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1 eaction in ethanol-treated mice (5 g/kg/day, intragastric, 10 days + 24 hours), rat brain slice cultu
2 thanol decreased both pancreatic blood flow (intragastric, 40% decrease, p < 0.05; intravenous, 34% d
3 eeding using intravenous -[1-13C]leucine and intragastric -[5,5, 5- 2H3]leucine.
4 34% decrease, p < 0.05) and interstitial pH (intragastric, 7.24 +/- 0.04 to 7.08 +/- 0.04, p < 0.05;
5         Gastric injury was induced by either intragastric absolute ethanol (1.0 mL) or subcutaneous i
6              Injury-induced sensitization to intragastric acid administration is consistent with a po
7     In awake rats, visceromotor responses to intragastric acid are quantifiable, reliable, and reprod
8 lcers enhanced the visceromotor responses to intragastric acid.
9 cinogen N-nitrosomethylbenzylamine (NMBA) by intragastric administration and killed 12 weeks later.
10 jury in male Sprague-Dawley rats after 3-day intragastric administration of 100 mg/kg or 300 mg/kg Ge
11      In the rat mammary tumorigenesis study, intragastric administration of 2 or 4 mg of deguelin/kg
12                                     A single intragastric administration of 250 mg/kg BR931 (4-chloro
13 le s.c. injection of T3 (2 mg/kg) and single intragastric administration of 9-cis RA (40 mg/kg) or 4-
14 ingual administration of Ag/CTB conjugate or intragastric administration of a >100-fold higher dose o
15                 In this report, we show that intragastric administration of a food allergen with a mu
16          Intraperitoneal injection of CCK or intragastric administration of a trypsin inhibitor to el
17 an agonistic monoclonal anti-Fas antibody or intragastric administration of alcohol is severely blunt
18  in rats in a model involving the continuous intragastric administration of an ethanol-containing, co
19 ous knockout mice were treated with a single intragastric administration of CCl(4).
20 etions were examined in conscious rats after intragastric administration of chopped rodent chow in th
21 and gut peptide secretion.We studied how the intragastric administration of DB affects interdigestive
22                                          The intragastric administration of DB decreased hunger (P =
23                                              Intragastric administration of either antagonist at 30 m
24                          Intraperitoneal and intragastric administration of ENT at a dose of 120 nmol
25                                              Intragastric administration of ethanol (45%, 5 ml/kg) re
26 gdala of rats treated with a binge protocol (intragastric administration of ethanol 3 times daily for
27           We investigated the effects of the intragastric administration of leucine and isoleucine on
28 s of the solitary tract (NTS) in response to intragastric administration of lipid in DIO-P (C57Bl6) a
29       Subcutaneous immunization, followed by intragastric administration of MAP peptide entrapped or
30                        We showed that, after intragastric administration of MSG, the MSG is preferent
31                                     A single intragastric administration of NAF resulted in a rapid i
32 nimals, in which oral tolerance was induced, intragastric administration of OVA did not result in a s
33 cific pathogen-free or germ-free conditions, intragastric administration of Pseudomonas aeruginosa, E
34 4% of pancreatic protein secretion evoked by intragastric administration of rodent chow.
35                                              Intragastric administration of soluble protein Ags resul
36                                              Intragastric administration of solutions prepared from o
37 ion, given by voluntary (pair) feeding or by intragastric administration, affected the peptidase acti
38 and the recording continued for 2 hours with intragastric air infusion of 15 mL/min.
39  6-fold increase in TNFR1 knockout mice with intragastric alcohol exposure for 4 weeks.
40 ion to hyperhomocysteinemia and ER stress in intragastric alcohol fed mice.
41 ial respiration increased with both oral and intragastric alcohol feeding despite extensive N-acetyla
42                                              Intragastric alcohol feeding to mice resulted in 1) incr
43 , lower than the striking increase caused by intragastric alcohol feeding.
44 lic liver injury was studied in the model of intragastric alcohol-fed mice.
45   Interestingly, exposure of mice to oral or intragastric allergen does not promote eosinophilic esop
46 ow was already only 60% of normal flow, both intragastric and intravenous ethanol decreased both panc
47 n mutants were severely attenuated following intragastric and intravenous inoculation of mice.
