ライフサイエンスコーパス: intracellular store

1 tly triggered mobilization of Ca(2+) from an intracellular store.

2 um, or inhibiting re-uptake of calcium by an intracellular store.

3 ay associated with rapid Ca(2+) release from intracellular stores.

4 ium channels or on calcium mobilization from intracellular stores.

5 d to the PLC pathway and Ca(2+) release from intracellular stores.

6 ed signaling upstream of Ca(2+) release from intracellular stores.

7 oval of extracellular Ca(2+) or depletion of intracellular stores.

8 t pathway) that stimulates Ca2+ release from intracellular stores.

9 hat the source of the Ca(2+) is release from intracellular stores.

10 channels, or mobilizing Ca(2+) release from intracellular stores.

11 4,5-triphosphate-stimulated Ca2+ efflux from intracellular stores.

12 nodine receptor-induced calcium release from intracellular stores.

13 but enhances stimulated Ca(2+) release from intracellular stores.

14 are a family of calcium release channels on intracellular stores.

15 associated transient release of Ca(2+) from intracellular stores.

16 ther dependent on the release of Ca(2+) from intracellular stores.

17 ulating factor induced BLyS release from the intracellular stores.

18 h Ca2+ influx, rather than Ca2+ release from intracellular stores.

19 (CRAC) channels following Ca2+ release from intracellular stores.

20 neurons and can mediate Ca(2+) release from intracellular stores.

21 elevation of cAMP or release of Ca(2+) from intracellular stores.

22 pathway, and reduce the Ca(2+)content of the intracellular stores.

23 4,5-trisphospate (IP3) and release Ca2+ from intracellular stores.

24 Ca2+ into the cytosol from extracellular or intracellular stores.

25 onists through augmented Ca(2+) release from intracellular stores.

26 the apical membrane from latent preexisting intracellular stores.

27 (2+) despite the limited available Ca(2+) in intracellular stores.

28 zyme secretion and is largely mobilized from intracellular stores.

29 trisphosphate and the release of Ca(2+) from intracellular stores.

30 il surface was rapidly replaced by CD93 from intracellular stores.

31 here they mediate the release of Ca(2+) from intracellular stores.

32 cium, showing that WIN releases calcium from intracellular stores.

33 es a rise in cytosolic Ca(2+) from undefined intracellular stores.

34 2+ transients by returning cytosolic Ca2+ to intracellular stores.

35 on through TRPM8 channels or by release from intracellular stores.

36 n to control the rate of ER Ca(2+) leak from intracellular stores.

37 conductance through release of calcium from intracellular stores.

38 ing the MAG source, due to Ca2+ release from intracellular stores.

39 -trisphosphate and mobilization of Ca2+ from intracellular stores.

40 plasma membrane often releases calcium from intracellular stores.

41 ysis of drugs that prevent Ca2+ release from intracellular stores.

42 usion via a PMCA and Ca2+ transport from the intracellular stores.

43 phosphate, which causes release of Ca2+ from intracellular stores.

44 LTX(N4C) involves mobilization of Ca2+ from intracellular stores.

45 sitol 1,4,5-trisphosphate receptor-sensitive intracellular stores.

46 at the initial increase in [Ca2+](i) is from intracellular stores.

47 ion via a PMCA and Ca(2+) transport from the intracellular stores.

48 vation of IP(3) mediated Ca(2+) release from intracellular stores.

49 ch is mediated by the release of Ca(2+) from intracellular stores.

50 he level of G17-stimulated Ca2+ release from intracellular stores.

51 (i) increase involves release of Ca(2+) from intracellular stores.

52 an signal by activating calcium release from intracellular stores.

53 P3 receptors, and release of Ca(2+) from the intracellular stores.

54 kainic acid or mobilization of calcium from intracellular stores.

55 flux to the cytosol from the apoplast and/or intracellular stores.

56 n previously, to trigger Ca(2+) release from intracellular stores.

57 by phospholipase C and release of Ca 2+ from intracellular stores.

58 nduced by IP(3)-mediated Ca(2+) release from intracellular stores.

59 mGluRs), and involved release of Ca(2+) from intracellular stores.

60 ough voltage-gated channels, or release from intracellular stores.

61 e cells is due to the release of Ca(2+) from intracellular stores.

62 igargin, suggesting it involves release from intracellular stores.

