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1 on to generate motility patterns linked with intestinal secretion.
2 ligands guanylin and uroguanylin, regulates intestinal secretion.
3 enteroid lumens, recapitulating ETEC-induced intestinal secretion.
4 (CFTR)-related chloride channel, triggering intestinal secretion.
5 tion routes to induce a response in serum or intestinal secretion.
6 associated chloride (Cl-) flux that underlie intestinal secretion.
7 , possibly representing a novel mechanism of intestinal secretion.
8 l toxin and second-messenger agonist-induced intestinal secretion.
9 permeabilizing effect of the toxin leads to intestinal secretion.
10 vation of a CCK-releasing peptide present in intestinal secretion.
11 and one third of this derived from recycled intestinal secretions.
12 (135-fold increase), in large but not small intestinal secretions.
13 1-6) abolished the CCK releasing activity of intestinal secretions.
14 ctive at induction of specific antibodies in intestinal secretions.
15 A) and IgG in the serum and increased IgA in intestinal secretions.
16 II) has been described in the regulation of intestinal secretion and absorption via angiotensin subt
18 previously reported inhibition of CT-induced intestinal secretion and CHO elongation by platelet-acti
20 R dsRNA pretreatment, TxA-induced changes in intestinal secretion and histopathologic inflammation we
21 ay a proinflammatory role in toxin A-induced intestinal secretion and inflammation and that CRH(1) re
22 ulating the severity of enterotoxin-mediated intestinal secretion and inflammation by activating both
23 partially protected against toxin A-induced intestinal secretion and inflammation, and this effect w
24 f cGMP concentration is necessary for proper intestinal secretion and intestinal epithelial cell prol
25 l enteric neural control and coordination of intestinal secretion and motility suggests that either t
26 l enteric neural control and coordination of intestinal secretion and motility, and acts as a common
27 he action of S. boulardii on toxin A-induced intestinal secretion and mucosal permeability to [3H]man
28 ence of secretory phospholipase A2 in tears, intestinal secretions, and leukocytes, this enzyme may p
29 lin and uroguanylin, are local regulators of intestinal secretion by activation of receptor-guanylate
30 n for the reported suppression of CT-induced intestinal secretion by inhibitors of protein synthesis.
32 le molecular mechanisms mediating ST-induced intestinal secretion have been defined, advancements in
36 and that the presence of these antibodies in intestinal secretions is sufficient for protection again
37 advantageous, as intraluminal nutrients and intestinal secretions might modulate the intestinal immu
38 ileal loops were used for the evaluation of intestinal secretion, mucosal disruption, and apoptosis.
39 n-CLR dsRNA pretreatment, TxA induced robust intestinal secretion, myeloperoxidase activity, and hist
40 t INT-767 on hepatic bile acid synthesis and intestinal secretion of glucagon-like peptide-1 (GLP-1)
46 f IgG in sera and IgA in tracheal, lung, and intestinal secretions, significantly higher (4-6 fold) t
47 utative luminal CCK-releasing factor (LCRF), intestinal secretions were collected by perfusing a modi
48 ited high levels of anti-SEB IgA in lung and intestinal secretions, whereas the toxoid without proteo
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