ライフサイエンスコーパス: inviability

1 re of hybrid inferiority (both sterility and inviability).

2 tive stress, floral bud abortion, and pollen inviability.

3 tem for investigating the genetics of hybrid inviability.

4 ation of glycoproteins in the Golgi leads to inviability.

5 es show Haldane's rule for sterility but not inviability.

6 obeys Haldane's rule for both sterility and inviability.

7 igh-copy-number suppressors of the mot1 spt3 inviability.

8 es whose overexpression also suppresses mec1 inviability.

9 bination with an spt3 null mutation, lead to inviability.

10 ssion of RED1 exacerbates the mek1-974 spore inviability.

11 hybrid male and hybrid female sterility and inviability.

12 ely to morphological abnormalities and adult inviability.

13 thus overestimating the frequency of hybrid inviability.

14 sult in reduced male fertility, sterility or inviability.

15 severe delay of anther dehiscence and pollen inviability.

16 n in the absence of histone synthesis causes inviability.

17 ially faster than female sterility or hybrid inviability.

18 s that are likely responsible for their high inviability.

19 tiations via elevated dnaA expression causes inviability.

20 sking two questions about its role in hybrid inviability.

21 with genetic studies of hybrid sterility and inviability.

22 ER-ER fusion pathways caused almost complete inviability.

23 ecessary nor sufficient for hybrid embryonic inviability.

24 one of the proximate causes of hybrid larval inviability.

25 xpression levels but relieved mec1 and rad53 inviability.

26 (2) Does Hmr(mel) cause hybrid larval inviability?

27 lanogaster (Hmr(mel)) cause hybrid embryonic inviability?

28 al races, predominantly in the form of spore inviability (23.7% F(1) spore viability).

29 initial pds1-1 allele was identified by its inviability after transient exposure to microtubule inhi

30 alternative screen for mutants that exhibit inviability after transient exposure to nocodazole and p

31 lex in ncr1Delta strains conferred anaerobic inviability and accumulation of multiple sterol intermed

32 results add to previous studies of F2 hybrid inviability and behavioral sterility, and indicate that

33 specially in contrast with the low degree of inviability and female sterility, is far greater than ex

34 ng isolating mechanisms (for example, hybrid inviability and hybrid sterility).

35 ssion of HOP1 partially suppresses the spore inviability and recombination defects of mek1-974; in co

36 premating behavioral isolation, postzygotic inviability and Wolbachia-induced cytoplasmic incompatib

37 ng both 3' to 5' and 5' to 3' decay leads to inviability, and conditional double mutants show extreme

38 of induced dnaC expression and a model that inviability arises by the binding of DnaC to DnaB to inh

39 The inviability associated with deletion of the yeast genes

40 Taken together, these data suggest that the inviability associated with inactivation of RNase E is n

41 oth 30 and 44 degrees C, suggesting that the inviability associated with the mrsC505 allele is not re

42 e biology, viable null mutations that confer inviability at 36 degrees have been identified for multi

43 tations affecting microtubules in yeast, and inviability at extreme temperatures (i.e., >/=37 degrees

44 re-accumulation of 4-carboxysterols and cell inviability at high temperature.

45 se IV mutants in Escherichia coli results in inviability at the permissive temperature.

46 gested by work on other species, that hybrid inviability between closely related species might be cau

47 uctive barriers, such as hybrid sterility or inviability between populations.

48 bias in the evolution of hybrid sterility or inviability but do find a very strong sex bias in the ev

49 This inviability can be suppressed in fission yeast by overex

50 The rapid loss of inviability displayed by a yku80delta est2delta strain w

51 arabiensis suffer from hybrid sterility, and inviability effects are sometimes present as well.

52 Generally, recessive inviability effects were found on the X chromosome of ga

53 ithin populations, may be aided by immigrant inviability, especially when a long period separates lar

54 dnaC suppressors of priA overcome this inviability, especially when RecF, RecO or RecR is inact

55 is complete when the gambiae and arabiensis inviability factors are hemi- or homozygous.

56 more rapidly than either female sterility or inviability factors.

57 or the histone deacetylase RPD3 gene, shows inviability following induction of an HO lesion that is

58 Hybrid seed inviability forms a potent reproductive barrier between

59 The relative paucity of "inviability genes" supports the idea, suggested by work

60 A single case of hybrid inviability has been tracked down to a 3-Kb element that

61 ividual genes that cause hybrid sterility or inviability have been identified in a few cases, my anal

62 Hybrid sterility and inviability have been unimportant in the early stages of

63 ed that different events are responsible for inviability in checkpoint-deficient cells harboring muta

64 one of the underlying causes of male hybrid inviability in Drosophila, wherein the fate of hybrid ma

65 compensation could contribute to male hybrid inviability in Drosophila.

66 ere we identify a gene that causes epistatic inviability in hybrids between two fruitfly species, Dro

67 l cycle-regulation gene as the cause of male inviability in hybrids resulting from a cross between Dr

68 netic incompatibilities causing sterility or inviability in interspecies hybrids grows faster than li

69 es to study the genetics of intrinsic hybrid inviability in male F2 hybrids of Nasonia giraulti and N

70 7, which encodes a flap endonuclease, causes inviability in mre11 strains.

71 ads to loss of sister chromatid cohesion and inviability in nocodazole.

