ライフサイエンスコーパス: intestinal tract

1 community structure along the length of the intestinal tract.

2 of the gut microbiota and gene expression in intestinal tract.

3 hly complex bacterial assembly in the canine intestinal tract.

4 d a complete loss of ability to colonize the intestinal tract.

5 mplex microbial communities of the mammalian intestinal tract.

6 hey exit their usual reservoir in the host's intestinal tract.

7 both viable and sufficiently numerous in the intestinal tract.

8 n clearance of Trichinella spiralis from the intestinal tract.

9 selectively reduce lymphocyte homing to the intestinal tract.

10 role in bacterial colonization of the murine intestinal tract.

11 against helminth parasitic infections in the intestinal tract.

12 to control release of these cells across the intestinal tract.

13 ohn's disease, an inflammatory malady of the intestinal tract.

14 ay facilitate the shedding of prions via the intestinal tract.

15 colonization, and pathogenicity in the human intestinal tract.

16 salivary gland, urinary bladder, and distal intestinal tract.

17 e of the epithelial barrier integrity in the intestinal tract.

18 l delivery or direct administration into the intestinal tract.

19 tivation and production, particularly in the intestinal tract.

20 osity, and stenosis at various levels of the intestinal tract.

21 stasis and specific disease processes in the intestinal tract.

22 RNA and protein from epithelial cells of the intestinal tract.

23 treatments were effective at sterilizing the intestinal tract.

24 ications-especially in the brain, heart, and intestinal tract.

25 mal tumour is the most common sarcoma of the intestinal tract.

26 h bridge innate and adaptive immunity in the intestinal tract.

27 e their function as bacterial tethers in the intestinal tract.

28 riants were impaired for growth in the chick intestinal tract.

29 tenuates chronic inflammatory disease in the intestinal tract.

30 was provided to cells within the guinea pig intestinal tract.

31 am-positive commensal bacterium of the human intestinal tract.

32 me was stochastically activated in the mouse intestinal tract.

33 al pathogens that enter the host through the intestinal tract.

34 characterized by chronic inflammation of the intestinal tract.

35 ts ability to survive and proliferate in the intestinal tract.

36 ibute important to microbial survival in the intestinal tract.

37 mor (GIST) is the most common sarcoma of the intestinal tract.

38 of bioactive sphingolipid metabolites in the intestinal tract.

39 that binds phosphorus and bile acids in the intestinal tract.

40 ve in eradicating some MRSA strains from the intestinal tract.

41 vival in the changing conditions of the fish intestinal tract.

42 s may be revealed amid the complexity of the intestinal tract.

43 udying the process of translocation from the intestinal tract.

44 g roles for one or more effector Yops in the intestinal tract.

45 effects of bile salts and fatty acids in the intestinal tract.

46 mRNA was expressed most predominantly in the intestinal tract.

47 ect in order to regulate the fluidity of the intestinal tract.

48 t transcript is not detectable in kidney and intestinal tract.

49 d develop multiple adenomas throughout their intestinal tract.

50 in repair of other segments of the abdominal intestinal tract.

51 species, maintain an ecological niche in the intestinal tract.

52 between viral and bacterial pathogens of the intestinal tract.

53 n transport system for NOS inhibitors in the intestinal tract.

54 proliferating crypt epithelial cells of the intestinal tract.

55 8 gene activity was present primarily in the intestinal tract.

56 major cytochrome P450 proteins in the mouse intestinal tract.

57 xpression of Cdx1 and Cdx2 in the developing intestinal tract.

58 to those found within the environment of the intestinal tract.

59 the major CYP2C isoform expressed in murine intestinal tract.

60 d frequency for E. coli colonizing the human intestinal tract.

61 gammadelta T cells in the epithelium of the intestinal tract.

62 on in both the aquatic environment and human intestinal tract.

63 physiology of the innervation of the gastro-intestinal tract.

64 mmals in structures such as the limbs or the intestinal tract.

