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1 This led to the discovery of a multicystic intraocular tumor.
2 e growth of a highly vascularized angiogenic intraocular tumor.
3 agent on the growth of a highly vascularized intraocular tumor.
4 cedure, PLSU can achieve control of selected intraocular tumors.
5 herapy with sonoporation in the treatment of intraocular tumors.
6 the failure to induce Th17 cells within the intraocular tumors.
7 ated killing of tumor cells and rejection of intraocular tumors.
8 a significant decrease (P = 0.01) in viable intraocular tumors.
9 observed nor necessary for rejection of the intraocular tumors.
10 of CD8+ T cells in the pristine rejection of intraocular tumors.
11 e and prevents the rejection of NK-sensitive intraocular tumors.
12 IFN-gamma knockout mice that fail to reject intraocular tumors, 5) CD4(+) T cells and corneal endoth
13 nosis was obtained in 97.6% (n = 121) of the intraocular tumors, and chromosome 3 status could be det
14 delivery of paclitaxel effectively inhibits intraocular tumor burden in the LH beta-Tag model of ret
15 nited States are diagnosed as having a large intraocular tumor burden that requires intensive ocular-
18 her supported by investigations showing that intraocular tumors grew progressively in IFN-gamma KO (k
20 oplatin in serial doses effectively inhibits intraocular tumor growth in a dose-dependent manner in t
21 chemotherapy agents in a novel setting (ie, intraocular tumor) has been successful in treating all b
23 carboplatin safely and effectively controls intraocular tumors in a dose-dependent manner in this mu
24 d studied the methylome in the most frequent intraocular tumors in adults and children (uveal melanom
25 biopsies were performed in 123 patients with intraocular tumors in the posterior segment from January
26 r data indicate that the function of FasL on intraocular tumors is determined by the microenvironment
28 However, tumor LBD, tumor thickness, and intraocular tumor location also proved to be significant
32 d eyes injected with CD34(+) cells showed no intraocular tumor or abnormal tissue growth after 8 mont
33 redictive of extraocular tumor extension was intraocular tumor recurrence after TTT treated with addi
34 ells circumvent immune privilege and mediate intraocular tumor rejection by a TNF-alpha-dependent man
35 cells from tumor-rejector mice could mediate intraocular tumor rejection following adoptive transfer
44 step toward developing an immunotherapy for intraocular tumors, the present study was conducted to e
45 was used as a screening tool to evaluate the intraocular tumor, to evaluate for multi-organ metastati
50 es of transgenic mice that develop pigmented intraocular tumors were produced with the SV40 T and t a
51 lines of transgenic mice developed bilateral intraocular tumors with complete penetrance and without
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