コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ptor-mediated opening of a G-protein-coupled inwardly rectifying potassium channel.
2 activation of the intrinsic G-protein-gated inwardly rectifying potassium channel.
3 ls the G protein-dependent activation of the inwardly rectifying potassium channel.
4 mplementary DNA for a component of a cardiac inwardly rectifying potassium channel.
5 activation of a barium- and cesium-sensitive inwardly rectifying potassium channel.
6 es coexpressing mGluR8 and G protein-coupled inwardly rectifying potassium channels.
7 voltage-gated potassium channels and one for inwardly rectifying potassium channels.
8 t (at - 70 mV) consistent with activation of inwardly rectifying potassium channels.
9 oding Trpv4, Aqp4, and the Kir4.1 subunit of inwardly rectifying potassium channels.
10 through the activation of G-protein-coupled inwardly rectifying potassium channels.
11 ir3.1 and Kir3.4 subunits of G protein-gated inwardly rectifying potassium channels.
12 study, we focused on the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) gene tha
13 e pair substitution in the G protein-coupled inwardly rectifying potassium channel 2 gene, the cell d
14 e randomly mutagenized the G protein-coupled inwardly rectifying potassium channel 3.2 (GIRK2) bearin
15 o participate in the rectification of cloned inwardly rectifying potassium channels, a class of potas
17 consequent upon loss or reduced function of inwardly rectifying potassium channels affecting various
18 xpression of tetanus toxin light chain or an inwardly rectifying potassium channel also inhibits cond
20 rs via the activation of a G-protein-coupled inwardly rectifying potassium channel and is blocked by
21 mutations in KCNJ6 (GIRK2), which encodes an inwardly rectifying potassium channel and maps to the Do
22 controlling the time-dependent gating of an inwardly rectifying potassium channel and suggest a clos
23 ha(i/o)-coupled receptors, G protein-coupled inwardly rectifying potassium channels and adenylate cyc
25 We tested the hypothesis that activation of inwardly rectifying potassium channels and the sodium-po
26 receptors couple via G-proteins to activate inwardly rectifying potassium channels and to inhibit ca
27 , increases glutathione levels, activates an inwardly rectifying potassium channel, and delays loss o
28 TA neurons, NK receptor activation closes an inwardly rectifying potassium channel, and moreover inhi
31 that the rectification properties of native inwardly rectifying potassium channels are largely contr
32 ltage-dependent inward rectification of Kir (inwardly rectifying potassium) channels arises from bloc
33 ated via G-proteins and G-protein-activated, inwardly rectifying potassium channels, as evidenced by
34 -THCs enable CB1-mediated optical control of inwardly rectifying potassium channels, as well as adeny
35 bers of the closely related Kir2.0 family of inwardly rectifying potassium channels, as well as vario
37 hloroquine and related compounds can inhibit inwardly rectifying potassium channels by multiple poten
38 r and its downstream GIRK (G protein-coupled inwardly rectifying potassium channels) channels (IK,Ado
39 ylation sites along with G-protein activated inwardly rectifying potassium channels composed of K(ir)
40 ings of ligand activated G-protein-activated inwardly rectifying potassium channel currents in mouse
42 between filamin-A and Kir2.1, an isoform of inwardly rectifying potassium channel expressed in vascu
43 ea receptor (SUR) family and a member of the inwardly rectifying potassium channel family (Kir6.x).
44 ranscriptional regulation of a member of the inwardly rectifying potassium channel family, we have ch
45 xpressing rat MOR1 as well G protein-coupled inwardly rectifying potassium channel (GIRK) channel sub
46 r-mediated activation of a G-protein-coupled inwardly rectifying potassium channel (GIRK) is a common
47 ce that reversed at -93 mV, indicative of an inwardly rectifying potassium channel (GIRK) mechanism.