65 enewal of the epithelial cells that line the intestinal tract.

66 d this effect is especially important in the intestinal tract.

67 orce the functional specificity of the adult intestinal tract.

68 tion consistent with their origins along the intestinal tract.

69 seen in nerve fibres innervating the gastro-intestinal tract.

70 r force similar to peristaltic forces in the intestinal tract.

71 ronic relapsing inflammatory disorder of the intestinal tract.

72 thogens and initiate inflammation within the intestinal tract.

73 of antibiotic-mediated perturbations in the intestinal tract.

74 the microbial breakdown of tryptophan in the intestinal tract.

75 ucosal and epithelial tissues, including the intestinal tract.

76 ifferent bacterial species cohabit the human intestinal tract.

77 r jejuni is a natural commensal of the avian intestinal tract.

78 y VRE-colonized mice eliminates VRE from the intestinal tract.

79 liferative cell loss and inflammation in the intestinal tract.

80 lions of bacteria, most of which live in the intestinal tract.

81 e linked to the MIA dimerization observed in intestinal tracts.

82 mber of AIDS patients have M. avium in their intestinal tracts.

83 generalists with relatively undifferentiated intestinal tracts.

84 In the intestinal tract, AMPR genes were involved in early inte

85 show that both genes are coexpressed in the intestinal tract, an organ responsible for not only the

86 influence the bacterial colonisation of the intestinal tract and can be visualised non-destructively

87 , differentiated epithelial cells lining the intestinal tract and exhibits a tumor suppressive effect

88 e and adaptive leukocyte localization to the intestinal tract and GALT, and discuss their relevance t

89 pathogen exploits a unique niche within the intestinal tract and has developed unique strategies to

90 s zinc loss by regulating excretion into the intestinal tract and hence influences the dietary zinc r

91 impact immune cell function both within the intestinal tract and in peripheral tissues.

92 rom cell membranes, is abundant in the human intestinal tract and in processed foods.

93 specific pathogens are able to colonize the intestinal tract and invade, despite the presence of an

94 , endothelial, and muscle cells of the human intestinal tract and is activated in inflamed and fibrot

95 This tumor resistance affects the entire intestinal tract and is independent of the status of mod

96 ic bacterium that metabolizes oxalate in the intestinal tract and is present in a large proportion of

97 tinal epithelial cells that lines the host's intestinal tract and leads to mucosal damage and inflamm

98 nal mucosa comprises the inner lining of the intestinal tract and maintains close proximity with comm

99 olerae cycle between the nutrient-rich human intestinal tract and nutrient-poor aquatic environments

100 essed prominently in epithelial cells of the intestinal tract and other organs exposed to the environ

101 olerae challenge reduces colonization of the intestinal tract and prevents cholera-like diarrhea.

102 erfringens vegetative cells sporulate in the intestinal tract and produce an enterotoxin (CPE) that i

103 Due to the limited absorption from the intestinal tract and sensitivity to degradation, oral va

104 tes have abundant memory CD4+ T cells in the intestinal tract and spleen and that these are selective

105 t link between the microbes that inhabit the intestinal tract and the developing brain.

106 as a barrier to the luminal contents of the intestinal tract and to facilitate the bidirectional tra

107 ies of indole, which is widespread in animal intestinal tracts and in the rhizosphere.

108 the respiratory and, to a lesser extent, the intestinal tracts and internal organs; with limited hist

109 ncer, noncancer diseases of the upper gastro-intestinal tract, and a healthy cohort.

110 ncer, noncancer diseases of the upper gastro-intestinal tract, and a healthy cohort.

