ライフサイエンスコーパス: intravascular ultrasound

1 l plaque volume was measured by quantitative intravascular ultrasound.

2 maging is hampered by the invasive nature of intravascular ultrasound.

3 served in 16 of 42 patients (38%) undergoing intravascular ultrasound.

4 atheroma progression was evaluated by serial intravascular ultrasound.

5 dent predictors for future cardiac events by intravascular ultrasound.

6 prit lesions identified on virtual-histology intravascular ultrasound.

7 allograft vascular disease (CAV) assessed by intravascular ultrasound.

8 y endovascular treatment using tools such as intravascular ultrasound.

9 red to reduce atheroma volume as measured by intravascular ultrasound.

10 d severe coronary arteriopathy documented by intravascular ultrasound.

11 ared with catheter angiographic findings and intravascular ultrasound.

12 , allograft vasculopathy is best detected by intravascular ultrasound.

13 ive biopsies developed intimal thickening by intravascular ultrasound.

14 ary anatomy was evaluated by angiography and intravascular ultrasound.

15 tients undergoing 3-vessel virtual-histology intravascular ultrasound.

16 CAV was investigated using intravascular ultrasound.

17 by consecutive volumetric three-dimensional intravascular ultrasound.

18 evation myocardial infarction (NSTEMI) using intravascular ultrasound.

19 py with FFR, near-infrared spectroscopy, and intravascular ultrasound.

20 oherence tomography and 0.17+/-0.26 mm(2) on intravascular ultrasound.

21 15 mm vs 1.46 mm, p<0.0001) and quantitative intravascular ultrasound (2.85 mm(2)vs 3.60 mm(2), p<0.0

22 (mean 49 years old) using three-dimensional intravascular ultrasound (3-D IVUS) examination of the l

23 grade IV coronary allograft vasculopathy on intravascular ultrasound, 3 of whom had angiographic dis

24 formation were studied with angiography and intravascular ultrasound 6 months after the index PCI.

25 e in first-year maximal intimal thickness by intravascular ultrasound, a recognized surrogate for lon

26 Serial volumetric intravascular ultrasound analyses (poststent and follow-

27 Intravascular ultrasound analysis at 9 months and final

28 nsplant recipients had baseline and one-year intravascular ultrasound analysis done to assess the pro

29 Serial 3-dimensional intravascular ultrasound analysis in the left anterior d

30 At 3 months, intravascular ultrasound analysis revealed that lumen cr

31 Serial (pre-, post- and follow-up) intravascular ultrasound analysis was performed in 46 na

32 Methods and Results- Volumetric intravascular ultrasound analysis was performed in 70 IS

33 Intravascular ultrasound and cardiac magnetic resonance

34 Using serial studies with intravascular ultrasound and Doppler flow-wire measureme

35 Intravascular ultrasound and histology showed no effect

36 Intravascular imaging-intravascular ultrasound and more recently optical coher

37 Recent findings utilizing intravascular ultrasound and optical coherence tomograph

38 in could regress coronary atherosclerosis by intravascular ultrasound and quantitative coronary angio

39 e vessel wall are apparent on angiography or intravascular ultrasound and that it has a prognostic va

40 ated in vivo based on virtual histology (VH) intravascular ultrasound and whether PSS varied accordin

41 ged by means of invasive techniques, such as intravascular ultrasound (and derived techniques), optic

42 intravascular ultrasound (virtual histology intravascular ultrasound) and computational fluid dynami

43 ural infarction using angiographic analysis, intravascular ultrasound, and delayed-enhancement magnet

44 on, including fluoroscopy, echocardiography, intravascular ultrasound, and electron beam computed tom

45 allograft vasculopathy (CAV) assessed by 3D intravascular ultrasound, and incidence of cardiac adver

46 Patients underwent serial coronary intravascular ultrasound, and interstitial myocardial fi

47 9-17 years) underwent coronary angiography, intravascular ultrasound, and MRI.

