ライフサイエンスコーパス: intrahepatic bile duct

1 uction of the extrahepatic biliary tract and intrahepatic bile ducts.

2 from chronic inflammatory destruction of the intrahepatic bile ducts.

3 t culminates in fibrosis of extrahepatic and intrahepatic bile ducts.

4 e in the context of progressively developing intrahepatic bile ducts.

5 , the destruction in PBC is limited to small intrahepatic bile ducts.

6 volves formation of liver cysts derived from intrahepatic bile ducts.

7 ted by autoreactive T cells infiltrating the intrahepatic bile ducts.

8 tes and cholangiocytes, the cells lining the intrahepatic bile ducts.

9 med for disease conditions featuring loss of intrahepatic bile ducts.

10 accumulation of viscous mucoid secretions in intrahepatic bile ducts.

11 disease characterized by destruction of the intrahepatic bile ducts.

12 iver disease characterized by destruction of intrahepatic bile ducts.

13 biliary glands within extrahepatic and large intrahepatic bile ducts.

14 adolinium administration = 1 x dilatation of intrahepatic bile ducts + 2 x dysmorphy + 1 x portal hyp

15 Intrahepatic bile duct anatomy is complex with many comm

16 hex(d2,3/-)) caused irregular development of intrahepatic bile ducts and an absence of Hnf1beta in ma

17 wth retardation, impaired differentiation of intrahepatic bile ducts and defects in heart, eye and ki

18 the characteristic phenotype of high-density intrahepatic bile ducts and enlarged liver in Rosa(NICD/

19 ealed that NK cells populate the vicinity of intrahepatic bile ducts and overexpress several genes in

20 ized by progressive destruction of the small intrahepatic bile ducts and portal inflammation, leading

21 mpt surgical intervention, ongoing injury of intrahepatic bile ducts and progressive cholangiopathy l

22 Cystic dysplasia of the intrahepatic bile ducts and progressive portal fibrosis

23 aracterized by neonatal jaundice, paucity of intrahepatic bile ducts, and abnormalities of the heart,

24 embryonic Foxm1b -/- livers did not develop intrahepatic bile ducts, and these presumptive biliary h

25 In situ morphometry showed that (1) intrahepatic bile ducts are heterogeneous in external di

26 olangiocytes, the epithelial cells that line intrahepatic bile ducts, are composed of plasma membrane

27 Intrahepatic bile ducts (BD) are a critical target of in

28 ults provide direct functional evidence that intrahepatic bile ducts both secrete and absorb water in

29 Intrahepatic bile duct calculi are present in approximat

30 ancer, 91% of ampula of Vater cancer, 96% of intrahepatic bile duct cancer, and 94% of hepatocellular

31 Small and large intrahepatic bile ducts consist of small and large chola

32 signaling as a common etiologic mechanism of intrahepatic bile duct defects in BA.

33 genetic interactions of factors important to intrahepatic bile duct development and their effect on c

34 of cirrhotic patients with tumors exhibiting intrahepatic bile duct differentiation remains controver

35 We demonstrate that tubule formation of intrahepatic bile ducts during embryonic development as

36 The cell of origin of intrahepatic bile ducts during fetal development remains

37 presence and induction of CYP1A1/1A2 within intrahepatic bile duct epithelia.

38 Chronic injury to intrahepatic bile duct epithelial cells (BDECs) elicits

39 ting of hepatocytes and cholangiocytes (i.e. intrahepatic bile duct epithelial cells).

40 e a group of hepatobiliary diseases in which intrahepatic bile duct epithelial cells, or cholangiocyt

41 fined the relationship between ICAM-1 on the intrahepatic bile duct epithelium and the evolution of N

42 ithout these infections: cancer of liver and intrahepatic bile duct; fibrosis, cirrhosis, and other l

43 le of elf3 in hepatocyte differentiation and intrahepatic bile duct formation.

44 ith risks of hepatocellular carcinoma (HCC), intrahepatic bile duct (IBD), and gallbladder and biliar

45 tin expression in cholangiocytes of isolated intrahepatic bile ducts (IBDUs) and liver cysts was anal

46 hip between adult gallbladder stem cells and intrahepatic bile duct (IHBD) cells is not well understo

47 erozygous mice (Jag1(+/-) ) exhibit impaired intrahepatic bile duct (IHBD) development, decreased SOX

48 The potential for intrahepatic bile duct (IHBD) regeneration in patients w

49 AGS is chiefly characterized by a paucity of intrahepatic bile ducts (IHBD), but also includes cardia

50 iary disease, is characterized by paucity of intrahepatic bile ducts (IHBDs).

