ライフサイエンスコーパス: intracellular Ca2 concentration

1 s used to examine receptor-evoked changes in intracellular Ca2+ concentration.

2 ways characterized by an initial increase in intracellular Ca2+ concentration.

3 r, partially inhibit the MTX-induced rise of intracellular Ca2+ concentration.

4 technique to 'uncage' InsP3 while recording intracellular Ca2+ concentration.

5 a2+ sensitivity without necessarily altering intracellular Ca2+ concentration.

6 TPase and the resulting rise in steady state intracellular Ca2+ concentration.

7 amines was accompanied by an increase in the intracellular Ca2+ concentration.

8 uency of the foot flickering are affected by intracellular Ca2+ concentration.

9 magnitude of the activity-dependent rise in intracellular Ca2+ concentration.

10 ted the glutamic acid-evoked increase in the intracellular Ca2+ concentration.

11 f many enzymes in response to changes in the intracellular Ca2+ concentration.

12 ect on NO synthesis stimulated by increasing intracellular Ca2+ concentration.

13 mal growth factor receptor is an increase in intracellular Ca2+ concentration.

14 en triggering agonist-specific signatures of intracellular Ca2+ concentration.

15 rs and by extracellular signals that elevate intracellular Ca2+ concentration.

16 2+-dependent protein kinase via an increased intracellular Ca2+ concentration.

17 hibitors, as well as after alteration of the intracellular Ca2+ concentration.

18 tion was closely related to the decay of the intracellular Ca2+ concentration.

19 -gated Ca2+ channels leading to increases in intracellular Ca2+ concentration.

20 nscription can be stimulated by increases in intracellular Ca2+ concentrations.

21 es and is expressed in response to increased intracellular Ca2+ concentrations.

22 these effects are independent of changes in intracellular Ca2+ concentrations.

23 ere incubated with several agents that alter intracellular Ca2+ concentrations.

24 A modest increase in intracellular Ca2+ concentration also promotes survival

25 The resting level of intracellular Ca2+ concentrations also affects CaMKII/Ca

26 uced a significant and sustained increase in intracellular Ca2+ concentration, although vitronectin-i

27 y diverse agonists that transiently increase intracellular Ca2+ concentration and activate the endoth

28 mented that plant cell stimulation increases intracellular Ca2+ concentration and activates cytosolic

29 responded to SDF-1 with a transient rise of intracellular Ca2+ concentration and by undergoing chemo

30 Glutamate decreased arteriole myocyte intracellular Ca2+ concentration and dilated brain slice

31 Elevations of the intracellular Ca2+ concentration and generation of react

32 of cardiac myocytes, where it may respond to intracellular Ca2+ concentration and modulate SR Ca2+ io

33 h effects on the Na/Ca exchanger, raises the intracellular Ca2+ concentration and strengthens cardiac

34 owed much less sensitivity to alterations in intracellular Ca2+ concentration and underwent little ru

35 ccordingly, FcepsilonRI-induced elevation of intracellular Ca2+ concentrations and activation of prot

36 movement in the late phase through altering intracellular Ca2+ concentrations and K+ channel activit

37 e history of action potentials that increase intracellular Ca2+ concentrations and of inhibitory sign

38 inhibiting adenylyl cyclase, increasing the intracellular Ca2+ concentration, and activating protein

39 Ca2+ selective, activated by a reduction in intracellular Ca2+ concentration, and inactivated by hig

40 argin increased nuclear phospho-CREB levels, intracellular Ca2+ concentration, and transcription of c

41 ion channels, second messenger pathways and intracellular Ca2+ concentrations, and is influenced by

42 P3R1, and NFATC3 expression; calcium influx; intracellular Ca2+ concentration; and calcineurin activi

43 uggested that EM-induced fluctuations in the intracellular Ca2+ concentration are responsible for the

44 ing is critical for lymphocyte function, and intracellular Ca2+ concentrations are regulated by store

45 Hypotonic solution did not cause a rise in intracellular Ca2+ concentration as measured by simultan

46 Ca2+ amplification required a rise in intracellular Ca2+ concentration at or near the intracel

47 attenuated the [NaCl]L-dependent changes in intracellular Ca2+ concentration, but hydrochlorothiazid

