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1 ar to represent a unique form of prokaryotic intracytoplasmic inclusions.
2 e first step in the formation of fibrils and intracytoplasmic inclusions.
3 ons diminish microtubule binding and lead to intracytoplasmic inclusions.
5 f the protein, leading to the development of intracytoplasmic inclusion bodies and eventually to neur
7 disorder characterized by the appearance of intracytoplasmic inclusions called Lewy bodies (LB) in d
9 sease (PD) patients progressively accumulate intracytoplasmic inclusions formed by misfolded alpha-sy
10 tification, we used laser microdissection of intracytoplasmic inclusions identified in patient muscle
11 g beta-sheet-rich fibrils that accumulate as intracytoplasmic inclusions in Parkinson disease and cer
12 ntotemporal cortices and ubiquitin-positive, intracytoplasmic inclusions in the granule cells of the
13 llmark of the disease is the accumulation of intracytoplasmic inclusions known as Lewy bodies that co
17 , and endomysial fibrosis, in the absence of intracytoplasmic inclusions, rimmed vacuoles, or any evi
18 yn by nitration can promote the formation of intracytoplasmic inclusions that constitute the hallmark
19 On routine staining, there were numerous intracytoplasmic inclusions, which ranged from eosinophi
20 y demonstrated pale pink, oval to crescentic intracytoplasmic inclusions with a predilection for peri
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