111 ncer, noncancer diseases of the upper gastro-intestinal tract, and a healthy upper gastrointestinal t

112 ghtly regulated ion transport by the kidney, intestinal tract, and bone, mediated by calcaemic hormon

113 A was isolated systematically throughout the intestinal tract, and expression of Paneth cell alpha-cr

114 However, during in vivo infection of the intestinal tract, and likely in the tumor microenvironme

115 GISTs occur throughout the intestinal tract, and most harbor an activating mutation

116 ric (125)I-PrP(Sc) were transported from the intestinal tract, and protein misfolding cyclic amplific

117 23A1 is a major ascorbate transporter in the intestinal tract, and some of its genetic variants have

118 nly found in bile duct epithelial cells, the intestinal tract, and the cerebellum and is activated by

119 es spp. are the predominant organisms in the intestinal tract, and they also are important opportunis

120 colon in the event that the contents of the intestinal tract are purged following exposure to a path

121 that Cftr is a tumor suppressor gene in the intestinal tract as Cftr mutant mice developed significa

122 multisystem chronic infection, involving the intestinal tract as well as various other organs.

123 strong epidemiologic evidence that the human intestinal tract, as well as household pets, may be a re

124 tic parameter both for findings in the upper intestinal tract (AUC 0.730, 0.66-0.79) and for the colo

125 survive the harsh environment of a churning intestinal tract, bacteria attach to the host epithelium

126 a novel inflammation-promoting action in the intestinal tract, because loss of p53 or the upstream ac

127 n orally because they are broken down in the intestinal tract before they are absorbed.

128 REVIEW: Bacterial colonization of the infant intestinal tract begins at birth.

129 Epithelial cells lining the intestinal tract build an apical array of microvilli kno

130 PPARdelta is highly expressed in the intestinal tract but its physiological function in this

131 d induced during in vivo growth in the chick intestinal tract, but an absence of these genes did not

132 t junctions (TJs); many are expressed in the intestinal tract, but little is known about their functi

133 wed that there was colonization of the avian intestinal tract by a Campylobacter strain having a know

134 ficantly associated with colonization of the intestinal tract by Citrobacter freundii, Clostridium sp

135 III) complexes, to colonization of the mouse intestinal tract by Escherichia coli.

136 pylobacter jejuni colonization of the animal intestinal tract by mediating the efflux of bile acids.

137 ged intestinal colonization along the entire intestinal tract by the streptomycin-resistant V. paraha

138 ged, which is able to translocate across the intestinal tract, causing systemic infection and abortio

139 Epithelia of the vertebrate intestinal tract characteristically maintain an inflamma

140 was performed in all groups using the Human Intestinal Tract Chip.

141 nstrated an enhanced ability to colonize the intestinal tract compared to the ure mutant strain.

142 fficiency of biofilm formation in the murine intestinal tract confirmed the role of one of the fimH a

143 expression of murine MGAT2 protein along the intestinal tract, determined its subcellular localizatio

144 The brain, heart and gastro-intestinal tract develop distinct left-right (LR) asymme

145 start site was seen along the length of the intestinal tract; different start sites being utilized i

146 Bacteria that colonize the intestinal tract do so despite the development of a spec

147 ability of E. faecium to colonize the murine intestinal tract during antibiotic treatment.

148 ts the polarized epithelial cells lining the intestinal tract early in infection.

149 cause acute gastroenteritis and colonize the intestinal tract for prolonged periods.

150 in all six subspecies persisted in the mouse intestinal tract for several weeks in multiple repeat ex

151 shedding was detected in the respiratory and intestinal tract for up to 9 days.

152 E. faecalis colonizes the nematode intestinal tract, forming a persistent lethal infection.

153 Radioactivity entering the intestinal tract from the gallbladder constituted <10% o

154 The endodermal lining of the adult gastro-intestinal tract harbours stem cells that are responsibl

155 mechanisms of T. foetus pathogenicity in the intestinal tract have not been examined.

156 In the intestinal tract, IL-22 activates STAT3 to promote intes

157 emia initiated from translocation across the intestinal tract in an immunocompromised host is substan

158 ribution of these populations throughout the intestinal tract in healthy individuals remains unclear.

159 ent, and can originate from flagellin in the intestinal tract in inflammatory conditions in the intes

160 ce expression in colonizing pathogens in the intestinal tract in response to surgical stress.