48 ility have been described by CT angiography, intravascular ultrasound, and optical coherence tomograp

49 eds conventional magnetic resonance imaging, intravascular ultrasound, and optical coherence tomograp

50 rvation systems by quantitative angiography, intravascular ultrasound, and optical coherence tomograp

51 , such as quantitative coronary angiography, intravascular ultrasound, and optical coherence tomograp

52 , such as quantitative coronary angiography, intravascular ultrasound, and optical coherence tomograp

53 erence in luminal dimension was confirmed by intravascular ultrasound assessment of the minimum lumen

54 Intravascular ultrasound assessments showed a preservati

55 All patients underwent intravascular ultrasound at 1 month and 1 year after tra

56 y angiography and 90 grafts were examined by intravascular ultrasound at 1 year after CABG.

57 Follow-up included coronary angiography and intravascular ultrasound at 4 months and clinical assess

58 subgroup of patients (n=56) underwent serial intravascular ultrasound at baseline and 9 months indica

59 Intravascular ultrasound at one year demonstrated rapid

60 Intravascular ultrasound-based 3-dimensional reconstruct

61 Results- Intravascular ultrasound-based, geometrically correct 3-

62 laque volume in ACS patients, as assessed by intravascular ultrasound, but no clinical trials assessi

63 cular ultrasound (IB-IVUS), and conventional intravascular ultrasound (C-IVUS) for tissue characteriz

64 agnosis of ISA, initially only possible with intravascular ultrasound, can currently be performed wit

65 ent minus lumen) areas and source-to-target (intravascular ultrasound catheter to external elastic me

66 thod of transthoracic echocardiography (with intravascular ultrasound catheters) at baseline and on d

67 The coprimary end point of first-year intravascular ultrasound change demonstrated no differen

68 patients who had undergone virtual histology intravascular ultrasound characterization of coronary pl

69 nce assessed by quantitative angiography and intravascular ultrasound; composite clinical endpoints b

70 dicts a suboptimal result based on validated intravascular ultrasound criteria; however, an FFR >/=0.

71 from baseline of percent atheroma volume on intravascular ultrasound, CRP-modulating effects, or MAC

72 ubstudies, 103 patients (54 BMS, 49 PES) had intravascular ultrasound data >/=10 mm distal to the ste

73 Intravascular ultrasound data at the sites of PP were co

74 raft plaque was divided on virtual histology intravascular ultrasound-derived "inflammatory" (VHD-IP)

75 dy to Evaluate the Effect of Rosuvastatin on Intravascular Ultrasound-Derived Coronary Atheroma Burde

76 dy to Evaluate the Effect of Rosuvastatin on Intravascular Ultrasound-Derived Coronary Atheroma Burde

77 giographic disease (n=23) and NSTEMI (n=24), intravascular ultrasound-derived measures (percent ather

78 ), changes in plaque area, virtual histology intravascular ultrasound-derived plaque composition, and

79 Intravascular ultrasound detects stent edge dissections

80 theter thrombolysis regimen, the addition of intravascular ultrasound did not facilitate thrombus res

81 The Study of Coronary Atheroma by Intravascular Ultrasound: Effect of Rosuvastatin Versus

82 hanges in lipoprotein levels and the primary intravascular ultrasound end point, change in percent at

83 irty-two consecutive HT recipients underwent intravascular ultrasound evaluation at month 1 and year

84 media (EEM-lumen) areas were measured (using intravascular ultrasound) every 1 mm over 5-mm-long refe

85 ith coronary artery disease underwent serial intravascular ultrasound examination in 7 clinical trial

86 cipients between 1993 and 1995 who underwent intravascular ultrasound examination of the coronary art

87 d and received study drug; 502 had evaluable intravascular ultrasound examinations at baseline and af

88 tients with coronary bifurcation lesion, 120 intravascular ultrasound examinations of the MV were per

89 In 38 transplant recipients, serial intravascular ultrasound examinations were performed 3.7

90 After serial intravascular ultrasound examinations, only small remnan

91 y was performed regardless of performance of intravascular ultrasound examinations.

