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1 f the fiber cell-specific protein MIP (major intrinsic protein).
2 a membrane intrinsic proteins from tonoplast intrinsic proteins.
3 re not present in any of the known tonoplast intrinsic proteins.
4 a) and plants overexpressing plasma membrane intrinsic protein 1;4 (PIP1;4) and PIP2;5.
5  thaliana plants lacking the Plasma membrane Intrinsic Protein 2;1 (PIP2;1) aquaporin have a defect i
6 cluded expression of crystallins, lens major intrinsic protein 26 (MIP26), CP49, and filensin and mor
7 eptide antibodies specific for the tonoplast intrinsic proteins alpha-TIP, gamma-TIP, and delta-TIP i
8 e that antibodies to two different tonoplast intrinsic proteins, alpha-TIP and TIP-Ma27, label vacuol
9 bidopsis aquaporin, delta-TIP (for tonoplast intrinsic protein), and show that it is located in the t
10 ase), phosphoenolpyruvate carboxylase, major intrinsic protein, and alpha-tubulin were amplified by p
11 e directly from the ER; a specific tonoplast intrinsic protein; and a novel receptor-like RING-H2 pro
12 tis vinifera) VvPIP2;4N (for Plasma membrane Intrinsic Protein) aquaporin gene.
13            Nodulin 26, a member of the major intrinsic protein/aquaporin (AQP) channel family, is a m
14 cript abundance of Zea maize Plasma Membrane Intrinsic Protein aquaporins.
15                     In contrast, the granule-intrinsic proteins are prone to covalent modification, b
16                                          The intrinsic protein characteristics of globular proteins w
17 urfaces are readily patternable, incorporate intrinsic protein charge into the film, and able to cont
18 s critically dependent on chloride ions, and intrinsic protein charges also play a role.
19                     In green plants, several intrinsic protein components of the photosystem II (PS I
20 ess of specific cuts of meat, based on their intrinsic protein composition.
21                                The stem cell intrinsic proteins controlling this switch are largely u
22                     Vavouri et al. find that intrinsic protein disorder and promiscuous molecular int
23 lex showed that environmental control of the intrinsic protein disorder could in principle explain th
24                                              Intrinsic protein disorder is functionally implicated in
25                          The contribution of intrinsic protein disorder to the nature and function of
26 43%) and highlights functional importance of intrinsic protein disorder.
27    Since ligand binding may be influenced by intrinsic protein dynamical properties, we have characte
28  substrate-induced conformational change and intrinsic protein dynamics.
29          We conclude that individual granule intrinsic proteins exist as monomers and are not deposit
30 efine a new evolutionary branch of the major intrinsic protein family of aquaporin proteins and descr
31 n (BIB) has sequence identity with the major intrinsic protein family that includes the water- and io
32 aration of specimens of adequate quality and intrinsic protein flexibility, rather than imaging or im
33 pha-lactalbumin (alpha-LA) has been shown by intrinsic protein fluorescence and electron spin resonan
34                                      Altered intrinsic protein fluorescence and highly cooperative bi
35 e change is accompanied by a decrease of the intrinsic protein fluorescence and is essential to creat
36 resolving changes in the label fluorescence, intrinsic protein fluorescence as well as in the absorpt
37  we have taken advantage of the quenching of intrinsic protein fluorescence by bound metal ions to co
38 and van't Hoff enthalpy changes derived from intrinsic protein fluorescence changes were in agreement
39                             The quenching of intrinsic protein fluorescence confirmed that the curcum
40 he kinetic profiles for the quenching of the intrinsic protein fluorescence during the course of the
41  as well as a 20 % weaker, 10 nm red-shifted intrinsic protein fluorescence emission spectrum.
42 trate that assaying leuprolide release using intrinsic protein fluorescence in a 96-well format requi
43 3 muM as assessed by changes in DREAM/KChIP3 intrinsic protein fluorescence in the presence of CaM.