63 ombin-stimulated release of [Ca(2+)](i) from intracellular stores.

64 ch enhanced release of calcium from parasite intracellular stores.

65 c reticulum that allow efflux of Ca(2+) from intracellular stores.

66 IP3 receptor-regulated calcium release from intracellular stores.

67 the release and sequestration of Ca(2+) from intracellular stores.

68 l in mouse microglia was due to release from intracellular stores.

69 us, suggesting the bulk of the protein is in intracellular stores.

70 /or stimulate the efflux of transmitter from intracellular stores.

71 ar calcium influx or release of calcium from intracellular stores.

72 2Y1 receptor-mediated release of Ca(2+) from intracellular stores.

73 flux and Ca(2+) -induced Ca(2+) release from intracellular stores.

74 llular Ca(2+) influx and Ca(2+) release from intracellular stores.

75 by Ca(2+)-induced Ca(2+) release (CICR) from intracellular stores.

76 type VDCCs, or block of calcium release from intracellular stores.

77 it apoptosis and promote Ca(2+) release from intracellular stores.

78 provide counter ions for Ca(2+) handling in intracellular stores.

79 ic channels that mediate Ca(2+) release from intracellular stores.

80 cium influx rather than calcium release from intracellular stores.

81 spholipase C and release of calcium from the intracellular stores.

82 ositol 1,4,5-trisphosphate (IP(3))-sensitive intracellular stores.

83 hose activation is dependent on depletion of intracellular stores.

84 +), but at a lower dose released Ca(2+) from intracellular stores.

85 ), critical signals for calcium release from intracellular stores.

86 ignaling mobilize peptide:MHC-II export from intracellular stores.

87 hrombin-induced release of calcium ions from intracellular stores.

88 d and/or RyR1-independent Ca(2)(+) leak from intracellular stores.

89 s probably triggered by the depletion of the intracellular stores, a mechanism known as store-operate

90 schaemia: Ca(2+) influx, Ca(2+) release from intracellular stores, actin filament depolymerization, a

91 te hypoxia, by causing Ca2+ release from the intracellular stores, activates CCE in isolated canine P

92 CR to determine whether calcium release from intracellular stores affected action potential waveform,

93 ntaneous calcium (Ca(2+)) release (SCR) from intracellular stores after the end of a preceding action

94 2 activation and that release of Ca(2+) from intracellular stores alone, in the absence of extracellu

95 pulating calcium uptake into or release from intracellular stores also modulated the level of reverbe

96 phate production and release of calcium from intracellular stores also were augmented after myo-inosi

97 hat is shaped by the Ca(2+) release from the intracellular store and extracellular Ca(2+) influx.

98 DPR are calcium messengers that can mobilize intracellular stores and activate influx as well.

99 phate (IP3)-mediated release of calcium from intracellular stores and activation of protein kinase C

100 ion, as cADPR regulates calcium release from intracellular stores and ADPR controls cation entry thro

101 s (and/or glia) to evoke Ca(2+) release from intracellular stores and an increase in ventilation that

102 c levels of Bax can reduce Ca(2+) content in intracellular stores and Ca(2+) homeostasis.

103 activation-induced Ca(2+) mobilization from intracellular stores and Ca(2+) influx from the extracel

104 tor, which causes the release of Ca(2+) from intracellular stores and Ca(2+)-dependent activation of

105 induced Ca2+ transients involve release from intracellular stores and Ca2+ signaling is essential for

106 sible and were mediated by Ca2+ release from intracellular stores and calcineurin-mediated Kv2.1 deph

107 es release of calcium from the IP3-sensitive intracellular stores and calcium-calmodulin formation.

108 lifies the CRTH2-induced Ca(2+) release from intracellular stores and coincidentally forfeits its own

109 d the Ca(2+) content of ionomycin-releasable intracellular stores and decreased endoplasmic reticulum

110 l types are triggered by Ca(2+) release from intracellular stores and driven by store-operated Ca(2+)

111 cal agents that alter Ca2+ mobilization from intracellular stores and experiments involving injection

112 Calcium arising through release from intracellular stores and from influx across the plasma m

113 -entry process activated by the depletion of intracellular stores and has an important role in many c

114 in promoting the mobilization of lipids from intracellular stores and in the liver for assembling VLD

115 estingly, BMP-2 induced calcium release from intracellular stores and increased calcineurin phosphata

116 pecific channels mediating Ca2+ release from intracellular stores and influx across the plasma membra

117 both cases was due to release of Ca(2+) from intracellular stores and influx of extracellular Ca(2+).