92 ared serial (postintervention and follow-up) intravascular ultrasound findings in 66 patients with na

93 scribe near-infrared spectroscopy (NIRS) and intravascular ultrasound findings in pre-existing stents

94 Intravascular ultrasound findings suggesting thrombus we

95 At the site of LRP, intravascular ultrasound found no neointimal tissue in 3

96 nt coronary angiography followed by coronary intravascular ultrasound, fractional flow reserve, and i

97 A total of 4429 VH intravascular ultrasound frames from 53 patients were an

98 eration DES (HR=1.75, P=0.02), no procedural intravascular ultrasound guidance (HR=1.75, P=0.04), and

99 The frequency of ST may be reduced with intravascular ultrasound -guided stenting, assiduous adh

100 Intravascular ultrasound-guided stent overexpansion (fin

101 ave not systematically evaluated the role of intravascular ultrasound-guided stenting and high platel

102 Intravascular ultrasound-guided stenting has been shown

103 ified 989 consecutive patients who underwent intravascular ultrasound-guided stenting of 1,015 corona

104 SF defined by catheter angiography or intravascular ultrasound has been implicated in ISR.

105 Intravascular ultrasound has been used to gain important

106 ontemporary imaging technology, particularly intravascular ultrasound, has allowed the study of arter

107 In vivo studies with intravascular ultrasound have shown that complex plaque

108 nce tomography (OCT), integrated backscatter intravascular ultrasound (IB-IVUS), and conventional int

109 Intravascular ultrasound identified intramural hematomas

110 he left anterior descending coronary artery; intravascular ultrasound images and Doppler velocities w

111 Coronary angiography and intravascular ultrasound images were analyzed.

112 In each segment, intravascular ultrasound images were digitized at 1-mm i

113 Intravascular ultrasound images were digitized every 1 m

114 Intravascular ultrasound imaging before stenting may be

115 Intravascular ultrasound imaging is the most sensitive t

116 Baseline intravascular ultrasound imaging was performed 0.9 +/- 0

117 Repeat intravascular ultrasound imaging was performed after con

118 ed in 7 clinical trials that employed serial intravascular ultrasound imaging.

119 t coronary artery disease (CAD) using serial intravascular ultrasound imaging.

120 nt serial evaluation of atheroma burden with intravascular ultrasound imaging.

121 This is the first study of POT guided by intravascular ultrasound in patients with coronary bifur

122 s with symptomatic carotid disease, and with intravascular ultrasound in patients with stable angina.

123 Intima-media thickness was assessed by intravascular ultrasound in the coronary arteries at wee

124 Intravascular ultrasound is a more sensitive diagnostic

125 Intravascular ultrasound is an accurate method to assess

126 Intravascular ultrasound is invasive and costly.

127 Intravascular ultrasound is more sensitive than coronary

128 ases (SES: 72; BMS: 50) with complete serial intravascular ultrasound (IVUS) (baseline and 8-month fo

129 re final lumen cross-sectional area (CSA) by intravascular ultrasound (IVUS) (p = 0.001) and restenot

130 Arteries were imaged with intravascular ultrasound (IVUS) 5 and 10 min after ELIP

131 Serial volumetric intravascular ultrasound (IVUS) analysis was performed i

132 Intravascular ultrasound (IVUS) and cardiac MRI (CMR) we

133 Intravascular ultrasound (IVUS) and fractional flow rese

134 general, it has a good correlation with both intravascular ultrasound (IVUS) and histopathology for d

135 tent of coronary atherosclerosis assessed by intravascular ultrasound (IVUS) and its rate of progress

136 hereby assessed whether integrating EIS with intravascular ultrasound (IVUS) and shear stress (ISS) p

137 Concomitant OCT and intravascular ultrasound (IVUS) area measurements were p

138 rwent simultaneous endomyocardial biopsy and intravascular ultrasound (IVUS) at one year of transplan

139 We assessed the pre- and post-PCI intravascular ultrasound (IVUS) characteristics of subac

140 imize stenting by comparing it with standard intravascular ultrasound (IVUS) criteria.

141 sought to assess the validity of first-year intravascular ultrasound (IVUS) data as a surrogate mark

142 Serial intravascular ultrasound (IVUS) data from the Reversal o

143 Spectral analysis of backscattered intravascular ultrasound (IVUS) data has potential for r

144 In vivo intravascular ultrasound (IVUS) data were acquired from

145 nderwent coronary angiography and volumetric intravascular ultrasound (IVUS) evaluation of the left a

146 Patients underwent intravascular ultrasound (IVUS) evaluation of the target

147 s designed to examine the impact of repeated intravascular ultrasound (IVUS) examinations on transpla

148 thickness (CMIT) and plaque volume (CPV) by intravascular ultrasound (IVUS) examinations.