44              This suggests that solid state, intrinsic protein fluorescence measurements using the Ca
45 strated to contribute to the large change in intrinsic protein fluorescence observed when the enzyme
46 riophage T7 by monitoring alterations in the intrinsic protein fluorescence of RNA polymerase in stop
47 nto an EC as monitored by alterations in the intrinsic protein fluorescence of the core polymerase re
48 kinetic studies involving alterations in the intrinsic protein fluorescence of the core polymerase up
49 calculated from data in which changes in the intrinsic protein fluorescence of the enzyme associated
50 ration is followed either by a change in the intrinsic protein fluorescence on ligand release, or by
51                  Further experience with the intrinsic protein fluorescence quenching approach to mon
52 IT purified as a monomer and, as measured by intrinsic protein fluorescence quenching, bound Fe(2+) i
53                                              Intrinsic protein fluorescence studies were consistent w
54 s was monitored by following the increase in intrinsic protein fluorescence that occurs upon ligand d
55 n derivatives of ArsD exhibited quenching of intrinsic protein fluorescence upon binding of As(III) o
56 tifying the time-dependent increase in total intrinsic protein fluorescence was assessed.
57                                          The intrinsic protein fluorescence was attributed primarily
58          The effect of As(III) or Sb(III) on intrinsic protein fluorescence was used to examine the p
59                       (BIP)(2)B quenched NS3 intrinsic protein fluorescence with an apparent dissocia
60 ditions using a combination of solution NMR, intrinsic protein fluorescence, and chromophoric chelato
61 were determined by monitoring changes in the intrinsic protein fluorescence, in the fluorescence of f
62 uench fluorophores that are red-shifted from intrinsic protein fluorescence, such as acridone.
63 g far UV circular dichroism spectroscopy and intrinsic protein fluorescence.
64 e Glu-98 or on its potential contribution to intrinsic protein fluorescence.
65 the enzyme with concomitant quenching of the intrinsic protein fluorescence.
66 xtend the use of in vitro phasor analysis to intrinsic protein fluorescence.
67 change is associated with a repositioning of intrinsic protein fluorophores from a hydrophobic to a s
68 al features that distinguish plasma membrane intrinsic proteins from tonoplast intrinsic proteins.
69 the tonoplast aquaporin gamma-TIP (tonoplast intrinsic protein) from Arabidopsis.
70 tein stability, subcellular localization, or intrinsic protein function.
71 ive spatiotemporal expression and not by its intrinsic protein functions.
72  a green fluorescent protein-delta tonoplast intrinsic protein fusion.
73                  Here, three plasma membrane intrinsic protein genes, SlPIP2;1, SlPIP2;7 and SlPIP2;5
74 ; thus, the tonoplast marker delta-tonoplast intrinsic protein-green fluorescent protein delineates c
75              Aquaporin 0 (AQP0) is the major intrinsic protein in the lens and is essential for estab
76     Aquaporin-0 (AQP0) is the most prevalent intrinsic protein in the plasma membrane of lens fiber c
77                  The process of insertion of intrinsic proteins into phospholipid membranes conjures
78 xamer containing 24 +/- 3 helices, the major intrinsic protein is a tetramer containing 24 +/- 3 heli
79 s expressing fluorescently labeled tonoplast intrinsic protein isoforms reveal an altered tonoplast m
80 issue-specific localization of two tonoplast intrinsic protein isoforms, the small leaf vacuoles were
81 phosphorylated form of Chk1 possessed higher intrinsic protein kinase activity and eluted more quickl
82       Here we report that VprBP possesses an intrinsic protein kinase activity and is capable of phos
83 largest subunit of TFIID, TAF1, possesses an intrinsic protein kinase activity and is important for c
84                   Immunoprecipitated ATM had intrinsic protein kinase activity and phosphorylated p53
85        We report here that yeast TOR1 has an intrinsic protein kinase activity capable of phosphoryla
86      We now report that PIKfyve possesses an intrinsic protein kinase activity inseparable from its l
87 rylation, demonstrating that hSMG-1 exhibits intrinsic protein kinase activity.
88 d preparations, only CBP2 exhibited apparent intrinsic protein kinase activity.
89 of the receptor guanylate cyclase family has intrinsic protein kinase activity.
90  solvent, resulting in overestimation of the intrinsic protein-ligand binding contribution to the app
91 vTIP3;1 (alpha-TIP) is a member of the Major Intrinsic Protein membrane channel family.
