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1 cher-344 rats and was followed 6 hr later by intranigral 6-OHDA or saline.
2 imals whose DA autoreceptors were blocked by intranigral application of raclopride, our results sugge
3 nic, intraventricular and intraputamenal (or intranigral) delivery of GDNF to maximize the efficacy o
4  the context of the anticonvulsant action of intranigral fluoxetine, we examined the influence of 5-H
5 etely prevented the anticonvulsant action of intranigral fluoxetine, without modifying the anticonvul
6 tially reversed the anticonvulsant action of intranigral fluoxetine.
7                               Thus, although intranigral GDNF can prevent 6-OHDA-induced reductions i
8                                              Intranigral infusion of CPX (100 ng) or DMPX (500 ng), h
9                                              Intranigral infusion of freshly prepared GSNO (0-16.8 nm
10                              Prior bilateral intranigral infusions of LY306740 or SR142801 (0, 5.0 an
11                                   Unilateral intranigral infusions of the neurokinin-1 (NK1) antagoni
12 f GDNF or vehicle followed 6 h later with an intranigral injection of 6-hydroxydopamine.
13                 When injected 6 hr before an intranigral injection of 6-OHDA, GDNF provides complete
14  of 3, 18 and 24 months of age were given an intranigral injection of GDNF or vehicle followed 6 h la
15 ed the response to quinpirole, and (3) prior intranigral injection of the Gi-Go-protein inactivator p
16  and the striatum (CS) several moments after intranigral injection of the neurotoxin, with or without
17 A-modulatory effect of quinpirole, (2) prior intranigral injection of the receptor inactivator N-etho
18                                              Intranigral injections of [D-Ala2, N-Me-Phe4, Gly-ol5]-e
19                                Rats received intranigral injections of rAAV2/5 alpha-syn and three we
20  alpha-synuclein pathology using unilateral, intranigral injections of recombinant adeno-associated v
21 thyl-4-phenyl-1,2,3,6-tetra-hydropyridine or intranigral inoculation of aggregated toxic alpha-SYN.
22 back for engineered systems implies that the intranigral microcircuit, despite its absence from canon
23                    In both mice and monkeys, intranigral or intrastriatal inoculations of PD-derived
24                                              Intranigral perfusion of either a DA D1 or GABA-A recept
25 that either systemic administration or local intranigral perfusion of J-113397 and SNC-80 led to the
26 entrations was D1 receptor-dependent because intranigral perfusion of the D1 DA antagonist SCH23390 (
27                                              Intranigral perfusion of the GABA-A receptor antagonist,
28 arrier, intraventricular, intrastriatal, and intranigral routes of administration have been found to
29 thout modifying the anticonvulsant effect of intranigral TFMPP.

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