48 hypothesized that both GALT-depleting diets (intragastric and intravenous TPN) would impair immunity
49                                     Repeated intragastric antigen challenge induced a significant inc
50                                     Repeated intragastric antigen challenge induced MMC9 development
51                        Although conventional Intragastric Balloons (IGBs) have become an efficient an
52 trictive procedures, intestinal sleeves, and intragastric balloons have demonstrated short-term effic
53                              In normal cats, intragastric, but not intravenous, ethanol reduced both
54                    The results indicate that intragastric, but not voluntary, ethanol consumption dif
55 her high-fat control diet or ethanol diet by intragastric cannulation for 2 or 4 weeks.
56 ere fed ethanol or high-fat control diet via intragastric cannulation for 4 weeks.
57                Twenty-one rats that received intragastric CCl(4) for 0-12 weeks were imaged with resp
58 LTB, or a combination of LT and LTB prior to intragastric challenge with H. pylori.
59 ve immunization with a CPB antibody prior to intragastric challenge.
60 longed to 7 days with the addition of K76 of intragastric CP at 5 mg/kg per day begun 1 day before op
61 d with intramuscular tacrolimus (TAC) and/or intragastric cyclophosphamide (CP) in rat recipients of
62  with 796 +/- 45 kcal; P = 0.08) in 20 women.Intragastric DB administration decreases both antral mot
63  0.04) plasma concentrations decreased after intragastric DB administration, whereas total and octano
64                           In women (n = 10), intragastric DB switched the origin of phase III contrac
65                            Strikingly, using intragastric delivery into fetal mouse intestine, preven
66                                       Direct intragastric delivery of a diet, nutrient or test substa
67                          Both intranasal and intragastric delivery of EtxB were effective in preventi
68                 The intrauterine but not the intragastric delivery of misoprostol significantly worse
69 eased bioavailability may limit the value of intragastric delivery of PPIs because of the high freque
70 20 mutant successfully targeted tumors after intragastric delivery, opening up the oral route as an o
71 ly visualize "H. heilmannii" types and their intragastric distribution.
72 re removal, the PED group was given a single intragastric dose of 1 mL saline, and the ALA and ALA+Vi
73 LA and ALA+Vit-C groups were treated with an intragastric dose of 50 mg/kg ALA and ALA+Vit-C for 15 d
74  cells were isolated 0 to 24 hours after one intragastric dose of ethanol daily, and intracellular Ca
75 ng animals in each group were given a single intragastric dose of NMBA at 2 mg/kg and sacrificed 12 w
76             Almost all of the mice given two intragastric doses also developed mucosal anti-H1 IgA an
77            After 6 wk, all rats received two intragastric doses of aspirin (1.4 mumol/kg body wt).
78 ox+/- and Wwox+/+ mice were treated with six intragastric doses of N-nitrosomethylbenzylamine and obs
79                 Our data show that multiple, intragastric doses of native OVA inhibited priming of CD
80 A and ZD+NMBA groups were treated with three intragastric doses of NMBA.
81                                              Intragastric emulsion instability was associated with a
82 sity-based tertiary care hospital, placed on intragastric enteral tube feeding through nasogastric or
83                                     However, intragastric ethanol administration caused a 35% to 40%
84        Therefore, in this study, a long-term intragastric ethanol feeding model was used to test the
85  alcohol-induced liver injury, the long-term intragastric ethanol feeding protocol developed by Tsuka
86                       Therefore, a long-term intragastric ethanol feeding protocol was used here to t
87 lcoholic liver injury in the murine model of intragastric ethanol feeding.
88 ulation of LPS enterally administered to the intragastric ethanol infusion model.
89         The rodent Tsukamoto-French model of intragastric ethanol infusion was used.
90    Ethanol dependence was induced by passive intragastric ethanol infusions in C57BL/6J (B6) and DBA/
91                                              Intragastric exposure of germ-free mice to Sphingomonas
92                                       Direct intragastric exposure to infected meat (A/Vietnam/1203/0
93 inated motivation deficits during flavorless intragastric feeding and increased oral intake of low-fa
94 es with the degree of liver pathology in the intragastric feeding model, which leads to the hypothesi
95 antly in Ad.lacZ-treated animals by using an intragastric feeding model.
96 ed either a control or ethanol diet using an intragastric feeding model.
97 lesterol and saturated fat and the rest from intragastric feeding of alcohol diet without or with wee
98          We used a mouse model of continuous intragastric feeding of alcohol or an isocaloric diet.
99 oto-French method (which involves continuous intragastric feeding of an isocaloric diet or alcohol) i
100 rench mouse model, which involves continuous intragastric feeding of isocaloric diet or alcohol for 3
101                     Tolerance was induced by intragastric feeding of low-dose CII to DBA/1 mice durin
102 day-1) continuously for up to 4 weeks via an intragastric feeding protocol.
