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1 ragments, only those separated in the second intradiscal and third cytoplasmic regions formed noncova
2 tion studies of rhodopsin indicate that both intradiscal and transmembrane (TM) domains are required
3 h in the C-terminal peptide sequence and the intradiscal and transmembrane domains, only a single sha
4 naturally occurring single mutations of the intradiscal cysteines (C110F, C110Y, and C187Y) are asso
5 e further the role of every one of the three intradiscal cysteines, mutants containing single-cystein
7 of these mutations are located in the second intradiscal (D2) loop, highlighting the functional impor
8 s-retinal, has been shown to be caused by an intradiscal disulfide bond different from the above nati
9 ed P23H rod opsin and other mutations in the intradiscal domain are characterized by the formation of
10 t, if not all, of the point mutations in the intradiscal domain identified in ADRP cause partial or c
11 ed to affect protein structure in the second intradiscal domain in each patient (Arg172Trp, Gly208Asp
12 fide bond between Cys-110 and Cys-187 in the intradiscal domain is required for correct folding in vi
13 integral part of a tertiary structure in the intradiscal domain of native rhodopsin coupled to a tert
14 ved suggest that the integrity of the second intradiscal domain of peripherin/RDS is critical for nor
16 fide bond between Cys-110 and Cys-187 in the intradiscal domain, we conclude from the misfolding in m
17 ting from these mutations are relayed to the intradiscal domain, where they cause misfolding of the o
22 e mutation affected mainly the "plug" at the intradiscal (extracellular) side of Rho, which is possib
23 pigmentosa (RP) point mutations in both the intradiscal (ID) and transmembrane domains of rhodopsin
24 sess the efficacy of a single glucocorticoid intradiscal injection (GC IDI) in patients with chronic
25 roup received allogeneic bone marrow MSCs by intradiscal injection of 25 x 10 cells per segment under
26 ence that may be due to extracellular versus intradiscal localization of the RDS glycan in cones vers
28 ervation of a short stretch within the large intradiscal loop also suggests an important function for
29 in structure: an empty space provided by the intradiscal loop connecting transmembrane helices 4 and
30 The rhodopsin crystal structure reveals that intradiscal loop E-2 covers the 11-cis-retinal, creating
31 cated among a cluster of RP mutations in the intradiscal loop region, mediate dose-dependent rhodopsi
33 VD degeneration is associated with an acidic intradiscal pH but the response of NP cells to this aber
34 contradistinction to previous measurements, intradiscal pressure has been determined in vivo to be g
35 t multiple switches on the extracellular (or intradiscal) side of rhodopsin trigger structural change
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