ライフサイエンスコーパス: ionotropic

1 otor neurons express distinct populations of ionotropic acetylcholine receptors (iAChRs) requiring th

2 hibition results from the combined action of ionotropic acetylcholine receptors and associated calciu

3 The blockade of muscle ionotropic acetylcholine receptors by (+)-tubocurarine d

4 is released to supplement influx through the ionotropic ACh receptor.

5 Ionotropic activation of NMDA receptors (NMDARs) require

6 icity-induced neuronal death through the non-ionotropic activity of GluN2ARs and the neuroprotective

7 ing NMDARs (GluN2ARs), suggesting that a non-ionotropic activity of GluN2ARs mediates glycine-induced

8 nexpected role of glycine in eliciting a non-ionotropic activity of GluN2ARs to confer neuroprotectio

9 Dental pulpal nerve fibers express ionotropic adenosine triphosphate (ATP) receptors, sugge

10 amate receptor subunit B, glutamate receptor ionotropic AMPA 2 (GRIA2), modifies a codon, replacing t

11 begins with the binding of glutamate to the ionotropic AMPA receptors and metabotropic glutamate rec

12 long-lived changes is the remodeling of the ionotropic AMPA-type glutamate receptors that underlie f

13 In previous studies, stimulation of ionotropic AMPA/kainate glutamate receptors on cultured

14 a local Ca(2+) rise, even in the presence of ionotropic and cell surface metabotropic receptor inhibi

15 ion, several sequences representing putative ionotropic and gustatory receptors were also identified.

16 Moreover, we demonstrate that these ionotropic and metabotropic 5-HT receptors have a synerg

17 We identify that both ionotropic and metabotropic 5-hydroxytryptamine (5-HT) r

18 "MAG" PTLs for ionotropic and metabotropic glutamate receptors (GluRs)

19 oincided with transcriptionally dysregulated ionotropic and metabotropic glutamate receptors and glut

20 attenuated by antagonists targeting multiple ionotropic and metabotropic glutamate receptors, and int

21 citatory glutamatergic transmission, through ionotropic and metabotropic glutamate receptors, is nece

22 tes aberrant extrasynaptic signaling through ionotropic and metabotropic glutamate receptors, ultimat

23 MF-CA3 synapses, with implications for both ionotropic and metabotropic glutamatergic recruitment of

24 A similar loss of ionotropic and metabotropic KAR function was observed in

25 These ionotropic and metabotropic P2 purinergic receptors modu

26 ATP release triggers the activation of both ionotropic and metabotropic purinoceptors, with strong p

27 iguingly, a large number of genes coding for ionotropic and metabotropic receptors for various neurot

28 mate released from climbing fibers activates ionotropic and metabotropic receptors on Golgi cells thr

29 They express ionotropic and metabotropic receptors, and can release g

30 that glutamate and GABA signal through both ionotropic and metabotropic receptors.

31 ratively controlled by the interplay between ionotropic and metabotropic signals.

32 compounds were used: agonists of receptors (ionotropic and metabotropic) that alter cytoplasmic calc

33 mechanism for the neuronal serotonin 5-HT3A ionotropic channel receptor, in which the role of routin

34 combines the activity of an unusual class of ionotropic cholinergic receptor with that of nearby calc

35 d within an intron of the glutamate receptor ionotropic delta-1 gene (GRID1), but its ER stress-assoc

36 channels and is responsible for the positive ionotropic effect of adrenergic stimulation.

37 uingly, a recent report revealed a novel non-ionotropic function of the NMDAR in the regulation of sy

38 rstanding of disease-associated mutations in ionotropic GABA and glutamate receptor families, and dis

39 Because we could not localize ionotropic GABA receptors on cone axon terminals using e

40 the expression of previously uncharacterized ionotropic GABA receptors.

41 e hypotheses in mice by genetically removing ionotropic GABA(A) or metabotropic GABA(B) receptors fro

42 Feed-forward inhibition mediated by ionotropic GABA(A) receptors contributes to the temporal

43 citatory nicotinic ACh receptors, inhibitory ionotropic GABA(A) receptors, and inhibitory ionotropic

44 s by targeting both metabotropic GABA(B) and ionotropic GABA(A)/glycine receptors.

