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1 rs for the purpose of developing hyperimmune intravenous immunoglobulin.
2 cal agents such as infliximab, rituximab and intravenous immunoglobulin.
3 rm was searched in combination with the term intravenous immunoglobulin.
4 imus, pimecrolimus, and imiquimod as well as intravenous immunoglobulin.
5 s of natural human IgG antibodies present in intravenous immunoglobulin.
6 id not respond to treatment with steroids or intravenous immunoglobulin.
7 et of response is slower than that seen with intravenous immunoglobulin.
8 tivity was promptly reversed with additional intravenous immunoglobulin.
9 nd ecchymoses, and recovered after receiving intravenous immunoglobulin.
10 the majority of cases reporting success with intravenous immunoglobulin.
11 reduction of immunosuppression, and frequent intravenous immunoglobulin.
12 o 7 days after completion of the infusion of intravenous immunoglobulin.
13 eroids in addition to plasma exchange and/or intravenous immunoglobulin.
14 inating polyneuropathy (CIDP) need long-term intravenous immunoglobulin.
15 Of those treated, 99% were treated with intravenous immunoglobulin.
16 asculitis that is treated with high doses of intravenous immunoglobulin.
17 rly axonal involvement, and poor response to intravenous immunoglobulin.
18 h groups received dopamine or dobutamine and intravenous immunoglobulin.
19 idofovir, leflunomide, fluoroquinolones, and intravenous immunoglobulins.
20 were observed for other therapies, including intravenous immunoglobulins.
28 osal bleeding at diagnosis or treatment with intravenous immunoglobulin alone developed chronic ITP l
29 is regimen, the next step is the addition of intravenous immunoglobulin although this is not supporte
30 rugs, including biologic therapy, as well as intravenous immunoglobulin, although results have been m
31 nduced by injection of heat-aggregated human intravenous immunoglobulin and active systemic anaphylax
32 addition of infliximab to standard therapy (intravenous immunoglobulin and aspirin) in acute Kawasak
34 ously published trials evaluating the use of intravenous immunoglobulin and colony-stimulating factor
37 nd address the role of fluorinated steroids, intravenous immunoglobulin and hydroxychloroquine for pr
38 re incubated on glass coverslips coated with intravenous immunoglobulin and inactive complement compo
43 estigated the effect of desensitization with intravenous immunoglobulin and rituximab on the antibody
44 f four plasmapheresis treatments followed by intravenous immunoglobulin and splenectomy at the time o
45 ition was not treated with plasmapheresis or intravenous immunoglobulin and was not associated with p
46 Treatment with glucocorticoids-less so with intravenous immunoglobulins and plasma exchange-was asso
47 For 2 years, the patient was treated with intravenous immunoglobulins and steroids, with partial i
48 ral arm of the immune system by using either intravenous immunoglobulins and/or immunoadsorption will
49 ve therapy (azathioprine or methotrexate and intravenous immunoglobulin) and had normalization of str
50 ith antitumor necrosis factor agents, pooled intravenous immunoglobulin, and anti-B-cell therapies su
51 (using plasmapheresis followed by 100 mg/kg intravenous immunoglobulin, and anti-CD20 antibody), and
53 nts, and was reversible with plasmapheresis, intravenous immunoglobulin, and increasing immunosuppres
54 nsisted of plasma exchange, corticosteroids, intravenous immunoglobulin, and other immunosuppressive
59 ination of anticoagulation, corticosteroids, intravenous immunoglobulin, and plasma exchange; there i
60 immunomodulatory agents including steroids, intravenous immunoglobulin, and plasmapheresis have show
61 yte antigen antibodies using plasmapheresis, intravenous immunoglobulin, and rituximab has been repor
62 ived previous treatment with plasmapheresis, intravenous immunoglobulin, and rituximab that was ineff
65 ing plasmapheresis preconditioning, low-dose intravenous immunoglobulin, and standard maintenance imm
66 e prophylaxis with steroids, pentoxifylline, intravenous immunoglobulin, and total body irradiation).
