ライフサイエンスコーパス: intracellular calcium

1 tion caused by frequency-induced increase in intracellular calcium.

2 ges in membrane potential and an increase in intracellular calcium.

3 caused a rapid and synergistic elevation of intracellular calcium.

4 esponse to environmental stimuli to increase intracellular calcium.

5 ence of the betaAR agonist isoproterenol and intracellular calcium.

6 oxic leading to ER-stress and an increase in intracellular calcium.

7 enced by Grx6 activity is the homeostasis of intracellular calcium.

8 ) mice showed no hypoxia-induced increase of intracellular calcium.

9 onged oscillations in membrane potential and intracellular calcium.

10 GluA1 which is dependent on alpha7-nAChR and intracellular calcium.

11 tion and invasion, likely through changes in intracellular calcium.

12 ediated currents and the ensuing increase in intracellular calcium.

13 LCgamma1, Akt, MAPK p38, and the increase of intracellular calcium.

14 Application of Ang(1-7) had no effect on intracellular calcium.

15 A shortage of ATP also causes a rise in intracellular calcium.

16 ly inhibiting glutamate-induced increases in intracellular calcium.

17 n, apoptosis, proliferation and increases in intracellular calcium.

18 sine kinases, p38 MAPK, phospholipase C, and intracellular calcium.

19 regulators of NRG3 signaling: (1) release of intracellular calcium, (2) activation of the BACE1 beta-

20 Most importantly, experimental increase in intracellular calcium abolished Flunarizine's effect.

21 ic acid acetoxymethyl ester, an inhibitor of intracellular calcium abundance, blocked BMP-2-induced t

22 of action potentials and consequent rises of intracellular calcium activity ([Ca(2+)]i).

23 ism of alpha7 on cytoskeletal growth via the intracellular calcium activity of the receptor channel a

24 Intracellular calcium acts as a secondary messenger in a

25 ucible and reversible transient increases in intracellular calcium, allowing the generation of a conc

26 However, the automatization of global intracellular calcium analysis at the single-cell level

27 ion accompanied by 2.1-fold increase in free intracellular calcium and a corresponding increase in th

28 by sustained increases in concentrations of intracellular calcium and adenosine 3',5'-cyclic monopho

29 us positive feedback loop involving elevated intracellular calcium and enhanced mGluR1 function, a me

30 cence resonance energy transfer reporter for intracellular calcium and examined calcium flux both in

31 ated with a period of sustained elevation of intracellular calcium and formation of larger and more h

32 and contribute to pathological elevations of intracellular calcium and increased oxidative stress ass

33 s CCL5 and the HIV-1 gp120 protein increased intracellular calcium and induced growth of human and hu

34 Intracellular calcium and membrane potential were evalua

35 protein-alpha-mediated signaling, mobilizing intracellular calcium and Nf-kappaB signaling, leading t

36 lement attack limits sustained elevations in intracellular calcium and prevents mitochondrial injury.

37 ssion of genes involved in the regulation of intracellular calcium and proliferation, and preventing

38 Blue/green produced a bigger increase in intracellular calcium and reactive oxygen species (ROS).

39 We hypothesize that the feedback of intracellular calcium and sodium concentrations ([Na](i)

40 We measured resting intracellular calcium and store-operated calcium entry (

41 lectrical activity leading to an increase in intracellular calcium, and cause exocytosis of glucagon.

42 increased phosphatidylserine exposure, high intracellular calcium, and elevated osmotic fragility.

43 lls, we imaged ciliary beat frequency (CBF), intracellular calcium, and nitric oxide (NO).

44 e on both total [(3)H]inositol phosphate and intracellular calcium, and to induce DNA fragmentation a

45 In response to increased intracellular calcium, ANO4-transfected cells triggered

46 Thus, this study reveals intracellular calcium as a key regulator of NMD and has

47 that matrix deprivation leads to a spike in intracellular calcium as well as oxidant signaling, and

48 cell imaging coupled with temporally precise intracellular calcium buffering.

49 termined by the frequency of oscillations of intracellular calcium (Ca(2+) ) concentration.

50 termined by the frequency of oscillations of intracellular calcium (Ca(2+) ) concentration.