51 Preoperative knowledge of intrahepatic bile duct (IHD) anatomy is critical for pla

52 n 11, hemorrhage in 10, fat in four, dilated intrahepatic bile ducts in 17, and abdominal lymphadenop

53 iography completely demonstrated first-order intrahepatic bile ducts in 23 (92%) patients, the donor

54 opment of strategies to block progression of intrahepatic bile duct injury in patients with BA.

55 ed hepatic IL-17A production and ameliorated intrahepatic bile duct injury.

56 entification of annexin V with predominantly intrahepatic bile ducts, is of significant interest beca

57 ive fibroinflammatory disorder of extra- and intrahepatic bile ducts, is the most common identifiable

58 isease characterised by destruction of small intrahepatic bile ducts, leading to fibrosis and potenti

59 llowing bile duct ligation (BDL), increasing intrahepatic bile duct mass (IBDM) and fibrosis.

60 ), GABA(B), and GABA(C) receptor expression; intrahepatic bile duct mass (IBDM) and the percentage of

61 ry proliferation was evaluated by changes in intrahepatic bile duct mass and the expression of prolif

62 Treatment with GnRH increased intrahepatic bile duct mass as well as proliferation and

63 ivo and in vitro knockdown of GnRH decreased intrahepatic bile duct mass/cholangiocyte proliferation

64 est that chronic inflammatory insults in the intrahepatic bile ducts might shed light on the cystaden

65 hepatoblasts into cholangiocytes, premature intrahepatic bile duct morphogenesis, and biliary hyperp

66 s regulating hepatoblast differentiation and intrahepatic bile duct morphogenesis.

67 in a novel cellular compartment surrounding intrahepatic bile ducts, namely portal fibroblasts.

68 orectal, breast, pancreatic, prostate, liver/intrahepatic bile duct, non-Hodgkin lymphoma, head/neck,

69 bile ducts, and its expression is reduced in intrahepatic bile ducts of patients with cholestatic dis

70 onfirmed that miR-506 is up-regulated in the intrahepatic bile ducts of PBC livers, compared with nor

71 antly inhibited the cystic dilatation of the intrahepatic bile ducts of PCK rats, which was accompani

72 demonstrated intense inflammation focused at intrahepatic bile ducts, pathology analogous to that fou

73 While intrahepatic bile duct proliferation is universal at dia

74 Pkhd1(del4/del4) mice develop intrahepatic bile duct proliferation with progressive cy

75 ed from BECs isolated from distinct areas of intrahepatic bile ducts revealed important functional di

76 sis is a chronic inflammatory disease of the intrahepatic bile ducts that eventually leads to liver c

77 aspase-3 staining was strongest in the small intrahepatic bile ducts, the major site of tissue destru

78 he release of bile constituents from injured intrahepatic bile ducts, thereby limiting the progressio

79 erimental model, the enclosed, polarized rat intrahepatic bile duct unit (IBDU).

80 ffect of phenylephrine on lumen expansion in intrahepatic bile duct units (IBDUs) and cyclic adenosin

81 vels in cholangiocytes and duct expansion in intrahepatic bile duct units (IBDUs) in the absence or p

82 d HCO(3)(-) transport: the microperfusion of intrahepatic bile duct units (IBDUs) isolated from norma

83 in altering AQP1-mediated water transport in intrahepatic bile duct units (IBDUs) isolated from rat l

84 Small and large cholangiocytes or intrahepatic bile duct units (IBDUs) were isolated from

85 Intrahepatic bile duct units (IBDUs) were microdissected

86 orescent confocal microscopy of rat isolated intrahepatic bile duct units (IBDUs) were used to detect

87 ing purified rat cholangiocytes and perfused intrahepatic bile duct units (IBDUs), we found that TRPV

88 Secretion and cAMP levels were measured in intrahepatic bile duct units after stimulation with secr

89 A complete resection of congenital intrahepatic bile ducts was achieved in 90.5% of the 148

90 Adjacent intrahepatic bile ducts were dilated.

91 pressed in the periportal region surrounding intrahepatic bile ducts, whereas NTPDase1 was found in h

92 ing excluded owing to creation of an RYHJ to intrahepatic bile ducts with concomitant liver resection

93 rial antibodies and the destruction of small intrahepatic bile ducts with portal inflammation.

94 is involvement of both the extrahepatic and intrahepatic bile ducts, with small duct PSC being less