72 ic and epigenetic mechanisms underlie hybrid inviability in Peromyscus and hence have a role in the e

73 Much of the spore inviability in sgs1 results from PSSC, and these events

74 loss of anionic phospholipids leads to cell inviability in the absence of mitochondrial DNA.

75 studies of the developmental basis of hybrid inviability in the Drosophila melanogaster complex.

76 asm account for most of the intrinsic hybrid inviability in this cross.

77 a high-expression suppressor of Sir2-induced inviability in yeast cells.

78 ytological analyses to show that hybrid male inviability is associated with, and probably caused by,

79 Inviability is complete when the gambiae and arabiensis

80 ains of Saccharomyces cerevisiae, most spore inviability is due to precocious separation of sister ch

81 double-mutant phenotype, suggesting that the inviability is not solely the result of precocious expre

82 Our studies reveal that hybrid inviability is partially due to a maternally expressed X

83 We present a simple model in which hybrid inviability is partly or entirely caused by a mitotic de

84 One explanation for this inviability is that one or more sumoylated proteins accu

85 of fertilization success or hybrid sterility/inviability, is very common.

86 isolating mechanisms of hybrid sterility and inviability, little is known about the genetic basis of

87 isolating mechanisms of hybrid sterility and inviability, little is known about the genetic basis of

88 several genes that cause hybrid sterility or inviability-many of which have evolved rapidly under pos

89 The inviability of a dam lexA (Ind(-)) host was abrogated by

90 ne-rich (G-F) region, was able to rescue the inviability of a Deltasis1 strain.

91 Synthetic lethality is inviability of a double-mutant combination of two fully

92 ther cdc33 or ceg1 and no suppression of the inviability of a pab1 null mutation by xrn1Delta.

93 selection for mutations overcoming the spore inviability of a rad52 spo13 haploid strain.

94 tion, was found to specifically suppress the inviability of a ydr1 deletion, demonstrating that the e

95 olism is the primary underlying cause of the inviability of apn1Delta apn2Delta rad1Delta and apn1Del

96 tion but instead are capable of rescuing the inviability of bem2 mutants at 35 degrees C.

97 s Cdc28) phosphorylation also suppresses the inviability of Bir1 deletion.

98 Here we show that inviability of double-mutant offspring biased inferences

99 hat deletion of KIP2 could also suppress the inviability of dyn1Delta kar3Delta cells suggests that k

100 2-1 (D530N, V615E) alleles that suppress the inviability of erg26-1ts at high temperature, and cause

101 have established a requirement for Xist with inviability of female embryos that inherit an Xist delet

102 a close relative (human KChIP2), rescues the inviability of frq1 cells.

103 ted Frq1(G2A,C15A) double mutant rescued the inviability of frq1Delta cells.

104 The inviability of gcn5 yng2 double mutants suggests overlap

105 taxa, including the intrinsic sterility and inviability of hybrids.

106 The inviability of KLP64D mutations can be rescued by expres

107 egulator causes mitotic arrest and, thereby, inviability of male hybrid larvae.

108 nent nuclear accumulation and did not rescue inviability of pik1Delta cells.

109 ded from the nucleus and also did not rescue inviability of pik1Delta cells.

110 ts are viable, suggesting, together with the inviability of pta/ackA recBC mutants, that chromosomal

111 To determine the reason for the inviability of Saccharomyces cerevisiae with skeletal mu

112 The sterility and inviability of species hybrids can be explained by betwe

113 The sterility and inviability of species hybrids is thought to evolve by t

114 have long recognized that the sterility and inviability of species hybrids must involve incompatible

115 dane's rule - the preferential sterility and inviability of species hybrids of the heterogametic (XY)

116 2Delta and rqh1Delta mutants and rescues the inviability of srs2Delta rqh1Delta cells.

117 The supposed inviability of such intermediates gave pleuronectiforms

118 of Clns, because a sic1 deletion rescued the inviability of the cln1 cln2 cln3 triple mutant.

119 In contrast, the spore inviability of the mum4-1 mutant is rescued by the spo13

120 Overexpression of TRF5 complements the inviability of top1 trf4 double mutants.

121 The conditional inviability of two mutants, msh2-L560S pol3-01 and msh2-

122 stability through several diverse pathways, inviability of vertebrate cells that lack Mre11 has hind

123 tic stages of pollen development, leading to inviability of xri(-) pollen and abnormal segregation of

124 A screen for bypass suppressors of the inviability of xrn1 ski2 double mutants identified domin

125 iability suggest that bacteria contribute to inviability only because intrinsic hybrid dysfunction in

126 gent cid alleles are not sufficient to cause inviability or female sterility in hybrid crosses.

127 t postzygotic reproductive isolation (hybrid inviability or sterility) evolves by the accumulation of

128 cdc13-ts alleles argues that the accelerated inviability previously observed at 36 degrees in cdc13-1

129 ies or subspecies and on hybrid sterility or inviability rather than on ecologically based barriers t

130 c distances between taxa that exhibit female inviability/sterility show no differences between "large

131 tes that in instances of hybrid sterility or inviability, the heterogametic sex tends to be more seve

132 Complete hybrid inviability was also associated with some regions of the

133 otherwise nonessential genes cause cellular inviability when knocked out simultaneously.

134 bstantially suppresses the sid2-1 sic1 Delta inviability, while stabilizing Clb5 protein exacerbates

135 in which presumed nicks in DNA do not cause inviability with recA, suggesting that nicks stimulate h