ライフサイエンスコーパス: iron supplementation

1 on the change in iron status in response to iron supplementation.

2 sed as a safe and easily available source of iron supplementation.

3 All patients received iron supplementation.

4 nt iron uptake, since it can be corrected by iron supplementation.

5 but who suffer from fatigue may benefit from iron supplementation.

6 evere growth defect, which can be rescued by iron supplementation.

7 results, and have not accounted for maternal iron supplementation.

8 hemoglobin concentrations were improved with iron supplementation.

9 e risk factors under different conditions of iron supplementation.

10 Otherwise, predictors varied by iron supplementation.

11 a severe growth defect corrected by in vivo iron supplementation.

12 This impairment can be corrected with iron supplementation.

13 absorption had decreased significantly with iron supplementation.

14 ed women and that this can be corrected with iron supplementation.

15 eme-iron absorption from food in response to iron supplementation.

16 rapy is an accepted and convenient method of iron supplementation.

17 and should be complemented with school-based iron supplementation.

18 and, as such, are routinely recommended for iron supplementation.

19 factors when formulating recommendations on iron supplementation.

20 lacental transfer and impair the efficacy of iron supplementation.

21 prevention, including food fortification and iron supplementation.

22 a loss of appetite that can be restored with iron supplementation.

23 s by region and worsens in pregnancy without iron supplementation.

24 dly elevated growth in serum collected after iron supplementation.

25 tions that result from long-term intravenous iron supplementation.

26 intervention groups.LBW children who receive iron supplementation (1 or 2 mg Fe . d(-1)) in infancy h

27 9 days (n = 112) and 84 days (n = 115) after iron supplementation (60 mg iron as ferrous fumarate dai

28 ay) rats and in both groups after daily high-iron supplementation (8,000 microg/day) for 34 days.

29 In contrast, in cells grown with iron supplementation, a PerR-repressed gene is completel

30 ract agar with a reduced amount of its usual iron supplementation, a phenotype that could be compleme

31 f these studies was to determine how dietary iron supplementation affected the severity of allergic i

32 Iron supplementation after cecal ligation and puncture i

33 Iron supplementation after sham laparotomy did not cause

34 d systemic bacterial counts in animals given iron supplementation after the onset of sepsis.

35 Question: What are the benefits and harms of iron supplementation alone and as an adjunct to erythrop

36 We determined whether n-3 LCPUFAs and iron supplementation, alone or in combination, affected

37 The patients who received iron supplementation also had a more rapid return of iro

38 Prenatal iron supplementation among iron-replete, nonanemic women

39 can be used to predict effects of trials of iron supplementation and fortification and to design iro

40 alth problem despite decades of efforts with iron supplementation and fortification.

41 ematologic response to supervised, long-term iron supplementation and the relation of this response t

42 n labile iron pools within living cells with iron supplementation and/or depletion.

43 ew of randomized controlled trials (RCTs) of iron-supplementation and -fortification trials that asse

44 he risk of GDM associated with dietary iron, iron supplementation, and iron status as measured by blo

45 diets, caloric supplementation, calcium and iron supplementation, and various other vitamin and mine

46 aining micronutrient powders (MNPs) and oral iron supplementation are both effective strategies to in

47 New indications of intravenous iron supplementation are emerging.

48 The benefits of iron supplementation are likely to outweigh possible ris

49 Current programs of universal iron supplementation are unlikely to have much effect on

50 reated iron deficiency, as well as excessive-iron supplementation, are deleterious and emphasize the

51 meostasis with the response to and risk from iron supplementation as well as the need for indicators

52 Iron supplementation attenuated the decrement in iron st

53 Iron status is affected by BCT, and iron supplementation attenuates the decrement in indicat

54 rld Health Organization recommendations that iron supplementation be given in combination with malari

55 al damage and cell death can be prevented by iron supplementation, but cannot be fully blocked by a p

56 Adverse effects of intravenous iron supplementation by ferric carboxymaltose seem to be

57 Taken together, these studies show that iron supplementation can decrease the severity of allerg

58 Iron supplementation caused modest augmentation of iron

59 ngleton pregnancies, administration of daily iron supplementation, compared with administration of pl

60 deficient rats and rats receiving daily high-iron supplementation, compared with iron-normal rats (P

61 nors with normal hemoglobin levels, low-dose iron supplementation, compared with no supplementation,

62 We wanted to determine whether iron supplementation could prevent decrements in iron st

63 In vitro, iron supplementation decreased mast cell granule content

64 on depletion of culture medium enhanced, and iron supplementation decreased, the efficiency of infect