161 um suggests that regional differences in the intestinal tract in the frequency and nature of secondar

162 microbial communities that reside within the intestinal tract in vertebrates are complex and dynamic.

163 ls of BCO mRNA were observed along the whole intestinal tract, in the liver, and in the kidney, where

164 Hence, different to the intestinal tract, in the spleen, Nkrp1g is selectively e

165 that Wnt11 is expressed throughout the mouse intestinal tract including the epithelial cells.

166 All components of the Drosophila intestinal tract, including the endodermal midgut and ec

167 Early-life colonization of the intestinal tract is a dynamic process influenced by nume

168 cedure for reconstruction of the oesophageal-intestinal tract is a highly debated topic.

169 The intestinal tract is a lymphocyte-rich site that undergoe

170 Since the intestinal tract is a major site of early viral replicat

171 The intestinal tract is a site of intense immune cell activi

172 The human intestinal tract is colonised by a complex community of

173 The mammalian intestinal tract is colonized by trillions of beneficial

174 Because the intestinal tract is considered a major portal of entry f

175 The intestinal tract is in intimate contact with the commens

176 The intestinal tract is inhabited by a large and diverse com

177 The intestinal tract is inhabited by a large and diverse com

178 We show that mCRAMP expression in the intestinal tract is largely restricted to surface epithe

179 The intestinal tract is lined by a single layer of columnar

180 ormally harmless behavior of bacteria in the intestinal tract is maintained by community structure an

181 The succession of microbes colonizing the intestinal tract is most marked in early development, du

182 Maintenance of the epithelial barrier in the intestinal tract is necessary to protect the host from t

183 As occupation of the intestinal tract is often a prerequisite for ExPEC-media

184 he presence of normal bacterial flora in the intestinal tract is thought to protect against colonizat

185 gh concentrations of bile salts in the human intestinal tract is vital for the survival of enteric ba

186 this pathotype infects tissues distal to the intestinal tract, is a frequent cause of such infections

187 oderm, which predominantly gives rise to the intestinal tract, is competent to respond to FGFs by ind

188 uc5ac, a mucin not normally expressed in the intestinal tract, is induced in the cecum of mice resist

189 richia coli in its primary niche, the animal intestinal tract, is remarkably unexplored.

190 by mediating resistance to bile salts in the intestinal tract, is required for successful colonizatio

191 ng neovascularization in the murine inflamed intestinal tract (IT) during GvHD.

192 netically heterogeneous protist found in the intestinal tract (IT) of many vertebrates, and although

193 n the immunologically activated state of the intestinal tract, it is conceivable that locally produce

194 rains most of the substances coming from the intestinal tract, it may also play a role in the pathoge

195 t from host-pathogen interactions within the intestinal tract itself.

196 estinal microbiota by the premature infant's intestinal tract, leading to inflammation and injury.

197 with a model in which defective CFTR in the intestinal tract leads to nutritional deficiency which i

198 Since this transporter is expressed in the intestinal tract, lung and mammary gland, it is likely t

199 The intestinal tract maintains proper function by replacing

200 ced samples of the Metagenomics of the Human Intestinal Tract (MetaHit) project with 1,018 previously

201 e been shown to have profound effects on the intestinal tract microflora and intestinal function.

202 sized to be initiated and perpetuated by the intestinal tract microflora.

203 athogens that replicate possibly only in the intestinal tract, noroviruses have developed unique stra

204 le in transepithelial HCO3- secretion in the intestinal tract, null mutant (NBC1-/-) mice were prepar

205 The inoculation of ATCV-1 into the intestinal tract of 9-11-wk-old mice resulted in a subse

206 Its ability to persistently colonize the intestinal tract of a broad range of hosts, including fo

207 that C. albicans strains can persist in the intestinal tract of a healthy individual over a 4-year p

208 exist at the site of bacterial invasion, the intestinal tract of a host animal.