51 tion rate, and diminished the sensitivity to intracellular calcium (Ca(2+) ) in G protein-induced exo

52 uples the calcium-sensing receptor (CaSR) to intracellular calcium (Ca(2+) i) signaling, lead to fami

53 nel is remarkably sensitive to inhibition by intracellular calcium (Ca(2+) i) through binding of Ca(2

54 Intracellular calcium (Ca(2+)) alternans is a dynamical

55 ic oxide (NO)-cyclic nucleotide (CN)-coupled intracellular calcium (Ca(2+)) homeostasis that enhances

56 Ca(2+) entry (SOCE) mediates the increase in intracellular calcium (Ca(2+)) in endothelial cells (ECs

57 ons across cellular membranes in response to intracellular calcium (Ca(2+)) levels.

58 -R1 with CP-154526 caused an accumulation of intracellular calcium (Ca(2+)) over time and cell death.

59 The type-1 ryanodine receptor (RyR1) is an intracellular calcium (Ca(2+)) release channel required

60 ositol 1,4,5-triphosphate receptor-dependent intracellular calcium (Ca(2+)) release.

61 Intracellular calcium (Ca(2+)) signaling resulting from

62 Additionally, elevated intracellular calcium (Ca(2+)) stimulated Anxa6-pro-ANP

63 activated receptors (PAR) leads to increased intracellular calcium (Ca(2+)).

64 tors for neurotransmitters that can increase intracellular calcium (Ca(2+)).

65 rom vascular signals covary with oscillatory intracellular calcium (Ca(2+)i) and with local field pot

66 rom 7.4 to 7.2 rapidly inhibited CaR-induced intracellular calcium (Ca(2+)i) mobilization, whereas ra

67 Intracellular calcium ([Ca(2)(+)]i) signaling mediates p

68 (RyR), plays a major role in agonist-induced intracellular calcium ([Ca(2+)]cyt) dynamics in vascular

69 arized membrane potentials or with decreased intracellular calcium ([Ca(2+)]i) and recovered with dep

70 exchanger (NCX3) is crucial for maintaining intracellular calcium ([Ca(2+)]i) homeostasis in excitab

71 Our results show a rise in basal intracellular calcium ([Ca(2+)]i) in response to applica

72 The effects of intracellular calcium ([Ca(2+)]i) on this channel are on

73 normal brain oxygenation with elevations in intracellular calcium ([Ca(2+)]i).

74 Resting intracellular calcium ([Ca(2+)]r) and sodium ([Na(+)]r)

75 e of metabotropic glutamate receptor-induced intracellular calcium (Ca2+) waves, small-conductance K+

76 Spatio-temporal dynamics of intracellular calcium, [Ca2+]i, regulate the contractile

77 Using cryo-SEM, we show that the intracellular calcium carbonate deposits are contained i

78 2 (RyR2) macromolecular complex, which is an intracellular calcium channel and abundant in the brain.

79 slocate into cells and potently activate the intracellular calcium channel type 1 ryanodine receptor

80 Although intracellular calcium chelation almost completely blocke

81 Intracellular calcium chelation also inhibited EGF-induc

82 Attenuation of the calcium signal by intracellular calcium chelation significantly reduced ep

83 AChR antagonist methyllycaconitine (MLA) and intracellular calcium chelator BAPTA.

84 Addition of an intracellular calcium chelator or an AMPK inhibitor to e

85 n was required to accelerate reloading of an intracellular calcium compartment before each heartbeat.

86 POINTS: For the heart to function as a pump, intracellular calcium concentration ([Ca(2+) ]i ) must i

87 Transient elevations in intracellular calcium concentration ([Ca(2+)]i) and migr

88 , a specific alpha7 nAChR agonist, increases intracellular calcium concentration ([Ca(2+)]i) mainly r

89 In resistance arteries, coupling a rise of intracellular calcium concentration ([Ca(2+)]i) to endot

90 his was followed by a sustained elevation of intracellular calcium concentration ([Ca(2+)]i) which co

91 ted that hemichannel activity depends on the intracellular calcium concentration and is associated wi

92 mechanotransduction is often an increase in intracellular calcium concentration associated with intr

93 filament, but inhibits contractility at high intracellular calcium concentration by disrupting the ac

94 dary to reduced ROS levels and reduced basal intracellular calcium concentration compared with mock c

95 shing the SR calcium store, the evolution of intracellular calcium concentration during a train of lo

96 the hyperpolarization-activated current and intracellular calcium concentration in both normal contr

97 thick filament stress but are independent of intracellular calcium concentration in the physiological

98 ing large, transient, localized increases in intracellular calcium concentration near the calcium-con

