elicit strong T an

206 exaggerated inflammation in the skin and the intestinal mucosa leading to electrolyte disturbance, hy

207 molecular basis of gliadin interaction with intestinal mucosa leading to intestinal barrier impairme

208 nstrate that decreased expression of DMT1 in intestinal mucosa leads to compromised absorption and tr

209 Anti-microbial factors produced by the intestinal mucosa limit the translocation of both commen

210 , elevated copper levels are observed in the intestinal mucosa, liver, and blood.

211 terial to mice, GFP was observed in the mice intestinal mucosa, liver, and spleen in fluorescence and

212 ssembly, or "organoid," similar to the human intestinal mucosa, making it an ideal model for enteric

213 pathogenesis: infiltrated macrophages in the intestinal mucosa may promote local inflammation and tis

214 t an ineffective cellular immune response in intestinal mucosa might partially explain the failure of

215 along this axis in the histologically normal intestinal mucosa of Apc(1638N/+) mice before tumor deve

216 bserved an increase in DAF expression in the intestinal mucosa of Apc(Min+/-) mice treated with PGE(2

217 eptor) are similarly altered in the proximal intestinal mucosa of Cav1 null and wild-type mice follow

218 2-mediated antigen presentation in the small intestinal mucosa of Celiac Sprue patients therefore rep

219 be activated by dietary gluten in the small intestinal mucosa of celiac sprue patients, our findings

220 up isolates are a group of isolates from the intestinal mucosa of Crohn's disease patients that can i

221 arkedly fewer TH17 cells were present in the intestinal mucosa of Crtam(-/-) mice.

222 , that were differentially expressed between intestinal mucosa of fasted vs non-fasted mice.

223 ll trafficking, and inflammatory response in intestinal mucosa of HVL patients as compared to LTNP pa

224 Thus, we analyzed the intestinal mucosa of JAM-A-deficient (JAM-A(-/-)) mice f

225 )-tagged gammadelta T cells within the small intestinal mucosa of mice infected with DsRed-labeled S

226 posed novel mechanism was operational in the intestinal mucosa of mice treated with dexamethasone or

227 r aim was to compare 5-HT disposition in the intestinal mucosa of neonatal and adult guinea pigs.

228 rowth factor-beta, which is increased in the intestinal mucosa of patients with active Crohn's diseas

229 The inflammatory environment in the intestinal mucosa of patients with CD contributes to the

230 In vivo, more CD14(+) macrophages from the intestinal mucosa of patients with CD than from controls

231 ammadelta TCR are more abundant in the small intestinal mucosa of patients with celiac disease (CD) c

232 FoxP3(+) T cells were identified in inflamed intestinal mucosa of patients with Crohn disease (CD), b

233 The intestinal mucosa of patients with inflammatory bowel di

234 Mediators released by the intestinal mucosa of patients with irritable bowel syndr

235 trated interleukin (IL)-15 expression in the intestinal mucosa of seronegative symptomatic volunteers

236 evealed that zonulin is overexpressed in the intestinal mucosa of subjects with celiac disease.

237 Invasion of intestinal mucosa of the host by Mycobacterium avium is

238 ected in leukocyte DNA and/or non-neoplastic intestinal mucosa of these patients.

239 Peyer's patches, mesenteric lymph nodes, and intestinal mucosa of transgenic mice.

240 nse to FOLFOX was partially sustained in the intestinal mucosa of VCMsh2(LoxP/G674D) animals.

241 Neutrophil (PMN) infiltration of the intestinal mucosa often leads to severe epithelial injur

242 nflammatory process specifically targets the intestinal mucosa, patients may present with gastrointes

243 vation play a key modulatory roles in normal intestinal mucosa permeability and in inflammatory and h

244 CD103(+) dendritic cells (DCs) in the intestinal mucosa play a crucial role in tolerance to co

245 Vaccination through large intestinal mucosa, previously proven protective for both

246 The intestinal mucosa promotes T cell responses that might b

247 ger RNA targeting relationships in the small intestinal mucosa provides insight into the molecular me

248 Migration to intestinal mucosa putatively depends on local activation

249 er loss of anti-TNF agents through ulcerated intestinal mucosa reduces the efficacy of these drugs in

250 However, the effects of LPS on the fish intestinal mucosa remain unknown.

251 ognition of bacterial amyloid fibrils in the intestinal mucosa represents a novel mechanism of immuno

252 Retention of lymphocytes in the intestinal mucosa requires specialized chemokine recepto

253 rs from Apc(Min/+) mice compared with normal intestinal mucosa, respectively.

