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1 cal coherence tomography, 83% had fluid (61% intraretinal, 38% subretinal, and 36% sub-retinal pigmen
10 curately detect, differentiate, and quantify intraretinal and SRF using area under the receiver opera
12 on of brolucizumab-treated eyes had resolved intraretinal and subretinal fluid compared with afliberc
14 emorrhages involving the optic nerve sheath, intraretinal and subretinal hemorrhages, and macular fol
16 al telangiectasis') which results in massive intraretinal and subretinal lipid accumulation (exudativ
18 of VEGF in the retina is sufficient to cause intraretinal and subretinal NV and provides a valuable n
21 nt epithelium), type 2 (subretinal), type 3 (intraretinal), and mixed neovascularization (NV), respec
22 evaluated while performing retinal surface, intraretinal, and subretinal maneuvers in cadaveric porc
23 eeks of follow-up with gradual resolution of intraretinal- and subretinal fluid, and remained stable
24 uingly, a small population of M1 ipRGCs have intraretinal axon collaterals that project toward the ou
25 and Slit2, regulate two distinct aspects of intraretinal axon guidance in a region-specific manner.
29 requirement for Hedgehog (Hh) signaling for intraretinal axon pathfinding and show that Shh acts to
31 idance molecule ephrinB2, was increased, and intraretinal axons were disorganised resulting in a fail
37 gas tamponade can safely create an effective intraretinal barrier to fluid egress from the optic disc
42 tantial and boosts the membrane potential of intraretinal blood vessels to a suprahyperpolarized leve
43 ied quantum dots in the choriocapillaris and intraretinal capillaries upon i.v. injection and 1-h cir
44 distinctive traits included the presence of intraretinal cavitation that could affect all retinal la
50 strophy characterized by multiple glistening intraretinal crystals scattered over the fundus, a chara
52 plete closure of the FTMH with resolution of intraretinal cystic changes was confirmed on OCT at 16 m
53 cribed features such as fluorescein-negative intraretinal cystic changes, choroidal neovascularizatio
54 ic patterns of fluid presentation, including intraretinal cystic spaces (ICS), retinal pigment epithe
55 tical coherence tomography revealed multiple intraretinal cystic spaces and hyperreflective deposit i
57 earning to automatically detect and quantify intraretinal cystoid fluid (IRC) and subretinal fluid (S
58 Assessed morphologic parameters included intraretinal cystoid fluid (IRC), subretinal fluid (SRF)
59 y masked reading centers for the presence of intraretinal cystoid fluid (IRC), subretinal fluid (SRF)
62 d patients, and by poor baseline BCVA, large intraretinal cystoid spaces, renal disease, and absence
63 showing retinal morphologic changes, such as intraretinal cysts (IRCs), subretinal fluid (SRF), and p
64 was assessed by standardized OCT, including intraretinal cysts (IRCs), subretinal fluid (SRF), and p
65 esence of features of nAMD disease activity (intraretinal cysts [IRC], subretinal fluid [SRF], diffus
67 f active myopic CNV (either subretinal fluid/intraretinal cysts on SD OCT or dye leakage on fluoresce
69 coherence tomography can clearly demonstrate intraretinal cysts which may not be clinically detectabl
72 l 1-mm subfield thickness, the occurrence of intraretinal cysts, ellipsoid zone disruption, and disor
76 eration characterized by multiple glistening intraretinal dots scattered over the fundus, degeneratio
77 ing degrees of venous stasis retinopathy and intraretinal edema overlying the macular detachment.
