ライフサイエンスコーパス: ionotropic glutamate receptor

1 e the N-methyl-D-aspartate (NMDA) subtype of ionotropic glutamate receptor.

2 face maintenance of the N-methyl-D-aspartate ionotropic glutamate receptor.

3 trafficking, or inactivation of postsynaptic ionotropic glutamate receptors.

4 is resistant to blockade of metabotropic and ionotropic glutamate receptors.

5 ease of ascorbate is delicately regulated by ionotropic glutamate receptors.

6 is mediated by glutamate acting on AMPA-type ionotropic glutamate receptors.

7 nnels, suggesting a link in the evolution of ionotropic glutamate receptors.

8 azole propionate and kainate subtypes of the ionotropic glutamate receptors.

9 occurs primarily through AMPA- and NMDA-type ionotropic glutamate receptors.

10 es were largely insensitive to block of fast ionotropic glutamate receptors.

11 tion by signaling through platelet-expressed ionotropic glutamate receptors.

12 c processes are essential to the function of ionotropic glutamate receptors.

13 coupled to an altered expression profile of ionotropic glutamate receptors.

14 ve deficits by increasing internalization of ionotropic glutamate receptors.

15 necessary for plasma membrane expression of ionotropic glutamate receptors.

16 mmalian central nervous system by activating ionotropic glutamate receptors.

17 onstrating functional activation of specific ionotropic glutamate receptors.

18 ned potential interactions between EAAC1 and ionotropic glutamate receptors.

19 nd GRIK2, respectively, both of which encode ionotropic glutamate receptors.

20 ast synaptic potentials mediated by GABAA or ionotropic glutamate receptors.

21 which has not previously been described for ionotropic glutamate receptors.

22 ystemic elimination and the kidney expresses ionotropic glutamate receptors.

23 key neuronal signaling molecules, including ionotropic glutamate receptors.

24 provide target validation for this family of ionotropic glutamate receptors.

25 Blockers of ionotropic glutamate receptors, 2,3-dihydroxy-6-nitro-7-

26 ype of positive allosteric modulators of the ionotropic glutamate receptor A2 (GluA2) are promising l

27 Ionotropic glutamate receptors activate cell signaling i

28 Subsequent activation of ionotropic glutamate receptors activates MAP kinases, th

29 Here, we show that treatment of neurons with ionotropic glutamate receptor agonists causes CPEB4 to a

30 sion, but it remains uncertain whether these ionotropic glutamate receptors also have essential subun

31 studied whether a prolonged inactivation of ionotropic glutamate receptors also induces cholinergic

32 Using heterologous expression of excitatory ionotropic glutamate receptor AMPA subunits in Xenopus o

33 Among ionotropic glutamate receptors (AMPA, kainate, NMDA), AM

34 Among the three classes of ionotropic glutamate receptors (AMPA, NMDA, and kainate

35 tion of PCs involves two distinct classes of ionotropic glutamate receptors, AMPA receptors and GluR5

36 Numerous in vitro findings indicate that the ionotropic glutamate receptor, AMPAR, can rapidly traffi

37 rain is carried by AMPA and NMDA subtypes of ionotropic glutamate receptors (AMPARs and NMDARs).

38 orylation and dephosphorylation of AMPA-type ionotropic glutamate receptors (AMPARs) by kinases and p

39 AMPA-subtype ionotropic glutamate receptors (AMPARs) mediate fast exc

40 KEY POINTS: The AMPA-type ionotropic glutamate receptors (AMPARs) mediate the majo

41 eurotransmission is mediated by AMPA-subtype ionotropic glutamate receptors (AMPARs).

42 GRIA3 encodes GluA3, a subunit of AMPA-type ionotropic glutamate receptors (AMPARs).

43 ry synaptic signaling mediated by AMPA-class ionotropic glutamate receptors (AMPARs).

