戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1                 Tumor recurrence tends to be intraabdominal.
2 dent diabetes (8%), pancreatic fistula (5%), intraabdominal abscess (4%), small bowel obstruction (4%
3  12, 3%), wound infections (n = 12, 3%), and intraabdominal abscess (n = 9, 2%).
4 .025), infectious complications (p = 0.014), intraabdominal abscess (p = 0.022), and postoperative de
5          Four complications were observed: 1 intraabdominal abscess and 1 case of urinary retention i
6 eric anastomosis site, graft thrombosis, and intraabdominal abscess formation have been well document
7 more, the trxB mutant was not able to induce intraabdominal abscess formation in a mouse model, where
8 ysaccharides (Zps) confer protection against intraabdominal abscess formation in a T cell-dependent m
9 report that purified CP5 and CP8 facilitated intraabdominal abscess formation in animals when given i
10 o shown when TLR2-/- mice exhibited impaired intraabdominal abscess formation in response to B. fragi
11 rding the mechanism by which T cells control intraabdominal abscess formation.
12 tivated T cells modulated the development of intraabdominal abscess formation.
13                                              Intraabdominal abscess occurred in 14% (33/240) of patie
14 such as pancreatic fistula, wound infection, intraabdominal abscess, and need for reoperation.
15 in were more likely to develop a significant intraabdominal abscess, collection, or fistula.
16 jor complications, infectious complications, intraabdominal abscess, or pancreaticojejunal anastomoti
17 ith complications, infectious complications, intraabdominal abscess, or postoperative death.
18 ajor complication, infectious complications, intraabdominal abscess, pancreaticojejunal anastomotic l
19 ound infection (8%), cholangitis (5.7%), and intraabdominal abscess/biloma (2.9%).
20    T cells are critical for the formation of intraabdominal abscesses by Staphylococcus aureus.
21 ha- or ICAM-1-specific Abs failed to develop intraabdominal abscesses following challenge with purifi
22 eroides fragilis promotes the development of intraabdominal abscesses in experimental models of sepsi
23 the 5.0-kDa molecule, were protected against intraabdominal abscesses induced by challenge with viabl
24 to rats in vivo conferred protection against intraabdominal abscesses induced by viable bacterial cha
25 traumatic injuries more than 4 hours old, or intraabdominal abscesses were enrolled.
26 tudy were 1) to examine interrelations among intraabdominal adipose tissue (IAAT) and other adiposity
27                                              Intraabdominal adipose tissue (IAAT) is the body fat dep
28 d with dual-energy X-ray absorptiometry, and intraabdominal adipose tissue (IAAT) was determined with
29 However, the relation between fat intake and intraabdominal adipose tissue (IAAT), a risk factor for
30 try, subcutaneous abdominal tissue (SAT) and intraabdominal adipose tissue (IAF) by computed tomograp
31 luation of the various fat depots, including intraabdominal adipose tissue (IAF), subcutaneous adipos
32              The inverse association between intraabdominal adipose tissue and the postprandial CRP r
33 Endpoint trunk fat mass, total fat mass, and intraabdominal adipose tissue were all lower with MCT co
34 Despite these genetic differences in loss of intraabdominal adipose tissue, improvement in glucose di
35                                              Intraabdominal adiposity and insulin resistance are risk
36 n transverse and coronal interpretations for intraabdominal anatomic and pathologic findings (kappa=0
37 of disease after resection was predominantly intraabdominal and involved the original tumor site, per
38 - and preadipocyte-containing fractions from intraabdominal and subcutaneous adipose tissue of mice r
39                                              Intraabdominal and subcutaneous fat depots were two- to
40                                  Measures of intraabdominal and subcutaneous fat did not predict fast
41 etic resonance imaging to measure accurately intraabdominal and subcutaneous fat masses in 14 obese [
42 n the renal subcapsular space, intrahepatic, intraabdominal, and subcutaneous locations.
43 ients who required anesthesia for one of the intraabdominal aortic, gastric, biliary, or colon operat
44            In addition, intraparenchymal and intraabdominal bleeding did not complicate any PIT; 71%
45                      There were two cases of intraabdominal bleeding, one of which required surgical
46   The amount of adipose tissue stored in the intraabdominal cavity is an important, independent risk
47 lative distribution of adipose tissue in the intraabdominal compared with the subcutaneous abdominal
48 he need for laparotomy and the potential for intraabdominal complications have been cited as relative
49 patients required operative intervention for intraabdominal complications.