209 to function as a surrogate mucin within the intestinal tract of a stressed host by inhibiting key in

210 nd depletion of CD4(+) T cells occurs in the intestinal tract of acutely infected macaques.

211 or the final clearance of infection from the intestinal tract of adult mice.

212 differentiated tissues, including the gastro-intestinal tract of adult rats.

213 sting at the site of bacterial invasion, the intestinal tract of an animal host.

214 essential for commensal colonization of the intestinal tract of avian species and infection of human

215 Surprisingly, subspecies IIIb colonizes the intestinal tract of BALB/c mice normally yet does not sp

216 Absence of O. formigenes from the intestinal tract of CF patients appears to lead to incre

217 The mere presence of P. aeruginosa in the intestinal tract of critically ill patients is associate

218 crobiota communities that inhabit the gastro intestinal tract of free-range, broiler and feral chicke

219 Examination of the intestinal tract of Glut5(-/-) mice fed a high fructose

220 Trillions of beneficial bacteria inhabit the intestinal tract of healthy mammals from birth.

221 ore-forming bacterium that infects the lower intestinal tract of humans and is the most common known

222 re Gram-negative anaerobes indigenous to the intestinal tract of humans, and they are important oppor

223 lar environments, aquatic reservoirs and the intestinal tract of humans.

224 nt members of the microbial community in the intestinal tract of infants, and studies have shown that

225 Xenorhabdus nematophila colonizes the intestinal tract of infective-juvenile (IJ) stage Steine

226 The preferential expression of DPP4 in the intestinal tract of insectivorous bats, suggests that tr

227 in lung tissue, and were recovered from the intestinal tract of intranasally inoculated ferrets.

228 ce microscopy (LSFM) to visualize H2S in the intestinal tract of live zebrafish.

229 The intestinal tract of mammals is colonized by a large numb

230 ed to lower levels of myeloperoxidase in the intestinal tract of mice developing GvHD and a reduced m

231 aticus causes disease in the liver and lower intestinal tract of mice.

232 pletion occurs almost exclusively within the intestinal tract of simian immunodeficiency virus (SIV)-

233 he adaptation of Campylobacter jejuni in the intestinal tract of the chicken, a natural host and a ma

234 As part of its survival mechanism in the intestinal tract of the host, the worm produces a number

235 m its periplasm into the lumen of the gastro-intestinal tract of the host.

236 ajor virulence factor cholera toxin into the intestinal tract of the human host.

237 r, only the primary variant can colonize the intestinal tract of the IJ stage of the nematode and sup

238 Ambergris, a waxy substance excreted by the intestinal tract of the sperm whale, has been a highly p

239 ransfer (HGT) between bacteria occurs in the intestinal tract of their animal hosts and facilitates b

240 the effect of the conditioning itself on the intestinal tract of these mice.

241 x N-glycans are prominently expressed on the intestinal tract of various avian species.

242 of eight distinctive bacterial phylotypes in intestinal tracts of adult worker bees.

243 compounds, which are normally present in the intestinal tracts of animals.

244 B) are autochthonous bacteria inhabiting the intestinal tracts of many species, including humans.

245 s in humans and a commensal bacterium of the intestinal tracts of many wild and agriculturally signif

246 e to and invade INT407 cells and to colonize intestinal tracts of mice.

247 f 1,000 species of bacteria that inhabit the intestinal tracts of poultry and livestock.

248 ed Deltaproteobacteria widely distributed in intestinal tracts of termites and cockroaches.

249 promote commensal colonization of the avian intestinal tract or invasion of human intestinal cells r

250 ong B cells than occurred within the spleen, intestinal tract, or mesenteric lymph nodes and were pre

251 cteroides fragilis, is a highly aerotolerant intestinal tract organism that has evolved a complex oxi

252 her living on exposed surfaces or within our intestinal tract, our microbial inhabitants produce a re

253 sed changes in the microbiota throughout the intestinal tract over the time course of infection.

254 e collection of all bacterial members in the intestinal tract, plays a key role in health.

