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1 with light interstripes of xanthophores and iridophores.
2 d those cells instruct the patterning of the iridophores.
3 foxd3, a mitfa repressor, resulted in fewer iridophores.
4 ophores, yellow xanthophores, and iridescent iridophores.
5 ophores, yellow xanthophores, and iridescent iridophores.
6 melanocytes, yellow xanthophores and silver iridophores.
7 ut development but have increased numbers of iridophores.
8 region shows specific enhancer activities in iridophores, a type of pigment cells found in egg-spots,
11 consisting of alternating dark (melanophore, iridophore) and light (xanthophore, iridophore) horizont
12 ring embryogenesis, and cells of melanocyte, iridophore, and xanthophore lineages all express this ge
16 ophores, yellow xanthophores, and shimmering iridophores arranged in a characteristic pattern of gold
18 xd3 expression in xanthophore precursors and iridophores, but not in terminally differentiated melano
19 vidence for a model whereby melanophores and iridophores descend from a common precursor whose fate i
22 ocalized with pnp4a, a novel marker of early iridophore development, and was necessary for its expres
24 e Ltk in a tissue-specific context to induce iridophore differentiation from neural crest-derived cel
25 to develop normal numbers of melanocytes and iridophores during pigment pattern metamorphosis and exh
28 bit simple stripes of melanocytes and silver iridophores in a background of yellow xanthophores, this
29 including epidermis, melanocyte/xanthophore, iridophore, intraray glia, lateral line, osteoblast, der
32 and aug-beta results in strong impairment in iridophore patterning of embryonic and adult zebrafish t
34 igmentation defects in which xanthophore and iridophore pigmentation is almost completely absent, and
36 ett (sbr) mutants exhibit delayed changes in iridophore shape and organisation caused by truncations
37 , comprising a series of dark melanocyte and iridophore stripes, alternating with light stripes of ir
38 in xanthophores and melanophores, but not in iridophores, suggest that both connexins form heteromeri
40 ed only by a subset of adult melanocytes and iridophores, these analyses also reveal variation among
41 velopment, resulted in supernumerary ectopic iridophores while loss of foxd3, a mitfa repressor, resu
42 tripe pattern is a complex tissue containing iridophores, xanthophores and multiple melanocyte types.
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