99 Furthermore, the intracellular calcium concentration of isolated neuroepi

100 lex is a molecular switch that ties shifting intracellular calcium concentration to association and d

101 Also, the lysoPC-induced increase in intracellular calcium concentration was inhibited in ECs

102 Changes of intracellular calcium concentration were involved not on

103 e to 4-CMC or caffeine, similar increases in intracellular calcium concentration were observed in Sta

104 osphorylation of TCRzeta, ZAP70, and LAT and intracellular calcium concentration, as well as IL-2 gen

105 uscle cell membrane, a transient increase of intracellular calcium concentration, binding of calcium

106 virus for cell fusion induced an increase in intracellular calcium concentration, causing premature o

107 sed F-actin content, and increased the basal intracellular calcium concentration.

108 he transformation of membrane potential into intracellular calcium concentration.

109 thymocyte exhibited persistent elevations in intracellular calcium concentration.

110 ating is dictated by membrane voltage (Vm ), intracellular calcium concentrations ([Ca(2+) ]i ) and e

111 n neurons enabled simultaneous monitoring of intracellular calcium concentrations ([Ca(2+)]i) in mult

112 ond to mechanical stimulation with increased intracellular calcium concentrations and increased inwar

113 AChRs), and our data suggest that changes in intracellular calcium concentrations triggered by nAChR

114 Our findings suggest a key role for intracellular calcium cycling and excitation-transcripti

115 s study, we present a computational model of intracellular calcium cycling in three-dimensions (3-D),

116 Intracellular calcium cycling is a vital component of ca

117 maintain transcription of genes that control intracellular calcium cycling.

118 Calpains are a family of intracellular, calcium-dependent cysteine proteases invo

119 al of extracellular calcium, or chelation of intracellular calcium did not normalize the differences

120 lar calcium buffering system that determines intracellular calcium diffusion and influences the spati

121 Knocking down PMCA2 increases intracellular calcium, disrupts interactions between HER

122 Model results show that agonist-induced intracellular calcium dynamics can be modified by changi

123 The model reproduces intracellular calcium dynamics during control pacing and

124 escence filter were developed to capture the intracellular calcium dynamics in response to the activa

125 The intracellular calcium dynamics of the outgrowing cells w

126 Intracellular calcium dynamics of ventricular myocytes i

127 ltered levels of SERCA, IP3R, and RyR on the intracellular calcium dynamics of VSMC and to understand

128 onses with respect to their contributions to intracellular calcium dynamics, testing the 'unifying hy

129 isation of these structures tightly controls intracellular calcium dynamics.

130 d multi-photon optical microscopy imaging of intracellular calcium dynamics.

131 lipid scramblase 1 (PLSCR1) activity reduces intracellular calcium dysregulation, prevents PtdSer ext

132 Leptin increased intracellular calcium, elevated calmodulin and calmoduli

133 t pieces of living human cells, resulting in intracellular calcium elevation and eventual cell death.

134 which extracellular signals elicit prolonged intracellular calcium elevation to drive changes in fund

135 ing factor and MAPK activities, elevation of intracellular calcium, extrusion of a second polar body,

136 antly more severe, showing increased rise of intracellular calcium, faster loss of function, and high

137 n gene in HIV; TAT-4BB) affected LPS-induced intracellular calcium flux and excitation in sensory neu

138 Cgamma2 phosphorylation for the induction of intracellular calcium flux and the subsequent activation

139 r of NF-kappa-B ligand-induced activation of intracellular calcium flux in vitro.

140 engagement of TLR7 in CD4(+) T cells induced intracellular calcium flux with activation of an anergic

141 by altering IP3 receptor phosphorylation and intracellular calcium flux, and activating calcium-depen

142 trans retinoic acid, we measured chemotaxis, intracellular calcium flux, and alpha4beta7-mediated cel

143 that GCs suppress CCR9-mediated chemotaxis, intracellular calcium flux, and alpha4beta7-mediated cel

144 effect of isoproterenol on histamine-induced intracellular calcium flux, and significantly attenuates

145 ted membrane currents or by instabilities in intracellular calcium fluxes.