92  lens differentiation marker proteins, major intrinsic protein (MIP) and delta-crystallin, was also i
93 hown the involvement of the members of major intrinsic protein (MIP) family in controlling B transpor
94 - to 29-kD proteins that belong to the major intrinsic protein (MIP) family of channels.
95 gnificant similarity to members of the major intrinsic protein (MIP) family of membrane transporters.
96                     The NIPs belong to major intrinsic protein (MIP) family, members of which form ch
97                                    The major intrinsic protein (MIP) from bovine lens fibre membranes
98 uaporin-1 (AQP1) is a member of the membrane intrinsic protein (MIP) gene family and is known to prov
99  and the exon-intron borders of the membrane intrinsic protein (MIP) gene.
100 l protein alpha-TIP is a member of the major intrinsic protein (MIP) membrane channel family.
101                                    The major intrinsic protein (MIP) of the vertebrate eye lens is th
102                      Expression of the major intrinsic protein (MIP) PIP1 in Xenopus laevis oocytes r
103    Aquaporins (AQP) are members of the major intrinsic protein (MIP) superfamily of integral membrane
104                                        Major intrinsic protein (MIP), also called aquaporin-0, is ess
105                                        Major intrinsic protein (MIP), also known as aquaporin-0 (AQP0
106                                   Lens major intrinsic protein (MIP), exclusive to the vertebrate len
107 quaporin-0 (AQP0), previously known as major intrinsic protein (MIP), is the only water pore protein
108 rt of mouse chromosome 10 close to the major intrinsic protein (Mip), which is expressed only in cell
109 oding lens water channel protein AQP0 (major intrinsic protein, MIP).
110  growth factor receptor 1 (FGFR1), and major intrinsic protein (MIP26) in exponential growth.
111                                        Major intrinsic proteins (MIPs) are a diverse class of integra
112                                        Major intrinsic proteins (MIPs) are a family of membrane chann
113 ave characterized transcripts for nine major intrinsic proteins (MIPs), some of which function as wat
114 ns are members of the larger family of major intrinsic proteins (MIPs).
115                          The nodulin 26-like intrinsic protein (NIP) subfamily in Arabidopsis can be
116                             Plant nodulin-26 intrinsic proteins (NIPs) are members of the aquaporin s
117                                   Nodulin 26 intrinsic proteins (NIPs) are plant-specific, highly con
118 rbed as silicic acid through nodulin 26-like intrinsic proteins (NIPs).
119  the first ADC locus on chromosome 12; major intrinsic protein of lens fiber (MIP) is a candidate gen
120 mammalian aquaporins (AQP) 1-5 and the major intrinsic protein of lens fiber (MIP).
121              Aquaporin-0 (AQP0) is the major intrinsic protein of lens fiber cells and the founder me
122      Aquaporin 0 (AQP0), also known as major intrinsic protein of lens, is the most abundant membrane
123                                Insig-1 is an intrinsic protein of the endoplasmic reticulum (ER) that
124                Aquaporin 0 (AQP0), the major intrinsic protein of the eye lens, plays a vital role in
125 n of the first mutations affecting the major intrinsic protein of the lens, MIP, encoded by the gene
126 calized zeins of 10 to 27 kD and the granule-intrinsic proteins of 32 kD or higher.
127  the EAA loop sequence that defines membrane-intrinsic proteins of ABC transporters.
128 e suggests that high concentrations of major intrinsic proteins on membranes provide interaction and
129  conserved transcriptional coactivators with intrinsic protein phosphatase activity.
130 oscopy and expression of six plasma membrane intrinsic protein (PIP) aquaporin genes (VvPIP1;1, VvPIP
131 ss-induced redistribution of plasma membrane intrinsic protein (PIP) aquaporins from the plasma membr
132 hat members of the aquaporin plasma membrane intrinsic protein (PIP) subfamily are expressed in these
133          The plant aquaporin plasma membrane intrinsic proteins (PIP) subfamily represents one of the
134                              Plasma membrane intrinsic proteins (PIPs) are aquaporins facilitating th
135                        Plant plasma membrane intrinsic proteins (PIPs) are aquaporins that facilitate
136                              Plasma membrane intrinsic proteins (PIPs) are involved in the adjustment
137 out plants showed that three plasma membrane intrinsic proteins (PIPs) sharing expression in veins (P
138 ally homologous subfamilies: plasma membrane intrinsic proteins (PIPs), tonoplast intrinsic proteins
139 intrinsic proteins (TIPs) or plasma membrane intrinsic proteins (PIPs).
