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1 ome formation and IL-1beta production in the intima.
2 presence of atheroma plaques in the carotid intima.
3 proliferation of SMCs already present in the intima.
4 ipoproteins on proteoglycans in the arterial intima.
5 a dominance of dendritic cells (DCs) in the intima.
6 e into macrophage foam cells in the arterial intima.
7 cells during calcification in the media and intima.
8 synthetic smooth muscle cells in the aortic intima.
9 ress to preserve functional viability of the intima.
10 in the adventitia and media and least in the intima.
14 vents in atherosclerosis occur in the aortic intima and involve monocytes that become macrophages.
15 ons of collagen-stainable material in tunica intima and media among patients with diabetes mellitus.
17 igation in SJL mice, RpL17 expression in the intima and media decreased, but the number of proliferat
20 l facilitate their ability to migrate to the intima and proliferate to contribute to neointimal lesio
21 expression and activity of elastases in the intima and shifting the balance with their inhibitors to
22 y puncture site is associated with increased intima and total wall thickness and with modest decrease
25 functional DCs are dominant in normal aortic intima and, in contrast to macrophages, CD103(+) classic
27 iogenesis, reduced outgrowth of hyperplastic intima, and minimized CD4(+)CD103(+) tissue-resident mem
28 been known that monocyte recruitment to the intima contributes to the burden of lesion macrophages.
29 f the main LDL modifications in the arterial intima, contributes to massive intracellular cholesteryl
31 gely responsible for SMC accumulation in the intima during vascular remodelling such as neointimal hy
32 ligands for the endothelial-specific tunica intima endothelial receptor tyrosine kinase 2 (Tie2) rec
33 vation pathway for barrier protective tunica intima endothelial receptor tyrosine kinase 2 (Tie2).
37 ccumulation of myeloid cells in the arterial intima, including macrophages and dendritic cells (DCs),
38 hy stresses increased significantly from the intima layer to the interface between media and adventit
41 n all children including intimal thickening (intima/lumen cross-sectional area ratio>0.4), loss of th
42 +/- 1.1; p = 0.010) and not associated with Intima Media thickening Area (26 +/- 5.4% vs. 28 +/- 6.7
43 AH patients showed significant thickening of Intima Media Thickening Area compared to patients withou
44 cal atherosclerosis markers (CAC >0; carotid intima media thickness >/=1 mm) predicted CHD events.
45 d) intakes slows 12-mo common carotid artery intima media thickness (CCA IMT) progression, compared w
49 y on cardiovascular risk factors and carotid intima media thickness (cIMT) in later life in participa
51 and DI-RISK in 152 healthy subjects; carotid intima media thickness (IMT) was assessed as a marker of
52 rminations of cIMT from the IMPROVE (Carotid Intima Media Thickness [IMT] and IMT-Progression as Pred
53 osclerosis (NIMA), i.e., presence of plaque, intima media thickness and ankle-brachial index (ABI), f
54 um-dependent dilatation, arterial stiffness, intima media thickness and blood pressure, indicating im
55 nomic regions associated with common carotid intima media thickness and two different regions associa
56 3 to 6.1 +/- 0.3 m s(-1) (P = 0.03), carotid intima media thickness from 0.43 +/- 0.01 to 0.37 +/- 0.
57 tid arterial compliance, superficial femoral intima media thickness or endothelium-independent dilata
59 s, coronary artery calcium (CAC) and carotid intima media thickness were each separately added to the
60 lood pressure monitoring and measurements of intima media thickness were performed at baseline and af
61 measures (coronary calcium score and carotid intima media thickness) in an independent sample cohort
62 fect of improving dietary quality on carotid intima media thickness, a marker of subclinical atherosc
63 iastolic blood pressure, and reduced carotid intima media thickness, with changes all on par or great
66 otential mechanisms of crosstalk between the intima, media, and adventitia, specific contributions of
67 maging techniques cannot distinguish between intima, media, or atherosclerotic plaque in the carotid
68 sterol esters with apolipoprotein B near the intima-media border, whereas in the complicated lesions,
69 al to circumferential Cauchy stresses at the intima-media layer, which had the highest values at the
70 compared with RITA, leading to an increased intima-media ratio (intima-media ratio, 0.72 [0.53-0.91]
72 leading to an increased intima-media ratio (intima-media ratio, 0.72 [0.53-0.91] versus 0.23 [0.12-0
73 .0 mm Hg, P=0.03), a reduced rate of carotid intima-media thickening (31.5%, P=0.047), and a greater
74 was progression of mean-mean common carotid intima-media thickening (CIMT) measured by ultrasound.
