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1 3% accurate on a dataset of 16 patients with intractable epilepsy.
2 haly, movement disorders, and/or early-onset intractable epilepsy.
3 injury (TBI) is one of the leading causes of intractable epilepsy.
4 on of dysplastic cortex for the treatment of intractable epilepsy.
5 pic cortex, and is generally associated with intractable epilepsy.
6 ng seizures in people with pharmacologically intractable epilepsy.
7 injury (TBI) and a common cause of medically-intractable epilepsy.
8 wer antiepileptic drugs for the treatment of intractable epilepsy.
9 es in 11 patients for surgical evaluation of intractable epilepsy.
10 brain development, cognitive disability, and intractable epilepsy.
11 of the corpus callosum for the treatment of intractable epilepsy.
12 sporadic, nonsyndromic patients with HH and intractable epilepsy.
13 re, benign congenital tumors associated with intractable epilepsy.
14 H tissue in 55 patients with sporadic HH and intractable epilepsy.
15 t support its use in children with treatment-intractable epilepsy.
16 ical management, particularly in the case of intractable epilepsy.
17 n patients undergoing surgical treatment for intractable epilepsy.
18 e frequency is reduced in some patients with intractable epilepsy.
19 an effective and safe therapy for medically intractable epilepsy.
20 and may be efficacious in treating medically intractable epilepsy.
21 y is a valuable option for pharmacologically intractable epilepsy.
22 vailable as adjunctive therapy for medically intractable epilepsy.
23 is a common and important cause of medically intractable epilepsy.
24 implantation for the surgical management of intractable epilepsy.
25 of lissencephaly are mental retardation and intractable epilepsy.
26 quency in animal models and in patients with intractable epilepsy.
27 r indications for the treatment of medically intractable epilepsy.
28 rted in ictal SPECT studies of patients with intractable epilepsy.
29 plied for the treatment of pharmacologically intractable epilepsy.
30 tion, may be important in the development of intractable epilepsy.
31 of surgical techniques for the treatment of intractable epilepsy.
32 (DGCs) harvested during hippocampectomy for intractable epilepsy.
33 various pathologies and are associated with intractable epilepsy.
34 language lateralization in 12 patients with intractable epilepsy.
35 ight or left temporal lobe for the relief of intractable epilepsy.
36 wake humans undergoing surgery for medically intractable epilepsy.
37 most common cause of death in patients with intractable epilepsy.
38 s from 11 patients undergoing resections for intractable epilepsy.
39 igration disorders are often associated with intractable epilepsy.
40 atients who underwent temporal lobectomy for intractable epilepsy.
41 cted deaths are more common in patients with intractable epilepsy.
42 s a process that plays a significant role in intractable epilepsies.
43 aterial from patients surgically treated for intractable epilepsy (46/57), exhibited characteristics
44 r Dravet's Syndrome), which includes severe, intractable epilepsy and comorbidities of ataxia and cog
45 orized based on the effect of callosotomy on intractable epilepsy and dichotic listening research, re
47 erapy for potential neuropeptide delivery in intractable epilepsy and possibly other neurological dis
48 pe of severe neurological symptoms including intractable epilepsy and profound neurocognitive impairm
49 a 36-year-old woman with muscular dystrophy, intractable epilepsy, and bilateral temporo-occipital li
50 l EEG data from rare patients with medically intractable epilepsy, and found evidence for respiratory
51 order associated with mental retardation and intractable epilepsy, and Miller-Dieker syndrome (MDS) i
52 lepsy, one with focal cortical dysplasia and intractable epilepsy, and one dysmorphic term infant wit
53 he comorbidities due to decades of medically intractable epilepsy; and possible acceleration of commo
54 We recorded from patients with chronically intractable epilepsy as they performed a task that requi
55 This approach is of therapeutic interest for intractable epilepsy, as it spares cortical function bet
56 onsecutive temporal resections for medically intractable epilepsy associated with mesial temporal scl
58 n 136 pediatric patients who had surgery for intractable epilepsy at The Cleveland Clinic between Jan
59 (SUDEP) is the most common cause of death in intractable epilepsies, but physiological mechanisms tha
60 Dravet syndrome (DS) is an infantile-onset intractable epilepsy caused by heterozygous loss-of-func
61 racterized by abnormal brain development and intractable epilepsy, caused similar defects in Golgi lo
62 tissue resected from patients with medically intractable epilepsy demonstrated increased adenosine ki
63 phic (ECoG) data from 15 human patients with intractable epilepsy during a word completion task and e
66 ubjects were patients with pharmacologically intractable epilepsy implanted with depth electrodes to
67 rtical resection is effective in alleviating intractable epilepsy in children with tuberous sclerosis
69 ns in doublecortin (DCX) are associated with intractable epilepsy in humans, due to a severe disorgan
70 nate dimer SPECT studies in 43 patients with intractable epilepsy in whom seizure laterality could be
71 eservation of perisomatic GABAergic input in intractable epilepsies may be a key factor in the genera
77 e young patients (mean age, 10.8 years) with intractable epilepsy of neocortical origin underwent chr
78 s a new model for studying the mechanisms of intractable epilepsy of the complex partial seizure type
79 s gestation, one with hemimegalencephaly and intractable epilepsy, one with focal cortical dysplasia
80 related to duration of epilepsy, duration of intractable epilepsy, or continuation of medications.
83 ype, including profound developmental delay, intractable epilepsy, progressive microcephaly, severe h
84 y; still, its clinical role in children with intractable epilepsy remains unclear, largely because of
86 Analysis of hippocampi from patients with intractable epilepsy revealed that Bim levels were signi
88 tions from tissue removed during surgery for intractable epilepsy, we examined the human hippocampal
89 study a research participant with medically intractable epilepsy who had extensive bilateral frontot
90 lization in 10 children and adolescents with intractable epilepsy who sustained an early lesion in th
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