78 f these features include photoreceptor loss, intraretinal edema, and retinal thinning overlying choro
79 oreceptors overlying choroidal melanoma; and intraretinal edema, retinoschisis, and retinal thinning
83 l architecture, cystoid macular edema (CME), intraretinal exudates and subretinal lipid aggregation,
86 as found for eyes that displayed fluid, NSD, intraretinal flecks, and low reflectivity or undefined b
87 fluid; 68% and 88% for NSD; 81% and 83% for intraretinal flecks; 63% and 92% for undefined boundarie
88 8 mm2, P < .001), and higher proportions of intraretinal fluid (82.5% vs 51.0%, P < .001), subretina
89 ere created on the basis of baseline CME and intraretinal fluid (IRF) status: (1) CME, (2) IRF withou
90 dependently graded OCT scans for presence of intraretinal fluid (IRF), subretinal fluid (SRF), and su
91 tal thickness at the foveal center point and intraretinal fluid (IRF), subretinal fluid (SRF), and su
94 TA) signals corresponding to hyperreflective intraretinal fluid across various exudative maculopathie
95 teristics included subretinal fluid (n = 5), intraretinal fluid and cysts (n = 1), and subretinal hyp
97 differences in the presence of subretinal or intraretinal fluid at final evaluation, dye leakage on a
98 llow eye (aHR, 2.07; 95% CI, 1.40-3.08), and intraretinal fluid at the foveal center (aHR, 2.10; 95%
102 nts (50%) had edema resolution defined as no intraretinal fluid for 6 months or more after the last i
103 here was a reduction of either subretinal or intraretinal fluid in 18 of 36 (50.0%) of the treated ey
104 0% to 12% (P = .05), and the proportion with intraretinal fluid increased from 72% to 71% to 82% (P =
105 7 25-line raster scans confirmed subretinal/intraretinal fluid not identified by the 6-line radial (
107 RPE drusen complex abnormal thinning volume, intraretinal fluid or cystoid spaces, hyperreflective fo
108 nable immunoglobulins along with accumulated intraretinal fluid to flow into the subretinal space, cr
109 tion (RAP) lesion, GA in the fellow eye, and intraretinal fluid were associated with a higher risk of
111 e risk factors included poor VA, RAP, foveal intraretinal fluid, monthly dosing, and treatment with r
112 reasing age, increasing CST, the presence of intraretinal fluid, pigment epithelial detachment, and s
113 -scans of each cube scan for the presence of intraretinal fluid, subretinal fluid, and sub-retinal pi
118 >/=20/40), scar (OR 2.21, 95% CI:1.22-4.01), intraretinal foveal fluid on optical coherence tomograph
123 zing the PLR as an assay for the efficacy of intraretinal grafts has highlighted the significance of
124 subtle but significant mistakes during their intraretinal growth and inappropriately defasciculate al
130 le a vaso-occlusive event and include edema, intraretinal hemorrhage, and nonperfusion detected by fl
131 leads to impaired blood vessel sprouting and intraretinal hemorrhage, particularly during formation o
132 y caused focal columns of retinal injury and intraretinal hemorrhages from retinal vessel bleeding, w
133 g of the optic nerve, macular edema, diffuse intraretinal hemorrhages, and dilated and tortuous retin
135 e included multifocal choroiditis, vitritis, intraretinal hemorrhages, iritis, keratic precipitates,
136 This study sought to quantify the change in intraretinal HF distribution and its correlation with ag
137 presence of ultrastructural features such as intraretinal hyperreflective flecks and the inherent ref
139 e use of cholesterol-lowering medication and intraretinal hyperreflective foci attributable to RPE ce
142 tion was significantly associated with SDOCT intraretinal hyperreflective foci in the 314 study eyes
143 retinal anatomic changes and the pattern of intraretinal hyperreflective foci migration were documen
144 defined by the presence of depolarization at intraretinal hyperreflective foci on PS-SLO and PS-OCT i
152 irregularities, abnormal retinal thickness, intraretinal hyperreflective/hyporeflective features, an
153 r than FP for abnormal retinal thickness (or intraretinal hyporeflective features); similar as FP for
156 nimals were anterogradely labeled with small intraretinal injections of the lipophilic dye 1,1'-dioct
162 owerful approach for measuring alteration in intraretinal ion demand in models of ocular injury.
163 nhanced MRI (MEMRI), assesses alterations in intraretinal ion demand in models of ocular insult.