44 at may permit calcium influx such as certain ionotropic glutamate receptors and canonical transient r

45 Adenosine release required the activation of ionotropic glutamate receptors and could be evoked by lo

46 populated by glial progenitors that express ionotropic glutamate receptors and extend numerous proce

47 lts extend the influence of PIP2 to the NMDA ionotropic glutamate receptors and introduce a novel mec

48 nomycin D and tetrodotoxin, by inhibitors of ionotropic glutamate receptors and L-type voltage-gated

49 l function and pharmacological properties of ionotropic glutamate receptors and likely are important

50 These results identified ionotropic glutamate receptors and NMDA-Rs, specifically

51 in the PSD fraction, whereas the amounts of ionotropic glutamate receptors and other prominent PSD p

52 the dimeric NTD of GluN1 is unique among the ionotropic glutamate receptors and predicts that the str

53 elevated glutamate may subsequently activate ionotropic glutamate receptors and result in the dephosp

54 ltage-gated Ca2+ channels but persisted when ionotropic glutamate receptors and sodium spikes were bl

55 or understanding gating across the family of ionotropic glutamate receptors and the role of AMPA rece

56 We focus particularly on three classes of ionotropic glutamate receptors and their transmembrane i

57 henylborate (2APB) but not by antagonists of ionotropic glutamate receptors and voltage-dependent cal

58 on of voltage-gated Na(+) (NaV) channels and ionotropic glutamate receptors, and formation of synapse

59 contain beta-adrenergic receptors as well as ionotropic glutamate receptors, and retromer knockdown r

60 s ([glutamate]e) and was prevented by either ionotropic glutamate receptor antagonism or removal of L

61 structure-activity study of the broad-acting ionotropic glutamate receptor antagonist 1a.

62 cell degeneration, or in combination with an ionotropic glutamate receptor antagonist 6-cyano-7-nitro

63 no-4-phosphonobutyric acid (APB), and/or the ionotropic glutamate receptor antagonist cis-2,3 piperid

64 y-NH2 , d(CH2 )5 [D-Tyr(2) ,Thr(4) ]OVT, the ionotropic glutamate receptor antagonist kynurenate or t

65 Injection of kynurenic acid (Kyn, ionotropic glutamate receptor antagonist) into RVLM or t

66 agonist) or kynurenic acid (a non-selective ionotropic glutamate receptor antagonist) microinjected

67 ficantly inhibited by bath application of an ionotropic glutamate receptor antagonist, indicating tha

68 s been identified as a competitive AMPA-type ionotropic glutamate receptor antagonist, whilst (S)-3,4

69 ent of complex EPSCs was markedly reduced by ionotropic glutamate receptor antagonists (2-amino-5-pho

70 rolactin were abolished by coinfusion of the ionotropic glutamate receptor antagonists 6-cyano-7-nitr

71 Ionotropic glutamate receptor antagonists are valuable t

72 Since ionotropic glutamate receptor antagonists can partially

73 ics (1.5-2.0 ms) and was blocked by non-NMDA ionotropic glutamate receptor antagonists NBQX or CNQX.

74 increased proMMP-2 release, whereas non-NMDA ionotropic glutamate receptor antagonists reduced IL-6 p

75 rficial layers of the SC, in the presence of ionotropic glutamate receptor antagonists, evoked IPSCs

76 ves, MC calcium transients were inhibited by ionotropic glutamate receptor antagonists, indicating th

77 ions on hypocretin neurons were abolished by ionotropic glutamate receptor antagonists.

78 ate and not 5-HT because it was abolished by ionotropic glutamate receptor antagonists.

79 This can be blocked by ionotropic glutamate receptor antagonists.

80 Importantly, synchrony was resistant to ionotropic glutamate receptors antagonists but was stron

81 We conclude that the two types of ionotropic glutamate receptor are built in different way

82 chitecture and atomic structure of an intact ionotropic glutamate receptor are unknown.

