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1 ed with their ability to induce experimental intra-abdominal abscesses.
2 ortant virulence factors in the formation of intra-abdominal abscesses.
3 and are both able to induce the formation of intra-abdominal abscesses.
4 s the anaerobe most frequently isolated from intra-abdominal abscesses.
5 s included three anastomotic leaks and three intra-abdominal abscesses.
6 x inflammation did LA significantly increase intra-abdominal abscesses.
7 roparesis, intra-abdominal fluid collection, intra-abdominal abscess (10% vs 25%, P = 0.027), severe
8 icture was 10.3%, wound dehiscence 4.4%, and intra-abdominal abscess 5.8%, and did not significantly
9                The third complication was an intra-abdominal abscess after a TV appendectomy.
10 ving the enteric anastomosis site, including intra-abdominal abscess and enterocutaneous fistula form
11 linical symptoms and signs of peritonitis or intra-abdominal abscess and isolation of Candida species
12 s, and postoperative complications including intra-abdominal abscess and pancreatic fistula (all P <
13 ll response necessary for the development of intra-abdominal abscesses and underscore the role of IL-
14     RIM101 contributes to persistence within intra-abdominal abscesses, at least in part through acti
15 OR], 0.93; 95% CI, 0.38-2.32; P = .88) or on intra-abdominal abscess development (OR, 0.89; 95% CI, 0
16 .093), postoperative hemorrhage (P = 0.174), intra-abdominal abscess formation (P = 0.199), biliary l
17                                              Intra-abdominal abscess formation was more common follow
18 stoperative pyrexia, ileus, wound infection, intra-abdominal abscess formation, operative time, and p
19 management included CT-guided drainage of an intra-abdominal abscess in 23 patients, and total parent
20 vitro and in vivo they were attenuated in an intra-abdominal abscess infection model.
21 s necessary for maximal virulence in a mouse intra-abdominal abscess model.
22  after 90 days included wound infections and intra-abdominal abscess (n = 75) and failure to thrive (
23                                There were no intra-abdominal abscesses or other major complications a
24 r IBD recurrence (n = 66, 12%) or because of intra-abdominal-abscess or fistula (n = 93, 16%).
25 t colonic procedures, OR = 1.5), presence of intra-abdominal abscess (OR = 5.0) or enteric fistula (O
26  (OR, 4.90; 95% CI, 1.41-17.06; P = .01) and intra-abdominal abscess (OR, 7.46; 95% CI, 1.65-33.66; P
27 ileostomy closure (6.5%), trauma (2.7%), and intra-abdominal abscess/other peritonitis (1.9%).
28                      With CPG-directed care, intra-abdominal abscess rate decreased from 0.24 to 0.10
29 ndpoints were postoperative wound infection, intra-abdominal abscess, reoperation, length of hospital
30 ndpoints were postoperative wound infection, intra-abdominal abscess, reoperation, length of hospital
31 f surgery and postoperative wound infection, intra-abdominal abscess, reoperation, or readmission.
32 f surgery and postoperative wound infection, intra-abdominal abscess, reoperation, or readmission.
33 c fluid collections, one pseudocyst, and one intra-abdominal abscess; two donors underwent reoperatio
34 rapeutic antibiotics, and the development of intra-abdominal abscess was significantly lower in patie
35      Patients who had an anastomotic leak or intra-abdominal abscess were included in the infection g
36 ith Bacteroides fragilis is the formation of intra-abdominal abscesses, which are induced by the caps
37 are mortality and postoperative morbidities (intra-abdominal abscess, wound dehiscence, and intestina

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