118 riant responsible for Ca2+ mobilization from intracellular stores and influx through voltage-gated Ca

119 Produced by release of Ca2+ from intracellular stores and mediated by type 2 and perhaps

120 is related to more efficient FV release from intracellular stores and microparticle production driven

121 Intracellular stores and mitochondria were not involved

122 tween the physiological release of Ca2+ from intracellular stores and opening of Ca2+ influx channels

123 esses essential for LTD-calcium release from intracellular stores and phosphatase activation-were abn

124 phate, leading to the release of Ca(2+) from intracellular stores and protein kinase C (PKC) activati

125 required to induce depletion of Ca(2+) from intracellular stores and provide insights into the abili

126 edistribution of transporter protein between intracellular stores and the plasma membrane.

127 receptors leads both to calcium release from intracellular stores and to dendritic protein synthesis.

128 s of the BCM model, calcium as released from intracellular stores and triggered by M1 muscarinic acet

129 volved the release of Ca2+ from IP-sensitive intracellular stores and was expressed via the internali

130 clic ADP-ribose evoked a slow Ca2+ leak from intracellular stores and were able to increase the Ca2+-

131 bilization from caffeine/ryanodine-sensitive intracellular stores and were not due to inositol 1,4,5-

132 different Ca(2+) signatures from the Ca(2+) intracellular stores and whether the amplitude of Ca(2+)

133 he route of Ca(2+) entry (i.e., release from intracellular stores and/or influx across the plasma mem

134 ed by PLC activation, release of Ca(2+) from intracellular stores, and activation of store-operated C

135 and ryanodine receptors) release Ca(2+) from intracellular stores, and by depleting the stores trigge

136 ptotic stimuli, reduce the Ca(2+) content of intracellular stores, and regulate Ca(2+) fluxes.

137 el were [Ca(2+)] in the cytosol, [Ca(2+)] in intracellular stores, and smooth muscle membrane potenti

138 problem concerns the mechanism by which the intracellular stores are refilled subsequent to depletio

139 location of G protein-coupled receptors from intracellular stores are unexplored.

140 ligand-gated channels that release Ca2+ from intracellular stores--are emerging as key sites for regu

141 rough gap junctions and calcium release from intracellular stores as mediators of collective gradient

142 sphosphate-mediated Ca(2+) mobilization from intracellular stores as well as decreased beta-cell Ca(2

143 affected the initial release of Ca(2+) from intracellular stores as well as sustained Ca(2+) levels.

144 Release of Ca(2+) from intracellular stores at fertilization of mammalian eggs

145 +) influx for triggering Ca(2+) release from intracellular stores at presynaptic terminals during in

146 To understand the properties of these intracellular stores better we used pharmacological appr

147 e lipo-oxygenase blockers released Ca2+ from intracellular stores but this was not associated with su

148 the release from or re-uptake of Ca(2+) into intracellular stores but, through protein kinase G, both

149 ateaus are not mediated by Ca2+ release from intracellular stores, but rather by an NMDA-dependent sm

150 ors (IP3Rs) release calcium ions, Ca2+, from intracellular stores, but their roles in mediating Ca2+

151 LR2 ligands stimulate release of Ca(2+) from intracellular stores by activating TLR2 phosphorylation

152 or, demonstrated the liberation of zinc from intracellular stores by peroxynitrite.

153 ase demonstrated the liberation of zinc from intracellular stores by peroxynitrite.

154 er, neither stimulation of Ca2+ release from intracellular stores by photolysis of caged IP3, nor exp

155 ) influx by ionomycin or Ca(2+) release from intracellular stores by thapsigargin alone failed to ind

156 eceptor-coupled phospholipase C activates an intracellular store calcium channel, the IP(3)R.

157 ed calcium channels (VGCCs) and release from intracellular stores, causing an increase in cAMP-respon

158 hydrolase (CEH) catalyzes the hydrolysis of intracellular stored cholesteryl esters (CEs) and thereb

159 They also suggest that Ca2+ release from intracellular stores contributes significantly to increa

160 PC5) and Na+/Ca2+ exchanger, indicating that intracellular store deficiency was compensated for by Ca

161 oplasmic reticulum Ca(2+) sensor that senses intracellular store depletion and migrates to plasma mem

162 ors for voltage control of Ca2+ release from intracellular stores during inositol lipid signalling.

