ライフサイエンスコーパス: intoxicated

1 grade memory task of alcohol impairment when intoxicated.

2 lls are not able to take up TpeL and are not intoxicated.

3 tic brain-injured (TBI) patients are alcohol intoxicated.

4 Finally, apoptosis was observed in CNF1-intoxicated 5637 cells.

5 .1% vs 7.1%) or conviction for driving while intoxicated (7.9% vs 1.2%).

6 Intoxicated AM remained fully capable of B. anthracis sp

7 For this, thioacetamide (TAA)-intoxicated and bile-duct-ligated (BDL) rats were used a

8 Fibroblasts that were intoxicated and later stimulated to proliferate failed t

9 e drinking, physical fighting, driving while intoxicated, and alcohol-related health problems and wer

10 or driver age, sex, history of driving while intoxicated, and survival status, implementation of the

11 fects and clinical presentation if abused or intoxicated, and to know what anesthetic options would b

12 MPTP-intoxicated animals that received Cop-1 immune cells sho

13 n intoxication allowed the rescue of 100% of intoxicated animals.

14 om agonist-treated mice administered to MPTP-intoxicated animals.

15 stry values from buffer-treated versus ricin-intoxicated animals.

16 f-function mutants resemble those of ethanol-intoxicated animals.

17 ects on vulnerable neurons or mediated by GA-intoxicated astrocytes that fail to support neuron funct

18 unable to communicate clearly, and who were intoxicated at the time of screening.

19 However, intoxicated bees were as willing to engage in trophallax

20 0 adults who had blood alcohol measured were intoxicated (blood alcohol content >22 mmol/L [100 mg/dL

21 Intoxicated burned mice demonstrated a two-fold (p < 0.0

22 and sciatic nerves was reduced in galactose-intoxicated, but not streptozotocin-diabetic rats.

23 ouse diaphragm neuromuscular junctions fully intoxicated by BoNT serotype A.

24 In contrast, synapses fully intoxicated by BoNT serotypes D or E were refractory to

25 ndplate potentials in mouse diaphragms fully intoxicated by BoNT/A.

26 tantial redistribution in human airway cells intoxicated by ExoS, -T, and -Y.

27 in the evaluation of all patients who may be intoxicated by natural substances.

28 Dermonecrotic lesions of supernatant intoxicated CD36(-/-) mice are significantly larger, wit

29 ating that BFT did not injure HT29/C1 cells, intoxicated cells exhibited regulatory volume decrease,

30 capacity of Stxs to alter gene expression in intoxicated cells have been limited to individual genes.

31 p1-induced mitochondrial fission within VacA-intoxicated cells inhibited the activation of the proapo

32 uction of lethal mitochondrial damage in TNF-intoxicated cells is discussed.

33 e the mammalian cell, ExoU rapidly lyses the intoxicated cells via its phospholipase A(2) (PLA(2)) ac

34 In Stx-intoxicated cells, the NLRP3 inflammasome triggered the

35 Within intoxicated cells, vacuolation precedes cytochrome c rel

36 to quantify the cytosolic pool of PTS1 from intoxicated cells.

37 xoS trafficking to the perinuclear region of intoxicated cells.

38 ading to cell cycle arrest and distension of intoxicated cells.

39 , which inhibits the accumulation of cAMP in intoxicated cultured cells, significantly decreases the

40 1-556) plus anthrax toxin protective antigen intoxicated cultured mammalian cells and caused actin re

41 re significantly reduced or abolished in Stx-intoxicated D-THP-1 cells in which the expression of NLR

42 involvement in risky driving, riding with an intoxicated driver and being taken advantage of sexually

43 e validity of this test in the conviction of intoxicated drivers.

44 astrocytes in the hippocampus of 26 lethally intoxicated drug addicts and 35 matched controls are des

45 0,+)AT-rBAT transfectants became selectively intoxicated during exposure to Cys-S-Hg-S-Cys.

46 ere were 65 abnormal CT scans (10.3%) in the intoxicated group.

47 Pet-intoxicated HEp-2 and HT29 cells stained with fluorescei

48 plasma membrane to the perinuclear region of intoxicated host cells.

49 Alcohol-intoxicated humans have high levels of fatty acid ethyl

50 ifetime ounces of alcohol consumed and times intoxicated in lifetime estimated at visits 1 week or mo

51 2 are activated in vivo in the colon of Stx2-intoxicated infant rabbits, a model in which Stx2 induce

52 ght percent of trespassers were killed while intoxicated (median alcohol level, 56 mmol/L [260 mg/dL]

53 ary bacterial burden was observed in alcohol-intoxicated mice at 16 and 24 h and was associated with

54 Orally intoxicated mice could be rescued by MAb 11E10 6 h but n

55 etected Stx2a in kidney sections from orally intoxicated mice in the same region as the epithelial ce

56 stration 4 h after MRSA infection in alcohol-intoxicated mice rescued USA300 clearance in vivo.

