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1 oblastoma if administered locally via direct intra-arterial infusion.
2 man protein (rhVEGF-C; 500 microg) by direct intra-arterial infusion.
3  peripheral blood flow in CHF patients after intra-arterial infusion and after oral administration in
4 ar implantation appeared more effective than intra-arterial infusion, and mobilized peripheral blood
5 ycemia (300 mg/dL) created by dextrose (50%) intra-arterial infusion, and with coadministration of vi
6 ncept for CBL0137 and its administration via intra-arterial infusion as a superior treatment compared
7  5.0 mg/kg) were administered as a 10-minute intra-arterial infusion; control animals received vehicl
8 titute CBL0137 in the ILP protocol, using an intra-arterial infusion method, to safely achieve effect
9                                              Intra-arterial infusion of 17 beta-oestradiol significan
10  local hyperglycemia (300 mg/dL) achieved by intra-arterial infusion of 50% dextrose.
11  responses (strain-gauge plethysmography) to intra-arterial infusion of a selective blocker of ETA re
12  studied the forearm blood flow responses to intra-arterial infusion of acetylcholine (7.5 to 30 micr
13 on plethysmography and blood sampling during intra-arterial infusion of acetylcholine, bradykinin, so
14 lusion plethysmography in response to serial intra-arterial infusion of acetylcholine, sodium nitropr
15 osine kinase inhibitor-as well as continuous intra-arterial infusion of adenosine.
16 ar conductance (CalfVC) were measured during intra-arterial infusion of an alpha-adrenoreceptor agoni
17 ence of clinical therapeutic angiogenesis by intra-arterial infusion of an angiogenic protein.
18  the nanomolar range and were not reduced by intra-arterial infusion of an angiotensin-converting enz
19                                   Repetitive intra-arterial infusion of autologous BMMNCs into the co
20 alation study provides evidence of safety of intra-arterial infusion of autologous MSCs in patients w
21 ned to repetitive (3 times; 3-week interval) intra-arterial infusion of BMMNC or placebo.
22 is trial was to determine whether repetitive intra-arterial infusion of bone marrow mononuclear cells
23 (FBF) and net t-PA release before and during intra-arterial infusion of bradykinin (25 to 400 ng/min)
24            These effects are abrogated by an intra-arterial infusion of corticosterone.
25                                              Intra-arterial infusion of ET-1 significantly increased
26  found in stroke animals that received local intra-arterial infusion of human PRP lysate (p's<0.05).
27                    Forearm flow responses to intra-arterial infusion of increasing doses of acetylcho
28 was evaluated by measuring the effects of an intra-arterial infusion of incremental doses of carbacho
29 elium-dependent vasodilation was assessed by intra-arterial infusion of methacholine (0.3 to 10 micro
30  with and without NO synthase inhibition via intra-arterial infusion of N(G) -monomethyl-L -arginine
31                                     Using an intra-arterial infusion of N(G) -monomethyl-L -arginine
32 t nitric oxide synthase (NOS) inhibition via intra-arterial infusion of N(G)-monomethyl-L-arginine (L
33                                              Intra-arterial infusion of N(G)-monomethyl-L-arginine (L
34 ium-independent vasodilation was measured by intra-arterial infusion of nitroprusside (0.3 to 10 micr
35                                          The intra-arterial infusion of oncolytic adenoviruses warran
36 tra-arterial pressure via catheter) to local intra-arterial infusion of phenylephrine (PE; alpha1 -ad
37 ted changes in vascular conductance (FVC) to intra-arterial infusion of phenylephrine (PE; alpha1 -ag
38                                     Hindlimb intra-arterial infusion of pyridoxal phosphate-6-azophen
39  occlusion plethysmography before and during intra-arterial infusion of the arginase inhibitor N(omeg
40  resistance vessels (blood flow responses to intra-arterial infusion of the endothelium-dependent dil
41 hours after enalapril (20 mg) or placebo, an intra-arterial infusion of the NEP inhibitor thiorphan (
42 acologically enhanced with Mch or reduced by intra-arterial infusion of the nitric oxide inhibitor N(
43 y measuring the effects on venous tone of an intra-arterial infusion of the NO synthase inhibitor N-m
44 ed by use of forearm blood flow responses to intra-arterial infusions of acetylcholine (a vasodilator
45 e above parameters were also assessed during intra-arterial infusions of acetylcholine and bradykinin
46  Agonist-induced vasodilation to incremental intra-arterial infusions of acetylcholine and bradykinin
47 Forearm venous occlusion plethysmography and intra-arterial infusions of acetylcholine and N(G)-monom
48        Forearm blood flow (FBF) responses to intra-arterial infusions of acetylcholine and sodium nit
49        Forearm blood flow (FBF) responses to intra-arterial infusions of acetylcholine, bradykinin, s
50 d 18 black control subjects before and after intra-arterial infusions of acetylcholine, nitroprusside
51 ascular conductance (FVC) responses to local intra-arterial infusions of ACh, ATP, and sodium nitropr
52 ation, and forearm blood flow in response to intra-arterial infusions of endothelial-dependent and -i
53 w (Q, ultrasound Doppler) at rest and during intra-arterial infusions of endothelium-dependent (acety
54                Forearm vascular responses to intra-arterial infusions of endothelium-dependent and -i
55 s occlusion plethysmography before and after intra-arterial infusions of N(G)-monomethyl-l-arginine (
56 otocols, measuring leg vascular responses to intra-arterial infusions of NG-monomethyl-l-arginine (l-
57 sures were also recorded in seven men during intra-arterial infusions of normal saline, phentolamine
58  alpha- and beta-adrenoreceptor blockade via intra-arterial infusions of phentolamine and propranolol
59 and calculated vascular conductance (FVC) to intra-arterial infusions of phenylephrine (alpha(1)-agon
60 ascular conductance (FVC) responses to local intra-arterial infusions of phenylephrine (alpha(1)-agon
61 sis were randomly assigned (1:1:1) bilateral intra-arterial infusions of placebo on days 1 and 30 (n=
62                                  Prior close-intra-arterial infusions of tetrodotoxin or hexamethoniu
63 in-gauge plethysmography) responses to local intra-arterial infusions of tyramine (which evokes endog
64 d, first before start of, and then during an intra-arterial infusion to, the muscle preparation of th

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