45 electrophysiology to study the expression of ionotropic GABA, glutamate, and ATP receptors in oligode

46 the driving force for the chloride-permeable ionotropic GABAA receptor in mature neurons.

47 aptic inhibition in the brain is mediated by ionotropic GABAA receptors (GABAARs) and metabotropic GA

48 nsmission in the brain is mediated mostly by ionotropic GABAA receptors (GABAARs), but their essentia

49 e metabotropic GABAB receptor GBB-1, but not ionotropic GABAA receptors.

50 ers are, surprisingly, severe antagonists of ionotropic gamma-aminobutyric acid (GABA) receptors.

51 hibitory postsynaptic potentials mediated by ionotropic gamma-aminobutyric acid receptors (GABAARs) a

52 -beta-benzyloxyaspartic acid, but not by the ionotropic Glu receptor agonists, alpha-2-amino-3-(5-met

53 The fast acting ionotropic Glu receptors (iGluRs) are ligand gated ion c

54 ionotropic GABA(A) receptors, and inhibitory ionotropic GluCl (glutamate-gated chloride) receptors.

55 ective activation of glomerular mAChRs, with ionotropic GluRs and nAChRs blocked, increased IPSCs in

56 inases have been securely identified in many ionotropic glutamate (iGlu) receptor subunits, but which

57 nd wasps are potent open-channel blockers of ionotropic glutamate (iGlu) receptors.

58 ent but nonselective open-channel blocker of ionotropic glutamate (iGlu) receptors.

59 of angiotensin II receptor type 1, oxytocin, ionotropic glutamate and GABAA receptors.

60 Activation of ionotropic glutamate and/or GABA receptors along the GnR

61 In each case, infusion of the ionotropic glutamate antagonist kynurenic acid blocked t

62 us glutamate inputs as it was blocked by the ionotropic glutamate antagonist NBQX, but independent of

63 ia angiotensin II type 1 receptor, oxytocin, ionotropic glutamate or GABAA receptors but instead invo

64 ptic depression is modulated by postsynaptic ionotropic glutamate receptor (iGluR) activity.

65 Exposure to ionotropic glutamate receptor (iGluR) agonists, kainic a

66 dria in astrocytic processes were blocked by ionotropic glutamate receptor (iGluR) antagonists, tetro

67 bitory currents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

68 Ionotropic glutamate receptor (iGluR) channels control s

69 anisms regulating the extent of postsynaptic ionotropic glutamate receptor (iGluR) clustering have be

70 Ionotropic glutamate receptor (iGluR) family members are

71 as seen a revolution in our understanding of ionotropic glutamate receptor (iGluR) structure, startin

72 The GluD1 and GluD2 receptors form the GluD ionotropic glutamate receptor (iGluR) subfamily.

73 Loss of specific ionotropic glutamate receptor (iGluR) subunits during in

74 The amino terminal domain (ATD) of ionotropic glutamate receptor (iGluR) subunits resides a

75 Within ionotropic glutamate receptor (iGluR) subunits, this pro

76 lated GluD1 are classified as members of the ionotropic glutamate receptor (iGluR) superfamily on the

77 trated that a member of the newly discovered ionotropic glutamate receptor (IR) family, IR76b, functi

78 excitatory mammalian ion channel light-gated ionotropic glutamate receptor (LiGluR) in retinal gangli

79 ype of positive allosteric modulators of the ionotropic glutamate receptor A2 (GluA2) are promising l

80 Using heterologous expression of excitatory ionotropic glutamate receptor AMPA subunits in Xenopus o

81 structure-activity study of the broad-acting ionotropic glutamate receptor antagonist 1a.