146 line activation of alpha7 promotes a rise in intracellular calcium from local ER stores via Galphaq s

147 culum calcium ATPase (SERCA) establishes the intracellular calcium gradient across the sarcoplasmic r

148 leotide targeted pathways linked to abnormal intracellular calcium handling and cardiac neurotransmis

149 NAome, here we identify miRNAs that suppress intracellular calcium handling in heart muscle by intera

150 tractility of heart muscle cells by boosting intracellular calcium handling might be an effective the

151 erlying physiological and pathophysiological intracellular calcium handling phenomena at the whole-ce

152 bited distinct cardiac dysfunction, dampened intracellular calcium handling, alterations in cardiac m

153 on, abnormal electrophysiology, dysregulated intracellular calcium handling, and proarrhythmic behavi

154 improvements were correlated with changes in intracellular calcium handling, resulting in increased n

155 pathway with no need to postulate defects in intracellular calcium handling.

156 genes encoding ion channels/pumps that alter intracellular calcium homeostasis and cause renin-indepe

157 These factors combined lead to disruption of intracellular calcium homeostasis and isoproterenol-indu

158 ace these new developments in the context of intracellular calcium homeostasis and signaling.

159 n central nervous system, where it regulates intracellular calcium homeostasis in response to excitat

160 These data link defects in neuronal intracellular calcium homeostasis to the vulnerability o

161 anges in the autophagic/lysosomal system and intracellular calcium homeostasis, which underlie vulner

162 gh excessive cation influx and disruption of intracellular calcium homeostasis.

163 d mRNA expression of B2R and the increase of intracellular calcium (iCa) in response to bradykinin we

164 TRPM4 is activated by increased intracellular calcium in a voltage-dependent manner but,

165 IDs are associated with a strong increase of intracellular calcium in astrocytes and neurons.

166 e treatment caused a significant decrease in intracellular calcium in MDCK cells.

167 or indirectly by affecting ion channels and intracellular calcium in particular.

168 hymal arteriole tone significantly increased intracellular calcium in perivascular astrocyte processe

169 alcium-sensing receptor (CASR), and mobilize intracellular calcium in response to CASR activation.

170 dynamics, highlighting the critical role of intracellular calcium in shaping the pERK1/2 signal.

171 BjIP increased intracellular calcium in ventricular cardiomyocytes and

172 store-operated calcium entry contributes to intracellular calcium increase, leading to reactive oxyg

173 ies were able to antagonize chemerin-induced intracellular calcium increase.

174 Intracellular calcium increases induced by TRPV4 agonist

175 un-protonated and TMEM16A is activated when intracellular calcium increases; however, under acidic c

176 nduces a mitochondrion-dependent increase in intracellular calcium, indicative of cellular signaling.

177 tion model and transduction with fluorescent intracellular calcium indicator, GCaMP3, we nondestructi

178 so report that SMF stimulation increases the intracellular calcium influx in OPCs as well as the gene

179 podocytes can occur as a result of excessive intracellular calcium influx, and we have previously sho

180 nsequence of ER stress in SMCs was increased intracellular calcium ion concentration, resulting in in

181 of articular cartilage via the generation of intracellular calcium ion transients.

182 nodol are complex diterpenoids that modulate intracellular calcium-ion release at ryanodine receptors

183 Maintaining low intracellular calcium is essential to the functioning of

184 The oscillating concentration of intracellular calcium is one of the most important examp

185 ssing cells showed a significant increase in intracellular calcium level (p < 0.05), impaired AKT1 an

186 ot analysis revealed that Rap2B elevates the intracellular calcium level and further promotes extrace

187 tilbene (TMS) which selectively elevated the intracellular calcium level in gefitinib-resistant (G-R)

188 thlut might be due to its ability to inhibit intracellular calcium level increases, as well as nuclea

189 ation methods, as expected, induced rises in intracellular calcium levels and also triggered the coor

190 logic mechanisms, including dysregulation of intracellular calcium levels and cAMP signaling, mediate

191 Aberrant intracellular calcium levels and increased cAMP signalin

192 The effect of the compounds on MC intracellular calcium levels and nuclear factor kappaB a

193 ciated with the highest basal and stimulated intracellular calcium levels and with increased cellular

194 Using a DNA polymerase to record intracellular calcium levels has been proposed as a nove

195 r cheek skin, (ii) acidified buffer elevated intracellular calcium levels in dorsal root ganglion pru

196 ing oocyte maturation, and yet, manipulating intracellular calcium levels interferes with first-polar

197 aling cascade and demonstrate that a rise in intracellular calcium levels is sufficient to modulate t

