ライフサイエンスコーパス: intracranial pressure

1 added only if hypothermia failed to control intracranial pressure.

2 en with either large hemorrhage or increased intracranial pressure.

3 provement in survival, regardless of initial intracranial pressure.

4 OCT were correlated with invasively measured intracranial pressure.

5 t and an over 50% reduction effect on raised intracranial pressure.

6 minute-by-minute mean arterial pressure and intracranial pressure.

7 o why glaucoma develops in patients with low intracranial pressure.

8 of successfully treated episodes of elevated intracranial pressure.

9 than mannitol for the treatment of elevated intracranial pressure.

10 ve intraventricular blood, and management of intracranial pressure.

11 as the primary intervention to reduce raised intracranial pressure.

12 CT parameters and directly measured elevated intracranial pressure.

13 that microgravity reduces central venous and intracranial pressure.

14 , correlated with CT-edema and preceded peak intracranial pressure.

15 ity (95% CI, 41%-79%) for detecting elevated intracranial pressure.

16 patients with acute brain injury and raised intracranial pressure.

17 if all stage 2 treatments failed to control intracranial pressure.

18 ncreased inflammatory response and to reduce intracranial pressure.

19 osmotherapy) were added as needed to control intracranial pressure.

20 . 24+/-2 mmHg, p=0.006) but had no effect on intracranial pressure (14+/-1 vs. 15+/-1 mmHg, p=0.72).

21 Complications in CNS disease included raised intracranial pressure (42%), hydrocephalus (30%), neurol

22 group, fewer episodes of critically elevated intracranial pressure (92 vs. 167, p = .027) in fewer pa

23 ertension exposure: area under the curve for intracranial pressure above 20 mm Hg (area under the cur

24 s had fewer hours than medical patients with intracranial pressure above 25 mm Hg after randomization

25 The complexity index of intracranial pressure achieved the strongest statistical

26 w, cerebral perfusion pressure, and elevated intracranial pressure after fluid percussion brain injur

27 g, embolic stroke) or indirectly (eg, raised intracranial pressure after head injury).

28 CNS requires aggressive management of raised intracranial pressure along with standard antifungal the

29 ic participants had the largest increases in intracranial pressure (AMS present, Delta7mmHg, 95% CI =

30 ry, closed traumatic brain injury; increased intracranial pressure; an initial head injury less than

31 of patients had at least one episode of high intracranial pressure and 36% had a highest mean intracr

32 yr) with acute CNS infections having raised intracranial pressure and a modified Glasgow Coma Scale

33 gnificant relationship between the change in intracranial pressure and AMS symptom severity (R(2) = 0

34 f perfusion could contribute to increases in intracranial pressure and an associated impairment of vi

35 Intracranial pressure and arterial blood pressure wavefo

36 Waveforms of intracranial pressure and arterial blood pressure, basel

37 ons in each arm were specified and impact on intracranial pressure and brain tissue oxygenation measu

38 raumatic brain injury informed by multimodal intracranial pressure and brain tissue oxygenation monit

39 Treatment was guided by controlling intracranial pressure and cerebral perfusion pressure ac

40 sing brain tissue oxygenation in addition to intracranial pressure and cerebral perfusion pressure mo

41 high frequency correlated significantly with intracranial pressure and cerebral perfusion pressure, b

42 arming at a rate compatible with maintaining intracranial pressure and cerebral perfusion pressure.

43 uration astronauts develop signs of elevated intracranial pressure and have neuro-ophthalmological fi

44 negative intrathoracic pressure would lower intracranial pressure and increase cerebral perfusion, t

45 al perfusion pressures and lowered diastolic intracranial pressure and intracranial pressure rate dur

46 y two routinely monitored signals as inputs (intracranial pressure and mean arterial blood pressure)

47 erating curve; adding dynamic information of intracranial pressure and mean arterial pressure during

48 Adding information of the first 24 hrs of intracranial pressure and mean arterial pressure monitor

49 ng the dynamic characteristics of continuous intracranial pressure and mean arterial pressure monitor

50 adding dynamic characteristics of continuous intracranial pressure and mean arterial pressure monitor

51 a life-threatening disorder causing elevated intracranial pressure and papilledema.