57 Furthermore, kidney sections from Stx2a-intoxicated mice revealed multifocal, acute tubular necr

58 l-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-intoxicated mice where it inhibits parkin through tyrosi

59 istopathology, and full protection of orally intoxicated mice with monoclonal antibody (MAb) 11E10 di

60 s and protected dopaminergic neurons in MPTP-intoxicated mice, but at levels less than simvastatin.

61 itters, and improved motor functions in MPTP-intoxicated mice.

62 t against nigrostriatal degeneration in MPTP-intoxicated mice.

63 l-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-intoxicated mice.

64 of neuronal cell bodies and termini in MPTP-intoxicated mice.

65 unced effects in VIPR2 agonist-treated, MPTP-intoxicated mice.

66 nd that ricin rapidly reaches the kidneys of intoxicated mice.

67 itters, and improved motor functions in MPTP-intoxicated mice.

68 and GGTI also protected nigrostriata in MPTP-intoxicated mice.

69 istent and progressive disease in acute MPTP-intoxicated mice.

70 itters, and improved motor functions in MPTP-intoxicated mice.

71 n the substantia nigra pars compacta of MPTP-intoxicated mice.

72 to non-transplanted, X-irradiated cuprizone-intoxicated mice.

73 inuous impairment of motor functions in MPTP-intoxicated mice.

74 itters, and improved motor functions in MPTP-intoxicated mice.

75 nigra pars compacta of PD patients and MPTP-intoxicated mice.

76 e nigrostriatal dopaminergic pathway in MPTP-intoxicated mice.

77 l-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-intoxicated mice.

78 is downregulated in PD patients and in MPTP-intoxicated mice.

79 We used the progressively MPTP-intoxicated monkey model that expresses recovery from mo

80 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-intoxicated monkey model to further elucidate the nature

81 l-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-intoxicated monkeys (when motor symptoms are less appare

82 rior olive neurons obtained from chronically intoxicated monkeys revealed a significant up-regulation

83 only used antimalarial agent, endows anthrax-intoxicated murine peritoneal macrophages with a 50% and

84 vents by increasing risk-taking behaviors in intoxicated or impaired persons.

85 SH and GSSG in rat urine and plasma samples, intoxicated or not by lead.

86 chemical and histological parameters in mice intoxicated orally versus intraperitoneally with Stx2a.

87 We also propose that mice intoxicated orally with ricin likely die from distributi

88 ne clearance based on a normal CT scan among intoxicated patients with no gross motor deficits appear

89 en; mean age, 40.3 years), 13 age-matched CO-intoxicated patients without parkinsonism, and 13 age-ma

90 the cervical spine should not be cleared in intoxicated patients, resulting in prolonged immobilizat

91 mal CT scans are sufficient to clear CSIs in intoxicated patients.

92 on reduces GCS, thus limiting its utility in intoxicated patients.

93 e" outbreak because of the appearance of the intoxicated persons.

94 ompare the unadjusted mortality of the total intoxicated population and for specific intoxication sub

95 ared to both control (P<0.001) and galactose-intoxicated rats (P<0.05).

96 n bile duct-ligated and carbon tetrachloride intoxicated rats.

97 e-mediated function in LED-treated, methanol-intoxicated rats.

98 onses were profoundly attenuated in methanol-intoxicated rats.

99 ith unchanged metallothionein-I in manganese-intoxicated rats.

100 3-beta-glucanase mRNA was up-regulated in Al-intoxicated roots, with highest expression after 12 h.

101 ists that rescue neurotransmission in BoNT/A-intoxicated synapses provides compelling evidence for po

102 ocations of the individual subunits in cells intoxicated, under various conditions, with hybrid heter

103 ith toxins) and endogeneous (serum from mice intoxicated via oral, intranasal, and intravenous routes

104 mates of lifetime ounces of alcohol or times intoxicated were observed.

105 In addition, many trauma patients are also intoxicated, which generally worsens outcomes.

106 estrain violent subjects, who are frequently intoxicated with cocaine and other drugs of abuse.

107 odulate enterotoxin expression because cells intoxicated with heat-labile toxin overproduce and relea

108 Rats were intoxicated with methanol, and retinal function was asse

109 We observed similar results in aged monkeys intoxicated with MPTP: they developed severe DOPA-respon

110 WldS and wild-type mice were intoxicated with the cancer chemotherapeutic agent pacli