50 iring ICU and transitioned to comfort-care), intraabdominal conditions, and alcohol abuse/withdrawal.
51 ene insertional mutation, crsp, causing high intraabdominal cryptorchidism in homozygous males.
52 reat gene in mice causes infertile bilateral intraabdominal cryptorchidism.
53                           Massive unilocular intraabdominal cysts in children are rare.
54 nths), none of the 10 recipients experienced intraabdominal desmoid tumor recurrence or developed de
55 gan-based primary site are characteristic of intraabdominal desmoplastic small round cell tumor.
56 g 16 patients, it was used to stage possible intraabdominal disease.
57  procedures, to create an adequate length of intraabdominal esophagus to perform a wrap.
58                                          The intraabdominal failure pattern appears to be decreased c
59                                              Intraabdominal fat (IAF) area is often measured indirect
60 an its wild-type littermates, and has excess intraabdominal fat accumulation.
61 ages of change in body weight, fat mass, and intraabdominal fat area were assessed.
62                              In obese girls, intraabdominal fat but not BMI or waist-to-hip ratio was
63 Assessments at baseline, 1, and 2 y included intraabdominal fat by computed tomography scan and body
64                                     Two-year intraabdominal fat changes were 7.05 +/- 5.07% for the t
65 sment of fatness reinforced the concept that intraabdominal fat compartment is strongly correlated wi
66                     Fat-free mass as well as intraabdominal fat correlated to a similar extent with L
67 ercentage body fat increases and attenuating intraabdominal fat increases in overweight and obese pre
68                     MR imaging percentage of intraabdominal fat measurements (mean, 23%; 95% CI: 17%,
69 tin, resistin, retinol binding protein-4, or intraabdominal fat volume.
70 ar risk factors are related to the amount of intraabdominal fat.
71 njury can be induced by metabolically active intraabdominal fat.
72 o avoid increases in percentage body fat and intraabdominal fat.
73  tissue was redistributed, with reduction of intraabdominal fat.
74                            Subcutaneous- and intraabdominal-fat areas, thigh-muscle area and strength
75 itivity of abdominal radiography was 90% for intraabdominal foreign body and 49% for bowel obstructio
76  vein gas, extensive intraperitoneal gas and intraabdominal free fluid.
77 ally important complications: three cases of intraabdominal hemorrhage and one case each of gross hem
78                         The 12th patient had intraabdominal hemorrhage around the spleen and no obvio
79                          Gastrointestinal or intraabdominal hemorrhage occurred in approximately 5 pe
80 was associated with clinically insignificant intraabdominal hemorrhage.
81 metry, and subcutaneous abdominal (SAAT) and intraabdominal (IAAT) adipose tissue by computerized tom
82 mon diagnoses were appendicitis (33%), other intraabdominal infection (29%), and abscess (25%).
83 domly assigned 518 patients with complicated intraabdominal infection and adequate source control to
84                  The most common causes were intraabdominal infection and graft pancreatitis (38%), p
85                                Patients with intraabdominal infection enrolled in PRTs have an increa
86                  The successful treatment of intraabdominal infection requires a combination of anato
87                             The incidence of intraabdominal infection was slightly higher in the SE g
88          One hundred sixty-eight adults with intraabdominal infection were treated at a single instit
89           Surgical-site infection, recurrent intraabdominal infection, or death occurred in 56 of 257
90 posite of surgical-site infection, recurrent intraabdominal infection, or death within 30 days after
91 formation in an experimental animal model of intraabdominal infection.
92  or negative for acute appendicitis or other intraabdominal infection.
93 derwent relaparotomy, including two (7%) for intraabdominal infection.
94                                 Treatment of intraabdominal infections remains a challenge, mainly be
95                             The incidence of intraabdominal infections significantly decreased betwee
96                             In patients with intraabdominal infections who had undergone an adequate
97 clinafloxacin in the treatment of a range of intraabdominal infections, and in patients with a broad
98 s, Infections of the Gastrointestinal Tract, Intraabdominal Infections, Bone and Joint Infections, Ur
99 samples are indicated during soft tissue and intraabdominal infections, but cultures obtained through
100 rs were treated for bloodstream, complicated intraabdominal infections, or complicated urinary tract
101 s may not be applicable to all patients with intraabdominal infections.
102 as adjuncts in the management of complicated intraabdominal infections.
103 r the treatment of patients with complicated intraabdominal infections.
104 every 6 hours in the treatment of a range of intraabdominal infections.