52 forts should be made to aggressively control intracranial pressure and select a proper donor to minim

53 Continuous monitoring of intracranial pressure and temperature illustrates functi

54 erformed to evaluate the percent decrease in intracranial pressure and the 95% confidence intervals,

55 dies do not support the speculation that low intracranial pressure and the resulting pressure-depende

56 Cerebral blood flow, pial artery diameter, intracranial pressure, and autoregulatory index were det

57 e relationships between autonomic functions, intracranial pressure, and cerebral autoregulation.

58 Cerebral microdialysis, brain tissue oxygen, intracranial pressure, and cerebral blood flow were meas

59 nuous monitoring of arterial blood pressure, intracranial pressure, and cerebral perfusion pressure,

60 rmalities in intracerebral ventricle volume, intracranial pressure, and CSF electrolyte levels.

61 = 0.078; control vs 4-hr delay, p = 0.150), intracranial pressure, and microdialysis values.

62 intracranial pressure, and partial brain tissue oxygenat

63 ally throbs, intensifies with an increase in intracranial pressure, and presents itself in associatio

64 s per treatment dose, quantitative change in intracranial pressure, and prespecified adverse events.

65 Conversely, intracranial pressure appears to be elevated in patients

66 Brain edema and the associated increase in intracranial pressure are potentially lethal complicatio

67 Episodes of increased intracranial pressure are secondary injuries associated

68 oreflex sensitivity, heart rate variability, intracranial pressure, arterial blood pressure, cerebral

69 lts of this study suggest that complexity of intracranial pressure assessed by multiscale entropy was

70 njury, care focused on maintaining monitored intracranial pressure at 20 mm Hg or less was not shown

71 curve, 0.74 [95% CI, 0.61-0.87]), monitoring intracranial pressure + brain tissue PO2 (area under the

72 g characteristic curve, 0.84 [0.74-0.93]) or intracranial pressure + brain tissue PO2+ cerebral micro

73 Under general anesthesia, probes to measure intracranial pressure, brain oxygen tension (PbtO2), and

74 Intracranial pressure, brain tissue PO2, and cerebral mi

75 Brain multimodal monitoring-including intracranial pressure, brain tissue PO2, and cerebral mi

76 or to mannitol for the treatment of elevated intracranial pressure, but their impact on clinical prac

77 as recorded during the stepwise elevation of intracranial pressure by inflation of an epidural balloo

78 RECENT FINDINGS: Treatment of elevations in intracranial pressure can begin at the roadside and end

79 omputed tomography to detect the position of intracranial pressure catheter was 100% and 78%.

80 ve care because of the potential increase in intracranial pressure caused by the rise in cerebral blo

81 oring modalities included digitally recorded intracranial pressure, cerebral perfusion pressure, cere

82 y in real time as well as to monitor in vivo intracranial pressure continuously in proof-of-concept m

83 random-effects models, the relative risk of intracranial pressure control was 1.16 (95% confidence i

84 Intracranial pressure control was similar in both groups

85 Intracranial pressure correlated with maximal retinal ne

86 on is safe and might reduce the frequency of intracranial pressure crises and mortality rate.

87 Our algorithm can predict the onset of intracranial pressure crises with 30-minute advance warn

88 r scale, rebleeding, global cerebral oedema, intracranial pressure crisis, pneumonia and sepsis, hype

89 he downward slope of the decompression phase intracranial pressure curve was steeper (-60.3 +/- 12.9

90 Similar mortality rates and refractory intracranial pressure deaths suggest that children with

91 Intracranial pressure did not change significantly in th

92 tic hypothermia plus standard care to reduce intracranial pressure did not result in outcomes better

93 + impedance threshold device, mean diastolic intracranial pressure during decompression was lower (12

94 tress syndrome, status asthmaticus, elevated intracranial pressure, elevated intra-abdominal pressure

95 Intracranial pressure elevation and brain water accumula

96 Intracranial pressure elevation appears to be significan

97 Intracranial pressure elevation>20 mm Hg occurred during

98 We developed a model to predict increased intracranial pressure episodes 30 mins in advance, by us

99 Increased intracranial pressure episodes could be predicted 30 min

100 ess of the model to predict future increased intracranial pressure events 30 minutes in advance, in a

101 hic opening of the lower ventricular system, intracranial pressure events>20 mm Hg remained significa

102 ankin Scale score at 30 days were percent of intracranial pressure events>30 mm Hg per patient (p=.01

103 tic therapy may reduce the frequency of high intracranial pressure events.

