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1 u]-PTSM showed lower perfusion than [(14)Cu]-iodoantipyrine.
2 ke and subsequent bolus injection of [(14)C]-iodoantipyrine.
6 )F-FEtOH uptake corresponded well with (14)C-iodoantipyrine and reflected the tumor perfusion-modifyi
11 f glucose (lCMRglu) were measured using [14C]iodoantipyrine (IAP) and [14C]2-deoxyglucose (2-DG) auto
12 that of the classic perfusion tracer [(14)C]-iodoantipyrine in a rat model during treadmill walking.
16 rCBF measured autoradiographically with 14C iodoantipyrine (Kety method) in the mid-line thalamus (b
17 rtex with 10(-2) M NMDA, the blood flow (14C-iodoantipyrine method) increased from 98 +/- 11 ml/min/1
18 edated, nontethered animal using the ((14))C-iodoantipyrine method, in which the tracer was administe
19 found between the [(64)Cu]-PTSM and [(14)C]-iodoantipyrine patterns of cerebral activation in cortic
20 uptake was directly compared with the (14)C-iodoantipyrine perfusion reporter, and the perfusion-mod
21 d by laser Doppler flowmetry or by the (14)C-iodoantipyrine technique with quantitative autoradiograp
22 with the quantitative autoradiographic (14)C-Iodoantipyrine technique, in conscious animals, 4 days,
24 que, the bulk diffusion coefficient for [14C]iodoantipyrine was measured in brain tissue (2.1 x 10(-6
25 X-1(-/-) mice, resting CBF assessed by (14)C-iodoantipyrine was reduced (-13% to -20%) in cerebral co
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