149 We report intravascular ultrasound (IVUS) findings after crush-ste

150 era of drug-eluting stents, it is unknown if intravascular ultrasound (IVUS) guidance for percutaneou

151 Intravascular ultrasound (IVUS) guidance has been shown

152 to modest-sized studies suggest a benefit of intravascular ultrasound (IVUS) guidance in noncomplex l

153 l studies have indicated better outcome with intravascular ultrasound (IVUS) guidance when performing

154 Intravascular ultrasound (IVUS) has become an indispensa

155 Intravascular ultrasound (IVUS) has played an integral r

156 Intravascular ultrasound (IVUS) has the ability to detec

157 neously obtained endomyocardial biopsies and intravascular ultrasound (IVUS) images of coronary arter

158 Optical coherence tomography and intravascular ultrasound (IVUS) images of these sites we

159 Participants underwent coronary intravascular ultrasound (IVUS) imaging and were randomi

160 ultaneous optical coherence tomography (OCT)-intravascular ultrasound (IVUS) imaging at 72 frames per

161 scending artery and compared with volumetric intravascular ultrasound (IVUS) imaging.

162 Intravascular ultrasound (IVUS) is a valuable adjunct to

163 Intravascular ultrasound (IVUS) is being used to assess

164 It can only be detected if intravascular ultrasound (IVUS) is performed at follow-u

165 tent malapposition (LSM) is only detected if intravascular ultrasound (IVUS) is performed at implanta

166 Although intravascular ultrasound (IVUS) is widely used to detect

167 uantitative coronary angiographic and planar intravascular ultrasound (IVUS) measurements were perfor

168 T (0.75-mm collimation, 420-ms rotation) and intravascular ultrasound (IVUS) of one coronary artery w

169 who underwent stenting under the guidance of intravascular ultrasound (IVUS) or conventional angiogra

170 High-frequency intravascular ultrasound (IVUS) revealed atherosclerotic

171 Previous intravascular ultrasound (IVUS) studies have demonstrate

172 Coronary angiography and intravascular ultrasound (IVUS) studies were performed a

173 Serial 3-dimensional intravascular ultrasound (IVUS) studies were performed w

174 The 274 patients who completed the intravascular ultrasound (IVUS) substudy of the CAMELOT

175 mine the optimal minimum lumen area (MLA) by intravascular ultrasound (IVUS) that correlates with fra

176 We used intravascular ultrasound (IVUS) to assess 78 coronary ar

177 We used serial intravascular ultrasound (IVUS) to assess disease progre

178 The goal of this study was to use intravascular ultrasound (IVUS) to compare octogenarians

179 volumetric (post-irradiation and follow-up) intravascular ultrasound (IVUS) to compare the effective

180 The current study analyzed intravascular ultrasound (IVUS) to define the changes th

181 We used diagnostic and preintervention intravascular ultrasound (IVUS) to determine the inciden

182 We used intravascular ultrasound (IVUS) to evaluate recurrence a

183 This study used serial volumetric intravascular ultrasound (IVUS) to evaluate the effect o

184 m of this study was to use serial volumetric intravascular ultrasound (IVUS) to evaluate the effects

185 The aim of this study was to use serial intravascular ultrasound (IVUS) to evaluate the long-ter

186 We used serial intravascular ultrasound (IVUS) to study patients with l

187 Prior intravascular ultrasound (IVUS) trials have demonstrated

188 study purposes were to determine 1) whether intravascular ultrasound (IVUS) was more sensitive than

189 Intravascular ultrasound (IVUS) was used to evaluate 38

190 tics, quantitative coronary angiography, and intravascular ultrasound (IVUS) were evaluated in subjec

191 s the first intravascular catheter combining intravascular ultrasound (IVUS) with multispectral fluor

192 enal translesional pressure gradients (TPG), intravascular ultrasound (IVUS), and angiographic parame

193 Six-month angiographic, intravascular ultrasound (IVUS), and clinical follow-up

194 atients (B2) underwent coronary angiography, intravascular ultrasound (IVUS), and optical coherence t

195 to compare color-flow duplex imaging (CFDI), intravascular ultrasound (IVUS), and renal arteriography

196 Intravascular ultrasound (IVUS)-attenuated plaque is cha

197 Are progressive changes in intravascular ultrasound (IVUS)-derived indexes of plaqu

198 Intravascular ultrasound (IVUS)-derived lumen, outer ste

199 was to investigate the relationship between intravascular ultrasound (IVUS)-derived measures of athe

200 his study was to evaluate the efficacy of an intravascular ultrasound (IVUS)-guided strategy for pati

201 etes mellitus using 3-dimensional volumetric intravascular ultrasound (IVUS).