104 m Hg lasting at least 5 minutes and the mean intracranial pressure for every 12-hour interval were an

105 lysis estimated that the percent decrease in intracranial pressure from baseline to either 60 minutes

106 e suggested a converse causation with raised intracranial pressure giving rise to obesity.

107 ately 80%, as a result of the development of intracranial pressure gradients, brain tissue shift, and

108 Patients with highest mean intracranial pressure greater than 20 mm Hg had signific

109 Episodes of intracranial pressure greater than 20 mm Hg lasting at l

110 Highest mean intracranial pressure greater than 20 mm Hg was signific

111 an/peak intracranial pressure, proportion of intracranial pressure greater than 20 mm Hg, use of edem

112 A 90-day mortality in intracranial pressure group (38.2%) was significantly hi

113 perfusion pressure group in comparison with intracranial pressure group had significantly higher med

114 ) had evidence of intracranial hypertension (intracranial pressure > 25 mm Hg) and overall 21-day mor

115 ted intracranial pressure (new anisocoria or intracranial pressure >20 mm Hg for >/=20 mins), inhospi

116 severe intraventricular hemorrhage, although intracranial pressure >30 mm Hg predicts higher short-te

117 aumatic brain injury and refractory elevated intracranial pressure (>25 mm Hg) to undergo decompressi

118 An intracranial pressure>18 mm Hg was an exclusion criterio

119 ion pressure (CPP) concomitant with elevated intracranial pressure (ICP) after FPI were greater in ma

120 Mean arterial blood pressure, heart rate, intracranial pressure (ICP) and cerebral perfusion press

121 d treatment variables associated with raised intracranial pressure (ICP) and immune reconstitution in

122 improvement in blood pH and in parameters of intracranial pressure (ICP) and oxygenation.

123 Both intracranial pressure (ICP) and the cerebrovascular pres

124 rence between intraocular pressure (IOP) and intracranial pressure (ICP) at the level of lamina cribr

125 l is an anesthetic used for controlling high intracranial pressure (ICP) caused by brain surgery, bra

126 Intracranial pressure (ICP) control is a mainstay of tra

127 ctions did not mirror the large increases in intracranial pressure (ICP) during locomotion, indicatin

128 patient-specific in silico computer model of intracranial pressure (ICP) dynamics may predict the ICP

129 ure of the current methods for monitoring of intracranial pressure (ICP) has prevented their use in m

130 m and/or shed capsule is postulated to raise intracranial pressure (ICP) in cryptococcal meningitis (

131 Raised intracranial pressure (ICP) is common in cryptococcosis.

132 thological concepts relating to the field of intracranial pressure (ICP) monitoring and offers an up-

133 tudy is the largest on the use and effect of intracranial pressure (ICP) monitoring in pediatric trau

134 Intracranial pressure (ICP) monitoring is a mainstay of

135 Intracranial pressure (ICP), CBF, and mean arterial pres

136 itoring of arterial blood pressure (ABP) and intracranial pressure (ICP), were retrospectively analyz

137 Further, decreases in intracranial pressure (ICP), with postulated increases i

138 minal pressure may have a negative effect on intracranial pressure (ICP).

139 tis (CM) are commonly attributed to elevated intracranial pressure (ICP).

140 ertension is generally assessed by measuring intracranial pressure (ICP).

141 ss the lamina cribrosa (LC) related to a low intracranial pressure (ICP).

142 mary or secondary process relative to raised intracranial pressure (ICP).

143 ntation that resembles syndromes of elevated intracranial pressure (ICP).

144 have various causes, including disorders of intracranial pressure (ICP).

145 relationships between autonomic impairment, intracranial pressure, impaired cerebral autoregulation,

146 th acute CNS infection, management of raised intracranial pressure improves mortality and neuromorbid

147 Stage 3 treatments were required to control intracranial pressure in 54% of the patients in the cont

148 etween peak sulfonylurea receptor-1 and peak intracranial pressure in 91.7% of patients with intracra

149 re-targeted therapy for management of raised intracranial pressure in children with acute CNS infecti

150 Detecting elevated intracranial pressure in children with subacute conditio

151 nce current treatment paradigms for elevated intracranial pressure in children.