83 Ionotropic glutamate receptors are a family of tetrameri

84 Thus, VP ionotropic glutamate receptors are critical mediators of

85 Ionotropic glutamate receptors are functionally diverse

86 Ionotropic glutamate receptors are important for estradi

87 Ionotropic glutamate receptors are ligand-gated ion chan

88 Ionotropic glutamate receptors are ligand-gated ion chan

89 Ionotropic glutamate receptors are ligand-gated ion chan

90 The ionotropic glutamate receptors are localized in the pre-

91 Ionotropic glutamate receptors are postsynaptic tetramer

92 Positive allosteric modulators of ionotropic glutamate receptors are potential compounds f

93 The ionotropic glutamate receptors are primary mediators of

94 ng, and synaptic targeting of KARs and other ionotropic glutamate receptors are processes controlled,

95 and transporter knock-out mice revealed that ionotropic glutamate receptors are responsible for >40%

96 Ionotropic glutamate receptors are tetramers, the isolat

97 Ionotropic glutamate receptors are thought to play an es

98 Ionotropic glutamate receptors are widely distributed in

99 MPA) receptors, one subtype in the family of ionotropic glutamate receptors, are the main receptors r

100 Kainate receptors (KARs), a family of ionotropic glutamate receptors, are widely expressed in

101 s NL1 isoform-specific cis-interactions with ionotropic glutamate receptors as a key mechanism for co

102 was not due to a differential expression of ionotropic glutamate receptors, as evidenced by similar

103 eceptors (KARs), one of three subfamilies of ionotropic glutamate receptors, as well as the putative

104 -93, and SAP97, are scaffolding proteins for ionotropic glutamate receptors at excitatory synapses.

105 generation was blocked by OT antagonist and ionotropic glutamate receptor blockers or tetanus toxin.

106 einstated by 4-aminopyridine, and blocked by ionotropic glutamate receptor blockers.

107 ological diseases, is not mediated merely by ionotropic glutamate receptors but also by heteromeric T

108 al D1R/PKA/MEK1/2 pathway and independent of ionotropic glutamate receptors but blocked by antagonist

109 afficking is regulated by Ca2+ entry through ionotropic glutamate receptors, but the underlying mecha

110 Here, we design a light-controlled ionotropic glutamate receptor by genetically encoding a

111 We conclude that activation of ionotropic glutamate receptors by excitatory temporoammo

112 binding core (S1S2) of the GluR2 subtype of ionotropic glutamate receptors can be produced as a solu

113 The many divergent features of the different ionotropic glutamate receptor classes and different subu

114 Ionotropic glutamate receptors comprise two conformation

115 The N-methyl-d-aspartate (NMDA) subtype of ionotropic glutamate receptors comprises both NR1 and NR

116 rons after I(h) block required activation of ionotropic glutamate receptors; consistent with this was

117 The delta family of ionotropic glutamate receptors consists of glutamate del

118 -methyl-4-isoxazolepropionic acid subtype of ionotropic glutamate receptors consists of rapidly gatin

119 ow voltage-sensitive Ca channels (VSCCs) and ionotropic glutamate receptors contribute to Ca signals

120 Blockade of ionotropic glutamate receptors desynchronized onset of a

121 The overstimulated ionotropic glutamate receptors exert their neurotoxic ef

122 We used RT-PCR to profile ionotropic glutamate receptor expression in cultured SCs

123 The members of the ionotropic glutamate receptor family, namely, a-amino-3-

124 We focus here on the control of ionotropic glutamate receptor function by GPCR signaling

125 NMDA ionotropic glutamate receptors gate the cytoplasmic infl

126 ome abnormality disrupting the kainate class ionotropic glutamate receptor gene, GRIK4/KA1, in an ind