67 pies including corticosteroids, splenectomy, intravenous immunoglobulin, and various cytotoxic or imm
68 e impossible for mycophenolate, montelukast, intravenous immunoglobulins, and systemic glucocorticost
69 responses to treatment with glucocorticoids, intravenous immunoglobulins, and/or plasma exchange at s
70 patients received intravenous steroids; 43, intravenous immunoglobulin; and 13, plasma exchange; or
71 of the underling inflammatory condition with intravenous immunoglobulin, anti TNF agents, thalidomide
72 been suggested, including administration of intravenous immunoglobulin, apheresis, and combination t
73 manage, but antimalarials, methotrexate, and intravenous immunoglobulin are effective in small, often
77 r to those seen in treatment with polyclonal intravenous immunoglobulin, but anemia requiring blood t
78 ivalent efficacy of both plasma exchange and intravenous immunoglobulin, but not corticosteroids, in
79 tients improve with GABA-enhancing drugs and intravenous immunoglobulin, but some respond poorly and
80 trate that the anti-inflammatory activity of intravenous immunoglobulin can be recapitulated by the t
81 econdary causes, we treated the patient with intravenous immunoglobulin, considering primary neuromyo
82 g therapies, including glucocorticosteroids, intravenous immunoglobulins, cyclosporine, plasmapheresi
83 rticotropin hormone, corticosteroids, and/or intravenous immunoglobulin, develop long-term neurologic
86 ethylprednisolone for 5 days, 0.4 mg/kg/d of intravenous immunoglobulin for 5 days, and 2 doses of ri
87 rdiography is a criterion for treatment with intravenous immunoglobulin for incomplete Kawasaki disea
90 by individual human sera or by pooled human, intravenous immunoglobulin G (IVIG) were dispersed over
91 osomes (n=2) or commercially available human intravenous immunoglobulin G depleted of anti-Gal Ab (n=
93 aggressive management with corticosteroids, intravenous immunoglobulin G, or anti-D immune globulin.
94 reductions in immunosuppressive medication, intravenous immunoglobulin, ganciclovir, and rituximab.
97 usion of convalescent plasma (or hyperimmune intravenous immunoglobulin) has been reported to be an e
98 xisting therapeutics such as the delivery of intravenous immunoglobulin have led to interest in devel
99 esponses to anti-influenza virus hyperimmune intravenous immunoglobulin (hIVIG) were characterized.
100 mune anti-human immunodeficiency virus (HIV) intravenous immunoglobulin (HIVIG) were evaluated in the
101 a mouse model of EV-D68 infection: (1) human intravenous immunoglobulin (hIVIG), (2) fluoxetine, and
102 investigate the effect of Thymoglobulin and intravenous immunoglobulin (i.v.IG) therapy on the clini
104 unodeficiency (PI) is equally efficacious to intravenous immunoglobulin (IGIV), induces fewer systemi
105 -term follow-up data received immunotherapy (intravenous immunoglobulin in 10 and corticosteroids and
106 e from randomized trials supports the use of intravenous immunoglobulin in Guillain-Barre syndrome, c
109 ramer acetate in primary progressive MS, and intravenous immunoglobulin in secondary progressive MS).
110 thymoglobulin induction, plasmapheresis, and intravenous immunoglobulin in the highest risk groups.
111 echanisms of action, efficacy, and safety of intravenous immunoglobulins in rheumatic diseases demons
113 stored sera from 254 infected children in an intravenous immunoglobulin infection prophylaxis trial.