255 of parenteral piperacillin excreted into the intestinal tract, preserving colonization resistance of

256 s responsible for immune surveillance of the intestinal tract recognize and generate an antibody resp

257 Gal-4) and Gal-8, which are expressed in the intestinal tract, recognize and kill human blood group a

258 symptoms of VM, such as abnormal dilation of intestinal tracts, reduced gut motility, feeding defects

259 glial cells residing within the walls of the intestinal tract, regulate intestinal motility, a well-c

260 ues, including the enterocytes that line the intestinal tract, remodel their apical surface during di

261 The surface epithelium lining the intestinal tract renews itself rapidly by a coordinated

262 (CD), a chronic inflammatory disease of the intestinal tract, report tertiary lymphoid organs presen

263 The mucosa of the intestinal tract represents a finely tuned system where

264 ued to express a lethal phenotype within the intestinal tract reservoir-a hostile, nutrient scarce en

265 represented in nonhematopoietic cells of the intestinal tract, responds to microbial stimuli once bar

266 The infusion of feces into the intestinal tract shows great promise for treatment and m

267 helial cells of both the respiratory and the intestinal tract, similar to what has been reported for

268 In the intestinal tract, such derepression of toxin synthesis w

269 s intact and functional forms throughout the intestinal tract, suggesting that the peptides may media

270 y to represent those conditions found in the intestinal tract than conditions using batch culture tec

271 gen dispersed more widely through the gastro-intestinal tract than intranasally delivered antigen and

272 or remitting, inflammatory disorders of the intestinal tract that although somewhat similar clinical

273 gan system with distinct functions along the intestinal tract that are critical for health.

274 rated that conditions exist in the mammalian intestinal tract that permit a mode of respiration for E

275 s an enteric bacterial pathogen of the mouse intestinal tract that triggers inflammatory responses re

276 spleens) of live mice, and levels within the intestinal tract (the presumed origin of the gas) were f

277 jejuni invade the cells that line the human intestinal tract, the bacterial proteins that enable thi

278 ed at high levels in epithelial cells of the intestinal tract, the lung, and in cells of the immune s

279 Like the human intestinal tract, the mosquito midgut contains a diverse

280 In the intestinal tract, these protrusions play central roles i

281 EHEC colonizes the intestinal tract through a range of virulence factors en

282 dicate that p53 promotes inflammation in the intestinal tract through suppression of epithelium-prote

283 to transport pathogenic Salmonella from the intestinal tract to the mesenteric lymph nodes.

284 t of uropathogenic Escherichia coli from the intestinal tract to the urinary tract.

285 dependent microvascular beds (such as gastro-intestinal tract) to plasma volume regulation.

286 ctly quantitate sPLA2 activity in the murine intestinal tract utilizing a fluorescent BODIPY-labeled

287 s foetus but having a unique tropism for the intestinal tract was recognized as a significant cause o

288 the physicochemical conditions of the gastro-intestinal tract was used in association with a mathemat

289 hanisms by which this pathogen colonizes the intestinal tract, we used a recombinase gene fusion repo

290 differences in gene transcription along the intestinal tract were accompanied by major alterations i

291 transferred into RAG-2(-/-) recipients whose intestinal tracts were colonized with OVA-expressing or

292 for this receptor are largely unknown in the intestinal tract, where epithelial cells are normally ex

293 coli to thrive in the gallbladder and upper intestinal tract, where high bile concentrations are pro

294 , CaR has been identified in the stomach and intestinal tract, where it has been proposed to function

295 ILCs were first identified in the intestinal tract, where they contribute to epithelial ba

296 eins leads to the release of peptides in the intestinal tract, where they may exert a variety of func

297 and that the released bacteria move into the intestinal tract, where they pass into the environment a

298 s, and the increasing differentiation of the intestinal tract, which also creates new niches for micr

299 s) are chronic inflammatory disorders of the intestinal tract with unknown multifactorial etiology th

300 istent with the time for transit through the intestinal tract without colonization.