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1 5 other G-protein-coupled receptors or on 15 ionic channels.
2 and CCK-8, which operate through independent ionic channels.
3 ake, but may be due to a secondary effect on ionic channels.
4 ndent properties in two molecularly distinct ionic channels.
5 those of gating current records reported for ionic channels.
6 to variability in the types and densities of ionic channels.
7 n translocation into liposomes and to elicit ionic channel activity at the phospholipids low affinity
8 h-clamped, providing the first recordings of ionic channel activity, synaptic vesicle release, and ga
9 ese operations, involving synapses, membrane ionic channels and changes in membrane potential, are th
10                                              Ionic channels and gap junctions are remodeled in cells
11 teins, including receptors, transporters and ionic channels, and to be active mostly as a homodimer.
12                                              Ionic channels bathed in mixed solutions of two permeant
13 reproducible conductance levels expected for ionic channels, Bax, but not Bcl-xL, created arbitrary a
14 nd lipid uptake through a temporarily stable ionic channel) become dominant in model liposome systems
15 nlinear DeltaV(m), we studied the effects of ionic channel blockers on DeltaV(m) in geometrically def
16 in cell cultures by measuring the effects of ionic channel blockers on DeltaVm and measuring uptake o
17 n with the rapid movement of calcium through ionic channels can cause large external calcium fluctuat
18 changes include alterations at the levels of ionic channels, cellular energy balance, neurohormonal e
19 We incorporated these parameters into a nine ionic channel conductance model to obtain completed mode
20 h as adenosine)-mediated response and not in ionic channel coupled receptor (such as GABA(A))-mediate
21 tal data sets on AF-induced changes of major ionic channel currents (ICaL, IKur, Ito, IK1, IKs, INaCa
22 duced in the model by inhibiting appropriate ionic channel currents according to experimentally repor
23 ctifier potassium current) or block multiple ionic channels (e.g., ibutilide and azimilide) in order
24 systems, suggesting that the coregulation of ionic channel expression, by thus linking their variabil
25 is interpreted as due to its passage through ionic channels for both Na+ and K+.
26                    Kinetics of voltage-gated ionic channels fundamentally reflect the response of the
27 gate age- and chamber-specific expression of ionic channels in the developing fetal mouse.
28 merous ion channels, to simplified, grouping ionic channels into a minimal set of variables.
29 ionic transport through porous membranes and ionic channels is important in numerous applications ran
30            Selective and rapid regulation of ionic channels is pivotal to the understanding of physio
31                       Such a coregulation of ionic channels is uniquely observable in a cell speciali
32    In this review, we focus on the principal ionic channels (KATP, Nav, and Cav channels) involved in
33 Additional experiments examined the membrane ionic channels mediating these GluR activation effects.
34 biological "fusion pores" can be as small as ionic channels or gap junctions, one model posits a prot
35 hy causes remodelling, leading to changes in ionic channel, pump and exchanger densities and kinetics
36 t the cellular scale this remodelling of the ionic channels, pumps and exchangers gives rise to chang
37 or agents that are neuroprotective or affect ionic channels; straightforward transduction of gene exp
38  expressed in a "retrograde" manner with the ionic channel that is modulated appearing early in devel
39                 The various abnormalities in ionic channels, transporters, kinases and various signal
40                            The nature of the ionic channels underlying the excitatory actions of BK i

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