82 y-NH2 , d(CH2 )5 [D-Tyr(2) ,Thr(4) ]OVT, the ionotropic glutamate receptor antagonist kynurenate or t

83 Ionotropic glutamate receptor antagonists are valuable t

84 Since ionotropic glutamate receptor antagonists can partially

85 ves, MC calcium transients were inhibited by ionotropic glutamate receptor antagonists, indicating th

86 ate and not 5-HT because it was abolished by ionotropic glutamate receptor antagonists.

87 We conclude that the two types of ionotropic glutamate receptor are built in different way

88 einstated by 4-aminopyridine, and blocked by ionotropic glutamate receptor blockers.

89 Here, we design a light-controlled ionotropic glutamate receptor by genetically encoding a

90 We used RT-PCR to profile ionotropic glutamate receptor expression in cultured SCs

91 We focus here on the control of ionotropic glutamate receptor function by GPCR signaling

92 he responsible sensory receptor (the variant ionotropic glutamate receptor IR75b) and attraction-medi

93 The mechanism by which agonist binding to an ionotropic glutamate receptor leads to channel opening i

94 y SNAG-mGluR2 and excitatory light-activated ionotropic glutamate receptor LiGluR yielded a distribut

95 -shifted PTL, L-MAG0460, for the light-gated ionotropic glutamate receptor LiGluR.

96 ations in melanoma and the significance that ionotropic glutamate receptor signaling has in malignant

97 Electrophysiological testing at three ionotropic glutamate receptor subtypes reveals that two

98 onses, short-term dynamics and expression of ionotropic glutamate receptor subtypes.

99 N2A subunit of the NMDA receptor (NMDAR), an ionotropic glutamate receptor that has important roles i

100 NMDA receptors (NMDAR), a type of ionotropic glutamate receptor, mediate synaptic plastici

101 orylation and dephosphorylation of AMPA-type ionotropic glutamate receptors (AMPARs) by kinases and p

102 AMPA-subtype ionotropic glutamate receptors (AMPARs) mediate fast exc

103 KEY POINTS: The AMPA-type ionotropic glutamate receptors (AMPARs) mediate the majo

104 eurotransmission is mediated by AMPA-subtype ionotropic glutamate receptors (AMPARs).

105 GRIA3 encodes GluA3, a subunit of AMPA-type ionotropic glutamate receptors (AMPARs).

106 Ionotropic glutamate receptors (GluRs) are ligand-gated

107 Ionotropic glutamate receptors (iGluRs) are ligand-gated

108 Ionotropic glutamate receptors (iGluRs) are tetrameric c

109 Ionotropic glutamate receptors (iGluRs) are ubiquitous i

110 ation followed the arrival and clustering of ionotropic glutamate receptors (iGluRs) at NMJ synapses.

111 generated an extensive sequence alignment of ionotropic glutamate receptors (iGluRs) from diverse ani

112 hores have revealed the presence of numerous ionotropic glutamate receptors (iGluRs) in Mnemiopsis le

113 Ionotropic glutamate receptors (iGluRs) mediate excitato

114 Ionotropic glutamate receptors (iGluRs) mediate most exc

115 Ionotropic glutamate receptors (iGluRs) mediate neurotra

116 Ionotropic glutamate receptors (iGluRs) mediate the majo

117 Presynaptic ionotropic glutamate receptors (iGluRs) play important r

118 is comb jelly encodes homologs of vertebrate ionotropic glutamate receptors (iGluRs) that are distant

119 NMDA receptors (NMDARs) are a class of ionotropic glutamate receptors (iGluRs) that are essenti

120 Kainate receptors (KARs) are a subfamily of ionotropic glutamate receptors (iGluRs) that mediate exc

121 Kainate receptors (KARs) are ionotropic glutamate receptors (iGluRs) that modulate sy

122 Ionotropic glutamate receptors (iGluRs) transduce the ch

123 Ionotropic glutamate receptors (iGluRs) underlie rapid,

124 Ionotropic glutamate receptors (iGluRs), including the N

125 americ ion channels that together with other ionotropic glutamate receptors (iGluRs), the NMDA and ka

126 Here, we studied ionotropic glutamate receptors (iGluRs), which are ion c

127 stem is mediated by glutamate acting through ionotropic glutamate receptors (iGluRs).