48 ulation of the bradykinin-evoked increase in intracellular Ca2+ concentration by protein kinase C (PK

49 Preventing the increase in intracellular Ca2+ concentration by the inhibitor of Gal

50 uanylyl cyclase (RetGC) is stimulated at low intracellular Ca2+ concentrations by guanylyl cyclase ac

51 thresholds for Ca2+, we manipulated buffered intracellular Ca2+ concentrations by microinjecting Ca2+

52 Raising intracellular Ca2+ concentration ([Ca]i) from 10 to 200

53 prolonged, high-force maintenance at resting intracellular Ca2+ concentration ([Ca2+]) and very low e

54 racellular pH, cyclic nucleotide levels, and intracellular Ca2+ concentration ([Ca2+]).

55 elicited repetitive, transient increases in intracellular Ca2+ concentration ([Ca2+]i spikes) that r

56 ts is characterized by an initial decline in intracellular Ca2+ concentration ([Ca2+]i) (phase 0), fo

57 potential-induced Ca2+ influx increased the intracellular Ca2+ concentration ([Ca2+]i) above thresho

58 We measured intracellular Ca2+ concentration ([Ca2+]i) and contracti

59 t by initiating a rise in smooth muscle cell intracellular Ca2+ concentration ([Ca2+]i) and exert lit

60 opic glutamate receptors causes increases in intracellular Ca2+ concentration ([Ca2+]i) and intracell

61 suring the major cell signalling parameters, intracellular Ca2+ concentration ([Ca2+]i) and myosin re

62 Intracellular Ca2+ concentration ([Ca2+]i) and NO produc

63 effect of cortisol (200 nM for 48 h) on the intracellular Ca2+ concentration ([Ca2+]i) and parameter

64 tion of [Ca2+]0 also stimulates increases in intracellular Ca2+ concentration ([Ca2+]i) and this effe

65 Oscillations in intracellular Ca2+ concentration ([Ca2+]i) are thought t

66 el changes between capacitance responses and intracellular Ca2+ concentration ([Ca2+]i) at different

67 elease eCBs and requires a transient rise in intracellular Ca2+ concentration ([Ca2+]i) but not concu

68 The rate of intracellular Ca2+ concentration ([Ca2+]i) clearance was

69 tor FLUO-3, revealed a transient increase in intracellular Ca2+ concentration ([Ca2+]i) during brief

70 neurokinin A stimulated a rapid increase in intracellular Ca2+ concentration ([Ca2+]i) for both rece

71 pendent (EC50 = 6.0 +/- 0.3 nM) increases in intracellular Ca2+ concentration ([Ca2+]i) in a proporti

72 e determined the effects of (S)-albuterol on intracellular Ca2+ concentration ([Ca2+]i) in dissociate

73 n of C-protein in modulating contraction and intracellular Ca2+ concentration ([Ca2+]i) in intact car

74 sP3 receptor modulation and hence control of intracellular Ca2+ concentration ([Ca2+]i) in neuronal t

75 te several pathways leading to a rise in the intracellular Ca2+ concentration ([Ca2+]i) in pulmonary

76 of potentials studied, the peak increase in intracellular Ca2+ concentration ([Ca2+]i) in response t

77 the membrane currents during oscillations of intracellular Ca2+ concentration ([Ca2+]i) in single rat

78 Low nanomolar concentrations of NO increased intracellular Ca2+ concentration ([Ca2+]i) in somata, de

79 ow here that insulin causes an acute rise in intracellular Ca2+ concentration ([Ca2+]i) in these neur

80 acid to induce CCK secretion and changes in intracellular Ca2+ concentration ([Ca2+]i) in two entero

81 he TxA2 mimetic IBOP required to reduce peak intracellular Ca2+ concentration ([Ca2+]i) induced by a

82 n at Ser-843, indicating that an increase in intracellular Ca2+ concentration ([Ca2+]i) is a potentia

83 The free intracellular Ca2+ concentration ([Ca2+]i) is governed b

84 a direct, spatially restricted elevation of intracellular Ca2+ concentration ([Ca2+]i) on one side o

