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1 al reflux, which may be related to increased intra-abdominal pressure.
2 s occurred in transpulmonary pressure due to intra-abdominal pressure.
3 utes to continence during rapid increases in intra-abdominal pressure.
4 al organ dysfunction resulting from elevated intra-abdominal pressure.
5 trunk stabilization by indirectly increasing intra-abdominal pressure.
6 cing sphincteric closure during increases in intra-abdominal pressure.
7 al cavity, was filled with water to increase intra-abdominal pressure.
8 increase in pleural pressure with increasing intra-abdominal pressure.
9 re remeasured 30 mins after each increase in intra-abdominal pressure.
10                                        Above intra-abdominal pressure 5 mm Hg, plateau airway pressur
11 us muscle activity associated with increased intra-abdominal pressure and anal or vaginal EMG probes
12 dal ventilation in the presence of increased intra-abdominal pressure and in calculating true transpu
13 cs, and cardiac output 5 mins after each new intra-abdominal pressure and positive end-expiratory pre
14 niques are necessary to decompress increased intra-abdominal pressure and prevent or treat persistent
15  to minimize the risk of developing elevated intra-abdominal pressure and to aggressively treat intra
16  pressure is the gold standard for measuring intra-abdominal pressure, and several nonsurgical method
17 us, elevated intracranial pressure, elevated intra-abdominal pressure, and therapeutic hypothermia af
18                                    Increased intra-abdominal pressure appears to produce this effect
19 as well as minimization of intrathoracic and intra-abdominal pressure as clinically possible.
20                            Indeed, increased intra-abdominal pressure, as an extreme marker of abdomi
21                                     The mean intra-abdominal pressure at admission day was an indepen
22 city was significantly reduced by increasing intra-abdominal pressure at both positive end-expiratory
23                               Four levels of intra-abdominal pressure (baseline, 12, 18, and 22 mm Hg
24 arying degrees of matching the corresponding intra-abdominal pressure: baseline positive end-expirato
25 ra-abdominal pressure compared with baseline intra-abdominal pressure) but did not change cardiac out
26                            Acutely increased intra-abdominal pressure causes a significant increase i
27 ry lung volume (+119% [p < .001] at 22 mm Hg intra-abdominal pressure compared with 5 cm H2O positive
28 ry lung volume (+233% [p < .001] at 22 mm Hg intra-abdominal pressure compared with 5 cm H2O positive
29 and -8% [p < .05], respectively, at 22 mm Hg intra-abdominal pressure compared with baseline intra-ab
30                                         High intra-abdominal pressure could enhance the penetration o
31 raneous muscle) activity that would increase intra-abdominal pressure during EAS contraction, and (4)
32 njury and acute renal failure with regard to intra-abdominal pressure dynamics, preload limitation, a
33 ricardotomy negated all effects of increased intra-abdominal pressure except the decreased cardiac in
34  < 0.001), and intra-abdominal hypertension (intra-abdominal pressure &gt;/= 12 mm Hg) occurred in 19.9%
35 ission to the ICU was associated with higher intra-abdominal pressure, higher plasma C reactive prote
36                                     Elevated intra-abdominal pressure (IAP) is a frequent cause of mo
37                                     Elevated intra-abdominal pressure (IAP) is associated with ICP el
38 is considered the gold standard for indirect intra-abdominal pressure (IAP) measurements.
39 study sought to determine whether changes in intra-abdominal pressure (IAP) with aggressive diuretic
40 d can be present at relatively low levels of intra-abdominal pressure (IAP).
41 ate positive end-expiratory pressure (= half intra-abdominal pressure in cm H2O + 5 cm H2O), and high
42 and high positive end-expiratory pressure (= intra-abdominal pressure in cm H2O).
43 anges in positive end-expiratory pressure or intra-abdominal pressures in both conditions.
44 were studied: a) group 1 (n = 9) animals had intra-abdominal pressure increased to 25 mm Hg above bas
45 tched to intra-abdominal pressure to prevent intra-abdominal pressure-induced end-expiratory lung vol
46                    When compared with normal intra-abdominal pressures, intra-abdominal hypertension
47  positive end-expiratory pressure matched to intra-abdominal pressure led to a preservation of end-ex
48 ivided into three subgroups according to the intra-abdominal pressure measurement policy in different
49                                              Intra-abdominal pressure measurements in predefined at-r
50 dent predictor of mortality in patients with intra-abdominal pressure measurements started within the
51                             Whether limiting intra-abdominal pressure measurements to preselected at-
52                                       Serial intra-abdominal pressure measurements, nonoperative pres
53                             At each level of intra-abdominal pressure, moderate positive end-expirato
54         We aimed to clarify whether expanded intra-abdominal pressure monitoring results in an increa
55                             In patients with intra-abdominal pressure monitoring, the mean intra-abdo
56 tudied, and 1,241 patients (46.0%) underwent intra-abdominal pressure monitoring.
57                               Application of intra-abdominal pressures of 0 and 20 cm H2O at positive
58 hing positive end-expiratory pressure to the intra-abdominal pressure on cardio-respiratory parameter
59 ntra-abdominal pressure monitoring, the mean intra-abdominal pressure on the admission day is an inde
60           The detrimental impact of elevated intra-abdominal pressure, progressing to abdominal compa
61 ratios; (4) leak-point pressure, the minimum intra-abdominal pressure required to cause incontinence,
62  shock, and its resuscitation with increased intra-abdominal pressure results in many untoward hemody
63 cy of this condition, routine measurement of intra-abdominal pressure should be performed in high-ris
64 ay be why persons with chronically increased intra-abdominal pressure, such as the morbidly obese, su
65 t fluid, blood and protein losses, increased intra-abdominal pressure, systemic hyperthermia, and inc
66                             At each level of intra-abdominal pressure, three levels of positive end-e
67                                  Increase of intra-abdominal pressure to 25 mm Hg above baseline caus
68                 Expanding the measurement of intra-abdominal pressure to more than 50% of intensive c
69  positive end-expiratory pressure matched to intra-abdominal pressure to prevent intra-abdominal pres
70 cted by opening the abdomen, suggesting that intra-abdominal pressure transmission contributes little
71 e increased linearly by ~ 50% of the applied intra-abdominal pressure value, associated with commensu
72             Following baseline measurements, intra-abdominal pressure was increased by incrementally
73                                          The intra-abdominal pressure was measured in 31.7%, 55.6%, a
74 nism proposed is that central obesity raises intra-abdominal pressure, which increases pleural pressu
75 ring laparoscopic surgery produces increased intra-abdominal pressure, which potentially influences h
76 ethysmography (OEP), (ii) intra-thoracic and intra-abdominal pressures with a balloon catheter in eac

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