128 s excitatory synaptic transmission by gating ionotropic glutamate receptors (iGluRs).

129 neurons express either odorant receptors or ionotropic glutamate receptors (IRs).

130 The ionotropic glutamate receptors (N-methyl-D-aspartate rec

131 and memory and a reduction in the amounts of ionotropic glutamate receptors (NMDA and AMPA receptors)

132 Ionotropic glutamate receptors activate cell signaling i

133 Adenosine release required the activation of ionotropic glutamate receptors and could be evoked by lo

134 These results identified ionotropic glutamate receptors and NMDA-Rs, specifically

135 the dimeric NTD of GluN1 is unique among the ionotropic glutamate receptors and predicts that the str

136 or understanding gating across the family of ionotropic glutamate receptors and the role of AMPA rece

137 We focus particularly on three classes of ionotropic glutamate receptors and their transmembrane i

138 Importantly, synchrony was resistant to ionotropic glutamate receptors antagonists but was stron

139 Ionotropic glutamate receptors are a family of tetrameri

140 Ionotropic glutamate receptors are important for estradi

141 Ionotropic glutamate receptors are ligand-gated ion chan

142 Ionotropic glutamate receptors are postsynaptic tetramer

143 Positive allosteric modulators of ionotropic glutamate receptors are potential compounds f

144 The ionotropic glutamate receptors are primary mediators of

145 Ionotropic glutamate receptors are thought to play an es

146 Ionotropic glutamate receptors are widely distributed in

147 s NL1 isoform-specific cis-interactions with ionotropic glutamate receptors as a key mechanism for co

148 -93, and SAP97, are scaffolding proteins for ionotropic glutamate receptors at excitatory synapses.

149 ological diseases, is not mediated merely by ionotropic glutamate receptors but also by heteromeric T

150 al D1R/PKA/MEK1/2 pathway and independent of ionotropic glutamate receptors but blocked by antagonist

151 Ionotropic glutamate receptors comprise two conformation

152 The delta family of ionotropic glutamate receptors consists of glutamate del

153 The overstimulated ionotropic glutamate receptors exert their neurotoxic ef

154 NMDA ionotropic glutamate receptors gate the cytoplasmic infl

155 s been designed to enable optical control of ionotropic glutamate receptors in neurons via sensitized

156 discovery of 20 genes encoding for putative ionotropic glutamate receptors in the Arabidopsis (Arabi

157 e N-methyl-d-aspartate (NMDA) subtype of the ionotropic glutamate receptors is the primary mediator o

158 Therefore, our results suggest that ionotropic glutamate receptors may have been conserved t

159 N-methyl-d-aspartate (NMDA)-type ionotropic glutamate receptors mediate excitatory neurot

160 5-methyl-4-isoxazole propionic acid)-subtype ionotropic glutamate receptors mediate fast excitatory n

161 AMPA subtype ionotropic glutamate receptors mediate fast excitatory n

162 cumulation leading to overstimulation of the ionotropic glutamate receptors mediates neuronal injury

163 ced by systemic injections of antagonists of ionotropic glutamate receptors or metabotropic glutamate

164 ptors (IRs) are a large subfamily of variant ionotropic glutamate receptors present across Protostomi

165 enhanced by DEX (10 microM), and blockade of ionotropic glutamate receptors reduced the DEX effect on

166 e suggests that surface trafficking of other ionotropic glutamate receptors requires ligand binding f

167 -4-isoxazole-propionate receptor (AMPAR) are ionotropic glutamate receptors responsible for excitator

168 Kainate receptors (KARs) are ionotropic glutamate receptors that also activate noncan

169 NMDA receptors (NMDARs) are ionotropic glutamate receptors that are crucial for neur

170 NMDA receptors are ionotropic glutamate receptors that function as heterote

171 ptors (NMDARs) are a subtype of postsynaptic ionotropic glutamate receptors that function as molecula

