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1 possible, and the remaining 165 patients no invasive pulmonary aspergillosis.
2 drugs, and is avirulent in a murine model of invasive pulmonary aspergillosis.
3 ematopoietic cell transplant recipients with invasive pulmonary aspergillosis.
4 y avirulent in two distinct murine models of invasive pulmonary aspergillosis.
5 e models of haematogenously disseminated and invasive pulmonary aspergillosis.
6 us infection in vitro and in mouse models of invasive pulmonary aspergillosis.
7 yene may be antagonistic in the treatment of invasive pulmonary aspergillosis.
8 ions of lung tissue recovered from mice with invasive pulmonary aspergillosis.
9 istance of A. terreus to AmB in experimental invasive pulmonary aspergillosis.
10 ay represent a new strategy for treatment of invasive pulmonary aspergillosis.
11 id-treated animals against subsequent lethal invasive pulmonary aspergillosis.
12 ha is an important proximal signal in murine invasive pulmonary aspergillosis.
15 p. was recovered as the etiological agent of invasive pulmonary aspergillosis and had reduced in vitr
16 are unexplored targets for the treatment of invasive pulmonary aspergillosis and may potentiate both
17 Little is known about the pathogenesis of invasive pulmonary aspergillosis and the relationship be
18 idence that the inflammatory response during invasive pulmonary aspergillosis, and in particular the
20 depletion of neutrophils, animals developed invasive pulmonary aspergillosis, associated with delaye
21 pulmonary aspergillosis, two each with acute invasive pulmonary aspergillosis, chronic necrotizing pu
22 CalA antibody improves survival of mice with invasive pulmonary aspergillosis, demonstrating the pote
26 ergillus respiratory tract colonization from invasive pulmonary aspergillosis in critically ill patie
27 s fumigatus is an important pathogen causing invasive pulmonary aspergillosis in immunocompromised pa
28 nhanced fungal clearance during experimental invasive pulmonary aspergillosis in neutropenic mice.
29 le in simultaneous treatment of experimental invasive pulmonary aspergillosis in persistently neutrop
30 nd-generation triazole, against experimental invasive pulmonary aspergillosis in persistently neutrop
31 inate Aspergillus colonization from putative invasive pulmonary aspergillosis in this patient group.
33 e prognostic factors and optimal therapy for invasive pulmonary aspergillosis (IPA) after kidney tran
34 eolar lavage (BAL) fluid in the diagnosis of invasive pulmonary aspergillosis (IPA) among solid-organ
39 damage due to unresolved inflammation during invasive pulmonary aspergillosis (IPA) is associated wit
43 identification of the causative organism in invasive pulmonary aspergillosis (IPA) is recommended.
61 influx of mononuclear cells into the lung in invasive pulmonary aspergillosis is in part mediated by
62 l effectors, and antifungal drug therapy for invasive pulmonary aspergillosis may be further understo
64 ppo-silenced mutant was hypervirulent in the invasive pulmonary aspergillosis murine model system and
65 ion in fungal lung burdens in a rat model of invasive pulmonary aspergillosis (p<0.05) compared to tr
66 e is unlikely to serve as an S source during invasive pulmonary aspergillosis since a sulfate transpo
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