ライフサイエンスコーパス: intrapancreatic

1 the mechanisms responsible for the premature intrapancreatic activation of digestive enzyme zymogens.

2 osphatidylinositol 3-kinase is necessary for intrapancreatic activation of trypsinogen and regulating

3 3-kinase inhibitor wortmannin can reduce the intrapancreatic activation of trypsinogen that occurs du

4 n thoracic spinal dorsal horn segments after intrapancreatic administration of proteinase-activated r

5 quantified following transplantation via the intrapancreatic and subrenal routes.

6 Intrapancreatic angiotensin II generation has been impli

7 Wistar rats, we observed that switching off intrapancreatic artery insulin infusions in vivo during

8 Increased intrapancreatic autoactivation of trypsinogen mutants ha

9 ion, biliary strictures were confined to the intrapancreatic bile duct in 51%; the proximal extrahepa

10 number of experimental studies suggest that intrapancreatic calcium concentrations play an important

11 sured simultaneously from 33 preparations of intrapancreatic canine ganglia and pancreatic parenchyma

12 Intrapancreatic DCs acquired a distinct immune phenotype

13 ar cell injury, pancreatic inflammation, and intrapancreatic digestive enzyme (i.e., trypsinogen) act

14 cell differentiation from progenitors in the intrapancreatic duct (IPD).

15 We examined whether intrapancreatic duct infusion of AAV containing an NF-ka

16 or of endocrine progenitors in the zebrafish intrapancreatic duct.

17 emical analyses support a model in which the intrapancreatic ductal system develops from progenitors

18 tions of IgG1 isotype antibodies and reduced intrapancreatic expression of IFN-gamma, IL-6, and IL-17

19 The amount of intrapancreatic fat (IPF) is increased in obese patients

20 lted in oil red O-positive areas, resembling intrapancreatic fat.

21 circular muscle, and in fibers and cells in intrapancreatic ganglia.

22 These first simultaneous measurements of intrapancreatic ganglion activity and insulin secretion

23 reactivity was found in numerous enteric and intrapancreatic ganglion cells and in dense networks of

24 es in electrical activity in a population of intrapancreatic ganglion neurons.

25 nts with T1D exhibited pronounced inter- and intrapancreatic heterogeneity in signal intensity.

26 tumor angiogenesis and metastasis following intrapancreatic implantation with either PANC-1 or T3M4

27 e intestinal and pulmonary tracts were given intrapancreatic injections of Panc02 CEA(+) cells (expre

28 Intrapancreatic lavage catheter placement is essential t

29 te pancreatitis to account for nearly 15% of intrapancreatic leukocytes.

30 n of IFN-gamma, but not IL-4, was limited to intrapancreatic lymphocytes and was not detectable at ex

31 data distinguish macrophages within distinct intrapancreatic microenvironments and suggest how macrop

32 In 8 patients, an intrapancreatic nasobiliary lavage catheter was placed i

33 the demonstration of the essential role for intrapancreatic nerves in mediating meal-induced respons

34 In addition, the endocrine pancreas, intrapancreatic nerves, and some extrapancreatic neural

35 Perineural tumor invasion of intrapancreatic nerves, neurogenic inflammation, and tum

36 sults suggest that neurohormone release from intrapancreatic neurons could help synchronize islets in

37 sed from various cells within islets or from intrapancreatic neurons, are hypothesized to further adj

38 indirect and may be exerted at the level of intrapancreatic neurons.

39 ablished experimental evidence demonstrating intrapancreatic parasympathetic (cholinergic) ganglia an

40 Intrapancreatic prostaglandin E(2) levels were reduced i

41 Terms such as pancreatic abscess and intrapancreatic pseudocyst have been abandoned.

42 a single clone with subsequent intramural or intrapancreatic spread.

43 inically optimize trypsin inhibitors towards intrapancreatic target inhibition.

44 was associated with increased percentage of intrapancreatic Tregs.

45 eatitis by eliminating prematurely activated intrapancreatic trypsin.

46 these regulatory cleavages lead to increased intrapancreatic trypsinogen activation and cause heredit

47 The relative contributions of intrapancreatic trypsinogen activation and nuclear facto

48 epsin B is thought to play a central role in intrapancreatic trypsinogen activation and the onset of

49 is the dominant mechanism that can mitigate intrapancreatic trypsinogen activation.

50 in IAC and compare relapse rates in IAC with intrapancreatic vs proximal bile duct strictures.