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1 15 mm Hg), and simultaneous pleural effusion/intra abdominal hypertension.
2 ng results in an increased detection rate of intra-abdominal hypertension.
3 end-expiratory lung volume decline caused by intra-abdominal hypertension.
4 ions does not increase the detection rate of intra-abdominal hypertension.
6 al pleural effusion instillation (13 mL/kg), intra-abdominal hypertension (15 mm Hg), and simultaneou
8 udy period, the use of a continually revised intra-abdominal hypertension/abdominal compartment syndr
9 at risk significantly improves survival from intra-abdominal hypertension/abdominal compartment syndr
10 of an open abdomen to prevent development of intra-abdominal hypertension/abdominal compartment syndr
13 n often effectively affect lesser degrees of intra-abdominal hypertension and abdominal compartment s
14 larly crucial to the successful treatment of intra-abdominal hypertension and abdominal compartment s
15 medical training exists in the management of intra-abdominal hypertension and abdominal compartment s
16 ) and lung compliance (C(L)), in response to intra-abdominal hypertension and changes in positive end
20 considered mostly a postsurgical condition, intra-abdominal hypertension (IAH) and the abdominal com
22 pared with normal intra-abdominal pressures, intra-abdominal hypertension increased DP(AW), during bo
23 ond, and third time periods (p < 0.001), and intra-abdominal hypertension (intra-abdominal pressure >
28 with at least one additional risk factor for intra-abdominal hypertension (multiple trauma, abdominal
30 early abdominal decompression for refractory intra-abdominal hypertension or abdominal compartment sy
31 and DP(TP) were increased by the presence of intra-abdominal hypertension (p < 0.0001 and p = 0.0222,
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