77 rkedly increased aortic pulse-wave velocity, intima-media thickening, oxidized low-density lipoprotei
81 0.54 (SD, 0.12) for CVD, followed by carotid intima-media thickness <25th percentile (DLR, 0.65 [SD,
82 Coronary artery calcium score of 0, carotid intima-media thickness <25th percentile, absence of caro
84 cumference, needed more insulin, had greater intima-media thickness (+5%, P < 0.001 EDIC year 1, P =
85 .85], P=0.001), and high-risk carotid artery intima-media thickness (0.75 [0.60-0.94], P=0.01) in adu
86 .3x10(-4)), stiffness (12.5%, P<8.0x10(-4)), intima-media thickness (10.6%, P<7.9x10(-4)), and wall c
87 jBMI was also associated with higher carotid intima-media thickness (39%; 95% CI, 9%-77% per 1 SD).
88 -performance liquid chromatography), carotid intima-media thickness (a measure of arterial stiffness)
90 e performance of several measures of carotid intima-media thickness (C-IMT) as predictors of cardiova
91 5% confidence interval, 0.74-0.98) and lower intima-media thickness (carotid bulb beta=-0.024, P=0.00
92 luded changes in right common carotid artery intima-media thickness (CCA-IMT) and new focal carotid a
94 ted the association of common carotid artery intima-media thickness (CCA-IMT) with snoring sounds in
95 d the association with common carotid artery intima-media thickness (cCIMT) using multivariable linea
96 a prospective 3-year study of carotid artery intima-media thickness (CIMT) (AIDS Clinical Trials Grou
97 of coronary artery calcium (CAC) or carotid intima-media thickness (CIMT) among asymptomatic adults
98 red in newborn blood spot tests, and carotid intima-media thickness (CIMT) and blood pressure (BP) in
99 therosclerosis as measured by carotid artery intima-media thickness (CIMT) and coronary artery calcif
100 linical atherosclerosis, measured as carotid intima-media thickness (CIMT) and coronary artery calciu
101 particle (HDL-P) concentrations with carotid intima-media thickness (cIMT) and incident coronary hear
103 n to estimate percent differences in carotid intima-media thickness (CIMT) at baseline (2004) and use
104 years, we report the differences in carotid intima-media thickness (cIMT) at baseline between childr
106 dence that measurement of the common carotid intima-media thickness (CIMT) improves the risk scores i
107 ceptibility to arsenic exposure, and carotid intima-media thickness (cIMT) in 959 subjects from the H
113 ildren's Health Study on whom carotid artery intima-media thickness (CIMT) measurements were also col
114 point was progression of mean distal carotid intima-media thickness (cIMT) over 18 months in the modi
116 computed tomography, and common and internal intima-media thickness (cIMT) were measured by carotid u
118 ed cross-sectional associations with carotid intima-media thickness (CIMT), adjusting for subject-spe
119 ured by progression of common carotid artery intima-media thickness (cIMT), in adults with type 1 dia
121 ome was the rate of change in carotid-artery intima-media thickness (CIMT), which was measured every
124 to investigate whether NT was related to the intima-media thickness (IMT) and to atherosclerotic plaq
125 etween youth and adulthood on carotid artery intima-media thickness (IMT) and type 2 diabetes mellitu
126 ound assessment of the common carotid artery intima-media thickness (IMT) during or after PE has not
129 (CAC), carotid plaque, and increased carotid intima-media thickness (IMT) may indicate elevated cardi
131 he average yearly change in the mean maximal intima-media thickness (IMT) of the common carotid arter
132 Endothelial Pulse Amplitude Testing index), intima-media thickness (IMT) of the right common carotid
136 d with increased common carotid artery (CCA) intima-media thickness (IMT), a measure of subclinical a
137 pler ultrasound to assess plaque and carotid intima-media thickness (IMT), computed tomography, magne
138 surements included carotid- and femoral-wall intima-media thickness (IMT), flow-mediated vasodilatati
143 rol and Complications Trial (DCCT) underwent intima-media thickness (n = 1015) and coronary artery ca
144 (n(e) = 1,328); CAD (n(e) = 12,716), carotid intima-media thickness (n = 3,714), and angiographic CAD
145 pheral arterial stiffness (P = .02), carotid intima-media thickness (P = .04), and reduced ocular per
146 metrics was inversely associated with aortic intima-media thickness (P<0.0001) and directly associate
149 allele carriers also showed reduced carotid intima-media thickness (P=0.010) and lower prevalence of
150 CAC (P=1x10(-)(12)) and 1.4% reduced carotid intima-media thickness (P=4x10(-)(14)) in carriers compa
151 ficant increase in blood pressure and aortic intima-media thickness (systolic blood pressure in contr
152 an established biomarker of atherosclerosis (intima-media thickness [IMT] by echo-color Doppler) in a
153 and without CKD and contrasted with carotid intima-media thickness and ankle-brachial index (two oth
154 ividuals who underwent evaluation of carotid intima-media thickness and arterial rigidity (reflection
156 to moderately hypertensive patients, carotid intima-media thickness and cardiovascular outcomes were
157 mary outcomes included common carotid artery intima-media thickness and coronary artery calcium; seco
158 association between the concept and vascular intima-media thickness and elasticity in adolescence.
