ライフサイエンスコーパス: invasion

1 the conoid, parasite motility, and host cell invasion.

2 ompletely different lifestyles and routes of invasion.

3 homeostasis, antigen presentation, and cell invasion.

4 otential therapeutic targets to limit glioma invasion.

5 lationships between food web connectance and invasion.

6 cell-cell junction formation precede matrix invasion.

7 on cell proliferation but blocked tumor cell invasion.

8 edium from these cells failed to support CTB invasion.

9 eds in environments otherwise susceptible to invasion.

10 nce for niche conservatism during biological invasion.

11 sponded with the onset of seizures and tumor invasion.

12 pment, including angiogenesis, migration and invasion.

13 target this motor complex to block host cell invasion.

14 gical tumor stage and presence of lymph node invasion.

15 ferent ages, but share a common mechanism of invasion.

16 ing the cell cycle, apoptosis, migration and invasion.

17 lity and is sufficient to inhibit macrophage invasion.

18 protein (PfRipr), essential for erythrocyte invasion.

19 thermodynamics of cancer cell migration and invasion.

20 rtant for suppression of mTORC2 activity and invasion.

21 ase activity in the context of migration and invasion.

22 ntifying and anticipating potential areas of invasion.

23 ry response is pivotal to support growth and invasion.

24 g polarized secretion for cell migration and invasion.

25 her any of the RhopH proteins play a role in invasion.

26 ce against environmental attack and pathogen invasion.

27 omyosin-mediated contractility to facilitate invasion.

28 nd its activation induces cancer perineurial invasion.

29 us unknown proteins likely to be involved in invasion.

30 suppress cervical cancer cell migration and invasion.

31 ak intercellular adhesions and initiate cell invasion.

32 confirming the essential role for BSG during invasion.

33 s a virulence protein involved in chlamydial invasion.

34 ng the different phases of bacteria:bacteria invasion.

35 s epithelial-mesenchymal transition and cell invasion.

36 imal environment outside their niches during invasion.

37 d thus reducing a critical energy barrier to invasion.

38 litates cancer cell adhesion, migration, and invasion.

39 lgae nor herbivory was sufficient to prevent invasion.

40 and growth, and may protect against pathogen invasion.

41 ferentially alter local biotic resistance to invasion.

42 minants in mediating Pfalciparum erythrocyte invasion.

43 h of its core components to inhibit parasite invasion.

44 high/MITF-low state is insufficient to drive invasion.

45 lignant nature of the DCIS is defined before invasion.

46 lumen in CRC spheroids, and slowed CRC cell invasion.

47 ote cancer cell proliferation, migration and invasion.

48 ting the expression of genes involved in RBC invasion.

49 collagen promoted dispersive epithelial cell invasion.

50 ffering climatically suitable conditions for invasion.

51 uently failed to repress mTORC2 activity and invasion.

52 trand DNA substrate for RecA-mediated strand invasion.

53 hibitor of beta-catenin, which also promotes invasion.

54 gy with androgen receptor in mediating tumor invasion.

55 vity but was not sufficient to induce matrix invasion.

56 phic chondrocyte layer and impaired vascular invasion.

57 une system to protect the host from pathogen invasion.

58 larity determinants necessary for plant cell invasion.

59 tx4-mediated invadopodium formation and cell invasion.

60 vasive range, or in areas with similar shrub invasions.

61 n essential mechanistic insights into insect invasions.

62 to better understand demographic drivers of invasions.

63 revolutionized our perception of biological invasions.

64 hizal type is closely linked with understory invasions.

65 x sources) spread farther than single-source invasions.

66 he host immune system in response to foreign invasions.