103                                  We used the intragastric feeding rat model for alcoholic liver disea
104                                  We used the intragastric feeding rat model to assess the relationshi
105 ministered to critically ill patients during intragastric feeding to augment small intestinal nutrien
106               Test meals were fed through an intragastric feeding tube on Sprague-Dawley male rats af
107 gimens have taken more invasive routes using intragastric feeding tubes to infuse alcohol directly in
108  x d[-1]) continuously for up to 4 weeks via intragastric feeding using an enteral feeding model.
109 2 g/kg/d) continuously for up to 4 weeks via intragastric feeding using an enteral model developed by
110 -transferase activity (feeding of 3 mg/mouse intragastric for 4 days), whereas the elevation of hepat
111 eproduces the stress dampening, whereas oral intragastric gavage of sucrose is without effect.
112                          (54)Mn uptake after intragastric gavage was markedly reduced in ffe/+ mice (
113 c mice with a sealed anus were inoculated by intragastric gavage with type D isolates, 7 of 10 type D
114          When inoculated into BALB/c mice by intragastric gavage, 7 of 14 type C isolates were lethal
115 ombinant wildtype Cyp c 1 or carp extract by intragastric gavage.
116 n gravid and nongravid female mice following intragastric (gavage) inoculation, namely, (i) disease s
117  healthy, conscious baboons before and after intragastric glucose administration during infusions of
118 tion of GLP-1 impaired the disposition of an intragastric glucose meal and this was at least partly a
119 ated lymphoid tissue and in the periphery to intragastric (i.g.) and i.p. administration of SEB.
120                                           An intragastric (I.G.) infusion of alcohol (2.5 g/kg b.wt)
121  susceptibilities of A/J and C57BL/6 mice to intragastric (i.g.) inoculation with L. monocytogenes.
122                           Within hours after intragastric (i.g.) inoculation, virus appears in the ga
123 learn to prefer a taste that was paired with intragastric (IG) carbohydrate infusions during 22 hr/da
124  the water drinking response that follows an intragastric (ig) load of hypertonic NaCl.
125                                              Intragastric (IG) TPN maintains antiviral defenses but o
126 fter 4 days of feeding chow, a complex diet, intragastric (IG)-PN, or PN.
127  persisted for up to 6 months, whereas after intragastric immunization, responsive T cells were not f
128 esenteric lymph nodes (MLN) and spleen after intragastric immunization.
129                               The continuous intragastric in vivo enteral feeding model in the rat de
130 al sections taken from Peyer's patches after intragastric infection of mice showed that, in contrast
131  in rederived NF-kappaB-deficient mice using intragastric infection with Helicobacter hepaticus.
132 mal genes expressed in lymphoid tissue after intragastric infection.
133                                  This rodent intragastric infusion (iG) model has broad applications
134  were fed liquid control or ethanol diets by intragastric infusion for 4 weeks.
135 fed an ethanol-containing diet by continuous intragastric infusion for 42 days.
136 ssociated with ethanol hepatotoxicity in the intragastric infusion model of ethanol treatment.
137 learly dissociate the induction of CYP2E1 by intragastric infusion of ethanol from the generation of
138 lcohol-containing liquid diet and mice given intragastric infusion of ethanol).
139                                   Continuous intragastric infusion of ethanol-containing diets as par
140 urse experiment showed that after 2 weeks of intragastric infusion, a time point that results in isol
141 ated fat, palm oil, corn oil, or fish oil by intragastric infusion.
142 ning saturated fat, corn oil, or fish oil by intragastric infusion.
143 dematous children with PEM by using constant intragastric infusions of [2H3]leucine.
144                                Specifically, intragastric infusions of glucose induced significantly
145 nses in accumbens and dorsal striatum during intragastric infusions of glucose or serine.
146 eferences for flavors paired with concurrent intragastric infusions of maltodextrin or corn oil and f
147 fer a flavor that was paired with concurrent intragastric infusions of maltodextrin.