198 We demonstrate that increased intracellular calcium levels lead to histone hyperacetyl

199 nally show that the LKB1/CaMKK-AMPK axis and intracellular calcium levels play a critical role in anc

200 As adenosine is known to cause changes in intracellular calcium levels upon addition to cell cultu

201 ched in the brain and neurons that regulates intracellular calcium levels via signaling through the i

202 er resting conditions, with no difference in intracellular calcium levels, hydrogen peroxide (H2 O2 )

203 pound 130038) causes an increase in parasite intracellular calcium levels, leading to a calcium-depen

204 logical processes that include disruption in intracellular calcium levels, so amelioration of the cal

205 al growth factor receptor and an increase in intracellular calcium levels, under the permissive contr

206 ansient actin reset in response to increased intracellular calcium levels.

207 associated with an M3R-mediated increase in intracellular calcium levels.

208 plasma membrane and subsequent elevation of intracellular calcium levels.

209 nction was determined with tissue myography, intracellular calcium measurements, and regulatory myosi

210 ion of Galphaq-coupled CysLT1, and sustained intracellular calcium mobilisation and extracellular sig

211 3 and 10 +/- 0.18 mug/ml, respectively) with intracellular calcium mobility similar to amlodipine.

212 Since platelet intracellular calcium mobilization [Ca(t)]i controls gra

213 dose-dependently inhibited CP55,940-induced intracellular calcium mobilization and [(35)S]GTP-gamma-

214 fficacy as an antagonist of chemerin induced intracellular calcium mobilization and a much higher pot

215 s and in coordinating biased agonism between intracellular calcium mobilization and ERK1/2 phosphoryl

216 ng through S1P2 and S1P3 receptors activated intracellular calcium mobilization and extracellular sig

217 IL-1beta had no detectable effect on intracellular calcium mobilization or endothelial cell v

218 Survival promotion by OXB required intracellular calcium mobilization via inositol-1,4,5-tr

219 Increased intracellular calcium mobilization was observed in B cel

220 d that transcriptional changes indicative of intracellular calcium mobilization were significantly ov

221 islets resulted in a significant increase in intracellular calcium mobilization, an effect that was b

222 luding Fc receptor gamma-chain signaling and intracellular calcium mobilization.

223 gulation of calcium homeostasis; the resting intracellular calcium of extensor digitorum longus and s

224 Lowering SERCA level will enable intracellular calcium oscillations at low agonist concen

225 c development is initiated by sperm-mediated intracellular calcium oscillations, followed by activati

226 nd RyR need higher agonist concentration for intracellular calcium oscillations.

227 We also find that a ring of elevated intracellular calcium overlaps the region where membrane

228 shared arrhythmia mechanism, consistent with intracellular calcium overload and triggered activity.

229 t a unifying hypothesis whereby depletion of intracellular calcium pools by crude oil-derived PAHs di

230 eceptor (C5aR) and loaded with a fluorescent intracellular calcium probe: Fura-2 AM.

231 mechano- stimulation of these cells induced intracellular calcium propagation in both cell types; in

232 lular alkalinization was dependent on C5aR1, intracellular calcium, protein kinase C, and calmodulin,

233 serum treatment via a mechanism dependent on intracellular calcium, protein kinase C, and phosphatidy

234 gy transfer (FRET), confocal microscopy, and intracellular calcium quantitation.

235 The recirculation fraction, which indexes intracellular calcium recycling, was also depressed in S

236 sphate receptor (ITPR3) is the most abundant intracellular calcium release channel in cholangiocytes.

237 gets ryanodine receptors (RyRs), a family of intracellular calcium release channels essential for man

238 ed assay measuring inhibition of UTP-induced intracellular calcium release in 1321N1 astrocytoma cell

239 logical activity of SDF1-ELP, as measured by intracellular calcium release in HL60 cells was dose dep

240 f calcium-activated chloride conductances by intracellular calcium release is the key factor underlyi

241 ecently been demonstrated that ATP dependent intracellular calcium release leads to an increase of ne

242 jects and evaluated their ability to inhibit intracellular calcium release mediated by angiotensin II

243 ites where cisternal organelles, specialized intracellular calcium release membranes, come into close

244 is blocked by BAPTA chelation, and recruits intracellular calcium release on its way to activation o

245 een CMs, triggering membrane depolarization, intracellular calcium release, and actomyosin contractio

246 membrane voltage changes and then triggering intracellular calcium release.