67 ito infection by 50%, impedes salivary gland invasion 10-fold, and causes a complete absence of liver

68 istinct phenotypes with regard to macrophage invasion: a defect in matrix degradation, due to a disru

69 Cell invasion across basement membrane barriers is important

70 ases the speed and variability of replicated invasions: after 10 generations of range expansion, inva

71 ue features potentially associated with host invasion and adaptation, including genes for the complet

72 reducing CD82, KSHV miR-K6-5p expedites cell invasion and angiogenesis by activating the c-Met pathwa

73 down-regulating several genes important for invasion and angiogenesis.

74 bition on c-Met activation and inducing cell invasion and angiogenesis.

75 oenvironment that contributes to tumour cell invasion and angiogenesis.

76 -mediated LDHA activity promotes cancer cell invasion and anoikis resistance through redox homeostasi

77 M3A played a dual role in breast cancer cell invasion and apoptosis by demethylating histone and the

78 s to melanoma cells correlates with melanoma invasion and arises as a result of intimate cell-cell co

79 te-encoded RhopH complex contributes to both invasion and channel-mediated nutrient uptake.

80 and EMT-associated traits such as migration, invasion and chemoresistance.

81 is induction and resulted in attenuated cell invasion and elevated reactive oxygen species, whereas s

82 n is a surface glycoprotein related to virus invasion and host immune system response.

83 breast cancer cell lines with PNAs decreased invasion and increased chemotherapy sensitivity.

84 oma, inhibiting Wnt5a activity blocked brain invasion and increased host survival.

85 ficient to protect against Salmonella tissue invasion and involved a previously reported IEC expulsio

86 liver metastasis (CRLM), intrahepatic lymph invasion and lymph node metastasis are poor prognostic f

87 iven gene expression and clinically relevant invasion and metastasis gene signatures.

88 are required for OGT-mediated regulation of invasion and metastasis in breast cancer cells.

89 In line, WNT2 also promotes tumor growth, invasion and metastasis in vivo.

90 e determine that DOCK5 inhibition attenuates invasion and metastasis of MDA-MB-231 cells and prolongs

91 led receptors, is reported to be involved in invasion and metastasis of some cancers, but the role of

92 e product of the MET proto-oncogene, promote invasion and metastasis of tumor cells and have been con

93 ly responsible for Twist1-induced migration, invasion and metastasis, but less responsible for Twist1

94 gs interfering with all modes of cancer cell invasion and metastasis, to distinguish this class from

95 ctivates TGF-beta signaling to promote tumor invasion and metastasis.

96 ncers, where it has been linked to increased invasion and metastasis.

97 potency (50-100 nM) as modulators for cancer invasion and metastasis.

98 ppression of O-GlcNAcylation inhibits cancer invasion and metastasis.

99 riptional repressor Capicua (CIC) suppresses invasion and metastasis.

100 o the elevation of NSCLC cell proliferation, invasion and migration.

101 compound (38u) that represses breast cancer invasion and migration.

102 explain our findings of increased lymph node invasion and new metastatic sites in 30% of sunitinib-tr

103 ough the intracellular development following invasion and parasite growth.

104 ultures, oxamate also effectively suppressed invasion and proliferation.

105 etition assays in a murine abscess model and invasion and replication assays with human lung adenocar

106 mesenchymal transition) signaling, transwell invasion and soft agar colony formation, and in vivo pro

107 therefore consider the likely site of first invasion and the probabilistic position of additional fo

108 a critical regulator of RA-FLS migration and invasion and therefore represents an attractive target f

109 ially associated to rhoptry discharge during invasion and to host cell plasma membrane lysis during e

110 Furthermore, cell invasion and wound healing assays together with qRT-PCR

111 sistance that PAs provide against biological invasions and climate change on a continental scale and

112 sed to the interactive effects of biological invasions and climate change, with rising temperatures e

113 ental to effective predictions of viability, invasions and evolutionary pressures.

114 ing the Milan criteria, macroscopic vascular invasion, and AFP score>2 were independent predictors of

115 y toxins that facilitate human colonization, invasion, and dissemination.

116 i including secretion of adhesins, motility, invasion, and egress.