148 g following either 22 h water deprivation or intragastric injection of hypertonic saline (1.5 M), att
149                                              Intragastric injection of S. typhimurium 14028 and SR-11
150                     Adult male rats received intragastric injections of the vehicle or a moderately i
151  monoclonal anti-C5 i.p. on day 3 or 6 after intragastric inoculation and monitored for clinical sign
152 on in the liver and spleen of mice following intragastric inoculation and that mig-14 expression, whi
153 to colonize the gastrointestinal tract since intragastric inoculation did not rescue the mutants' col
154 ngolia/244/05, was able to infect mice after intragastric inoculation in liquid, whereas no evidence
155                         Here, we report that intragastric inoculation of a Shiga toxin 2 (Stx2)-produ
156                                              Intragastric inoculation of purified Stx2 also induced i
157 bacteria, whereas most control mice survived intragastric inoculation with 4 x 10(8) L. monocytogenes
158                                    Following intragastric inoculation with murine gamma-herpesvirus 6
159 ion genes during infection of mice following intragastric inoculation.
160 ng the gastrointestinal (GI) tract following intragastric inoculation.
161 ckling mouse GI tract at various times after intragastric inoculation.
162 e of infection was observed in ferrets after intragastric inoculation.
163 ere exposed to ethanol (0 or 6 g/kg/day) via intragastric intubation from gestational day 8-20.
164 p to 27 mg/kg/d) in a high-fat diet using an intragastric intubation model for 28 days to male mice f
165 f ethanol (4.5g/kg body weight) delivered by intragastric intubation on PN4, PN4-6, or PN7-9.
166 g/kg of alcohol or an isocaloric solution by intragastric intubation once a day from gestational day
167 fected with candidate bacteria by oral gauge intragastric intubation.
168 ine or vehicle from gestational days 8-22 by intragastric intubation.
169 r dose of lipopolysaccharide plus continuous intragastric KIC, PYR, or HCO3.
170      However, combining upper endoscopy with intragastric laparoscopic surgery offers advantages of b
171                                              Intragastric lipid administration activated neurons in t
172                                              Intragastric lipid administration to CD36 mice released
173                                    Assessing intragastric meal distribution (IMD) during gastric empt
174 rulent type C strain CN3685 avirulent in the intragastric model and nearly nonlethal in the intraduod
175                            Here, a sublethal intragastric mouse infection model using wild-type and c
176  pseudotuberculosis to cause mortality in an intragastric mouse model of infection, and a strain lack
177   After 4 weeks, animals were given a single intragastric NMBA dose and were sacrificed 25 and 77 day
178 eks later, the mice were randomized to chow, intragastric Nutren (Clintec, Chicago, IL), intravenous
179                                 We show that intragastric nutrient infusion rapidly and durably inhib
180 ke peptide-1 levels before and for 4 h after intragastric nutrients during a control study and on 2 d
181 ive absorption) and 5 h (steady state) after intragastric olive oil (70% triolein).
182 se liver or spleen at 2 and 4 days following intragastric or intraperitoneal infection.
183 nt strains from livers and spleens following intragastric or intraperitoneal infection.
184 markably, however, within 20 min after acute intragastric or intravenous glucose delivery (with blood
185 ro on epithelial cells and in vivo following intragastric or intravenous infusion in parenterally fed
186                  In anesthetized mice, after intragastric or intravenous saline delivery, pancreatic
187  we immunized female mice by the intranasal, intragastric, or intraperitoneal route with purified OMV
188  ethyl alcohol was administered by the oral, intragastric, or intravenous route.
189    Eritoran administration via intracolonic, intragastric, or intravenous routes significantly reduce
190 l vaccination regimens involving intranasal, intragastric, or rectal routes of immunization and found
191 VA/alum-sensitized mice to repeated doses of intragastric OVA induced genetically restricted, dose-de
192 characterized steatohepatitis (SH) caused by intragastric overfeeding in mice.
193          Results for percentage of time with intragastric pH <3 were similar.
194 rion was 24-h percent of time post dose with intragastric pH above 3; secondary criteria were percent
195   Acid-suppressive regimens that elevate the intragastric pH and maintain the pH over time have the p
196                                              Intragastric pH and serum gastrin were monitored 1 hour
197 pump inhibitors can elevate and maintain the intragastric pH at >6.
198 H 5 to mimic the fasted-to-fed transition in intragastric pH in rats.
199  data suggested that agents that elevate the intragastric pH may increase the susceptibility of patie
200           However, the resulting increase of intragastric pH may predispose to gastric cancer by allo
201        Twelve volunteers underwent overnight intragastric pH monitoring after 7 days of treatment wit
202  bedtime reduced the percentage of time with intragastric pH of <4 from 48% to 31% (P < 0.005) compar
203  through the mucus may be appropriate at the intragastric pH of the fasted, but not fed, animal.
