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1             Rabbits were killed 5 days after iontophoresis.
2 f magnitude greater than that seen following iontophoresis.
3 en delivered across the skin with the aid of iontophoresis.
4 ty of concentrations delivered by controlled iontophoresis.
5 h duration was evaluated following histamine iontophoresis.
6 7 mug/g in epi-on, and 15.0 +/- 5.1 mug/g in iontophoresis.
7 olonged recovery of barrier disrupted due to iontophoresis.
8 ites of mature adult-born GCs following NMDA iontophoresis.
9 izable groups to render propofol amenable to iontophoresis.
10 e immediate vicinity of the neurons by micro-iontophoresis.
11 eness of activating presynaptic receptors by iontophoresis.
12 ock adrenergic function, was administered by iontophoresis.
13 ically, by local intradermal injection or by iontophoresis.
14 set was faster than could be reproduced with iontophoresis.
15 nts of human interstitial fluid extracted by iontophoresis.
16                               Application of iontophoresis (0.5mA/cm(2)) to SCD-propofol solution enh
17                               Prolonged AMPH iontophoresis (2-3 min; 5-30 nA) inhibited both spontane
18 crease in the average pain rating during ATP iontophoresis (22.7 +/- 3.1) compared with pre-capsaicin
19 (alpha) in vivo has been tetramethylammonium iontophoresis, a technically challenging method develope
20 des that had been transdermally delivered by iontophoresis across hairless mouse skin.
21 vidual cells with Lucifer Yellow dextran via iontophoresis and by analyzing patterns of fluorescence
22                                              Iontophoresis and pressure ejection were employed to loc
23 on, agonists and antagonists were applied by iontophoresis and single neurone activity was recorded e
24              To study this, we use glutamate iontophoresis and synaptic stimulation to position pairs
25 ng pain, which began within 20 s of starting iontophoresis and was maintained for several minutes.
26 f solvent into the skin, which is induced by iontophoresis and which contributes significantly to the
27 hat transscleral iontophoresis, transcorneal iontophoresis, and intravitreal injection deliver ions t
28 compare transscleral and transcorneal ocular iontophoresis, and monitor the distribution of a probe i
29                     Amethocaine, tetracaine, iontophoresis, and the S-caine patch, a product on the h
30                                           An iontophoresis approach was implemented to explore the ef
31 cular reactivity (laser Doppler imaging with iontophoresis), arterial stiffness (pulse wave velocity
32 cular reactivity (laser Doppler imaging with iontophoresis), arterial stiffness [pulse wave velocity,
33 hree types of stimuli (electrical, glutamate iontophoresis, bath-applied kainate) produced biphasic f
34 popular in the clinical setting (pilocarpine iontophoresis being a prominent exception).
35 ed to reactivate by transcorneal epinephrine iontophoresis, but others were not induced.
36 ntal fluorescent profiles to demonstrate how iontophoresis can generate near-uniform concentration di
37  carboplatin delivered by Coulomb-controlled iontophoresis (CCI; 14 mg/mL carboplatin, 5.0 mA/cm(2),
38 rget sites of ion delivery in the eye during iontophoresis, compare transscleral and transcorneal ocu
39 , but this effect can easily be caused by an iontophoresis current artifact.
40 nstant current transscleral and transcorneal iontophoresis (current density, 10 mA/cm(2)) was perform
41 ered the ion into the vitreous, transcorneal iontophoresis delivered the ion into the anterior chambe
42                         Whereas transscleral iontophoresis delivered the ion into the vitreous, trans
43            T-ionto CL was performed using an iontophoresis device with dextran-free 0.1% riboflavin-5
44         Patients were randomized into 1 of 4 iontophoresis dose groups (1.6, 4.8, 10.0, or 14.0 mA-mi
45                                          ACh iontophoresis either excited or inhibited striatal and a
46 hloride concentrations are unperturbed, GABA iontophoresis elicits spikes and increases excitability
47                Transdermal administration by iontophoresis (enhanced transport via the skin using the
48                Transscleral and transcorneal iontophoresis experiments were conducted with the electr
49 ate for the first time power generation from iontophoresis extracts obtained from pig skin.
50  acetylcholine (ACh) (endothelium dependent) iontophoresis, flicker-light-induced retinal vasodilatat
51 ntly delivered into and across the sclera by iontophoresis for drug delivery.
52 up was about 2-fold greater than that of the iontophoresis group (50.5 +/- 5.3 mug/g and 23.6 +/- 2.5
53 al enhancers, noncavitational ultrasound and iontophoresis have also resulted in clinical products; t
54                  While pressure ejection and iontophoresis have been used to deliver picoliter volume
55        Corneal cross-linking transepithelial iontophoresis imbibition yielded greater and deeper ribo
56  laser-Doppler flowmetry (LDF) combined with iontophoresis in 15 low-flow POTS patients, 17 normal-fl
57 ed extracellular single-unit recordings with iontophoresis in awake animals.
58 ity, single-unit recording was combined with iontophoresis in awake, unrestrained rats.
59  for sustained drug delivery in transscleral iontophoresis in vitro.