127 NMDA receptor subunit NR2B/Grin2B and other ionotropic glutamate receptor genes.

128 Ionotropic glutamate receptors (GluRs) are ligand-gated

129 Ionotropic glutamate receptors (GluRs) are ligand-gated

130 We investigated whether directly stimulating ionotropic glutamate receptors (GluRs) within the nucleu

131 Desensitization of ionotropic glutamate receptors (GluRs), specifically the

132 lence of somatic mutations within one of the ionotropic glutamate receptors, GRIN2A, in malignant mel

133 The kainate (KA) subtype of ionotropic glutamate receptor has been shown to potently

134 e structure of the agonist binding domain of ionotropic glutamate receptors has led to an improved un

135 the S1S2 ligand-binding domain of the GluR2 ionotropic glutamate receptor have been studied by NMR s

136 hypothesis of addiction and the role of the ionotropic glutamate receptors have been emphasized.

137 Structural and functional studies of ionotropic glutamate receptors have offered detailed ins

138 nnels, cyclic nucleotide-gated channels, and ionotropic glutamate receptor homologs.

139 ptic depression is modulated by postsynaptic ionotropic glutamate receptor (iGluR) activity.

140 Exposure to ionotropic glutamate receptor (iGluR) agonists, kainic a

141 ficial cerebrospinal fluid, or a cocktail of ionotropic glutamate receptor (iGluR) antagonists each b

142 near 0 mV or by recording in the presence of ionotropic glutamate receptor (iGluR) antagonists to iso

143 dria in astrocytic processes were blocked by ionotropic glutamate receptor (iGluR) antagonists, tetro

144 bitory currents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

145 Ionotropic glutamate receptor (iGluR) channels control s

146 anisms regulating the extent of postsynaptic ionotropic glutamate receptor (iGluR) clustering have be

147 reconfiguration of the subunits constituting ionotropic glutamate receptor (iGluR) complexes.

148 Ionotropic glutamate receptor (iGluR) desensitization ca

149 High-resolution studies of ionotropic glutamate receptor (iGluR) extracellular doma

150 Ionotropic glutamate receptor (iGluR) family members are

151 as seen a revolution in our understanding of ionotropic glutamate receptor (iGluR) structure, startin

152 The GluD1 and GluD2 receptors form the GluD ionotropic glutamate receptor (iGluR) subfamily.

153 postmortem study examined mRNA expression of ionotropic glutamate receptor (iGluR) subunits and PSD95

154 Loss of specific ionotropic glutamate receptor (iGluR) subunits during in

155 The amino terminal domain (ATD) of ionotropic glutamate receptor (iGluR) subunits resides a

156 Within ionotropic glutamate receptor (iGluR) subunits, this pro

157 lated GluD1 are classified as members of the ionotropic glutamate receptor (iGluR) superfamily on the

158 , the bilobate ligand-binding domains of the ionotropic glutamate receptors (iGluR) undergo a cleft c

159 determine the efficacy of PTL gating in the ionotropic glutamate receptor iGluR6 using a family of p

160 Ionotropic glutamate receptors (iGluRs) are glutamate-ga

161 Ionotropic glutamate receptors (iGluRs) are ligand-gated

162 Ionotropic glutamate receptors (iGluRs) are ligand-gated

163 Ionotropic glutamate receptors (iGluRs) are tetrameric c

164 Ionotropic glutamate receptors (iGluRs) are ubiquitous i

165 ation followed the arrival and clustering of ionotropic glutamate receptors (iGluRs) at NMJ synapses.

166 e protein synthesis critical for trafficking ionotropic glutamate receptors (iGluRs) at synapses.