117 ic delivery of miR-194 stimulated migration, invasion, and epithelial-mesenchymal transition in human

118 induces marked inhibition of cell migration, invasion, and glycolysis through suppression of microRNA

119 promoting cell survival, anoikis resistance, invasion, and metastasis in several cancers.

120 d are often implicated in tumor progression, invasion, and metastasis.

121 xcision of PMIX revealed its crucial role in invasion, and recombinantly active PMIX and PMX cleave e

122 eletion of MCT1 in vivo inhibited migration, invasion, and spontaneous metastasis.

123 ance of prosocial rewarding but prevents its invasion, and that spatial structure can sometimes selec

124 at liver transplantation (LT), microvascular invasion, and the sum of the largest viable tumor and nu

125 ting ecological responses to climate change, invasion, and their interaction must rely on understandi

126 stasis staging systems; had no macrovascular invasion; and showed the lowest metastasis-specific gene

127 ibition of cell adhesion, wound healing, and invasion are demonstrated; near-infrared fluorescent psi

128 nchymal genes involved in cell migration and invasion are repressed.

129 Biological invasions are a key component of global change, and unde

130 fold, and causes a complete absence of liver invasion as mutants fail to attach to host cells.

131 SOCS1 silencing inhibited cell migration and invasion as well as in vitro growth by cell cycle arrest

132 nfirmed by colony formation assay, transwell invasion assay, and tumor xenograft model.

133 Using adhesion/invasion assays and immunofluorescent and transmission e

134 In the absence of vivax invasion assays, binding-inhibitory activity of antibodi

135 utionary time, is cornerstone for biological invasion assessments.

136 switches three-dimensional endothelial cell invasion between two distinct modes: single-cell migrati

137 m pHe7.4 to 6.4 decreases cell migration and invasion but increases single cell detachment from the s

138 e interactions play crucial roles in species invasions but are rarely investigated at the intraspecif

139 argets both merozoite egress and erythrocyte invasion, but crucially, also blocks transmission of mat

140 om many causes, some closely associated with invasions, but others occurring across a wide range of e

141 ferences in native species richness prior to invasion by a non-native zooplankter, Daphnia lumholtzi.

142 MLN4924 also induced anti-migration and anti-invasion by activating E-cadherin and repressing Vimenti

143 associated with afforestation processes and invasion by alien woody plants, significantly incresed.

144 helial-mesenchymal transition, motility, and invasion by cancer cells.

145 issociated bound proteins are susceptible to invasion by competitor proteins in solution.

146 chanistically, miR-520f inhibited tumor cell invasion by directly targeting ADAM9, the TGFbeta recept

147 ese native ecosystems subjected to prolonged invasion by exotic plants may be instrumental in disting

148 Mechanistically, SMARCE1 drives invasion by forming a SWI/SNF-independent complex with t

149 ade that sub-cellular resolution of red cell invasion by the malaria parasite Plasmodium falciparum h

150 s light on mechanisms via which it regulates invasion.Calcium dependent protein kinase 1 (CDPK1) play

151 ese findings reveal that cells refractory to invasion can still be injected, thus extending the panel

152 Globally, biological invasions can have strong impacts on biodiversity as wel

153 volved in modulating tumor cell motility and invasion, cancer stem cell viability and differentiation

154 rk of YAP1 effectors with executive roles in invasion, chemotaxis and adhesion downstream of the ROCK

155 d functionally replaced RhoA with respect to invasion, clonogenic growth and survival.

156 ression of genes implicated in migration and invasion compared to commonly used, immortalized TB cell

157 had increased levels of PPP1R11 at areas of invasion, compared with other areas of the tumor; increa

158 We used invasion-deficient S.

159 a 'leader-follower' mode of collective cell invasion, demonstrating that matrix remodeling and creat

160 ession in GC were positively correlated with invasion depth, lymph node metastasis and negatively cor

161 The mechanisms that direct invasion, despite their importance in normal and disease

162 uman specificity, we established an in vitro invasion-differentiation trophoblast model.