60 ned electrophysiologically and marked by dye iontophoresis in vivo.
61 -437 (dexamethasone phosphate formulated for iontophoresis) in patients with noninfectious anterior u
62                                           NE iontophoresis inhibited spontaneous firing and decreased
63                  A wearable and miniaturized iontophoresis interface is an excellent solution to over
64         The challenge remains in devising an iontophoresis interface that can extract sufficient amou
65 ough realizing an electrochemically enhanced iontophoresis interface, integrated in a wearable sweat
66  ability to reactivate following epinephrine iontophoresis into the cornea.
67                                              Iontophoresis is now established as one of the methods o
68          With this information, quantitative iontophoresis is possible for electrochemically inactive
69 ximum increase in calcein fluorescence after iontophoresis is proportional to the aqueous volume into
70                                              Iontophoresis is the movement of charged molecules in so
71 irment using microneedles (MNs) coupled with iontophoresis (ITP) may broaden the range of drugs suita
72 low-mediated dilatation (FMD), laser Doppler iontophoresis (LDI), and digital volume pulse (DVP).
73 nanoparticulates, liposomes, or transscleral iontophoresis may provide the solution.
74 t-source paradigm, realized in the real-time iontophoresis method using tetramethylammonium, as well
75 onditions, we used the established real-time iontophoresis method with tetramethylammonium (TMA(+)) t
76 bition with epi-on (n = 3), epi-off (n = 3), iontophoresis (n = 3), and saline exposure (control; n =
77 erm systemic exposure to dexamethasone after iontophoresis; no nonocular systemic corticosteroid-medi
78 odilation in the forearm skin in response to iontophoresis of 1% acetylcholine chloride (Ach) (endoth
79  putative pyramidal cells were attenuated by iontophoresis of 5-HT2A antagonists, which primarily pro
80 ecording of reflectance properties following iontophoresis of a potent vasoconstrictor, noradrenaline
81 es, measured using laser Doppler imaging, to iontophoresis of acetylcholine (ACh) and sodium nitropru
82 n the dorsal foot in response to heating and iontophoresis of acetylcholine (endothelium-dependent) a
83 esponses in the fingers were assessed during iontophoresis of acetylcholine and sodium nitroprusside,
84 ase in skin blood flow induced following the iontophoresis of ACh and to investigate possible contrib
85 ood cell flux (P < 0.001) in response to the iontophoresis of ACh minus the response to ACh vehicle w
86 kin perfusion (P < 0.001) in response to the iontophoresis of ACh minus the response to ACh vehicle w
87  in perfusion (P < 0.001) in response to the iontophoresis of ACh vehicle was significantly inhibited
88                                  Conversely, iontophoresis of acid over a wide range of skin temperat
89  significantly inhibited the pain induced by iontophoresis of acid, suggesting an involvement of acid
90                                              Iontophoresis of ACPD had different effects in different
91 hermal burn, and the pain ratings during the iontophoresis of adenosine triphosphate (ATP) and acid.
92                                              Iontophoresis of alpha4beta2-nAChR agonists also enhance
93                                              Iontophoresis of alpha4beta2-nAChR agonists increased th
94 activity in cortical layer 2 was silenced by iontophoresis of AMPA and NMDA receptor antagonists.
95            We compared the sweat response to iontophoresis of an alpha-adrenergic agent (phenylephrin
96                                              Iontophoresis of ATP caused an increase in blood flow, a
97 ation of presynaptic purinergic receptors by iontophoresis of ATP decreased evoked IPSC amplitudes an
98                                   Persistent iontophoresis of ATP led to desensitization within 12 mi
99                                              Iontophoresis of ATP produced a higher pain rating than
100   Interictal spikes (IISs) were induced with iontophoresis of bicuculline methiodide in rat neocortex
101 e modulation (IPM) before, during, and after iontophoresis of bicuculline, an antagonist to the inhib
102                                              Iontophoresis of Ca2+ at various membrane potentials all
103                                    Following iontophoresis of CtB into the PVN, retrogradely labeled
104 ter infection, rabbits received transcorneal iontophoresis of epinephrine.
105 and spontaneous firing could be inhibited by iontophoresis of ergometrine (9/10 cells), sumatriptan (
106 ning retrogradely labeled cells following LC iontophoresis of Fluoro-Gold and anterograde LC labeling
107 puts, single-unit recording was coupled with iontophoresis of GLU and GABA in rats under two conditio
108                                              Iontophoresis of glutamate NMDA receptor agonists increa
109 tivity also decreases the response to direct iontophoresis of glutamate onto postsynaptic receptors.
110 lectrodes combined with pressure ejection or iontophoresis of ions from a micropipette to quantify di
111 ny change in intracellular calcium following iontophoresis of l-aspartate or two-photon uncaging of g
112                                              Iontophoresis of l-aspartate, an NMDAR agonist, onto bas
113                                              Iontophoresis of L-glutamate or vasopressin into the vic
114                            In ambulant rats, iontophoresis of low concentrations of dopamine (DA) enh
115 ses to the chemoreflex excitation and direct iontophoresis of N-methyl-D-aspartic acid (NMDA) but wit
116 arbors in layer IV of area 17 were filled by iontophoresis of Phaseolus lectin into lamina A of the l
117  (cRCF) was recorded in dorsal finger during iontophoresis of phenylephrine (PE) or clonidine (0.5 mm
118 logical methods and was visibly marked using iontophoresis of pontamine sky blue dye.