202 ivo, and results were compared with those of intravascular ultrasound (IVUS).

203 ut not in SVGs have been well described with intravascular ultrasound (IVUS).

204 ents measured atherosclerosis progression by intravascular ultrasound (IVUS).

205 ment of coronary flow reserve (CFR), and (3) intravascular ultrasound (IVUS).

206 cebo on coronary atheroma burden measured by intravascular ultrasound (IVUS).

207 hic correlates of plaque rupture detected by intravascular ultrasound (IVUS).

208 ses of qDES compared with BMS as assessed by intravascular ultrasound (IVUS).

209 ith decreased coronary plaque progression by intravascular ultrasound (IVUS).

210 l LAD plaque volume was determined by use of intravascular ultrasound (IVUS).

211 ssed in 108 native coronary lesions by using intravascular ultrasound (IVUS).

212 plaque burden as assessed by preintervention intravascular ultrasound (IVUS).

213 study the progression of GVD by using serial intravascular ultrasound (IVUS).

214 al coronary angiography (CCAG) alone or with intravascular ultrasound (IVUS).

215 g coronary three-dimensional (3D) volumetric intravascular ultrasound (IVUS).

216 l (postirradiation and follow-up) volumetric intravascular ultrasound (IVUS): 1) to evaluate the actu

217 s include fractional flow reserve; grayscale intravascular ultrasound (IVUS); IVUS radiofrequency tis

218 dy comparing SES and BMS, serial qualitative intravascular ultrasound (IVUS; at stent implantation an

219 tissue at the site of LRP detected by NIRS, intravascular ultrasound may provide some insight into t

220 ed quantitative angiography and morphometric intravascular ultrasound measurements pre and post proce

221 tin Versus Atorvastatin (SATURN) used serial intravascular ultrasound measures of coronary atheroma v

222 LGE scores correlate well with traditional intravascular ultrasound measures.

223 In atherosclerosis, 2D NIRF, intravascular ultrasound-NIRF fusion, microscopy, and im

224 We investigated the role of POT guided by intravascular ultrasound on the main vessel (MV) stent e

225 and a subgroup of patients was scheduled for intravascular ultrasound, optical coherence tomography,

226 hod among quantitative coronary angiography, intravascular ultrasound, optical coherence tomography,

227 ral care were examined, including the use of intravascular ultrasound, optical coherence tomography,

228 re available (eg, fractional flow reserve or intravascular ultrasound) or being validated (eg, instan

229 ive measures of restenosis (angiographic and intravascular ultrasound) or its clinical sequelae.

230 clerosis was evaluated by B-mode ultrasound, intravascular ultrasound, or angiography.

231 compared with baseline (0.54+/-1.09 mm(2) on intravascular ultrasound, P=0.003 and 0.77+/-1.33 m(2) o

232 imus-eluting stent groups, respectively, and intravascular ultrasound percent neointimal hyperplasia

233 CAV was diagnosed through intravascular ultrasound performed 1 month and 1 year af

234 Intravascular ultrasound plaque rupture strongly correla

235 nd points, such as quantitative angiography, intravascular ultrasound, plasma biomarkers, and functio

236 We investigated 3D intravascular ultrasound (postprocedure and 6 to 9 month

237 Intravascular ultrasound provided coregistered anatomica

238 Atheroma volume was determined in serial intravascular ultrasound pullbacks in matched arterial s

239 gnificantly correlated with plaque volume by intravascular ultrasound (r=0.69; P<0.0001) but not with

240 (Fractional Flow Reserve and Intravascular Ultrasound Relationship Study [FIRST]; NCT

241 FIRST (Fractional Flow Reserve and Intravascular Ultrasound Relationship Study) aimed to de

242 Although angiographic and intravascular ultrasound results at 8 months demonstrate

243 For each virtual histology intravascular ultrasound segment (n=2249), changes in pl

244 tive randomized trials using serial coronary intravascular ultrasound, serial changes in coronary per