167 generated an extensive sequence alignment of ionotropic glutamate receptors (iGluRs) from diverse ani

168 hores have revealed the presence of numerous ionotropic glutamate receptors (iGluRs) in Mnemiopsis le

169 Ionotropic glutamate receptors (iGluRs) mediate excitato

170 Ionotropic glutamate receptors (iGluRs) mediate fast exc

171 Ionotropic glutamate receptors (iGluRs) mediate most exc

172 Ionotropic glutamate receptors (iGluRs) mediate most exc

173 Ionotropic glutamate receptors (iGluRs) mediate neuronal

174 Ionotropic glutamate receptors (iGluRs) mediate neurotra

175 Ionotropic glutamate receptors (iGluRs) mediate the majo

176 Ionotropic glutamate receptors (iGluRs) mediate the majo

177 OL excitotoxicity is mediated by ionotropic glutamate receptors (iGluRs) of the alpha-ami

178 Ionotropic glutamate receptors (iGluRs) play important r

179 Presynaptic ionotropic glutamate receptors (iGluRs) play important r

180 is comb jelly encodes homologs of vertebrate ionotropic glutamate receptors (iGluRs) that are distant

181 NMDA receptors (NMDARs) are a class of ionotropic glutamate receptors (iGluRs) that are essenti

182 ing in the brain is mediated by postsynaptic ionotropic glutamate receptors (iGluRs) that are gated o

183 Kainate receptors (KARs) are a subfamily of ionotropic glutamate receptors (iGluRs) that mediate exc

184 A) receptors (AMPARs) are a major subtype of ionotropic glutamate receptors (iGluRs) that mediate rap

185 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors (iGluRs) that mediate the

186 Kainate receptors (KARs) are ionotropic glutamate receptors (iGluRs) that modulate sy

187 Ionotropic glutamate receptors (iGluRs) transduce the ch

188 Ionotropic glutamate receptors (iGluRs) underlie rapid,

189 lso attenuated by the blockade of the spinal ionotropic glutamate receptors (iGLURs) which was accomp

190 Ionotropic glutamate receptors (iGluRs), a family of lig

191 Ionotropic glutamate receptors (iGluRs), including the N

192 ory neurotransmission is mediated largely by ionotropic glutamate receptors (iGluRs), tetrameric, lig

193 americ ion channels that together with other ionotropic glutamate receptors (iGluRs), the NMDA and ka

194 Here, we studied ionotropic glutamate receptors (iGluRs), which are ion c

195 s excitatory synaptic transmission by gating ionotropic glutamate receptors (iGluRs).

196 oduces a robust learning that is mediated by ionotropic glutamate receptors (iGluRs).

197 citatory synaptic transmission by activating ionotropic glutamate receptors (iGluRs).

198 stem is mediated by glutamate acting through ionotropic glutamate receptors (iGluRs).

199 Glutamate-gated ion channels (ionotropic glutamate receptors, iGluRs) sense the extrac

200 Ionotropic glutamate receptors (IGR), including NMDA, AM

201 ed by activation of either NMDA or AMPA-type ionotropic glutamate receptors in a calcium-dependent ma

202 selective genetic inactivation of NMDA-type, ionotropic glutamate receptors in DA neurons.

203 s been designed to enable optical control of ionotropic glutamate receptors in neurons via sensitized

204 discovery of 20 genes encoding for putative ionotropic glutamate receptors in the Arabidopsis (Arabi

205 s strongly correlated with calcium-permeable ionotropic glutamate receptors in the neurons of the sen

206 Ionotropic glutamate receptors in the spinal cord are in

207 s of anti-BDNF were abolished by blockade of ionotropic glutamate receptors, indicating a role for gl

208 e effects were blocked by the antagonists of ionotropic glutamate receptors, indicating the involveme

209 d Ca(2+) imaging, we show that activation of ionotropic glutamate receptors induces a selective inter

210 that a long-term decrease in the activity of ionotropic glutamate receptors induces cholinergic activ

211 In the presence of ionotropic glutamate receptor inhibitors and tetrodotoxi

212 nfusion of kynurenic acid (an antagonist for ionotropic glutamate receptors) into core but not shell

213 trated that a member of the newly discovered ionotropic glutamate receptor (IR) family, IR76b, functi

214 he responsible sensory receptor (the variant ionotropic glutamate receptor IR75b) and attraction-medi

215 neurons express either odorant receptors or ionotropic glutamate receptors (IRs).