172 N-methyl-D-aspartate-receptors (NMDARs) are ionotropic glutamate receptors that function in synaptic

173 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors that mediate a majority o

174 NMDA receptors are ionotropic glutamate receptors that mediate a slow, Ca2+

175 NMDA receptors are ionotropic glutamate receptors that mediate excitatory s

176 During apnea, induced by blockade of ionotropic glutamate receptors within the same region, m

177 nfusion of kynurenic acid (an antagonist for ionotropic glutamate receptors) into core but not shell

178 Blockers of ionotropic glutamate receptors, 2,3-dihydroxy-6-nitro-7-

179 contain beta-adrenergic receptors as well as ionotropic glutamate receptors, and retromer knockdown r

180 Kainate receptors (KARs), a family of ionotropic glutamate receptors, are widely expressed in

181 eceptors (KARs), one of three subfamilies of ionotropic glutamate receptors, as well as the putative

182 lence of somatic mutations within one of the ionotropic glutamate receptors, GRIN2A, in malignant mel

183 Glutamate-gated ion channels (ionotropic glutamate receptors, iGluRs) sense the extrac

184 s of anti-BDNF were abolished by blockade of ionotropic glutamate receptors, indicating a role for gl

185 at central synapses depends on the number of ionotropic glutamate receptors, particularly the class g

186 structural basis of allosteric inhibition in ionotropic glutamate receptors, providing key insights i

187 inate receptors (KARs) consist of a class of ionotropic glutamate receptors, which exert diverse pre-

188 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors, which mediate most excit

189 Correspondingly, AMPA-subtype ionotropic glutamate receptors, which mediate the majori

190 Ionotropic glutamate receptors, which underlie a majorit

191 ystemic elimination and the kidney expresses ionotropic glutamate receptors.

192 key neuronal signaling molecules, including ionotropic glutamate receptors.

193 provide target validation for this family of ionotropic glutamate receptors.

194 trafficking, or inactivation of postsynaptic ionotropic glutamate receptors.

195 is resistant to blockade of metabotropic and ionotropic glutamate receptors.

196 ease of ascorbate is delicately regulated by ionotropic glutamate receptors.

197 is mediated by glutamate acting on AMPA-type ionotropic glutamate receptors.

198 nnels, suggesting a link in the evolution of ionotropic glutamate receptors.

199 azole propionate and kainate subtypes of the ionotropic glutamate receptors.

200 All three ionotropic glutamate subfamilies (i.e. AMPA-type, kainat

201 gle sub-psychotomimetic dose of ketamine, an ionotropic glutamatergic n-methyl-D-aspartate (NMDA) rec

202 nt clinical studies report that ketamine, an ionotropic glutamatergic N-methyl-D-aspartate (NMDA) rec

203 ngle subpsychotomimetic dose of ketamine, an ionotropic glutamatergic N-methyl-D-aspartate (NMDA) rec

204 ingle sub-psychomimetic dose of ketamine, an ionotropic glutamatergic NMDAR (N-methyl-D-aspartate rec

205 We show that ionotropic glutamatergic receptor activation is required

206 signalling in PF-PC spines does not involve ionotropic glutamatergic receptors because postsynaptic

207 N-methyl-d-aspartate receptors (NMDARs) are ionotropic glutamatergic receptors that have been implic

208 ovel analgesic strategy is to restore spinal ionotropic inhibition by enhancing KCC2-mediated chlorid

209 ng KCC2 may be a tenable method of restoring ionotropic inhibition not only in neuropathic pain but a

210 s on chromosome 1 (GRIK3 (glutamate receptor ionotropic kainate 3)), chromosome 4 (KLHL2 (Kelch-like

211 Although a G-protein-dependent non-ionotropic mechanism has been suggested to underlie this

212 Owing to the dual ionotropic/metabotropic nature of alpha7 receptors, sign

213 ngs demonstrate that Ppk1 can function as an ionotropic molecular sensory transducer capable of trans

214 mplification) of the gene glutamate receptor ionotropic N-methyl D-aspertate as a potential new thera

215 based antidepressants by not only modulating ionotropic (N-methyl-D-aspartate and alpha-amino-3-hydro

216 ted ion channel (pLGIC) family that mediates ionotropic neurotransmission.