114 ressive protocol consisted of plasmapheresis/intravenous immunoglobulin infusion before LDLT followed
117 vel anti-metabolites; combination therapies; intravenous immunoglobulin; intravitreally inserted cort
120 ry aneurysms; this risk is reduced 5-fold if intravenous immunoglobulin is administered within 10 day
122 bocytopenic bleeding in the third trimester, intravenous immunoglobulin is an appropriate first-line
125 enolate mofetil, dehydroepiandrosterone, and intravenous immunoglobulin is increasing, but plasmapher
131 phosphamide combined with plasmapheresis and intravenous immunoglobulins is an option for patients wi
132 infants born to mothers following antenatal intravenous immunoglobulin (IVIG) +/- prednisone therapy
134 rtiary centers compared the effectiveness of intravenous immunoglobulin (IVIg) and corticosteroids in
135 ctiveness of desensitization using high-dose intravenous immunoglobulin (IVIG) and rituximab to impro
136 e than 5 years to investigate the effects of intravenous immunoglobulin (IVIG) and rituximab treatmen
138 reatment unnecessarily; there was overuse of intravenous immunoglobulin (IVIg) as first-line therapy
139 anaphylactoid reactions on administration of intravenous immunoglobulin (IVIg) associated with the pr
142 of administering during pregnancy high-dose intravenous immunoglobulin (IVIG) derived from pooled se
144 neglobulin (anti-D) and 6 of 8 responding to intravenous immunoglobulin (IVIG) did not have correspon
146 trial showed short and long-term efficacy of intravenous immunoglobulin (IVIG) for the treatment of C
150 evels and improvement of transplant rates by intravenous immunoglobulin (IVIG) in a randomized, doubl
151 were conducted to investigate the effects of intravenous immunoglobulin (IVIG) in a rat model of immu
152 nized paradigm for the therapeutic action of intravenous immunoglobulin (IVIG) in immune thrombocytop
153 espite the beneficial therapeutic effects of intravenous immunoglobulin (IVIg) in inflammatory diseas
165 ith rabbit antithymocyte globulin (RATG) and intravenous immunoglobulin (IVIG) may allow successful t
168 ntibodies received monthly courses of either intravenous immunoglobulin (IVIg) or plasmapheresis, in
171 althy people and routinely in pharmaceutical intravenous immunoglobulin (IVIG) purified from serum po
173 IgG-Fc receptor gene FcgammaRIIB influences intravenous immunoglobulin (IVIG) response in Kawasaki d
174 cytic cells, and a commercial preparation of intravenous immunoglobulin (IVIG) served as the source o
175 Treatment with high-dose (400 mg/kg/day) intravenous immunoglobulin (IVIg) shows benefit in many
177 rpose of this study was to determine whether intravenous immunoglobulin (IVIG) therapy could prevent
184 patient received one plasmapheresis (PP) and intravenous immunoglobulin (IVIg) treatment, anti-CD25,
185 ers as well as fetal inflammatory responses, intravenous immunoglobulin (IVIG) was evaluated as preve
186 provement on the disability grade scale with intravenous immunoglobulin (IVIg) was very similar to th
187 ith AHR treated with plasmapheresis (PP) and intravenous immunoglobulin (IVIG) with allograft surviva
188 compared with treatment with zidovudine and intravenous immunoglobulin (IVIG) without HIV antibody.
191 olysis, and cumulative doses of steroids and intravenous immunoglobulin (IVIG), and ameliorates neuro
193 ents, such as glucocorticoids, vitamin D, or intravenous immunoglobulin (IVIG), are discussed, as the
194 ioperative treatment with plasmapheresis and intravenous immunoglobulin (IVIG), combined with a tacro
195 Other options include plasmapheresis and intravenous immunoglobulin (IVIg), coupled with cytotoxi
196 The US Food and Drug Administration approved intravenous immunoglobulin (IVIg), extracted from the pl
197 tandard practice, aerosolized ribavirin plus intravenous immunoglobulin (IVIG), is extremely expensiv
199 otrexate antibody (AMI)-coated liposomes and intravenous immunoglobulin (IVIG)-coated liposomes (15,
200 ernal-infant pairs enrolled in a randomized, intravenous immunoglobulin (IVIG)-controlled trial of HI
214 is of HBV serology in 16 patients commencing intravenous immunoglobulin (IVIG); and pre- and post-inf
215 n G (IgG) purified from pooled human plasma [intravenous immunoglobulin (IVIG)] confer anti-inflammat
218 pretreatment and 1 year after treatment with intravenous immunoglobulin [IVIG]) from 26 children with
219 have shown that pooled human gammaglobulin (intravenous immunoglobulin [IVIG]) inhibits the mixed ly
220 ingent selection with pooled human antisera (intravenous immunoglobulin [IVIG]) then led to the selec
224 the immune system of the neonate, including: intravenous immunoglobulins, myeloid hematopoietic growt
225 Treatment included corticosteroids (n = 7), intravenous immunoglobulin (n = 3), and plasma exchange
226 rapy consisted of steroids (n = 61/74; 82%), intravenous immunoglobulins (n = 71/74; 96%), and plasma
227 y [cPRA], 34%-99%), desensitization included intravenous immunoglobulin on days 0 and 30 and a single
229 consensus was reached concerning the use of intravenous immunoglobulin or corticosteroids as first-l
230 immunoglobulin in 10 and corticosteroids and intravenous immunoglobulin or other immunosuppressors in
231 lled in the larger study where they received intravenous immunoglobulin or placebo as intervention.