163 ated effect likely does not explain impaired invasion displayed by the tmeA strain of Chlamydia, sinc

164 eciate the specific conditions of the Mongol invasion, do not offer new or different climatic data, a

165 ong-standing hypothesis that the waves of TE invasions endured by organisms for eons have catalysed t

166 After invasion, establishment success and effects of the non-n

167 logy Group performance status, macrovascular invasion, extrahepatic disease, and alpha-fetoprotein le

168 nantly active PMIX and PMX cleave egress and invasion factors in a 49c-sensitive manner.

169 There is growing evidence that invasions foster eutrophication.

170 ly discriminated districts with high risk of invasion from others with a lower risk (area under the c

171 Cancer cell invasion from primary tumors is mediated by a complex in

172 tracellular matrix (ECM) and increased local invasion from the primary tumour.

173 variability in population density behind the invasion front can produce fluctuations in spreading spe

174 odel of tumour growth to investigate how the invasion front is delayed by resection, and how this dep

175 alysis of invading leader cells at the tumor invasion front is of significant interest as these cells

176 moroid culture which recapitulated the tumor invasion front, allowing for both quantification of inva

177 ed unrivalled spatiotemporal data across the invasion front.

178 rse further, suggesting a sociability-biased invasion front.

179 M cells to radiation, reducing postradiation invasion gains.

180 Critical factors involved in invasion have been identified using biochemical and gene

181 Paleontological data show that older marine invasions have consistently ended in extinction.

182 egression analysis showed that macrovascular invasion (hazard ratio [HR], 4.8; P < 0.001), pre-LT wai

183 Mismatch correction of strand invasion heteroduplex DNA is strongly polar, favouring c

184 nce on each other across a chronosequence of invasion history.

185 ess (HR, 2.6; P = 0.01) and hilar lymph node invasion (HR = 2.2; P = 0.03), but not pre-LT extrahepat

186 ockout was completely refractory to parasite invasion in a strain-transcendent manner, confirming the

187 d thus suppressed mTORC2 kinase activity and invasion in colon tumor cells.

188 l time-lapse confocal imaging of anchor-cell invasion in live Caenorhabditis elegans.

189 nized as a key element of cell migration and invasion in lung cancer; however, the underlying mechani

190 XCR4/CXCR7 on SC in the initiation of neural invasion in the cancer precursor stage and the resulting

191 trated their synergy with PTEN in preventing invasion in vitro and confirmed their clinical relevance

192 f ER and IKKbeta promoted cell migration and invasion in vitro and drove experimental metastasis in v

193 de decreased invadopodium formation and cell invasion in vitro Of note, cells expressing the Stx4 N-t

194 nd overexpression of NNMT blocked tumor cell invasion in vitro.

195 circCCDC66 inhibited tumor growth and cancer invasion in xenograft and orthotopic mouse models, respe

196 ixture may be a common feature of biological invasions in nature, but that these effects can easily g

197 s traits that commonly facilitate biological invasions in terrestrial systems may not be as represent

198 ld more variable spread dynamics relative to invasions in which spatial sorting is suppressed.

199 ted mechanisms mediating tumor viability and invasion, including potassium channel function and EPH r

200 Instead, invasion increased due to increased bacterial adhesion t

201 During invasion, individuals can become isolated from the bulk

202 ssembled FN are directly proportional to the invasion induced by fibroblast populations.