164 irst-time evidence for changes (P < 0.05) in intraretinal ion regulation before and during pathologic
165 tive foci (HRF), average and largest area of intraretinal (IR) cysts, and extent of disruption of ext
169 eflectance and fractal dimension) of various intraretinal layers extracted from optical coherence tom
170 ne photoreceptor length and the thickness of intraretinal layers were measured and compared to previo
173 In Akita mouse retinas, diabetes increased intraretinal levels of oxidized LDL and glycated LDL, in
174 inal injection of 1% hyaluronic acid and the intraretinal levels of the autophagy proteins LC3 and At
175 acute retinal injury (consisting of abnormal intraretinal light scattering) were visualized in vivo i
177 of young and aged mice revealed a subnormal intraretinal manganese uptake (P < 0.05) in aged DBA/2J
178 provide proof-of-concept that the extent of intraretinal manganese uptake after systemic MnCl(2) inj
185 raphy showed a normal foveal contour without intraretinal microcystic spaces and a resolution of the
189 the percent of capillary length involved in intraretinal microvascular abnormalities, expressed as h
190 ose of the present study was to evaluate the intraretinal migration of the retinal pigment epithelium
191 potential of these resident cells to act as intraretinal modulators of immune and inflammatory respo
192 dal anastomosis was found, 3 patients showed intraretinal neovascularization connected with a pigment
194 tion of Srf in adult murine vessels elicited intraretinal neovascularization that was reminiscent of
195 pithelial detachment, 2 patients showed only intraretinal neovascularization, and in 2 patients flow
197 nting extrinsic macrophages, were present in intraretinal ON region, but not in the retroscleral (iso
198 t was worst for subretinal fluid compared to intraretinal or sub-retinal pigment epithelial fluid.
199 within 6 months with classic features of new intraretinal or sub-retinal pigment epithelium infiltrat
200 es with and without persistent fluid (cystic intraretinal or subretinal fluid at all 4 initial visits
202 eneration who exhibit recurrent or resistant intraretinal or subretinal fluid following multiple inje
203 line at the last follow-up and/or persistent intraretinal or subretinal fluid or detectable choroidal
206 nthly with intravitreal bevacizumab until no intraretinal or subretinal fluid was observed on optical
207 ts with resistant or multiple recurrences of intraretinal or subretinal fluid while receiving monthly
208 r irregularity of each category (epiretinal, intraretinal, or RPE/choroidal irregularity), 3D-OCT was
209 n of tempol, a superoxide scavenger, reduced intraretinal oxidized LDL and glycated LDL levels, PGIS
214 e qualitatively and quantitatively identical intraretinal pathfinding errors to those reported previo
217 nation features included macular edema, mild intraretinal pigment migration, and widespread atrophy i
219 eposits, differing from the classic spicular intraretinal pigmentation observed in other individuals
220 ensitive microelectrodes were used to record intraretinal Po(2) profiles from healed photocoagulation
222 does not cause the immediate degeneration of intraretinal portions of axons or the immediate death of
224 s used to calculate the fractal dimension in intraretinal regions of interest identified in the image
227 extent of preretinal neovascularization and intraretinal revascularization was quantified by image a
228 rovascular networks were analyzed to examine intraretinal revascularization, capillary sprouting, and
230 retinal hyperreflective foci correlated with intraretinal RPE and lipid-filled cells of probable mono
235 results identify retinal Muller cells as an intraretinal source of TNF alpha and IL-6 and support th
240 and progressing to the subretinal space with intraretinal, subretinal, and choroidal angiogenic stage
241 in optical coherence tomography (presence of intraretinal, subretinal, and subretinal pigment epithel
242 omic improvement in patients with persistent intraretinal, subretinal, or subretinal pigment epitheli
243 aflibercept injection due to the presence of intraretinal/subretinal fluid and pigment epithelial det
244 macular thickness (CMT), and the presence of intraretinal/subretinal fluid and the height and presenc
245 and 25-line raster scans were evaluated for intraretinal/subretinal fluid and, when applicable, vitr
247 ver a mean of 13 months follow-up, there was intraretinal tumor recurrence (n = 1), subretinal seed r
249 , and a surrogate of retinal ion regulation (intraretinal uptake of manganese) were assessed from MEM
253 cell dysfunction induces alterations to the intraretinal vasculature and substantial visual deficits
254 e that specific retinal interneurons and the intraretinal vasculature are highly interdependent, and
255 required for generating and maintaining the intraretinal vasculature through precise regulation of h
258 cytes, and staining was increased around new intraretinal vessels in mouse OIR and rat retinopathy of
259 el, while not significantly affecting normal intraretinal vessels, it holds therapeutic potential for
260 ark adaptation of M-cones driven by both the intraretinal visual cycle and the retinal pigmented epit
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