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1 ive hippocampal neurons by overexpressing an inward-rectifier potassium channel.
2 ceptor channels, and up-regulation of muscle inward rectifier potassium channels.
3 exchange, KATP potassium channels, and other inward rectifier potassium channels.
4 hmias can be perpetuated by up-regulation of inward rectifier potassium channels.
5 ecific residues of the cytoplasmic domain of inward rectifier potassium channels.
6 expressing either connexin-43 (Cx43 HEK) or inward rectifier potassium channel 2.1 (Kir2.1) and Cx43
8 annels are thought to be composed of Kir6.2 (inward-rectifier potassium channel 6.2) and SUR2A (sulfo
9 ductance on KOUT is a well-known property of inward rectifier potassium channels and is a property of
10 HERG (human eag-related gene) encodes an inward-rectifier potassium channel formed by the assembl
11 s a major contributor to G protein-activated inward rectifier potassium channels in the mammalian bra
12 scarinic m2 ACh receptors (mAChRs) linked to inward rectifier potassium channels (K(ir)) evenly distr
13 ulated potassium (KATP) channel complexes of inward rectifier potassium channel (Kir) 6.2 and sulfony
14 effects of e-LXA4 on signaling and on Kv and inward rectifier potassium channels (Kir) in mice bone m
16 itional experiments using the PIP2-sensitive inward rectifier potassium channel Kir2.1 as a biosensor
17 nding partners, kainate receptor GluR6/7 and inward rectifier potassium channel Kir2.1, closely assoc
20 e m2 muscarinic receptor and G-protein-gated inward rectifier potassium channels show that RGS9-2, vi
22 of the sulfonylurea receptor 1, SUR1, and an inward rectifier potassium channel subunit, Kir6.2, regu
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