203 Tumor cell invasion involves targeted localization of proteins requ

204 Invasion is a hallmark of advanced head and neck squamou

205 Fs is preserved, but their ability to induce invasion is abrogated.

206 Species invasion is an important disturbance to ecosystems world

207 A key step in merozoite invasion is the essential binding of PfRh5/CyRPA/PfRipr

208 Understanding biological invasions is crucial for their control and prevention.

209 re, we hypothesized that excessive leukocyte invasion leads to heart failure and death during acute m

210 -binding protein homologue 2b (PfRh2b) is an invasion ligand that is a potential blood-stage vaccine

211 However, nonnative invasion may overwhelm the effect of immigration timing

212 om SANS are higher than those from two fluid-invasion methods, due to the ability of neutrons to prob

213 required for TG2-dependent cancer stem cell invasion, migration and tumour formation.

214 various solid tumors, promoting cancer cell invasion, migration, and metastasis.

215 ciated with tumour aggressiveness, including invasion, migration, and oxygen consumption.

216 g aggressive growth phenotypes, such as cell invasion, migration, and xenograft tumors, in nude mice.

217 on and provide conditions for the successful invasion of a population.

218 mporal changes in connectivity driven by the invasion of an exotic food resource, illustrating the ch

219 Invasion of ancestral mammals into nocturnality has long

220 and their function in motility and host cell invasion of apicomplexan parasites.

221 r capacity to contract the matrix and induce invasion of cancer cells has been well documented.

222 omotes cell viability, colony formation, and invasion of cancer cells in vitro and in vivo, which wer

223 Blocking Nrp-2 significantly reduced invasion of Colo320 cells through an LEC monolayer.

224 The asexual blood stage involves invasion of erythrocytes by merozoites, in which they gr

225 Invasion of erythrocytes by Plasmodial merozoites is a c

226 WA inhibited growth, migration and invasion of HCC cells.

227 he in vitro defect was reflected by impaired invasion of host cells.

228 ributes to bacterial adhesion to host cells, invasion of host tissues, and evasion of the immune syst

229 er, PRELP reduces Moraxella adherence to and invasion of human lung epithelial A549 cells.

230 exosomes enhance the growth, migration, and invasion of malignant cells, demonstrating the capacity

231 and metastatic cancers is the migration and invasion of malignant cells.

232 protein A (AmOmpA; AM854) contributes to the invasion of mammalian and tick host cells.

233 t the characterization of PIMMS2 (Plasmodium invasion of mosquito midgut screen candidate 2), a Plasm

234 ction, inhibits clonal growth, migration and invasion of ovarian cancer cells, whereas silencing in v

235 rowth, cytoskeleton remodeling and motility, invasion of PDAC cells-all collectively contributing to

236 ng Schwann cells in the injured spinal cord; invasion of peripheral myelinating (P0+) Schwann cells m

237 brin binding to actin filaments, reduced the invasion of prostate cancer cell lines in 3D in vitro as

238 With elevated awareness, the recent invasion of Pterois miles was quickly detected in the Me

239 ctural similarities to a protein crucial for invasion of red blood cells.

240 ating that UPF1-augmented TumiD promotes the invasion of T24 cells in part by degrading anti-invasive

241 GLI1/GLI2 inhibitor Gant61 led to decreased invasion of the melanoma cells in a three-dimensional sk

242 The initial step of metastasis is the local invasion of tumor cells into the surrounding tissue.

243 1327 1328 References 1328 Invasions of alien plants are typically studied as invas

244 Genetic admixture propels invasions of Callosobruchus maculatus across experimenta

245 Global climate change may increase invasions of exotic plant species by directly promoting

246 ons of alien plants are typically studied as invasions of individual species, yet interactions betwee

247 We present a hybrid approach for modelling invasions of populations with two sexes that links indiv

248 phenomenon, particularly for carcinoma cell invasion on aligned collagen fibres.

249 Diffuse type (P < 0.001), macro vascular invasion (P < 0.001) and late stage tumours (P < 0.001)

250 ica and has been associated with alternative invasion pathway usage.

251 s relative to the well-described EBA-175/GPA invasion pathway, we used an ex vivo erythrocyte culture

252 ls have suggested the evolution of alternate invasion pathways that may be mediated by the PvRBPs.