152 re no observed increases in serum lactate or intracranial pressure in either group.

153 ns to mannitol for the treatment of elevated intracranial pressure in human subjects undergoing quant

154 ric hypoxia causes elevated brain volume and intracranial pressure in individuals with symptoms consi

155 It has been suggested that a low intracranial pressure in patients with normal intraocula

156 Hypothermia reduces brain edema and intracranial pressure in patients with traumatic brain i

157 oring allows to accurately predict increased intracranial pressure in the neuro-ICU.

158 l pressure-targeted approach to treat raised intracranial pressure in these children.

159 for early detection of episodes of increased intracranial pressure in traumatic brain injury patients

160 injuries that would be predicted to increase intracranial pressure, including inflammation, head trau

161 Intracranial pressure increased to a mean maximum of 19

162 bital area and the eye, and intensifies when intracranial pressure increases.

163 n damage is influenced by the speed at which intracranial pressure increases.

164 The authors postulate that elevated intracranial pressure induces forces in the retrolaminar

165 ansion, perihaematomal oedema with increased intracranial pressure, intraventricular extension of hae

166 High intracranial pressure is a common complication in the fi

167 tension (IIH) is a condition in which raised intracranial pressure is associated with a high body mas

168 Mean intracranial pressure is associated with the severity of

169 Recent studies have shown that intracranial pressure is lower in patients with glaucoma

170 Recent studies have reported that intracranial pressure is lower in patients with NTG when

171 Intracranial pressure is not frequently elevated during

172 This being said, intracranial pressure is not reduced to the levels obser

173 Elevated intracranial pressure is one of the proposed mechanisms

174 The natural history indicates that increased intracranial pressure is transient in survivors.

175 This low intracranial pressure, leading to an abnormally high tra

176 Elevated intracranial pressure leads to structural changes in the

177 oward effects (including promoting increased intracranial pressure), little is known about the regula

178 ssure-targeted therapy (n = 55) (maintaining intracranial pressure < 20 mm Hg using osmotherapy while

179 disease, a finding that suggests that raised intracranial pressure may contribute to a fatal outcome.

180 elationship between intraocular pressure and intracranial pressure may play a fundamental role in the

181 hat increased brain volume leading to raised intracranial pressure may play a role.

182 elationship between intraocular pressure and intracranial pressure may play an important role in the

183 No significant difference in intracranial pressure, mean cerebral artery transcranial

184 ol patients, underwent direct intraoperative intracranial pressure measurement.

185 re in human subjects undergoing quantitative intracranial pressure measurement.

186 serve as an effective surrogate for invasive intracranial pressure measurement.

187 ting characteristic curve of 0.86 using only intracranial pressure measurements and time since last c

188 Outcomes included CT edema, intracranial pressure measurements, therapies targeting

189 g 112 patients with 184 episodes of elevated intracranial pressure met our inclusion criteria.

190 opriate indication underwent placement of an intracranial pressure monitor and only 134 of 335 (45.6%

191 ift of ~12 mm were most likely to receive an intracranial pressure monitor and probabilities decrease

192 The use of intracranial pressure monitor in acetaminophen acute liv

193 nophen status and adjusting for confounders, intracranial pressure monitor placement did not impact 2

194 Hemorrhagic complications from intracranial pressure monitor placement were uncommon an

195 However, intracranial pressure monitor was associated with increa

196 Intracranial pressure monitored (n = 140) versus nonmoni

197 Overall 21-day mortality was similar (intracranial pressure monitored 33% vs controls 38%, p =

198 able, hemorrhagic complications were rare in intracranial pressure monitored patients (4 of 56 [7%];

199 Forty-one percent of intracranial pressure monitored patients received liver

200 During the first 7 days, intracranial pressure monitored patients received more i

201 Intracranial pressure monitored patients were younger th

202 In intracranial pressure monitored patients with acute live

203 Of 87 intracranial pressure monitored patients with detailed i

204 outcomes whereas white race (p=0.01), use of intracranial pressure monitoring (p=0.001), and increasi

205 ion in continuous mean arterial pressure and intracranial pressure monitoring allows to accurately pr

206 Compared with intracranial pressure monitoring alone (area under the r

207 erebral microdialysis--is more accurate than intracranial pressure monitoring alone in detecting cere

208 s brain tissue oxygenation monitoring versus intracranial pressure monitoring alone.