216 The N-methyl-d-aspartate (NMDA) subtype of ionotropic glutamate receptor is an important mediator o

217 The NMDA ionotropic glutamate receptor is ubiquitous in mammalian

218 vidence that a family of proteins related to ionotropic glutamate receptors is a previously unrecogni

219 Intracellular trafficking of ionotropic glutamate receptors is controlled by multiple

220 Whereas overactivation of ionotropic glutamate receptors is neurotoxic, the role o

221 Intracellular trafficking of ionotropic glutamate receptors is regulated predominantl

222 e N-methyl-d-aspartate (NMDA) subtype of the ionotropic glutamate receptors is the primary mediator o

223 Among the three classes of ionotropic glutamate receptors, kainate receptors (KARs)

224 The mechanism by which agonist binding to an ionotropic glutamate receptor leads to channel opening i

225 y SNAG-mGluR2 and excitatory light-activated ionotropic glutamate receptor LiGluR yielded a distribut

226 -shifted PTL, L-MAG0460, for the light-gated ionotropic glutamate receptor LiGluR.

227 excitatory mammalian ion channel light-gated ionotropic glutamate receptor (LiGluR) in retinal gangli

228 We show that the engineered light-gated ionotropic glutamate receptor (LiGluR), when introduced

229 and bradycardic responses via activation of ionotropic glutamate receptors located on secondary mNTS

230 Therefore, our results suggest that ionotropic glutamate receptors may have been conserved t

231 N-methyl-d-aspartate (NMDA)-type ionotropic glutamate receptors mediate excitatory neurot

232 5-methyl-4-isoxazole propionic acid)-subtype ionotropic glutamate receptors mediate fast excitatory n

233 AMPA subtype ionotropic glutamate receptors mediate fast excitatory n

234 Ionotropic glutamate receptors mediate fast excitatory s

235 Ionotropic glutamate receptors mediate fast synaptic tra

236 Ionotropic glutamate receptors mediate most excitatory n

237 AMPA-type ionotropic glutamate receptors mediate the majority of f

238 Ionotropic glutamate receptors mediate the majority of v

239 Ionotropic glutamate receptors mediate the majority of v

240 Ionotropic glutamate receptors mediate the majority of v

241 possibility that plant homologs of neuronal ionotropic glutamate receptors mediate these neuron-like

242 NMDA receptors (NMDAR), a type of ionotropic glutamate receptor, mediate synaptic plastici

243 isoxazole-4-propionic acid (AMPA) subtype of ionotropic glutamate receptors mediates much of the fast

244 cumulation leading to overstimulation of the ionotropic glutamate receptors mediates neuronal injury

245 Presynaptic ionotropic glutamate receptors modulate transmission at

246 The ionotropic glutamate receptors (N-methyl-D-aspartate rec

247 and memory and a reduction in the amounts of ionotropic glutamate receptors (NMDA and AMPA receptors)

248 the central network by glutamate binding to ionotropic glutamate receptors on second-order barorecep

249 distension (CRD), and the potential role of ionotropic glutamate receptors on sensitization.

250 n addition, the effect of antagonists to the ionotropic glutamate receptors on TAI was evaluated.

251 the activation of N-methyl-d-aspartate-type ionotropic glutamate receptors opposed increases in stri

252 ced by systemic injections of antagonists of ionotropic glutamate receptors or metabotropic glutamate

253 at central synapses depends on the number of ionotropic glutamate receptors, particularly the class g

254 Ionotropic glutamate receptors perform diverse functions

255 Some forms of ionotropic glutamate receptors permit passage of Ca++ io

256 ptors (IRs) are a large subfamily of variant ionotropic glutamate receptors present across Protostomi

257 Ionotropic glutamate receptors principally mediate fast

258 structural basis of allosteric inhibition in ionotropic glutamate receptors, providing key insights i

259 enhanced by DEX (10 microM), and blockade of ionotropic glutamate receptors reduced the DEX effect on

260 e suggests that surface trafficking of other ionotropic glutamate receptors requires ligand binding f

261 -4-isoxazole-propionate receptor (AMPAR) are ionotropic glutamate receptors responsible for excitator

262 ations in melanoma and the significance that ionotropic glutamate receptor signaling has in malignant

263 Electrophysiological testing at three ionotropic glutamate receptor subtypes reveals that two

264 onses, short-term dynamics and expression of ionotropic glutamate receptor subtypes.