245 Intravascular ultrasound showed a significantly greater

246 Intravascular ultrasound showed atheroma volume regressi

247 Intravascular ultrasound showed that early failure lesio

248 Intravascular ultrasound shows significant reference seg

249 Intravascular ultrasound studies have demonstrated that

250 these patients subsequently underwent two 3D intravascular ultrasound studies in 2004 to 2006 12 mont

251 Postmortem and intravascular ultrasound studies of arterial remodeling

252 one orthotopic heart transplantation, serial intravascular ultrasound studies of the proximal left an

253 Three-dimensional intravascular ultrasound studies were performed at basel

254 Three-dimensional intravascular ultrasound studies were performed at basel

255 Intravascular ultrasound studies with motorized pullback

256 S or stable angina, consistent with previous intravascular ultrasound studies.

257 n, and time from transplantation to baseline intravascular ultrasound study were not different (P>0.2

258 iren Quantitative Atherosclerosis Regression Intravascular Ultrasound Study) comparing aliskiren with

259 stents (BMS) on distal vessels in the serial intravascular ultrasound substudies of TAXUS IV, V, and

260 In their intravascular ultrasound substudies, 103 patients (54 BM

261 The intravascular ultrasound substudy enrolled 241 patients

262 METHODS AND A formal intravascular ultrasound substudy enrolled 464 patients

263 e associated with more high-risk features on intravascular ultrasound than those without uptake: posi

264 patients (2433 lesions) were evaluated with intravascular ultrasound to characterize the morphologic

265 We used intravascular ultrasound to determine the incidence, mor

266 m of this study was to use serial volumetric intravascular ultrasound to evaluate the effect of gamma

267 creased the positive predictive value for VH intravascular ultrasound to identify clinical presentati

268 SS improved the ability of virtual-histology intravascular ultrasound to predict MACE in plaques with

269 Virtual-histology intravascular ultrasound (VH-IVUS) and optical coherence

270 Recent studies show that virtual histology intravascular ultrasound (VH-IVUS) can identify plaques

271 therosclerotic plaque with virtual histology intravascular ultrasound (VH-IVUS) imaging to assess the

272 vivo CT coregistered with virtual histology intravascular ultrasound (VH-IVUS) in 108 plaques from 5

273 We used virtual histology intravascular ultrasound (VH-IVUS) to investigate the na

274 nderwent baseline and 6-month radiofrequency intravascular ultrasound (virtual histology intravascula

275 Mean neointimal area by intravascular ultrasound was higher in PF-PES than in PB

276 Intravascular ultrasound was more sensitive than angiogr

277 n lumen size by quantitative angiography and intravascular ultrasound was observed in nonballoon dene

278 Intravascular ultrasound was performed after stent deplo

279 Intravascular ultrasound was performed at baseline and f

280 In a subset of these patients (n=1107), intravascular ultrasound was performed at follow-up.

281 Intravascular ultrasound was performed during angiograph

282 Six-month intravascular ultrasound was performed in 20% of the pat

283 Intravascular ultrasound was performed in 24 lesions wit

284 Intravascular ultrasound was performed in 262 heart tran

285 AND Combined near-infrared spectroscopy and intravascular ultrasound was performed in 57 vessels in

286 serial (baseline and 1-year post-transplant) intravascular ultrasound was performed in the first 50 m

287 s, 3-vessel gray-scale and virtual histology intravascular ultrasound was performed in the proximal-m

288 Intravascular ultrasound was performed on 121 patients w

289 Intravascular ultrasound was performed within 2 weeks fo

290 PSS derived from virtual-histology intravascular ultrasound was subsequently estimated in n

291 by NIRS in a cohort of pre-existing stents, intravascular ultrasound was used to determine the prese

292 Intravascular ultrasound was used to measure progression

293 ar profiling, using coronary angiography and intravascular ultrasound, was used to reconstruct each a

294 Coronary angiography and three-dimensional intravascular ultrasound were performed at baseline and

295 ne and 6-12 months) coronary angiography and intravascular ultrasound were performed in 2931 lesions

296 uation, serial quantitative angiography, and intravascular ultrasound were performed periprocedurally

297 Changes in atheroma burden monitored by intravascular ultrasound were studied in 3,437 patients

298 mography and near-infrared spectroscopy with intravascular ultrasound were used to characterize NA in

299 erity of GVD was determined every 3 weeks by intravascular ultrasound, which quantified intimal area

300 eserve, endothelial function assessment, and intravascular ultrasound with volumetric analysis were p