217 d by UNC-3 also regulating the expression of ionotropic neurotransmitter receptors and putative stret

218 Ionotropic neurotransmitter receptors mediate fast synap

219 Ionotropic neurotransmitter receptors mediate fast synap

220 eptors, induces physical association between ionotropic (NMDA) and metabotropic (mGlu5a) synaptic glu

221 ging and time-lapse imaging to show that non-ionotropic NMDAR signaling can drive shrinkage of dendri

222 Here, we tested whether non-ionotropic NMDAR signaling could also play a role in dri

223 tivation of NMDARs, whereas costimulation of ionotropic non-NMDAR glutamate receptors transiently ant

224 lthough pharmacological activation of either ionotropic or cAMP-dependent pathways acted in synergy w

225 trains the high-power state because blocking ionotropic or metabotropic glutamate receptors results i

226 Actions of ATP are mediated through both ionotropic P2X receptors and metabotropic P2Y receptors.

227 P2Y receptors are G-protein-coupled, whereas ionotropic P2X receptors are ATP-gated ion channels.

228 nd P1 receptors for adenosine in addition to ionotropic P2X receptors for ATP.

229 osphate (ATP) induces pain via activation of ionotropic P2X receptors while adenosine mediates analge

230 This response depends on both ionotropic (P2X) and metabotropic (P2Y) purinergic recep

231 ATP-gated ionotropic P2X2 receptors are widely expressed in neuron

232 ATP-gated ionotropic P2X4 receptors are up-regulated in activated

233 nel/pannexin1 (Panx1) channel and purinergic ionotropic P2X7 receptor (P2X7R) blockers.

234 The ATP-gated ionotropic P2X7 receptor (P2X7R) modulates glial activat

235 mechanism involving autocrine activation of ionotropic P2X7 receptors (P2X7R) by ATP.

236 The lymphocytic ionotropic purinergic P2X receptors (P2X1R-P2X7R, or P2X

237 ing cell damage/activation, is sensed by the ionotropic purinergic receptor P2X7 (P2X7R) on lymphocyt

238 is emerging consensus that P2X(4) and P2X(7) ionotropic purinoceptors (P2X(4)R and P2X(7)R) are criti

239 Taste buds release ATP to activate ionotropic purinoceptors composed of P2X2 and P2X3 subun

240 n the hippocampal CA1 region is dependent on ionotropic, rather than metabotropic, NMDAR signaling.

241 ials required several members of the variant ionotropic receptor (IR) family (IR25a, IR62a, and IR76b

242 express IR92a, a member of the chemosensory ionotropic receptor (IR) family and project to a pair of

243 All sour GRNs prominently express two Ionotropic Receptor (IR) genes, IR76b and IR25a, and we

244 The Ionotropic Receptor (IR) superfamily is best known for i

245 odor receptor (Or), gustatory receptor (Gr), ionotropic receptor (IR), Pickpocket (Ppk), and Trp fami

246 Here we show that Drosophila Ionotropic Receptor 25a (IR25a) is required for behaviou

247 Immunolocalization and analysis using the ionotropic receptor channel-permeant cation agmatine ind

248 rs and demonstrate that they form functional ionotropic receptor channels.

249 acts in mechanosensory neurons by modulating ionotropic receptor currents, the initiating step of cel

250 enriched in female antennae, thus making the ionotropic receptor family the largest of antennae-rich

251 These findings identify TRPV1 as an ionotropic receptor for retinoids and provide cellular a

252 y monocytes but does not stimulate canonical ionotropic receptor functions.

253 GRN activation requires the function of the Ionotropic Receptor genes IR25a, IR76b and IR56d.

254 Most odorant and ionotropic receptor genes seemed to be expressed in all

255 of a distinct repertoire of metabotropic and ionotropic receptor genes was identified in both NPS neu

256 A neuron responding to moist air and its ionotropic receptor have been identified in Drosophila m

257 Neurons expressing the ionotropic receptor IR40a have been implicated in the se

258 regarding its mode of action: activation of ionotropic receptor IR40a vs. odorant receptor(s).