265 thdrawal from cocaine self-administration on ionotropic glutamate receptor subunit (iGluRs) protein l

266 The distribution of ionotropic glutamate receptor subunit 4 (iGluR4) was exa

267 tations disrupt a previously uncharacterized ionotropic glutamate receptor subunit, named here "GluRI

268 Other ionotropic glutamate receptor subunits are expressed nor

269 ellular amino-terminal domains (ATDs) of the ionotropic glutamate receptor subunits form a semiautono

270 dings and showed expression of mRNA encoding ionotropic glutamate receptor subunits NR2A, NR2D, GluR4

271 y advance our physiological understanding of ionotropic glutamate receptor subunits, but is generally

272 a(v)beta(3) integrin-Lyn kinase complex with ionotropic glutamate receptor subunits, GluR2 and GluR4,

273 he expression of non-NMDA (AMPA and kainate) ionotropic glutamate receptor subunits.

274 viously observed for the AMPA subtype of the ionotropic glutamate receptors, suggesting a similar mec

275 ivo; and (3) constitutive desensitization of ionotropic glutamate receptors suppresses their ability

276 Here we report the discovery of an ionotropic glutamate receptor that combines the typical

277 xazoleproprionic acid receptor (AMPAR) is an ionotropic glutamate receptor that governs most of excit

278 N2A subunit of the NMDA receptor (NMDAR), an ionotropic glutamate receptor that has important roles i

279 We developed a K+-selective ionotropic glutamate receptor that reversibly inhibits n

280 Kainate receptors (KARs) are ionotropic glutamate receptors that also activate noncan

281 NMDA receptors (NMDARs) are ionotropic glutamate receptors that are crucial for neur

282 NMDA receptors (NMDARs) are a major class of ionotropic glutamate receptors that can undergo activity

283 sts of N-methyl D-aspartate (NMDA)-selective ionotropic glutamate receptors that contain the (E)-3-ph

284 NMDA receptors are ionotropic glutamate receptors that function as heterote

285 ptors (NMDARs) are a subtype of postsynaptic ionotropic glutamate receptors that function as molecula

286 N-methyl-D-aspartate-receptors (NMDARs) are ionotropic glutamate receptors that function in synaptic

287 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors that mediate a majority o

288 NMDA receptors are ionotropic glutamate receptors that mediate a slow, Ca2+

289 NMDA receptors are ionotropic glutamate receptors that mediate excitatory s

290 After suppressing this component by blocking ionotropic glutamate receptors, the entire a-wave up to

291 ction and its proposed role in AMPA and NMDA ionotropic glutamate receptor trafficking, we believe th

292 biophysical characteristics of postsynaptic ionotropic glutamate receptors via changes in subunit co

293 inate receptors (KARs) consist of a class of ionotropic glutamate receptors, which exert diverse pre-

294 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors, which mediate most excit

295 Correspondingly, AMPA-subtype ionotropic glutamate receptors, which mediate the majori

296 Ionotropic glutamate receptors, which underlie a majorit

297 king the N-methyl-d-aspartate (NMDA) type of ionotropic glutamate receptor with D-AP5 attenuated this

298 Kainate receptor subunits, which form ionotropic glutamate receptors with diverse roles in the

299 During apnea, induced by blockade of ionotropic glutamate receptors within the same region, m

300 The role of ionotropic glutamate receptors within the ventral pallid