209 vel compliance ranged from 9.6% to 65.2% for intracranial pressure monitoring and 6.7% to 76.2% for c

210 with Brain Trauma Foundation guidelines for intracranial pressure monitoring and craniotomy.

211 cal characteristics with a propensity score, intracranial pressure monitoring guideline compliance wa

212 The role of intracranial pressure monitoring has been challenged; ho

213 he complex nature of examining the effect of intracranial pressure monitoring in observational studie

214 onclusively confirm or refute the utility of intracranial pressure monitoring in patients with acute

215 Intracranial pressure monitoring is a widely but inconsi

216 Intracranial pressure monitoring is standard of care aft

217 otocol based on brain tissue oxygenation and intracranial pressure monitoring reduced the proportion

218 amount of sustained osmolar therapy without intracranial pressure monitoring suggest opportunities t

219 ular perforation rat model under guidance by intracranial pressure monitoring to investigate whether

220 Intracranial pressure monitoring was not associated with

221 ive and nonaggressive based on the frequency intracranial pressure monitoring) on outcome was assesse

222 atic brain injury (Glasgow Coma Scale </= 8; intracranial pressure monitoring).

223 mobile medical team, mechanical ventilation, intracranial pressure monitoring, vasopressors, acute ne

224 the first week of therapy, 29% did not have intracranial pressure monitoring.

225 els in those patients with an indication for intracranial pressure monitoring.

226 Intracranial-pressure monitoring is considered the stand

227 ents had a higher proportion of samples with intracranial pressure more than 20 mm Hg (13% vs 30%), b

228 acranial pressure and 36% had a highest mean intracranial pressure more than 20 mm Hg.

229 yzed the frequency of episodes with elevated intracranial pressure (new anisocoria or intracranial pr

230 ue oxygenation crisis events were defined as intracranial pressure of greater than or equal to 20 mm

231 In patients with an intracranial pressure of more than 20 mm Hg after trauma

232 We randomly assigned adults with an intracranial pressure of more than 20 mm Hg despite stag

233 ntation that resembles syndromes of elevated intracranial pressure on Earth.

234 entobarbital infusion, or markedly increased intracranial pressure on interruption of continuous seda

235 remains controversy about how best to manage intracranial pressure on the ICU; we review the recent l

236 tissue oxygenation data were recorded in the intracranial pressure -only group in blinded fashion.

237 after severe traumatic brain injury (0.45 in intracranial pressure-only group and 0.16 in intracrania

238 of patients with good recovery compared with intracranial pressure-only management; however, the stud

239 r mortality and more favorable outcomes than intracranial pressure-only treatment.

240 s complications as related to an increase in intracranial pressure or as a direct result from cranial

241 caused by the seizure disorder or increased intracranial pressure or by the underlying disorder (tha

242 eflex sensitivity showed no correlation with intracranial pressure or cerebral perfusion pressure; th

243 s between autonomic impairment and increased intracranial pressure or impaired cerebral autoregulatio

244 m to be independent of age, trauma severity, intracranial pressure, or autoregulatory status, and thu

245 rsening hydrocephalus, evidence of increased intracranial pressure, or necessity for surgical resecti

246 cs, type of intracranial pathology, baseline intracranial pressure, osms per treatment dose, quantita

247 romise as surrogate, noninvasive measures of intracranial pressure, outperforming other conventional

248 ore likely to receive treatment for elevated intracranial pressure (P < .001).