140                         Both plasma membrane intrinsic protein (PMIP) subgroups partitioned identical
141 yields large-scale data sets that can reveal intrinsic protein properties, protein behavior within ce
142 e 17 and 23 kDa extrinsic proteins and other intrinsic proteins remaining bound to the reaction cente
143                                     Here the intrinsic protein S-glutathionylation (PrSSG) at the 70-
144                 Transmembrane receptors with intrinsic protein serine kinase activity bind ligand in
145 wo conserved sequences associated with major intrinsic proteins (SGxHxNPA at residues 78 to 85, NPA r
146 ic membrane proteins (NIPs), and small basic intrinsic proteins (SIPs).
147 ) storage tissue contain two prominent major intrinsic protein species of 31 and 27 kD.
148 tion than to make a positive contribution to intrinsic protein stability.
149 r understanding of the basic determinants of intrinsic protein structure and dynamics.
150 3 and AtTIP5;1, two members of the Tonoplast Intrinsic Protein subfamily of aquaporins.
151 ngs to the nodulin-like members of the major intrinsic protein superfamily branch of the aquaporin (m
152 n superfamily branch of the aquaporin (major intrinsic protein) superfamily.
153 hat the two highly conserved plasma membrane intrinsic protein surface loops are structural features
154                Nodulin 26 (nod26) is a major intrinsic protein that constitutes the major protein com
155                Aquaporin-1 (AQP1) is a major intrinsic protein that facilitates flux of water and oth
156       Trg-31 belongs to a family of membrane intrinsic proteins that play a role in facilitating inte
157  50 hemi-channel; a water channel, the major intrinsic protein (the aquaporin 0); and a cotransporter
158 coincident with insertion of a new tonoplast intrinsic protein (TIP), delta-TIP, into their membranes
159 re marked by the presence of alpha-tonoplast intrinsic protein (TIP), whereas lytic vacuoles (LV) are
160       A comparison of the NIPs and tonoplast-intrinsic proteins (TIP) shows that the H2 residue can p
161                                    Tonoplast intrinsic proteins (TIPs) have been implicated in the pr
162 uaporins are categorized as either tonoplast intrinsic proteins (TIPs) or plasma membrane intrinsic p
163 mbrane containing alpha- and delta-tonoplast intrinsic proteins (TIPs), markers for protein storage v
164 embrane intrinsic proteins (PIPs), tonoplast intrinsic proteins (TIPs), nodulin 26-like intrinsic mem
165 ace has been eludicated in a recent study of intrinsic protein transport signals within potassium cha
166 epidermal growth factor receptor (EGFR), the intrinsic protein tyrosine kinase (PTK) activity of one
167 F), which act through receptors that possess intrinsic protein tyrosine kinase activity, raising ques
168  through cell surface receptors that possess intrinsic protein tyrosine kinase activity.
169        Although neither receptor contains an intrinsic protein tyrosine kinase, such activity has bee
170 s and in vitro indicate that Eyes absent has intrinsic protein tyrosine phosphatase activity and can
171 r suppressor function may associate with its intrinsic protein tyrosine phosphatase activity and its
172 ture motif, and we show that it possesses an intrinsic protein tyrosine phosphatase activity.
173 g protein-protein interactions and possesses intrinsic protein tyrosine phosphatase activity.
174 efficient at MHC-II presentation due to poor intrinsic protein uptake capability.
175  heat shock proteins, chaperonins, and major intrinsic proteins, were the largest class of genes regu
176     Cox25 is an inner mitochondrial membrane intrinsic protein with a hydrophilic C terminus protrudi
177                                            X-Intrinsic Proteins (XIP) were recently identified in a n
178 uses on the maize (Zea mays) plasma membrane intrinsic protein (ZmPIP) aquaporin subfamily, which is

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