259 Neither metabotropic nor ionotropic receptor mechanisms alone are sufficient for

260 y protein subunits) or NMDARs [via glutamate ionotropic receptor NMDA-type subunit 2B (GluN2B) subuni

261 dependent on the subunit composition of the ionotropic receptor or channel as well as the GPCR subty

262 icroglia can be influenced by the purinergic ionotropic receptor P2X7 via a coupling with Pannexin-1.

263 The P2X7 receptor is an ionotropic receptor predominantly expressed on the surfa

264 RNA levels for glycinergic and glutamatergic ionotropic receptor subunits, confirming a switch from G

265 ct activation of an odorant receptor, not an ionotropic receptor, is necessary for DEET reception and

266 h the synaptic activation of the fast-acting ionotropic receptor, LGC-55, and extrasynaptic activatio

267 t volatile molecules using olfactory (OR) or ionotropic receptors (IR) and in some cases gustatory re

268 Ionotropic Receptors (IRs) are a large subfamily of vari

269 ceptors (ORs), gustatory receptors (GRs) and ionotropic receptors (IRs) function to interface the ins

270 at Drosophila hygrosensation relies on three Ionotropic Receptors (IRs) required for dry cell functio

271 e show that DOCC cool-sensing is mediated by Ionotropic Receptors (IRs), a family of sensory receptor

272 ne in Drosophila a group of approximately 35 ionotropic receptors (IRs), the IR20a clade, about which

273 the odorant receptors (ORs) and the variant ionotropic receptors (IRs).

274 (Rs) constitute a subclass of ATP-sensitive ionotropic receptors (P2X1-P2X7).

275 lear hair cells via alpha9alpha10-containing ionotropic receptors and associated calcium-activated (S

276 Axons can be depolarized by ionotropic receptors and transmit subthreshold depolariz

277 ments show that functional glutamate or GABA ionotropic receptors are expressed on human subplate (SP

278 Ion channels and ionotropic receptors can also be reconstituted from memb

279 a simple behavioral assay, we find that the ionotropic receptors IR40a, IR93a, and IR25a are all req

280 Ionotropic receptors of gamma-aminobutyric acid (GABAAR)

281 alter two other classes of calcium-permeable ionotropic receptors on the same neurons.

282 of intracellular Ca(2+) via metabotropic and ionotropic receptors or direct UV-uncaging.

283 Identification of ionotropic receptors required for hygrosensation in Dros

284 tion of most representative Ca(2+)-permeable ionotropic receptors similarly regulate T-type current p

285 l-d-aspartate (NMDA) receptors are glutamate ionotropic receptors that play critical roles in synapti

286 Transcripts for as many as 43 ionotropic receptors were enriched in female antennae, t

287 w' (G-protein-coupled receptors) and 'fast' (ionotropic receptors) neurotransmission converging on th

288 s that arises from activation of presynaptic ionotropic receptors, or somatic depolarization, can enh

289 uences on ion channel signaling via specific ionotropic receptors, providing a window on the hidden s

290 ions in the subunit composition of glutamate ionotropic receptors.

291 e rapid excitatory transmission sustained by ionotropic receptors.

292 luding the feature and surface expression of ionotropic receptors.

293 y with regard to targets on metabotropic and ionotropic receptors.

294 noamine transporters should be considered as ionotropic receptors.

295 annel in vitro, identifying it as a probable ionotropic sensory receptor.

296 The ionotropic serotonin receptor, 5-HT3 , is expressed by m

297 x through the NMDAR normally overcomes a non-ionotropic shrinkage signal to drive spine growth.

298 TRPM3 channels form ionotropic steroid receptors in the plasma membrane of p

299 lly and this connection uses glutamate as an ionotropic transmitter.

300 Thus, to explore the role of ionotropic versus metabotropic NMDAR signaling in LTD, w