ライフサイエンスコーパス: intracranial aneurysm

1 ith FMD with intracranial imaging who had an intracranial aneurysm.

2 hese 86 patients, 25 (53.8%) had more than 1 intracranial aneurysm.

3 (12.9%; 95% CI, 10.3%-15.9%) had at least 1 intracranial aneurysm.

4 five putative risk loci are associated with intracranial aneurysm.

5 associated with arterial diseases, including intracranial aneurysm.

6 he pathophysiology, diagnosis and therapy of intracranial aneurysm.

7 , as well as those with PKD1, are at risk of intracranial aneurysm.

8 ween 3.6 and 6% of the population harbour an intracranial aneurysm.

9 culature to study the molecular pathology of intracranial aneurysms.

10 ts in whom the coils were implanted to treat intracranial aneurysms.

11 rior cerebral artery is a common location of intracranial aneurysms.

12 diction of the risk of rupture of incidental intracranial aneurysms.

13 reasonably safe and effective treatment for intracranial aneurysms.

14 of surgical and endovascular treatments for intracranial aneurysms.

15 ascular and surgical treatments for ruptured intracranial aneurysms.

16 ed in management of patients with unruptured intracranial aneurysms.

17 the spindle-shaped cells of the wall of the intracranial aneurysms.

18 in 32 of 74 patients (43.2%), and 24 of 128 intracranial aneurysms (18.8%) were in the posterior com

19 In 199 consecutive patients with 234 clipped intracranial aneurysms, 273 intraoperative angiographic

20 oking history vs 163 of 564 patients without intracranial aneurysm (28.9%; P < .001).

21 of smoking was significantly associated with intracranial aneurysm: 42 of 78 patients with intracrani

22 Intracranial aneurysms 5 mm or larger occurred in 32 of

23 ntracranial aneurysm: 42 of 78 patients with intracranial aneurysm (53.8%) had a smoking history vs 1

24 ion in PKD1 is predictive for development of intracranial aneurysms (59 mutations are more commonly a

25 h TAAD alone, along with families with TAAD, intracranial aneurysms, abdominal aortic and bilateral i

26 SAH, mostly due to rupture of an intracranial aneurysm, accounts for a quarter of cerebro

27 One cause of stroke, intracranial aneurysm, affects approximately 2% of the p

28 s from the International Study of Unruptured Intracranial Aneurysms aimed to assess the natural histo

29 to craniotomy and clipping for some ruptured intracranial aneurysms, although the relative benefits o

30 These findings identify a Mendelian form of intracranial aneurysm and map the location of the underl

31 of ADPKD, most significant of which include intracranial aneurysms and cystic liver diseases.

32 ed data on the natural history of unruptured intracranial aneurysms and on the morbidity and mortalit

33 manifestations include an increased level of intracranial aneurysms and polycystic liver disease (PLD

34 We enrolled 2143 patients with ruptured intracranial aneurysms and randomly assigned them to neu

35 The International Study of Unruptured Intracranial Aneurysms and the International Subarachnoi

36 to assess the natural history of unruptured intracranial aneurysms and to measure the risk associate

37 ecause they included individuals with either intracranial aneurysms and/or very-early-onset disease.

38 ey disease (ADPKD) are at risk of developing intracranial aneurysms, and subarachnoid haemorrhage is

39 Embolization of intracranial aneurysms, angioplasty and stenting of extr

40 In specimens of intracranial aneurysms, aortic dissections, and dolichoe

41 Saccular intracranial aneurysms are balloon-like dilations of the

42 brain arteriovenous malformations (AVM) and intracranial aneurysms are both intracranial vascular di

43 rrhage and surgical obliteration of ruptured intracranial aneurysms are frequently associated with ne

44 Unruptured intracranial aneurysms are increasingly being detected a

45 c stroke, carotid and intracranial stenosis, intracranial aneurysms, arteriovenous malformations, mal

46 a large kindred that segregates nonsyndromic intracranial aneurysm as a dominant trait with high pene

47 arachnoid hemorrhage secondary to a ruptured intracranial aneurysm, at our institution.

48 ent's knowledge that they have an unruptured intracranial aneurysm can lead to substantial stress and

49 Intracranial aneurysms can be treated with endovascular

50 ch and Japanese cohorts including over 2,100 intracranial aneurysm cases and 8,000 controls.

51 sign of subarachnoid haemorrhage, unruptured intracranial aneurysm, cerebral venous sinus thrombosis,

52 ies affecting the cerebrovasculature include intracranial aneurysms, cervical artery dissection, fibr

53 To assess ferromagnetic properties of intracranial aneurysm clips reported to be nonferromagne

54 -dominant polycystic kidney disease, such as intracranial aneurysms, cluster in families.