85 Changes in intracellular Ca2+ concentration ([Ca2+]i) play an impor

86 was to directly test if acute hypoxia causes intracellular Ca2+ concentration ([Ca2+]i) rises through

87 ion to 0.1 mM evoked a repetitive pattern of intracellular Ca2+ concentration ([Ca2+]i) spiking that,

88 ere shown to produce a sustained increase in intracellular Ca2+ concentration ([Ca2+]i) that was depe

89 In this way, the kinetics of intracellular Ca2+ concentration ([Ca2+]i) transients an

90 We have measured intracellular Ca2+ concentration ([Ca2+]i) using indo-1.

91 age-clamped guinea-pig ventricular myocytes; intracellular Ca2+ concentration ([Ca2+]i) was measured

92 The effect of these blockers on intracellular Ca2+ concentration ([Ca2+]i) was studied w

93 eous measurements of whole cell currents and intracellular Ca2+ concentration ([Ca2+]i) were made in

94 xtracellular pH were measured with SNARF and intracellular Ca2+ concentration ([Ca2+]i) with indo-1.

95 Fura-2 acetoxymethyl ester to measure their intracellular Ca2+ concentration ([Ca2+]i), they were sh

96 NMDA receptor activation leads to increased intracellular Ca2+ concentration ([Ca2+]i), we studied t

97 rpolarization depended solely on the rise in intracellular Ca2+ concentration ([Ca2+]i), whereas the

98 Agonist-mediated oscillations in intracellular Ca2+ concentration ([Ca2+]i), which are dr

99 nerate synchronous oscillations in beta cell intracellular Ca2+ concentration ([Ca2+]i), which lead t

100 are both triggered by a postsynaptic rise in intracellular Ca2+ concentration ([Ca2+]i).

101 m 5 to 40 mM evoked a 98+/-12 nM increase in intracellular Ca2+ concentration ([Ca2+]i).

102 ysia californica causes an increase in their intracellular Ca2+ concentration ([Ca2+]i).

103 , and a corresponding transient rise in free intracellular Ca2+ concentration ([Ca2+]i).

104 , or are up-regulated, by an increase in the intracellular Ca2+ concentration ([Ca2+]i).

105 KC-theta) and an appropriately elevated free intracellular Ca2+ concentration ([Ca2+]i).

106 s requires CFTR and a cAMP-dependent rise in intracellular Ca2+ concentration ([Ca2+]i).

107 position and activity of FDSs and on global intracellular Ca2+ concentration ([Ca2+]i).

108 rane current, cell shortening and changes in intracellular Ca2+ concentration ([Ca2+]i).

109 ith fluo-3-acetoxymethyl ester to quantitate intracellular Ca2+ concentrations ([Ca2+]i) by spectrofl

110 Intracellular Ca2+ concentrations ([Ca2+]i) in prolifera

111 scle cells (VSMCs) is poorly understood, but intracellular Ca2+ concentrations ([Ca2+]i) in the 2 cel

112 d PACAP-dependent elevation of both cAMP and intracellular Ca2+ concentrations ([Ca2+]i).

113 staglandin F(2alpha) (PGF(2alpha)) increases intracellular Ca2+ concentration [Ca2+]i in vascular smo

114 edict, based on these findings, that at high intracellular Ca2+ concentrations, Ca2+-synaptotagmin I

115 t fertilisation, repetitive increases in the intracellular Ca2+ concentration, [Ca2+]i, drive the com

116 alize changes in mitochondrial potential and intracellular Ca2+ concentration, [Ca2+]i, in thick slic

117 sigargin also induces a biphasic rise in the intracellular Ca2+ concentration, [Ca2+]i, which is comp

118 eased glycolytic flux, ATP-to-ADP ratio, and intracellular Ca2+ concentration) can dramatically enhan

119 ty of ATF1 and CREB to respond to changes in intracellular Ca2+ concentrations depending on differenc

120 ukotriene C4 generation or alter C5a-induced intracellular Ca2+ concentration elevations.