163 osphate (NAADP)-mediated Ca(2+) release from intracellular stores during stimulus-secretion coupling

164 Ca(2+) release from intracellular stores, elicited by caffeine or carbachol,

165 lux from extracellular space or release from intracellular stores, eliminates fast inactivation induc

166 cascades, resulting in calcium release from intracellular stores, ERK1/2 activation, and long term c

167 harmacologically induced Ca(2+) release from intracellular stores evoked a significant extracellular

168 s respond is through release of calcium from intracellular stores followed by store refilling from ex

169 ntracellular Ca2+ caused by its release from intracellular stores, followed by Ca2+ influx, possibly

170 cess is the excessive release of Ca(2+) from intracellular stores, followed by excessive entry of Ca(

171 calcium increase-the release of calcium from intracellular stores following activation of an IP3-depe

172 +) and decreased mobilization of Ca(2+) from intracellular stores following ionophore addition.

173 ar molecule, which is rapidly mobilized from intracellular stores following treatment with interferon

174 endocytic recycling compartment are critical intracellular stores for the rapid recycling of internal

175 eutrophils, both on the cell membrane and in intracellular stores; however, BLyS release from each of

176 h TZDs increased calcium concentrations from intracellular stores; however, only ciglitazone produced

177 However, the concentration of Ca(2+) in the intracellular stores (i.e., mitochondria and granules) w

178 in induces a modest increase in calcium from intracellular stores, IBTU was unable to block the respo

179 ation; (ii) BCR-mediated Ca(2+) release from intracellular stores; (iii) Ca(2+) entry from the extrac

180 nist caffeine evoked Ca(2+) release from the intracellular store in activated T cells but not in rest

181 ansient increase in [Ca(2+)](i) derived from intracellular stores in a Na(+)-rich environment.

182 ) formation and consequent Zn2+ release from intracellular stores in cerebrocortical neurons.

183 i) and the calcium that can be released from intracellular stores in HEp-2 or VAX-3 cells overexpress

184 x induces a transient release of Ca(2+) from intracellular stores in human neutrophils.

185 a role for the release of calcium ions from intracellular stores in mediating spatially compartmenta

186 t demonstration of mobilization of Ca2+ from intracellular stores in neurons by depolarization withou

187 ld enhancement in the release of Ca(2+) from intracellular stores in response to agents that release

188 IP(3)Rs) regulate the release of Ca(2+) from intracellular stores in response to IP(3).

189 rkA mobilization to the plasma membrane from intracellular stores in response to proton challenge.

190 modulin (CaM) regulates calcium release from intracellular stores in skeletal muscle through its asso

191 s, and depends on functional IP(3)-sensitive intracellular stores in support cells.

192 The nicotine-induced release of Ca(2+) from intracellular stores in T cells did not require extracel

193 n dependence of exocytic Ca(2+) signaling on intracellular stores in these cells.

194 second messenger for mobilizing Ca(2+) from intracellular stores in various cell types.

195 enerating metabolites that release Ca2+ from intracellular stores, including nicotinic acid adenine d

196 trocytic processes and found to arise via an intracellular store-independent process.

197 ncentration ([Ca(2+)]i) mainly released from intracellular stores, indicating that alpha7 nAChR is fu

198 ryotes, GAAPs regulate the Ca(2+) content of intracellular stores, inhibit apoptosis, and promote cel

199 presented that constitutive Ca(2+) flux from intracellular stores into mitochondria is required for b

200 e the rapid release of calcium (Ca(2+)) from intracellular stores into the cytosol, which is essentia

201 he hippocampus release calcium (Ca(2+)) from intracellular stores intrinsically and in response to ac

202 (V(m)) and the release of calcium (Ca) from intracellular stores is a crucial ingredient of heart fu

203 a2+-ATPase (SERCA)-mediated Ca2+ uptake into intracellular stores is also accelerated by PKC activati

204 Ca2+ signaling from intracellular stores is also essential for ERK1/2 activa

205 Postsynaptic release of Ca(2+) from intracellular stores is an important means of cellular s

206 a2+]i caused by the release from presynaptic intracellular stores is coincident with an enhancement o

207 rough voltage-gated calcium channels or from intracellular stores is critical for proper AChE inserti

208 n: an initial transient release of Ca2+ from intracellular stores is followed by a sustained phase of

209 Spontaneous Ca(2+) release from intracellular stores is important for various physiologi

210 The release of Ca from intracellular stores is key to cardiac muscle function;

211 that receptor-regulated release of Ca2+ from intracellular stores is likely to be involved.

