ライフサイエンスコーパス: intrathoracic pressure

1 is, hypothermia, hypervolemia, and increased intrathoracic pressure).

2 n (CPR) by increasing the degree of negative intrathoracic pressure.

3 airflow and helps maintain higher levels of intrathoracic pressure.

4 y are influenced by physiological changes in intrathoracic pressure.

5 intraabdominal pressure leading to increased intrathoracic pressure.

6 usion also occurs in the absence of negative intrathoracic pressure.

7 iratory muscle function to generate elevated intrathoracic pressures.

8 magnitude of respiratory phasic variation of intrathoracic pressures.

9 to exercise requires substantial changes in intrathoracic pressure and in the work output and metabo

10 al of patients of cardiac arrest by lowering intrathoracic pressure and increasing cardiac output.

11 on rates resulted in significantly increased intrathoracic pressure and markedly decreased coronary p

12 a-induced increase in CFV; however, negative intrathoracic pressure and the small amount of oxyhaemog

13 ely, by mechanical effects of respiration on intrathoracic pressure and/or cardiac filling; (3) BP va

14 this effect by augmenting pleural and other intrathoracic pressures and causing a functional obstruc

15 wall compliance both increase the change in intrathoracic pressures and the value of the dynamic ind

16 ransfusion, mechanical ventilation with high intrathoracic pressure, and acidosis, among others.

17 It may relate to increased intrathoracic pressure associated with retching and vomi

18 ves were compared between data obtained with intrathoracic pressure at atmospheric and with a phasic

19 on, hypoxia, hypoventilation, and changes in intrathoracic pressure can lead to severe hemodynamic in

20 negative inspiratory and positive expiratory intrathoracic pressures cancel each other out, so averag

21 Intrathoracic pressure changes are of particular importa

22 tions, with closed-chest and phasic negative intrathoracic pressure changes similar to those associat

23 onary arterial baroreceptors were altered by intrathoracic pressure changes similar to those encounte

24 acic pressure was at atmospheric, the phasic intrathoracic pressure decreased the pulmonary arterial

25 This new device enhances negative intrathoracic pressure during chest wall recoil or the d

26 d neck tissues as the generation of negative intrathoracic pressure during inspiration increases veno

27 Increased negative intrathoracic pressure during spontaneous inspiration th

28 Generation of negative intrathoracic pressure during the decompression phase of

29 in end-expiratory lung volume and increased intrathoracic pressure, eventually exacerbated by expira

30 Application of phasic negative intrathoracic pressures further reduced the threshold an

31 pulmonary resuscitation (CPR) with decreased intrathoracic pressure in the decompression phase can le

32 d and resealed, and (c) with phasic negative intrathoracic pressures in the resealed chest.

33 olume (4, 6, 8, and 10 mL/kg), the change in intrathoracic pressures increased linearly with 0.9 +/-

34 downward flow of venous blood due to reduced intrathoracic pressure is counterbalanced by an upward m

35 Elevated intrathoracic pressure may be similarly associated with

36 12, 20, and 30 breaths per minute, the mean intrathoracic pressure (mm Hg/min) and coronary perfusio

37 An increase in intrathoracic pressure played a deleterious role in Font

38 ic pressure at atmospheric and with a phasic intrathoracic pressure ranging from atmospheric to aroun

39 A novel device, the intrathoracic pressure regulator (ITPR), combines an ins

40 r with active compression-decompression plus intrathoracic pressure regulator compared with active co

41 w with active compression-decompression plus intrathoracic pressure regulator plus epinephrine were s

42 r, and active compression-decompression plus intrathoracic pressure regulator plus epinephrine.

43 t with active compression-decompression plus intrathoracic pressure regulator significantly improved

44 evice, active compression-decompression plus intrathoracic pressure regulator, and active compression

45 t with active compression-decompression plus intrathoracic pressure regulator.

46 t with active compression-decompression plus intrathoracic pressure regulator; and group C-3 minutes

47 n very severe COPD, the impressive swings in intrathoracic pressure resulting from deranged ventilato

48 ompression CPR with augmentation of negative intrathoracic pressure should be considered as an altern

49 as exchange was achieved at lower airway and intrathoracic pressures than those that developed during

50 g expiration to take advantage of changes in intrathoracic pressure that assist in postural maintenan

51 disease (COPD) may contribute to changes in intrathoracic pressure that increase LV wall stress.

52 ic vascular resistance and abrupt changes in intrathoracic pressure that occur with resistive exercis

53 Because obstructive events generate negative intrathoracic pressure that reduces left ventricular (LV

54 t the ITD would result in a greater negative intrathoracic pressure to enhance cardiac venous return,

55 on-decompression CPR with augmented negative intrathoracic pressure (via an impedance-threshold devic

56 Compared to the values obtained when intrathoracic pressure was at atmospheric, the phasic in

57 Furthermore, qualitative changes in intrathoracic pressure were without influence on the res

58 Piglets' hemodynamic and intrathoracic pressures were continuously monitored duri

59 These results have shown that a phasic intrathoracic pressure, which simulates respiratory osci

60 se findings suggest that increasing negative intrathoracic pressure with ITD breathing improves heart

61 tory variation is due to increased change in intrathoracic pressure with respiration in chronic obstr

62 s/min combined with augmentation of negative intrathoracic pressure would lower intracranial pressure

63 ttern in the superior vena cava (affected by intrathoracic pressure) would be different in these two