121 la densa exhibit spontaneous oscillations in intracellular Ca2+ concentration, enhanced by tubular fl

122 Both agonists inhibited the increase in free intracellular Ca2+ concentration evoked by depolarizatio

123 ccurs over 10-15 msec and linearly increases intracellular Ca2+ concentrations for up to 40 action po

124 ors, multiple measurements cannot be made of intracellular Ca2+ concentration from the same cell usin

125 Insulin also elicits increases in intracellular Ca2+ concentration in beta cells but has m

126 Sphingosine 1-phosphate (S1P) increases intracellular Ca2+ concentration in many cell types, but

127 4,5-trisphosphate significantly elevated the intracellular Ca2+ concentration in mouse morula-stage e

128 h the amplitude of Ca2+ oscillations and the intracellular Ca2+ concentration in perimacular cortical

129 receptors) and increased the peak change in intracellular Ca2+ concentration in response to this lip

130 icromol/L), hydrogen peroxide did not affect intracellular Ca2+ concentration in subconfluent, indo 1

131 Measurement of intracellular Ca2+ concentrations in Fura2-loaded cells

132 e of 2-APB on capacitative calcium entry and intracellular Ca2+ concentrations in rat basophilic leuk

133 d the spontaneous elevations of [Ca2+]i (the intracellular Ca2+ concentration) in neurons and induced

134 , which, in turn, regulated the differential intracellular Ca2+ concentration increase across the gro

135 The mechanism by which intracellular Ca2+ concentrations increase remains unkno

136 In intact HEK 293 cells, increases in the intracellular Ca2+ concentration induced by ionomycin, a

137 imilarly, carbachol-induced elevation of the intracellular Ca2+ concentration inhibited NO-stimulated

138 Although an increased intracellular Ca2+ concentration is expected to reduce g

139 ls by neuronal activity-dependent changes in intracellular Ca2+ concentration may contribute to short

140 cemaker cells, an increase in subsarcolemmal intracellular Ca2+ concentration occurred concomitantly

141 At intracellular Ca2+ concentrations of 250 nm, current act

142 sensor capable of responding to increases in intracellular Ca2+ concentration over the narrow dynamic

143 4-calmodulin is responsive to changes in the intracellular Ca2+ concentration over the physiological

144 n LLC-PK1 cells by preventing an increase in intracellular Ca2+ concentration, potentiating ERK activ

145 fter estradiol addition, a transient rise in intracellular Ca2+ concentration precedes eNOS transloca

146 By contrast, increases in intracellular Ca2+ concentration provoked by cell depola

147 In noncontractile cells, increases in intracellular Ca2+ concentration serve as a second messe

148 Fura-2 measurements of intracellular Ca2+ concentrations showed that the releas

149 sures, large SMI-32(+) neurons showed higher intracellular Ca2+ concentrations than most spinal neuro

150 ss preconditioning prevented the increase in intracellular Ca2+ concentration that normally follows H

151 PMN provoked rapid and transient changes in intracellular Ca2+ concentrations that were blocked by t

152 vates salvage purinergic pathways that raise intracellular Ca2+ concentration to stimulate an alterna

153 erstimulation of NMDA receptors may increase intracellular Ca2+ concentrations to lethal levels in ne

154 normal physiological responses (increases in intracellular Ca2+ concentrations) to acid taste stimuli

155 -attached patch conditions or an increase in intracellular Ca2+ concentration under inside-out patch

156 Perturbations in normal intracellular Ca2+ concentrations underlie many common p

157 se agents using fluorometric measurements of intracellular Ca2+ concentration, unidirectional Mn2+ en

158 Basal intracellular Ca2+ concentration was equivalent in Fura-

159 Inactivation was also reversed when the intracellular Ca2+ concentration was lowered to 100 nM a

160 in HF.We induced HF by tachypacing in sheep; intracellular Ca2+ concentration was measured in voltage

161 The intracellular Ca2+ concentration was raised by high gluc

162 express GJs, GJs mediated the propagation of intracellular Ca2+ concentration waves in supporting cel

163 rane Cl- channels were blocked or changes in intracellular Ca2+ concentration were prevented with a C

164 d apoptosis, even if associated increases in intracellular Ca2+ concentration were prevented.

165 Intracellular Ca2+ concentrations were optically recorde

166 it excitability based on variations of their intracellular Ca2+ concentrations, which leads to glutam

167 ptosis was largely blocked by increasing the intracellular Ca2+ concentration with NMDA (N-methyl-D-a