160 ystatin C was associated with higher carotid intima-media thickness and epicardial adipose tissue ind
162 (lumen diameter [LD], wall layer thickness [intima-media thickness and intima-media-adventitia thick
163 and measures of cardiovascular risk (carotid intima-media thickness and levels of high-sensitivity C-
164 onary artery calcium and mean carotid artery intima-media thickness and long-term exposure to ambient
165 acetylcholine receptor gene family with both intima-media thickness and plaque score independent of k
166 ltiple SNPs showed marginal association with intima-media thickness and plaque score individually, on
167 ical atherosclerosis, as measured by carotid intima-media thickness and plaque score, in 3665 America
170 strong inverse association with both carotid intima-media thickness and the likelihood of angiographi
171 ons between ECAS, carotid plaque and carotid intima-media thickness and the new occurrence of cardiov
172 source of dietary cholesterol, with carotid intima-media thickness and the risk of incident CAD in m
173 ential association of menopause with carotid intima-media thickness as well as with occurrence and st
175 as noted between efflux capacity and carotid intima-media thickness both before and after adjustment
176 e by 24 Agatston units per year (SD 58), and intima-media thickness by 12 mum per year (10), before a
180 ociated with the rate of increase of carotid intima-media thickness during adulthood in individuals w
182 Moreover, there was no difference in carotid intima-media thickness in adulthood between MHO children
184 [interquartile range, 0.45-0.56 mm]; aortic intima-media thickness in ART, 0.64 mm [interquartile ra
185 circumference and blood pressure had greater intima-media thickness in both EDIC years (P = 0.02 to <
186 uartile range, 75-94 mm Hg]; P<0.001; aortic intima-media thickness in controls, 0.52 mm [interquarti
187 ated with both the effects of dalcetrapib on intima-media thickness in dal-PLAQUE-2 (P=0.009) and eve
188 ion and childhood blood pressure and carotid intima-media thickness in the Children's Health Study.
189 sted P=0.004) and with common carotid artery intima-media thickness in the Framingham Heart Study (P=
190 sociated with the 6-y progression of carotid intima-media thickness in those with impaired fetal grow
191 ed with a slower rate of increase in carotid intima-media thickness in those with impaired fetal grow
193 and 52 869 participants with common carotid intima-media thickness measured by ultrasonography withi
194 ardiovascular outcomes, but only the maximum intima-media thickness of (and presence of plaque in) th
195 ernal carotid artery (7.6%, P<0.001) but not intima-media thickness of the common carotid artery (0.0
197 arbon, a correlate of traffic particles, and intima-media thickness of the common carotid artery (CIM
199 lar disease with a 1-SD increase in the mean intima-media thickness of the common carotid artery was
200 ex increased significantly after addition of intima-media thickness of the internal carotid artery (7
201 of the common carotid artery and the maximum intima-media thickness of the internal carotid artery in
202 With the presence of plaque, defined as intima-media thickness of the internal carotid artery of
203 e corresponding hazard ratio for the maximum intima-media thickness of the internal carotid artery wa
204 d Institute HIV-CVD Collaborative to measure intima-media thickness of the right far wall of the comm
207 r coronary artery calcium score than carotid intima-media thickness or ankle-brachial index in subjec
210 es of change in either common carotid artery intima-media thickness or coronary artery calcium nor di
211 as >/=1 of the following: increased carotid intima-media thickness or stenosis, left ventricular hyp
215 orted to correlate more closely with carotid intima-media thickness than HDL cholesterol concentratio
217 g/m(3) higher long-term exposure to PM2.5 in intima-media thickness was -0.9 mum per year (95% CI -3.