48 KOR-induced tyrosine phosphorylation of the inwardly rectifying potassium channel (GIRK) subunit Kir
50 immunoreactivity (IR) for a G protein-gated, inwardly rectifying potassium channel (GIRK1) was presen
51 ctural determinants of a G protein-activated inwardly rectifying potassium channel, GIRK1 (KIR3.1), i
53 mutation in the gene encoding the G-protein inwardly rectifying potassium channel Girk2, exhibits a
55 ostsynaptic inhibition via G protein-coupled inwardly rectifying potassium channels (GIRKs) and presy
57 d postsynaptic activation of G-protein-gated inwardly rectifying potassium channels (GIRKs) electroph
59 ought to determine whether G protein-coupled inwardly rectifying potassium channels (GIRKs) modulate
61 ave investigated the role of G-protein-gated inwardly rectifying potassium channels (GIRKs), a recent
62 trimers to interact with G-protein regulated inwardly rectifying potassium channels (GIRKs), and we s
66 ytes and the activation of G-protein-coupled inwardly rectifying potassium channels (GIRKs, also name
67 crystallization conditions for a prokaryotic inwardly rectifying potassium channel (>130 different co
68 specific amino acid mutations in the Kir2.1 inwardly rectifying potassium channel have been found to
70 er L-glutamate or expressing G-protein-gated inwardly rectifying potassium channels, hepatic lineage
71 eased the conductance of G protein-activated inwardly rectifying potassium channels in oocytes co-exp
72 -inducible promoter drives the expression of inwardly rectifying potassium channels in polycistronic
76 tyrosine phosphorylation of G-protein-gated inwardly rectifying potassium channels (K(ir)3 or GIRK)
78 l, cytoplasmic domain of the G-protein-gated inwardly rectifying potassium channel, K(ir)3.1 facilita
80 gate the subunit stoichiometry of endogenous inwardly rectifying potassium channel Kir 2.2 (IRK2) fro
81 pling of the receptor to the potentiation of inwardly rectifying potassium channel (KIR) currents and
82 ated vasodilatation occurs via activation of inwardly rectifying potassium channels (KIR ), and synth
83 Mutations that disrupt function of the human inwardly rectifying potassium channel KIR2.1 are associa
84 use visual system to ectopically express the inwardly rectifying potassium channel Kir2.1 in individu
85 us activity due to the overexpression of the inwardly rectifying potassium channel Kir2.1 in the olfa
88 iesterase (PDE) 1C, and PDE9A; and channels: inwardly rectifying potassium channel Kir2.4, transient
89 ore, whether Cav-1 regulates the function of inwardly rectifying potassium channels Kir2.1 that play
92 mediated via the G protein-coupled receptor inwardly rectifying potassium channel Kir3.Our findings
94 r-mediated activation of G-protein-activated inwardly rectifying potassium channel (Kir3.X) (GIRK) co
97 e primary conductance in Muller cells is the inwardly rectifying potassium channel Kir4.1 (BIR10 and
98 addressed the question of whether the glial inwardly rectifying potassium channel Kir4.1 associates
102 potassium, which are largely mediated by the inwardly-rectifying potassium channel Kir4.1, and to tak
105 units sulfonylurea receptor 1 (SUR1) and the inwardly rectifying potassium channel Kir6.2 cause persi
107 ng the sulfonylurea receptor 1 (SUR1) or the inwardly rectifying potassium channel Kir6.2, respective
108 multimeric protein complex composed of four inwardly rectifying potassium channel (Kir6.2) and four
109 subunits sulfonylurea receptor 1 (SUR1) and inwardly rectifying potassium channel (Kir6.2) of the be
111 -targeted CaMKII directly phosphorylates the inwardly-rectifying potassium channel, Kir6.2 (alpha sub
115 [(3)H]CGP 54626A, activate G-protein coupled inwardly rectifying potassium channels, or inhibit forsk
116 eosinophils resulted from the presence of an inwardly rectifying potassium channel, probably Kir2.1.
117 of the gene encoding the G-protein-activated inwardly rectifying potassium channel protein, GIRK2.
119 larke et al. now present 11 structures of an inwardly rectifying potassium channel, providing evidenc
120 P(a) < 0.08), as well as with ATP-sensitive inwardly rectifying potassium channel subunit Kir6.2 (KC
121 ultimers of sulfonylurea receptors (SUR) and inwardly rectifying potassium channel subunits (K(IR)6.x
123 oncentrations of ethanol, but other, related inwardly rectifying potassium channels were not affected
124 sulted in increased outward currents through inwardly rectifying potassium channels while intracellul
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。