254 Complete deletion of Klf5 from the intestinal mucosa resulted in neonatal lethality that co

255 Long-term increase in 5-HT in the intestinal mucosa results in increased 5-HT(3)R internal

256 Damage to the intestinal mucosa results in the translocation of microb

257 sm in which blood monocytes recruited to the intestinal mucosa retain avid scavenger and host defense

258 ixizumab, which induces immune tolerance, in intestinal mucosa samples from patients.

259 The animal and human intestinal mucosa secretes an assortment of compounds to

260 viremia; tonsil, mesentery lymph nodes, and intestinal mucosa served as major target sites of viral

261 uman IgA-secreting B cells were found in the intestinal mucosa, suggesting reconstitution of human mu

262 issues migrated promiscuously, except to the intestinal mucosa, supporting the concept that distinct

263 resence of one or more substances within the intestinal mucosa that directly modulate renal phosphate

264 wn about less typical endocrine cells in the intestinal mucosa that do not contain secretory granules

265 eased expression of Th2 cytokines within the intestinal mucosa that was significantly reduced in CCR6

266 on and differentiation of these cells in the intestinal mucosa, the functional consequences of cross-

267 ecause of their low permeability through the intestinal mucosa, the released protein would be soon de

268 The induction of protective immunity in the intestinal mucosa therefore represents a potentially des

269 odeoxycholate has a beneficial effect on the intestinal mucosa through an increase in resistance to a

270 M. avium subsp. paratuberculosis enters the intestinal mucosa through enterocytes in the absence of

271 e interaction between the microbiota and the intestinal mucosa through Toll-like receptors (TLRs) is

272 are key players in antimicrobial defense in intestinal mucosa, through interleukin 17 and interleuki

273 poptotic response of epithelial cells in the intestinal mucosa to cisplatin, which was defective in M

274 es to the permissiveness of both gastric and intestinal mucosa to colonization by persistent resident

275 tudy we report that villin is cleaved in the intestinal mucosa to generate a pro-apoptotic fragment t

276 /extracellular matrix from fresh gastric and intestinal mucosa to generate stroma-conditioned media.

277 and OT2 T cells reduced the capacity of the intestinal mucosa to make IFN-gamma and IL-17 after eith

278 activate sensory reflex mechanisms from the intestinal mucosa to stimulate or inhibit exocrine pancr

279 In the intestinal mucosa trefoil factors (TFF) and mucins (Muc)

280 Epithelial cells of the intestinal mucosa undergo a continual process of prolife

281 The intestinal mucosa undergoes a continual process of proli

282 ermined the miRNA transcriptome of the mouse intestinal mucosa using ultrahigh throughput sequencing.

283 TLR4 in the intestinal mucosa was inhibited with adenoviruses expres

284 Human intestinal mucosa was modeled using Caco-2 cells.

285 interleukin 2 and 4 mRNA within the inflamed intestinal mucosa was quantified by real-time PCR.

286 lous height/crypt depth (Vh/Cd) in the small-intestinal mucosa was significantly lower and the intrae

287 Since OPN is protective for the intestinal mucosa, we postulated that enhancing OPN expr

288 induction of p47 GTPase IGTP (Irgm3) in the intestinal mucosa were dependent upon functional MyD88.

289 a modest impact on the ability to enter the intestinal mucosa when compared with the wild-type contr

290 need to stimulate protective immunity in the intestinal mucosa, where HIV-1 infection causes severe C

291 nclude that B-cell development occurs in the intestinal mucosa, where it is regulated by extracellula

292 CTLs at the site of colorectal tumors (i.e., intestinal mucosa), which might efficiently eliminate CE

293 linase (NPP7) is an ecto-enzyme expressed in intestinal mucosa, which hydrolyses sphingomyelin (SM) t

294 e is a chronic inflammatory condition of the intestinal mucosa whose etiology is unclear but is likel

295 EAEC infection comprises colonization of the intestinal mucosa with elaboration of enterotoxins and c

296 atic, and reversible remodeling of the small intestinal mucosa with significant villus shortening.

297 reduction of immune activation in blood and intestinal mucosa, with the latter maintained through 8

298 elicits acute neutrophil influx in the human intestinal mucosa within 1 or 2 days after infection, re

299 K974, drives rapid tumour clearance from the intestinal mucosa without effects on normal intestinal c

300 is critical for self-rejuvenation of normal intestinal mucosa, wound repair, and cancer metastasis.