232 or one immunosuppressive therapy and chronic intravenous immunoglobulin or plasma exchange for 12 mon
234 within 12 months before screening, or use of intravenous immunoglobulin or plasma exchange within 4 w
237 with a combination of methylprednisolone and intravenous immunoglobulin (OR 2.67, 95% CI 1.44-4.96).
238 esection and immunotherapy (corticosteroids, intravenous immunoglobulin, or plasma exchange) respond
240 Responses to immunotherapy (corticosteroids, intravenous immunoglobulin, plasma exchange, and immunos
241 on protocol starting at day 0 with high-dose intravenous immunoglobulin, plasma exchanges, and eventu
242 included first-line immunotherapy (steroids, intravenous immunoglobulin, plasmapheresis), second-line
243 blood group incompatible transplant using an intravenous immunoglobulin/plasmapheresis preconditionin
244 more than four pretransplant plasmapheresis/intravenous immunoglobulin (PP/IVIg) had a greater likel
245 ines in producing a functional, high-titered intravenous immunoglobulin preparation for clinical use.
252 underwent treatment with corticosteroids and intravenous immunoglobulin, resulting in clinical improv
255 gents, such as steroids, plasmapheresis, and intravenous immunoglobulin, seem to offer substantial im
259 to a third of KD patients fail to respond to intravenous immunoglobulin, the standard therapy, and al
262 exception of juvenile rheumatoid arthritis, intravenous immunoglobulin therapy appeared ineffective
263 e antibodies must be in some doubt, although intravenous immunoglobulin therapy has been shown to be
264 anti-inflammatory properties associated with intravenous immunoglobulin therapy require the sialic ac
265 and a lack of response to corticosteroid and intravenous immunoglobulin therapy, a 3-day course of pl
266 itioning regimen using plasmapheresis and/or intravenous immunoglobulin therapy, but underlying mecha
267 oved, new, and controversial indications for intravenous immunoglobulin therapy, with special emphasi
269 ibodies were affinity purified by passage of intravenous immunoglobulin through purified, type-specif
271 d with thymocytes from ITP mice treated with intravenous immunoglobulin, thymocytes from untreated IT
272 treated with corticosteroids in addition to intravenous immunoglobulin to improve their outcomes.
273 randomized, controlled trial of prophylactic intravenous immunoglobulin to prevent ICU-associated inf
278 h definite or probable CIDP who responded to intravenous immunoglobulin treatment were eligible.
279 otection, as well as the limited efficacy of intravenous immunoglobulin treatments against human dise
280 nced by immunoglobulin production, decreased intravenous immunoglobulin use, and antibody response af
281 d first-line immunotherapy with steroids and intravenous immunoglobulins vs. late immunotherapy), and
284 ble regimes of steroids, plasma exchange and intravenous immunoglobulin were associated with variable
285 though a consensus agreed that biologics and intravenous immunoglobulin were considered appropriate i
287 ki disease (KD) and 5% of those treated with intravenous immunoglobulin will develop coronary artery
289 e patients were treated with steroids and/or intravenous immunoglobulin, with variable positive respo
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