55 The presence of intracranial aneurysm did not vary with location of extr

56 vational studies of patients with unruptured intracranial aneurysms, discharge outcomes were better a

57 reatment are the most important drawbacks of intracranial aneurysm embolization.

58 nt are being used for patients with ruptured intracranial aneurysms: endovascular detachable-coil tre

59 They play a vital role in the assessment of intracranial aneurysms, especially in evaluation of the

60 In patients with a ruptured intracranial aneurysm, for which endovascular coiling an

61 the recent International Study of Unruptured Intracranial Aneurysms found that the rupture rate of sm

62 Intracranial aneurysms from patients without ADPKD also

63 Intracranial aneurysms have been reported in PKD2, as we

64 The pathogenesis of intracranial aneurysm (IA) formation and rupture is comp

65 As endovascular approaches to intracranial aneurysm (IA) treatment continue to evolve,

66 omewide screen of 48 sib pairs affected with intracranial aneurysms (IAs) that revealed suggestive li

67 bdominal aortic aneurysms (AAAs) but not for intracranial aneurysms (IAs), and that hypertension is a

68 y disease (ADPKD) have a higher incidence of intracranial aneurysms (ICA) than the general population

69 lycystic kidney disease (ADPKD) and saccular intracranial aneurysms (ICA), the risk of MRA-defined gr

70 The prevalence of intracranial aneurysm in patients with fibromuscular dys

71 though the clinical benefit of screening for intracranial aneurysm in patients with FMD has yet to be

72 or endovascular coiling after rupture of an intracranial aneurysm in the follow-up of the Internatio

73 The prevalence of intracranial aneurysm in women diagnosed with FMD is sig

74 To examine the prevalence of intracranial aneurysm in women diagnosed with FMD.

75 erformed prospectively 10 years after EVT of intracranial aneurysms in a single institution.

76 showing strong evidence for association with intracranial aneurysms in the combined dataset, includin

77 sensitivity and specificity for detection of intracranial aneurysms, including small aneurysms, in pa

78 technique are presented in the management of intracranial aneurysms, including: preoperative aneurysm

79 Intracranial aneurysm is a common neurological problem;

80 The rupture of an intracranial aneurysm is a sudden devastating event with

81 Once an unruptured intracranial aneurysm is detected, decisions regarding o

82 EVT of intracranial aneurysm is effective for prevention of lon

83 The pathogenesis of intracranial aneurysm is unknown, and because catastroph

84 The management of unruptured intracranial aneurysms is controversial.

85 572); venous thromboembolism (n(e) = 4,607); intracranial aneurysm (n(e) = 1,328); CAD (n(e) = 12,716

86 normal elastic and intracranial arteries and intracranial aneurysms obtained at autopsy from patients

87 Surgery for intracranial aneurysm often results in postoperative neu

88 lies with multiple members that had TAAD and intracranial aneurysms or TAAD and intracranial and abdo

89 nous thromboembolism (OR: 0.97; p = 0.63) or intracranial aneurysm (OR: 0.85; p = 0.15).

90 lation, early angiogenesis, atherosclerosis, intracranial aneurysm, or interstitial flow.

91 In patients with ruptured intracranial aneurysms, rates of aneurysm obliteration w

92 types, including coronary artery disease and intracranial aneurysms (rs10757278 and rs1333040).

93 al analysis of consecutive attendances at an intracranial aneurysm screening clinic.

94 In clinical practice, people undergoing intracranial aneurysm screening had stronger family hist

95 e: (1) The International Study of Unruptured Intracranial Aneurysms studied the natural history and t

96 Familial clustering of intracranial aneurysms suggests that genetic factors are

97 In patients with ruptured intracranial aneurysms suitable for both treatments, end

98 The only known risk factor was an intracranial aneurysm that was found on her grandmother'

99 itive, specific studies for the diagnosis of intracranial aneurysms that are of sufficient size to be

100 Small unruptured intracranial aneurysms (UIAs) are increasingly diagnosed

101 Unruptured intracranial aneurysms (UIAs) are increasingly diagnosed

102 go, the treatment of incidentally discovered intracranial aneurysms was straightforward with a good e

103 2143 patients with ruptured intracranial aneurysms were enrolled between 1994 and 20

104 When intracranial aneurysms were identified, people were refe

105 2143 patients with ruptured intracranial aneurysms, who were admitted to 42 neurosur

106 um of 14 years after treatment of a ruptured intracranial aneurysm with either neurosurgical clipping

107 nd 9p that show significant association with intracranial aneurysm with odds ratios 1.24-1.36.

108 y continues to evolve which now implies that intracranial aneurysms with complex vascular architectur

109 ness and safety of endovascular treatment of intracranial aneurysms with the use hydrogel coils.