212 little or opposite effects, but release from intracellular stores is required for maximal acceleratio

213 T-3, indicating that release of calcium from intracellular stores is required.

214 cells, receptor-induced Ca(2+) release from intracellular stores is usually accompanied by sustained

215 h Ca2+ influx, rather than Ca2+ release from intracellular stores, is essential for growth factor-ind

216 y frequency involves Ca(2+) recruitment from intracellular stores leading to increases in intracellul

217 ic receptor and stimulates Ca2+ release from intracellular stores, leading to the enhancement of syna

218 , in addition to gating calcium release from intracellular stores, mAChR activation facilitates volta

219 tential channels in addition to release from intracellular store mechanisms.

220 llows rapid measurement of Ca2+ release from intracellular stores mediated by IP3R.

221 found throughout the brain, where they show intracellular store-mediated Ca(2+) signals.

222 ets that could be manipulated to enhance the intracellular store of A3 and potentially enhance A3 ant

223 l array implicates mitochondria as the major intracellular store of Ca2+ involved in IR-evoked respon

224 e conditions, wild-type cells accumulated an intracellular store of Mg that supported growth under de

225 ells not only express NKp80 but also contain intracellular stores of AICL colocalizing with the Golgi

226 t, thapsigargin, is dependent on emptying of intracellular stores of Ca(2+) and the ensuing influx of

227 Inositol trisphosphate mobilizes intracellular stores of Ca(2+), resulting in photorecept

228 haemic sIPSCs were not affected by depleting intracellular stores of calcium or by blocking the neuro

229 icromol/L) caused profound Ca2+ release from intracellular stores of intact or permeabilized cells.

230 has been shown to trigger Ca2+ release from intracellular stores of invertebrate eggs and mammalian

231 Intracellular stores of MHC class I partially localized

232 Intracellular stores of MMP12 are mobilized to macrophag

233 human airway cell Duox activity by depleting intracellular stores of NADPH, as it generates intracell

234 nge of abilities to mobilize Ca(2+) from the intracellular stores of permeabilized hepatocytes and ar

235 ed an enhanced capacity to produce acid from intracellular stores of polysaccharides, could grow fast

236 with tumor necrosis factor alpha to mobilize intracellular stores of receptor.

237 Due to its rapid turnover, the intracellular stores of SOCS3 seem insufficient to contr

238 Thus, replenishing the intracellular stores of SOCS3 with CP-SOCS3 effectively

239 ated in a convertase-independent manner from intracellular stores of the complement component C3.

240 clopiazonic acid, which mobilize Ca(2+) from intracellular stores of the endoplasmic reticulum, evoke

241 We found that dead cells not only released intracellular stores of uric acid but also produced it i

242 molecule can mobilize release of Ca(2+) from intracellular stores on the endoplasmic reticulum.

243 to bFGF without affecting Ca2+ release from intracellular stores or 1-oleoyl-2-acetyl-sn-glycerol-in

244 eatments that prevented release of Ca2+ from intracellular stores or activation of PKA blocked ATPgam

245 cal neurons by sustained Ca(2+) release from intracellular stores or by a brief episode of oxygen and

246 control mechanism in which Ca2+ release from intracellular stores plays a central role.

247 ly that cAMP potentiates Ca(2+) release from intracellular stores, primarily because of a protein kin

248 es were found to be due to mobilization from intracellular stores rather than by Ca(2+) entry.

249 obably reflects mobilization of [Ca2+]i from intracellular stores rather than entry via voltage-gated

250 gh voltage-gated channels or by release from intracellular stores, reduced primarily the NMDA compone

251 rocessing of caspases-3 and -7 occurred when intracellular store release was the sole Ca(2+) perturba

252 stimuli of cytosolic [Ca(2+)] elevation and intracellular store release.