253 nge, and understanding the drivers of global invasion patterns will aid in assessing and mitigating t

254 ion is based on tumor number, size, vascular invasion, performance status, functional liver reserve,

255 dia, since AHNAK-deficient cells revealed no invasion phenotype.

256 aggressive tumor cell growth, migration, and invasion phenotypes is mediated in part by the constitut

257 may offer redundancy and generally limit the invasion potential of mutualistic microbes in insects.

258 e of environmental filters during the entire invasion process for the facilitation or inhibition of i

259 shifts occur at all commonly as part of the invasion process, is indispensable to identifying and an

260 eptor is vital for junction formation in the invasion process.

261 izing these interaction motifs, parallels in invasion processes between pathogen and mutualist fungi

262 integrin signaling to promote cell motility, invasion, proliferation, and survival.

263 Salmonella invasion protein A (SipA) is a dual-function effector pr

264 ily of recombinases catalyzes the DNA strand invasion reaction that takes place during homologous rec

265 ypothesized that this may alter biodiversity-invasion relationships.

266 However, the factors that promote DCIS invasion remain poorly understood.

267 migration and the multicellular, strand-like invasion required for angiogenesis.

268 Epithelial invasion required matrix anchorage as well as signaling

269 However, that invasions result instead in the persistence of cooperati

270 l to identifying niche shifts during species invasion robustly, but also in applications of ENM to un

271 tive tract (FRT) is one of the major mucosal invasion sites for HIV-1.

272 ns: after 10 generations of range expansion, invasions subject to spatial sorting spread 8.9% farther

273 iological traits, and reproduction and hence invasion success.

274 rget of ABHD5, impeded the proliferation and invasion, suggesting an ATGL-independent role of ABHD5 i

275 or change in our concept of malaria parasite invasion, suggesting it is, in fact, a balance between p

276 s events ranging from starvation to pathogen invasion, the cell activates one or more forms of macroa

277 After erythrocyte invasion, the intracellular pathogen must increase host

278 -an observation that is consistent with the "invasion" theories of urban sociology.

279 asia, by promoting cell proliferation, micro-invasion, tissue inflammation, and expression of acknowl

280 to mediate repair via homologous DNA strand invasion to form D-loops.

281 -13Ralpha2 serves as a molecular switch from invasion to proliferation, and suggest that targeting bo

282 hanisms linking MITF-dependent inhibition of invasion to suppression of guanylate metabolism.

283 combined threat posed by climate change and invasions to existing PAs and the most susceptible speci

284 fungal-driven dynamics behind observed plant invasion trajectories.

285 tients with biopsy-proven SUSCC without bone invasion treated by wide surgical excision of the nail u

286 he course of spatial expansion, the stage of invasion typically associated with the greatest ecologic

287 nked habitat patches to compare experimental invasions using individuals from single population sourc

288 cell proliferation but increased tumor cell invasion via greater mitochondrial trafficking to the co

289 y of Red Sea individuals, thus indicating an invasion via the Suez Canal.

290 Vascular invasion was apparent at the time of bone formation but

291 Tumor cell invasion was studied using organotypic raft cultures and

292 s landcover preferences before and after the invasion, we demonstrate that this species experienced a

293 ma cells with NA in vitro results in reduced invasion, which is accompanied by a loss of mesenchymal

294 the adjacent epithelium allows for bacterial invasion, which may lead to peri-implantitis.

295 of the merozoite proteome during erythrocyte invasion, while identifying numerous unknown proteins li

296 d indigenous species suggest that biological invasions will exacerbate the impacts of climate change

297 , we consider methods that combine Wolbachia invasion with mating disruption tactics to enhance the p

298 Over six generations, invasions with any amount of genetic mixture (two, four

299 as been proposed as a mechanism facilitating invasion, with more flexible species better able to inva

300 e development and expressed primarily in the invasion zone of mature nodules.