ライフサイエンスコーパス: intracarotid

1 ere randomized to receive bolus injection of intracarotid (3-5 mg) or intravenous (10-20 mg) 1% propo

2 s, activation of carotid chemoreceptors with intracarotid administration of 100 nmol sodium cyanide r

3 -1 (HO-1) was studied in the rat brain after intracarotid administration of hyperosmolar mannitol.

4 rent oHSV variants (MSC-oHSV) and found that intracarotid administration of MSC-oHSV, but not of puri

5 Intracarotid administration of NAC resulted in high deli

6 reduce cellular glutathione levels prior to intracarotid alkylator administration.

7 out interfering with the CNS cytotoxicity of intracarotid alkylators, even after BSO depletion of CNS

8 al confrontational naming activation with an intracarotid amobarbital (Amytal) injection procedure (I

9 Language lateralization testing by intracarotid amobarbital injection showed no evidence of

10 aging can become a viable alternative to the intracarotid amobarbital procedure (IAP), the current go

11 ion and BRS before and during left and right intracarotid amobarbital procedure (IAP).

12 e focal epilepsies against the results of an intracarotid amobarbital procedure (IAP).

13 Functional MRI (fMRI) and the intracarotid amobarbital test (IAT) are generally congru

14 had bilateral language representation by the intracarotid amobarbital test.

15 Intracarotid amobarbital testing (IAT) is currently the

16 Memory profiles were obtained from intracarotid amobarbital testing and non-invasive verbal

17 significantly with memory lateralization by intracarotid amobarbital testing.

18 antly reduced declarative memory capacities (intracarotid amobarbital testing: P < 0.001; verbal memo

19 ing form intracranial electrodes, use of the intracarotid amobarbitol perfusion test, and the role of

20 xplored as alternatives to the more invasive intracarotid amytal procedure.

21 Many studies have compared fMRI with the intracarotid amytal test for establishing language domin

22 itive mapping and may be able to replace the intracarotid amytal test for language and memory lateral

23 tes of administration, such as craniotomy or intracarotid arterial infusion of noxious agents causing

24 ments were undertaken to investigate whether intracarotid arterial injection of CsA reduces brain dam

25 A recent study showed that a single intracarotid arterial injection of cyclosporin A (CsA) c

26 al FMT signal was disrupted after unilateral intracarotid artery (ICA) MPTP infusion which reduced ip

27 C6 tumors were significantly increased after intracarotid bradykinin infusion (2.8- and 2.2-fold, res

28 sed permeability across the BTB, compared to intracarotid bradykinin infusion alone.

29 in tumors and normal brain, with or without intracarotid bradykinin infusion, using RIA.

30 Intracarotid cold saline infusion (ICSI) is potentially

31 ing responses of the carotid sinus nerves to intracarotid cyanide.

32 Intracarotid fluorescein isothiocyanate-BSA infusion was

33 vate their sympathetic nervous system during intracarotid glucose infusion but their brains are gener

34 Here, 1 h intracarotid glucose infusions (4 mg/kg/min) selectively

35 lar infusions of mannitol, 60 min unilateral intracarotid glucose infusions at 4 mg/kg/min in awake r

36 Intracarotid glucose infusions stimulated c-fos expressi

37 lucose and the insulin secretory response to intracarotid glucose injection are blunted in TRPC3-defi

38 out 10% of PVN neurons with FLI activated by intracarotid glucose.

39 PA administration were used: i.p. injection, intracarotid (i.c.) injection [with and without blood-br

40 apture therapy could be enhanced by means of intracarotid (i.c.) injection of sodium borocaptate (BSH

41 However, when given by the intracarotid (i.c.) route after only 5 min of recirculat

42 changes in tissue NADH concentrations, after intracarotid (IC) infusion of equal volumes of normal sa

43 A single unilateral intracarotid infusion of 1-methyl-4-phenyl-1,2,3,6-tetra

44 n brain tumor microvessel permeability after intracarotid infusion of bradykinin in the RG2 rat gliom

45 Intracarotid infusion of bradykinin selectively increase

46 Intracarotid infusion of bradykinin selectively increase

47 Intracarotid infusion of bradykinin selectively increase

48 The coupled intracarotid infusion of mannitol and CsA (10 mg/kg) was

49 mg/kg i.v.), administered following rostral intracarotid infusion of mannitol to disrupt the blood-b

50 ucose were more variable and widespread than intracarotid infusions.

51 in vivo single-unit recordings revealed that intracarotid injection of hypertonic NaCl produced a con

52 a nigra of monkeys 4 days after a unilateral intracarotid injection of N-methyl-4-phenyl-1,2,3,6-tetr

53 mma-MSH (1.25 nmol) are not inhibited by the intracarotid injection of SHU9119 (1.25-12.5 nmol) or th

54 in unlesioned and MPTP-lesioned (unilateral intracarotid injection) rhesus monkeys following an intr

55 In contrast, intracarotid injections of fibroblasts or U87 glioma cel

56 yes of isoflurane-anesthetized piglets using intracarotid injections of sodium fluorescein.

57 Intracarotid low dose bradykinin infusion can selectivel

58 avenous CsA dose, or by osmotic opening with intracarotid mannitol.

59 Intracarotid or intracerebroventricular infusion of hype

60 inistered at the time of reperfusion via the intracarotid route to maximize their localization in the

61 ffects, which are much more pronounced after intracarotid than after intravenous administration.