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1 rement (mGFR) by the plasma disappearance of iohexol.
2 Iodixanol, 8 (16%) to Iomeprol and 4 (8%) to Iohexol.
3 in GFR, measured by the plasma clearance of iohexol.
4 s were injected with 0.5, 1.0, and 2.0 mL of iohexol.
5 inst induction of pancreatic inflammation by iohexol.
8 omography (CT) of the abdomen with 100 mL of iohexol (300 mg iodine per milliliter, Omnipaque; GE Hea
11 logram of body weight [mg I/kg]), CTAP (with iohexol [600 mg I/kg]), triglyceride-enhanced CT (126 mg
12 tion rate (GFR) was directly measured (using iohexol) along with 12 markers of inflammation in Multic
13 GFR measured by the plasma disappearance of iohexol among 391 participants in the Chronic Kidney Dis
16 particles, prepared through cross-linking of iohexol and hexamethylene diisocyanate followed by copre
17 d with 1.5 mL per kilogram of body weight of iohexol and imaged between 2.5 and 10 minutes after inje
18 rved in 37 (14.3%) of 258 subjects receiving iohexol and in seven (2.5%) of 281 subjects in the contr
19 equilibrium CT protocol was developed using iohexol at 300 mg of iodine per milliliter (bolus of 1 m
21 ermined by measuring the plasma clearance of iohexol) between the baseline value and the last availab
27 five helical CT examinations: unenhanced CT, iohexol-enhanced CT (600 mg iodine per kilogram of body
28 ual-contrast-enhanced CT (112.4 HU +/- 1.2), iohexol-enhanced CT (97.9 HU +/- 2.2), triglyceride-enha
29 nhanced CT was significantly greater than at iohexol-enhanced CT (P < .05), and attenuation differenc
33 demonstrated similar liver opacification to iohexol-enhanced scans obtained with 600 mg of iodine pe
36 most precise at 0.41, and were within 30% of iohexol GFR, 89.3 and 96% of the time, respectively.
38 nstrated identical concentrations of Na+ and iohexol in ureteral effluent (UE) compared with circulat
39 y contrast media (ICM) iopamidol, iopromide, iohexol, iomeprol, and diatrizoate was examined in purif
42 lar, iodinated intravenous products, such as iohexol, is unlikely to have a clinically important effe
43 ar cells, but not HEK293 or COS7 cells, with iohexol led to a peak and then plateau in Ca(2+) signali
44 s GFR as measured by plasma disappearance of iohexol, likely a result of a change in methods used to
45 ntional small-molecule contrast agents, poly(iohexol) nanoparticles exhibited substantially protracte
47 fter intravenous administration of 120 mL of iohexol (Omnipaque 350; GE Healthcare, Princeton, NJ) in
48 uman acinar cells to the radiocontrast agent iohexol (Omnipaque; GE Healthcare, Princeton, NJ) and me
49 ith 150 mL of intravenous contrast material (iohexol, Omnipaque; Amersham Healthcare, Cork, Ireland)
51 contrast material (59 who underwent CT with iohexol or iodixanol and 81 who underwent MR imaging wit
52 , 150 mL of 60% iodinated contrast material (iohexol or iothalamate meglumine) was injected at either
55 tatin c-based formulas with a gold standard (iohexol plasma clearance) in 193 renal transplant recipi
58 triglyceride (especially when combined with iohexol), sensitivity values and liver-to-lesion attenua
59 is diluted into a 50% or greater strength of iohexol, the signal intensity curve shifts so that the m
60 he plasma clearance of the endogenous marker iohexol, to compare performance of existing equations of
61 t of urine from neonates who did not receive iohexol was 5.6 HU +/- 3.9, and that from neonates witho
65 gested nonionic iodinated contrast material (iohexol) with a concentration of 300 mg per milliliter w
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