105  adequate surgical management of complicated intraabdominal infections.
106 biotic regimens for the empiric treatment of intraabdominal infections.
107 he plethora of microorganisms encountered in intraabdominal infections.
108 ith pharmacobehavioral techniques (including intraabdominal injections of active compounds and a comp
109                                Patients with intraabdominal injuries were identified retrospectively.
110       Seventy-five (10%) of 744 patients had intraabdominal injuries, and US depicted free fluid in 4
111 e patient with adrenal hematoma had no other intraabdominal injuries.
112                          CT findings were no intraabdominal injury (n = 932), solid organ injury only
113 l perforation following BAT without signs of intraabdominal injury on initial imaging and extensive p
114                             For detection of intraabdominal injury, US was less sensitive in pregnant
115 sonography may identify children at risk for intraabdominal injury.
116 graphy and computed tomography) in detecting intraabdominal injury.
117 aparotomy were commonly utilized to diagnose intraabdominal injury.
118 e were 328 pregnant patients, 23 of whom had intraabdominal injury.
119 , but moderately sensitive, for detection of intraabdominal injury.
120                  Together they determine the intraabdominal, intrathoracic, and subglottic pressure,
121                                              Intraabdominal malignancy was the leading provisional di
122 diac disease, extremity fracture, knee pain, intraabdominal malignancy, and stroke.
123 nts, unresectability is mainly the result of intraabdominal metastases.
124                    They are at high risk for intraabdominal metastatic spread.
125 tin, resistin, retinol binding protein-4, or intraabdominal obesity, suggesting that these factors do
126                    Because intrathoracic and intraabdominal organs are usually recovered before the k
127 sually recovered after the intrathoracic and intraabdominal organs, careful palpation of the kidneys
128 ity as a means of diagnosing mass lesions in intraabdominal organs.
129 ormation about the heart, vessels, lungs and intraabdominal organs.
130 l findings (n=35) about the intrathoracic or intraabdominal organs.
131               With regard to the presence of intraabdominal pathologic findings, coronal reformations
132 d ventilatory consequences such as increased intraabdominal pressure and hypercarbia.
133                                    Increased intraabdominal pressure associated with central obesity
134 cally, morbidly obese patients have a higher intraabdominal pressure at 2 to 3 times that of nonobese
135                                The increased intraabdominal pressure enhances venous stasis, reduces
136   Hemodynamic insults secondary to increased intraabdominal pressure include increased afterload and
137  probably secondary to a chronic increase in intraabdominal pressure leading to increased intrathorac
138  volume to minimize the effects of increased intraabdominal pressure on renal and cardiac function.
139 insufflation with CO2 and an increase in the intraabdominal pressure up to 15 mm Hg.
140 prone positioning, hemodynamic, respiratory, intraabdominal pressure, and echocardiographic data were
141  pressure over inspired oxygen fraction, the intraabdominal pressure, and the right and left cardiac
142 and avoidance of events leading to increased intraabdominal pressure.
143  increased variceal pressure with increasing intraabdominal pressure.
144  Combining extended hepatectomy with another intraabdominal procedure increases the risk of postopera
145 tivariate analysis showed that a synchronous intraabdominal procedure was the only factor associated
146 ion, and 40 patients underwent a synchronous intraabdominal procedure.
147 cipants had undergone one or more additional intraabdominal procedures.
148  specifically influence fat accretion in the intraabdominal region.
149 y polysaccharides in preventing experimental intraabdominal sepsis in the absence of antimicrobial th
150 omplication rates to include reoperation and intraabdominal sepsis were markedly increased in those p
151 ication for LTx, cytomegalovirus status, and intraabdominal sepsis) and donor factors (donor age, col
152 ls migrate into the peritoneal cavity during intraabdominal sepsis, but the trafficking of NKT and T
153 iologic drainage or surgical exploration for intraabdominal sepsis.
154 scess formation associated with experimental intraabdominal sepsis.
155 nt of abscesses associated with experimental intraabdominal sepsis.
156 y mediate CpG-inducible host defenses during intraabdominal sepsis.
157 that beta 2M-/- mice are resistant to lethal intraabdominal sepsis.
158 esion formation associated with experimental intraabdominal sepsis.
159 citis is the most common condition requiring intraabdominal surgery in infancy and childhood.
160                                    Increased intraabdominal (visceral) fat is associated with a high
161 elf occupying virtually all of the available intraabdominal volume.

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top