253 Ca(2+) mobilization from intracellular stores represents an important cell signal

254 Caffeine-induced Ca(2+) release from intracellular stores resulted in translocation of the C-

255 d transient rise in [Ca(2+)](i), mostly from intracellular stores, simultaneously with a drop in pH(i

256 Ca(2+) release from intracellular stores specifically through inositol 1,4,5

257 mission-evoked calcium (Ca(2+)) release from intracellular stores stabilizes dendrites during the per

258 PLC-gamma2, and of Ca(2+) mobilization from intracellular stores, stimulated by muH chain crosslinki

259 lcium channels and calcium mobilization from intracellular stores strongly implicates a role for calc

260 ate sufficient to induce Ca(2+) release from intracellular stores substantially activate TRPC3.

261 in response to agents that release Ca2+ from intracellular stores (such as arachidonic acid, C2-ceram

262 In contrast, the nature of the intracellular stores targeted by NAADP and the molecular

263 enesis; and stimulate release of Ca(2+) from intracellular stores that appears independent of IP(3) a

264 and dGal-1 independently release Ca(2+) from intracellular stores that cooperate to induce optimal re

265 oth forms of LTD involve Ca(2+) release from intracellular stores, the Ca(2+) sensors involved are di

266 renoceptors and mGluRs mobilize calcium from intracellular stores, the mechanisms and pools of calciu

267 te receptors and the release of calcium from intracellular stores, the NPY-IR puncta fuse with the ce

268 nstitutively between the plasma membrane and intracellular stores, thereby providing a protected rece

269 ymphocytes triggers the release of Ca2+ from intracellular stores; this release of Ca2+ results in th

270 l Ca2+ release, the graded Ca2+ release from intracellular stores through inositol 1,4,5-trisphosphat

271 r responses by inducing calcium release from intracellular stores through its preferred neurokinin 1

272 s calcium influx and release of calcium from intracellular stores to activate ERK1/2.

273 y on the controlled release of Ca(2)(+) from intracellular stores to activate stage-specific Ca(2)(+)

274 ) channels that regulate Ca(2+) release from intracellular stores to control a diverse array of cellu

275 calcium channels of the plasma membrane and intracellular stores to determine what sources of calciu

276 mic reticulum to promote Ca(2+) release from intracellular stores to increase the concentration of cy

277 cose transporter-4 (GLUT4) redistribute from intracellular stores to the cell periphery in response t

278 te receptor, AMPAR, can rapidly traffic from intracellular stores to the plasma membrane, altering ne

279 GLUT4 facilitative glucose transporter from intracellular stores to the plasma membrane.

280 cation of the glucose transporter GLUT4 from intracellular stores to the plasma membrane.

281 hain fatty acid (LCFA) transporter CD36 from intracellular stores to the sarcolemma.

282 Mobilization of this increased CXCL1 from intracellular stores to the venular surface triggered be

283 , a protein that "gates" Ca(2+) release from intracellular stores, triggers Ca(2+) cell influx and th

284 receptor type 1 (RyR1) releases Ca(2+) from intracellular stores upon nerve impulse to trigger skele

285 ion leads to robust Ca(2+) mobilization from intracellular stores via activation of phospholipase C a

286 lified by Ca(2+)-induced Ca(2+) release from intracellular stores via activation of ryanodine recepto

287 lly, ouabain induced Ca(2+) release from the intracellular stores via the activation of IP3 receptors

288 hate (PIP2) hydrolysis and Ca2+ release from intracellular stores via the phospholipase C (PLC)-inosi

289 An initial Ca(2+) mobilization from intracellular stores was followed by movement of extrace

290 Carbachol-mediated release of Ca(2+) from intracellular stores was significantly higher in Bax tra

291 Release of Zn(II) from intracellular stores was validated in human epithelial p

292 a2+]i by releasing Ca2+ from buffer sites or intracellular stores, we examined in detail the effect o

293 ents (Ca2+ syntillas) caused by release from intracellular stores were found in isolated nerve termin

294 secretion dependent upon Ca(2+) release from intracellular stores, whereas sustained secretion requir

295 , arising from both extracellular medium and intracellular stores, which induces the activation of ad

296 in B lymphocytes causes Ca(2+) release from intracellular stores, which, in turn, activates ion chan

297 tors (GPCRs) that cause calcium release from intracellular stores while other stimuli depolarize tast

298 Depletion of intracellular stores with 10 mM caffeine also significan

299 The discharge of intracellular stores with thapsigargin stimulated mTORC1

300 ary for the release of free fatty acids from intracellular stores within neutrophil precursor cells.