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1 15 mm Hg), and simultaneous pleural effusion/intra abdominal hypertension.
2 ng results in an increased detection rate of intra-abdominal hypertension.
3 end-expiratory lung volume decline caused by intra-abdominal hypertension.
4 ions does not increase the detection rate of intra-abdominal hypertension.
5                                Air-regulated intra-abdominal hypertension (0-25 mm Hg).
6 al pleural effusion instillation (13 mL/kg), intra-abdominal hypertension (15 mm Hg), and simultaneou
7                                  In managing intra-abdominal hypertension, 33% of pediatric intensivi
8 udy period, the use of a continually revised intra-abdominal hypertension/abdominal compartment syndr
9 at risk significantly improves survival from intra-abdominal hypertension/abdominal compartment syndr
10 of an open abdomen to prevent development of intra-abdominal hypertension/abdominal compartment syndr
11 phylactic use of the open abdomen to prevent intra-abdominal hypertension after DC laparotomy.
12              The diagnosis and management of intra-abdominal hypertension and abdominal compartment s
13 n often effectively affect lesser degrees of intra-abdominal hypertension and abdominal compartment s
14 larly crucial to the successful treatment of intra-abdominal hypertension and abdominal compartment s
15 medical training exists in the management of intra-abdominal hypertension and abdominal compartment s
16 ) and lung compliance (C(L)), in response to intra-abdominal hypertension and changes in positive end
17                                              Intra-abdominal hypertension decreased end-expiratory lu
18                                              Intra-abdominal hypertension decreased end-expiratory tr
19                                              Intra-abdominal hypertension decreased total respiratory
20  considered mostly a postsurgical condition, intra-abdominal hypertension (IAH) and the abdominal com
21                During bilateral lung injury, intra-abdominal hypertension increased both DP(AW) (at p
22 pared with normal intra-abdominal pressures, intra-abdominal hypertension increased DP(AW), during bo
23 ond, and third time periods (p < 0.001), and intra-abdominal hypertension (intra-abdominal pressure >
24                                              Intra-abdominal hypertension is common in critically ill
25  patients allows for sufficient detection of intra-abdominal hypertension is unclear.
26                                    Relief of intra-abdominal hypertension may be instrumental to the
27                                              Intra-abdominal hypertension may contribute to a poor ou
28 with at least one additional risk factor for intra-abdominal hypertension (multiple trauma, abdominal
29                              The presence of intra-abdominal hypertension negates most of the positiv
30 early abdominal decompression for refractory intra-abdominal hypertension or abdominal compartment sy
31 and DP(TP) were increased by the presence of intra-abdominal hypertension (p < 0.0001 and p = 0.0222,
32 sion was 29.8% versus 18.6% in those without intra-abdominal hypertension (p < 0.001).
33                            In a pig model of intra-abdominal hypertension, positive end-expiratory pr
34               The mortality of patients with intra-abdominal hypertension was 29.8% versus 18.6% in t
35 ural effusion was present and by 184 mL when intra-abdominal hypertension was present.
36                    When pleural effusion and intra-abdominal hypertension were simultaneously applied
37 abdominal pressure and to aggressively treat intra-abdominal hypertension when identified.

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