249 ure-reactivity index and complexity index of intracranial pressure (P < 0.0001; P = 0.001; P < 0.0001

250 0.048), lactate/pyruvate ratio (P = 0.044), intracranial pressure (P = 0.006) and cerebrovascular pr

251 0.024), lactate/pyruvate ratio (P = 0.016), intracranial pressure (P = 0.029), cerebrovascular press

252 g cerebral perfusion pressure (p = 0.03) and intracranial pressure (p = 0.06) seen after seizure onse

253 d mean middle cerebral artery flow velocity (intracranial pressure, PaCO2, hematocrit, sedation, feve

254 The number of patients with high intracranial pressure peaked 3 days after subarachnoid h

255 or age, initial Glasgow Coma Scale, and mean intracranial pressure, percentage of time with cerebral

256 intracranial pressure-only group and 0.16 in intracranial pressure plus brain tissue oxygenation grou

257 re randomized to treatment protocol based on intracranial pressure plus brain tissue oxygenation moni

258 al to assess impact on neurologic outcome of intracranial pressure plus brain tissue oxygenation-dire

259 values (+/- SDs) of arterial blood pressure, intracranial pressure, pressure reactivity index, and ot

260 dently of age, admission Glasgow Coma Scale, intracranial pressure, pressure reactivity index, or cer

261 s classified by the Glasgow Outcome Scale in intracranial pressure, pressure-reactivity index and com

262 rmation extraction from radiographic images, intracranial pressure processing, low back pain and real

263 n/peak sulfonylurea receptor-1 and mean/peak intracranial pressure, proportion of intracranial pressu

264 lowered diastolic intracranial pressure and intracranial pressure rate during the decompression phas

265 Percentage of intracranial pressure readings per patient>30 mm Hg and

266 Intracranial pressure readings were analyzed at predefin

267 Of 2576 intracranial pressure readings, 91.5% (2359) were </=20

268 rval, 1.00-1.33), and the difference in mean intracranial pressure reduction was 2.0 mm Hg (95% confi

269 ta-analysis of the effect of 23.4% saline on intracranial pressure reduction.

270 Elevated intracranial pressure requiring acute intervention is a

271 Elevated intracranial pressure requiring acute intervention was a

272 H due to vascular malformation, and elevated intracranial pressure requiring urgent intervention woul

273 The intracranial pressure response to hypoxia varied between

274 AMS symptomology is explained by a variable intracranial pressure response to hypoxia.

275 h post-traumatic cerebral edema and elevated intracranial pressure resulting from TBI.

276 Intracranial pressure significantly increases with abdom

277 sure-targeted approach with the conventional intracranial pressure-targeted approach to treat raised

278 py-dopamine, and if needed noradrenaline) or intracranial pressure-targeted therapy (n = 55) (maintai

279 ventilation and osmotherapy, was superior to intracranial pressure-targeted therapy for management of

280 toward an early warning system for increased intracranial pressure that can be generally applied.

281 ension is a disorder characterised by raised intracranial pressure that predominantly affects young,

282 edema develops quickly after trauma, raising intracranial pressure that results in a decrease of bloo

283 ions in the gradient between intraocular and intracranial pressures that direct the movement of fluid

284 sed a direct link between obesity and raised intracranial pressure through a specific fat distributio

285 he area under the curve from high-resolution intracranial pressure-time plots was calculated to repre

286 gs in eyes with papilledema caused by raised intracranial pressure to findings in eyes with optic dis

287 ypothermia as a first line measure to reduce intracranial pressure to less than 20 mm Hg is harmful i

288 optic nerve head (ONH) resulting from raised intracranial pressure, using high definition optical coh

289 bral blood flow, regional brain volumes, and intracranial pressure, using high-resolution magnetic re

290 injury based on brain tissue oxygenation and intracranial pressure values was consistent with reduced

291 The highest mean intracranial pressure was analyzed in relation to demogr

292 An intraparenchymal intracranial pressure was inserted.

293 al diagnosis of cerebral edema and increased intracranial pressure was made.

294 disability at 6 months were pooled, however, intracranial pressure was not an independent predictor o

295 Intracranial pressure was recorded every 4 hrs in all pa

296 At the end of the experiment, the intracranial pressure was significantly higher in c-ECPR

297 h a protocol for monitoring intraparenchymal intracranial pressure was used (pressure-monitoring grou

298 te the correlation between the complexity of intracranial pressure waveform and outcome after traumat

299 ese women, producing a syndrome of increased intracranial pressure without identifiable cause.

300 uggested that the counterbalance provided by intracranial pressure would be an important factor in th