119                    We found that transdermal iontophoresis of protons consistently caused moderate pa
120                   We used a novel technique, iontophoresis of protons, to investigate acid-induced pa
121 essed this issue by examining the effects of iontophoresis of selective receptor ligands on prefronta
122  in perfusion (P < 0.001) in response to the iontophoresis of SNP vehicle was significantly inhibited
123                                 In addition, iontophoresis of the alpha-2 agonist clonidine, footshoc
124 archistriatum were therefore investigated by iontophoresis of the anterograde tracer Phaseolus vulgar
125 on-specific effect of the drug, since (1) co-iontophoresis of the D2 antagonist YM 09151-2 antagonize
126 ropic glutamate agonist ACPD was examined by iontophoresis of the drugs close to the cell body.
127              Inactivating a single barrel by iontophoresis of the GABA(A) agonist muscimol abolished
128              Reactivating a single barrel by iontophoresis of the GABA(A) antagonist bicuculline whil
129                                              Iontophoresis of the GABAA agonist muscimol (MUS) into t
130 nd LC stimulation mimicked the effects of NE iontophoresis on spontaneous activity.
131 ts demonstrate the importance of the reverse iontophoresis operation and validate the sensor specific
132                         Likewise, repetitive iontophoresis (or puffs) of glutamate (or AMPA) onto the
133 r, and sweating in response to acetylcholine iontophoresis over the calf and forearm.
134  ion in the aqueous and vitreous humor after iontophoresis, passive delivery, and intravitreal inject
135               Surprisingly, the elevated dye iontophoresis persists in shaking B mutants that elimina
136 ly unexpected and highly robust neuronal dye iontophoresis phenotype in the well mapped giant fiber (
137                                          The iontophoresis process involves delivery of stimulating a
138                                              Iontophoresis produced no meaningful increase in deliver
139 or agonist, applied by either superfusion or iontophoresis, produced an atropine-sensitive increase o
140 e during and after iontophoresis, such as in iontophoresis protocol and device testing.
141                With the present transscleral iontophoresis protocol, the ion penetrated the sclera an
142 smic and occurs due to a highly elevated dye iontophoresis rate.
143 nvestigation indicates that the tattoo-based iontophoresis-sensor platform holds considerable promise
144             Understanding how FMRP regulates iontophoresis should reveal new molecular factors underp
145                              Five hours post-iontophoresis, sites were irradiated with broad-band yel
146 ion was measured before and during carbachol iontophoresis stimulated accommodation, a total of seven
147  probe compounds in the eye during and after iontophoresis, such as in iontophoresis protocol and dev
148  transepithelial corneal cross-linking using iontophoresis (T-ionto CL) and standard corneal cross-li
149                                       During iontophoresis, the current delivered into the eye was mo
150 resolving perhaps the greatest limitation of iontophoresis, the inability to determine the concentrat
151                             For transcorneal iontophoresis, the ion penetrated the cornea and filled
152 lutinin and cholera toxin B were injected by iontophoresis through a single glass micropipette and de
153 esulted in clinical products; the ability of iontophoresis to control delivery rates in real time pro
154                            Here we have used iontophoresis to deliver ATP to the forearm skin of volu
155 investigational PCA transdermal system using iontophoresis to deliver fentanyl provided postsurgical
156   We used single-unit recording coupled with iontophoresis to examine impulse activity and responses
157 lied to a site pretreated with bretylium via iontophoresis to inhibit noradrenergic neurotransmission
158      Single-unit recording was combined with iontophoresis to monitor basal impulse activity of dorsa
159     With MRI, it was shown that transscleral iontophoresis, transcorneal iontophoresis, and intravitr
160  Forty of 42 randomized patients received an iontophoresis treatment in 1 qualifying eye and complete
161  zero within 28 days, after only receiving 1 iontophoresis treatment.
162                                              Iontophoresis uses a current to eject solution from the
163                                              Iontophoresis uses electricity to deliver solutes into l
164  To address this, a method called controlled iontophoresis was developed which employs a carbon-fiber
165                         Afterwards histamine iontophoresis was performed and skin blood flow and itch
166 cy was established, transcorneal epinephrine iontophoresis was performed.
167                               Moreover, NMDA iontophoresis was sufficient to induce the formation of
168                                        Using iontophoresis, we demonstrated that ET-1 is a potent, pa
169                                        Using iontophoresis, we developed a rapid and quantifiable sys
170                  The current pathways during iontophoresis were mainly from the electrode into the ey
171 -200 ms) activation of mGluRs with aspartate iontophoresis were suppressed by superfusion of agonists
172 es above the pore opening is observed during iontophoresis, which is a consequence of the separation

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