204 he United States, is as effective in raising intragastric pH on the first day as a continuous infusio
205 enting with overt bleeding due to ulcers had intragastric pH probes placed after endoscopy and baseli
206                                              Intragastric pH studies have demonstrated that, whereas
207 proton pump inhibitors to raise and maintain intragastric pH suggest that, although both can raise th
208            Acid breakthrough, or decrease in intragastric pH to <4 for an hour or longer, occurs in t
209 volunteers to document regional variation in intragastric pH under both fasting and postprandial cond
210                                              Intragastric pH was > 6 for > 60% of the study period in
211                                              Intragastric pH was > 6 for 67.8% of the study period wi
212                                              Intragastric pH was maintained at 4.0 by in vivo intraga
213 th sucralfate, an agent that does not affect intragastric pH.
214         Multi-channel pH-monitoring assessed intragastric pH.
215 tion and colonization over the wide range of intragastric pHs experienced by the organism.
216 ring the first 4 hours after intravenous and intragastric Pi loading in rats.
217          This was accomplished by monitoring intragastric pressure (gastric tone) and contractility o
218 timulated gastric contractions and increased intragastric pressure (IGP).
219 ndex (BMI) and waist circumference (WC) with intragastric pressure (inspiration, BMI [r = 0.57], WC [
220                           The belt increased intragastric pressure by a median of 6.9 mmHg during fas
221                            Without the belt, intragastric pressure correlated with waist circumferenc
222 LES relaxation pressure, and intraesophageal-intragastric pressure gradient.
223 tric distension caused a smaller increase in intragastric pressure in response to gastric distension
224 stric distension (6 ml) evoked a much larger intragastric pressure in the denervated, vascularly isol
225 ) evoked an increase of 9.0 +/- 1.0 cmH2O of intragastric pressure in vivo.
226                                          The intragastric pressure rise inducing this effect is well
227                                 In addition, intragastric pressure was lower in the prone position (p
228 ate of 100 mL/min, with pauses for measuring intragastric pressure, until no further distension was t
229 ition of gastric motility and an increase in intragastric pressure.
230 sally, and simultaneous intra-esophageal and intragastric pressures were measured over 6-8 respirator
231 ong strains; often correlates with different intragastric regions and evolves during chronic infectio
232 tect BALB/c mice against intraperitoneal and intragastric ricin challenges.
233  S. typhimurium for infection of mice by the intragastric route but not the intraperitoneal route, an
234 e to be an important virulence factor by the intragastric route of infection in mice.
235 re immunized by either the intranasal or the intragastric route with a single dose of 10(9) or 10(10)
236 the prototype arenavirus, implicate oral and intragastric routes as natural routes of infection.
237 f the current study was to establish whether intragastric self-administration of fat emulsions, that
238 aintenance, extinction, and reinstatement of intragastric self-administration of lipid emulsions to d
239 linical models of acute toxin-associated and intragastric, spore-induced colonic disease.
240   We aimed to visualize the influence of the intragastric stability of fat emulsions on their dynamic
241                                              Intragastric stimulation of dopamine release seemed to d
242 and provide insight into mechanisms used for intragastric survival.
243 agastric pH was maintained at 4.0 by in vivo intragastric titration with 0.3N sodium bicarbonate for
244                         Both intravenous and intragastric total parenteral nutrition (TPN) produce GA
245 e complex enteral diet group (n = 5) and the intragastric TPN group (n = 5) after 30 minutes of gut i
246 r 30 minutes of gut ischemia, the IV-TPN and intragastric TPN groups showed a higher death rate than
247 in leak between the complex enteral diet and intragastric TPN groups.
248 emia (30 minutes) eliminated any benefits of intragastric TPN on survival.
249                                              Intragastric TPN partially preserved this respiratory im
250 trition (TPN) produce GALT atrophy, but only intragastric TPN preserves established antiviral immunit
251        Mice were randomized to chow, IV-TPN, intragastric TPN, or complex enteral diet for 5 days' fe
252 e randomized to chow, complex enteral diets, intragastric TPN, or intravenous TPN.
253  (Clintec, Chicago, IL), intravenous TPN, or intragastric TPN.
254                                 In contrast, intragastric treatment evoked a brief, modest elevation
255                          Mice fed alcohol by intragastric tube were assayed for serum alanine aminotr
256 ion via different routes (intraperitoneal vs intragastric), we noted a difference in the time course
257  response that was protective against lethal intragastric Y. pseudotuberculosis challenge.
258 nc-replenished esophagi after treatment with intragastric zinc compared with zinc-sufficient esophagi

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