247 The increase of intracellular calcium requires Cav1.3a channels, as a Ca

248 e cavitation-induced injury while evoking an intracellular calcium response, may be particularly usef

249 S1P elicited heightened intracellular calcium responses and enhanced S1P-trigger

250 However, the analysis of global intracellular calcium responses both at the single-cell

251 quantitation of cholesterol composition and intracellular calcium responses to CCK.

252 y in vitro, without influencing NMDA-induced intracellular calcium responses.

253 When the dosing of oligomers was stopped the intracellular calcium returned to basal levels or below.

254 s work establishes an important role for the intracellular calcium signal in the induction of EMT in

255 results showed that neuronal CALHM1 controls intracellular calcium signaling and cell excitability, t

256 alistic biophysical model of glutamate-based intracellular calcium signaling in astrocytes, we sugges

257 1 activity through phospholipase C (PLC) and intracellular calcium signaling in vivo.

258 r translocation of MEF2C was associated with intracellular calcium signaling induced by beta-catenin.

259 In both cell types, intracellular calcium signaling that links membrane depo

260 Detecting intracellular calcium signaling with fluorescent calcium

261 Systems as varied as blood clotting, intracellular calcium signaling, and tissue inflammation

262 The process relies on intracellular calcium signaling, PDZ [postsynaptic densi

263 ated calcium entry is a central regulator of intracellular calcium signaling.

264 requires activation of protein kinase C and intracellular calcium signaling.

265 expression of CysLT1 in LUVA cells augmented intracellular calcium signalling induced by LTE4 but did

266 Intracellular calcium signalling was recorded to compare

267 We show that intracellular calcium signals are critical for the regul

268 nels to independently modulate the resulting intracellular calcium signals in a physiologically relev

269 ith the generation and modulation of the key intracellular calcium signals that initiate and control

270 Using in vivo two-photon imaging of intracellular calcium signals, we measure the receptive

271 face sarcolemma and transverse-tubules), the intracellular calcium store (the sarcoplasmic reticulum)

272 BCR signaling by LPA5 manifests by impaired intracellular calcium store release and most likely by i

273 Since intracellular calcium stores are finite and readily exha

274 ory responses depend on calcium release from intracellular calcium stores, and run down rapidly at re

275 rane potentials (RMPs) reflects depletion of intracellular calcium stores, while mAChR-driven excitat

276 action molecule 1 (STIM1) after depletion of intracellular calcium stores.

277 d reinforcing signals mediated by mGluRs and intracellular calcium stores.

278 onent of the action potential is the rise in intracellular calcium that activates both small conducta

279 diated, IP3 receptor-dependent elevations of intracellular calcium that gated surface-membrane calciu

280 stress (200 mum H2 O2 ) for 20 min increased intracellular calcium to 4-fold greater levels in endoth

281 ly protonated and works synergistically with intracellular calcium to activate the channel.

282 s with dose-dependent detrimental effects on intracellular calcium transient amplitude, contractility

283 llected to assess sympathetic postganglionic intracellular calcium transients ([Ca(2+) ]i ) and immun

284 the main factor responsible for the reduced intracellular calcium transients and contractility in VS

285 Intracellular calcium transients are a universal phenome

286 e present an instrument capable of recording intracellular calcium transients from the majority of ne

287 revealed a 4-fold faster decay of ATP-evoked intracellular calcium transients than GCaMP6f.

288 is dependent on and occurs coordinately with intracellular calcium transients, which are tightly asso

289 oincident with those of mutants deficient in intracellular calcium transporters, such as the Golgi Pm

290 Increased intracellular calcium up-regulated ERK1/2 via calmodulin

291 Intracellular calcium was higher after 420 nm and 540 nm

292 Intracellular calcium was higher after blue/green, and c

293 A sustained aggregation of intracellular calcium was observed upon these stimuli, w

294 Phenylephrine-stimulated free intracellular calcium was significantly higher in the RI

295 llular calcium concentration associated with intracellular calcium waves (ICWs) in various physiologi

296 Since estrogen regulates intracellular calcium, we investigated the interaction b

297 tein kinase (AMPK) is regulated, in part, by intracellular calcium, we postulated that AMPK participa

298 In rods, light triggers a decline in intracellular calcium, which exerts a well studied negat

299 , governing ITPKC protein levels and thereby intracellular calcium, which in turn regulates NLRP3 exp

300 Buffering intracellular calcium with EGTA-AM or BAPTA-AM reduced a