219 024, ankle-brachial index was 0.036, carotid intima-media thickness was 0.102, family history was 0.1
222 iable models, the 6-y progression of carotid intima-media thickness was inversely associated with die
228 s significantly (P<0.001) faster and carotid intima-media thickness was significantly (P<0.0001) grea
230 d hyperlipidemia was associated with greater intima-media thickness with intensive but not convention
231 (n = 214) showed a 2.01% decrease in carotid intima-media thickness with statins, compared with 1.02%
232 ial artery, pulse-wave velocity, and carotid intima-media thickness) and pulmonary (pulmonary artery
233 s (coronary artery calcification and carotid intima-media thickness) at year 20, after adjustment for
235 s who underwent assessment of carotid artery intima-media thickness, 442 patients with angiographical
237 ncreased aortic pulse wave velocity, carotid intima-media thickness, and circulating markers of endot
238 s including coronary artery calcium, carotid intima-media thickness, ankle-brachial index, brachial f
239 s included vascular function, carotid artery intima-media thickness, augmentation index, central bloo
240 Both toxins were associated with carotid intima-media thickness, brachial artery reactivity-glyce
241 osclerosis, such as increased carotid artery intima-media thickness, carotid arterial wall stiffness,
242 noglobulin G (IgG) level with carotid artery intima-media thickness, carotid artery distensibility, Y
243 ng electrocardiogram, measurement of carotid intima-media thickness, echocardiography, measurement of
244 to cardiovascular disease, including carotid intima-media thickness, left atrial volume index, monocy
245 ther drug had significant effects on carotid intima-media thickness, other cardiovascular markers, th
246 oronary artery calcium [CAC] scores, carotid intima-media thickness, peripheral arterial disease, and
248 PA variants were not associated with carotid intima-media thickness, they were associated with the nu
259 R( 2) range, 0.37-0.47 for LD; 0.09-0.35 for intima-media thickness; 0.21-0.41 for intima-media-adven
260 imum internal and mean common carotid-artery intima-media thicknesses both predict cardiovascular out
262 35 for intima-media thickness; 0.21-0.41 for intima-media-adventitia thickness; and 0.23 for CAWS; al
263 layer thickness [intima-media thickness and intima-media-adventitia thickness], and carotid artery w
266 phy was normal; however, intimal thickening (intima/media cross-sectional area ratio>1) was seen in 9
267 ular adhesion molecule-1 expression, reduced intima/media neutrophil infiltration, and increased DHCR
269 TA grafts show intimal thickening, increased intima/media ratio, and maintained endothelium-derived v
270 7-positive cells was not correlated with the intima/media ratio, but was negatively correlated with t
273 associations between the progression of the intima-medial thickness (IMT) of the common carotid arte
274 using computed tomography and carotid artery intima-medial wall thickness using ultrasonography.
276 expressed by infiltrating leukocytes in the intima of arteries with TV, and the majority of IL-17-po
277 on hydrogels matching the elasticity of the intima of compliant, young, or stiff, aging arteries.
278 primary inflammatory events occur within the intima of the blood vessel and contribute to both the in
279 clerosis occurs in the subendothelial space (intima) of medium-sized arteries at regions of disturbed
280 These included a subset of lungs lacking intima or, most prominently, media remodeling, which had
281 opopliteal segments exhibited 180mum thinner intima (p<0.001), 45% less plaque calcification, and 2 l
284 ce developed severe medial wall hypertrophy, intima proliferation, and various forms of obliterative
285 cellular lipids) in the first 200 mum of the intima provide important biochemical information that ca
286 et the endothelial cell surface and inflamed intima-related regions of rabbit and human tissue sectio
287 ng was defined as a folding/gathering of the intima, resulting in multiple kinks in the luminal conto
288 intimal stiffening and/or the EC response to intima stiffening clinically may improve vascular health
289 ved apparent copulatory damage to the female intima, suggesting a mechanism for entry of seminal prot
292 ver-operating characteristic curve analysis, intima thickness and I/M were strongly predictive of pre
294 ificantly thicker mean common carotid artery intima, thinner media, and higher I/M ratio than in norm
297 , we find that migrating SMCs must enter the intima very slowly compared to cells in dermal wounds.
299 ortic endothelial cells and in murine aortic intima with a concomitant rise in arginase activity.
300 CyAm7 primarily deposited in the superficial intima within plaque macrophages, endothelial cells, and
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