65 Maternal iron supplementation did not affect infant iron status a

66 In most of the studies, iron supplementation did not affect the biochemical stat

67 ive risk [RR], 1.03; 95% CI, 0.65-1.65), and iron supplementation did not significantly affect birth

68 Iron supplementation directly restores PHD catalytic act

69 s a sign of adequate iron nutrition, because iron supplementation does not increase hemoglobin higher

70 children in each group who received 28 d of iron supplementation during antimalarial treatment with

71 status, indicators of iron sufficiency, and iron supplementation during inflammation and how it may

72 There is limited evidence on the safety of iron supplementation during pregnancy in these areas.

73 The effect of iron supplementation during pregnancy may depend on init

74 In one randomized trial, iron supplementation during pregnancy reduced child mort

75 vidence supports the advisability of routine iron supplementation during pregnancy.

76 stfed infants and tested the hypothesis that iron supplementation enhances iron status.

77 +MMN, and placebo groups, respectively.Daily iron supplementation for 12 wk increased hemoglobin in n

78 All children received iron supplementation for 6 weeks; children in the interv

79 We hypothesized that iron supplementation for 6 wk would significantly improv

80 ve malaria control can mitigate the risks of iron supplementation for children in areas of malaria tr

81 Furthermore, iron supplementation for iron deficient children in mala

82 d insufficient evidence to recommend routine iron supplementation for pregnant women or routine scree

83 the balance of benefits and harms of routine iron supplementation for pregnant women to prevent adver

84 da and the United States recommend universal iron supplementation for pregnant women.

85 On the basis of 11 trials, routine maternal iron supplementation had inconsistent effects on rates o

86 Prenatal iron supplementation had no effects on infant iron statu

87 ive iron supplementation, those who received iron supplementation had shortened time to 80% hemoglobi

88 ormone concentrations, patients who received iron supplementation had significantly higher circulatin

89 Iron supplementation has been associated with greater su

90 The practice of intravenous iron supplementation has grown as nephrologists have gra

91 ctional iron deficiency, but its response to iron supplementation has not been investigated in margin

92 nantly due to iron deficiency, but antenatal iron supplementation has uncertain health benefits and c

93 ll as alternative therapies, including diet, iron supplementation, herbal medications, and neurofeedb

94 o control IDA include daily and intermittent iron supplementation, home fortification with micronutri

95 otransfusion, duration of hospital stay, and iron supplementation; however, differences between the g

96 n children with poor iron and n-3 FA status, iron supplementation improved verbal and nonverbal learn

97 non-anaemic iron-deficient adolescent girls, iron supplementation improved verbal learning and memory

98 Among women, iron supplementation improves physical and cognitive per

99 e on hematologic responses and malaria after iron supplementation in anemic (hemoglobin: 70-109 g/L)

100 Iron supplementation in anemic HIV-infected children has

101 Although the benefits of iron supplementation in anemic women are well recognized

102 The long-term benefits of antenatal iron supplementation in child survival are not known.

103 and neonatal benefits and harms of universal iron supplementation in developed countries as evidenced

104 bal health applications such as guiding safe iron supplementation in developing countries with high i

105 The value of prophylactic iron supplementation in elderly blood donors was evaluat

106 ficacy of inexpensive readily available oral iron supplementation in heart failure is unknown.

107 multicenter trials exploring the use of oral iron supplementation in heart failure, a therapy that is

108 Thus, iron supplementation in HIV-infected children living in

109 It is unknown whether iron supplementation in human immunodeficiency virus (HI

110 ifies the assessment of the effectiveness of iron supplementation in improving conventional iron stat

111 aining reduces the apparent effectiveness of iron supplementation in improving sFer and calls into qu

112 Multivariate logistic regression showed that iron supplementation in infancy reduced the odds of havi

113 he mean SBP in LBW children who had received iron supplementation in infancy was 2.2 mm Hg (95% CI: 0

114 termine whether there are adverse effects of iron supplementation in iron-replete women.

115 Calculations of body iron in trials of iron supplementation in Jamaica and iron fortification i

116 re protective against severe malaria, whilst iron supplementation in malaria endemic regions is with

117 have raised concerns regarding the safety of iron supplementation in malaria-endemic regions.

118 Although iron supplementation in malaria-free areas mostly reduce

119 These results do not support use of oral iron supplementation in patients with HFrEF.

120 ITDI represents a more efficacious method of iron supplementation in PD patients receiving rhEPO.

121 Iron supplementation in preventive programs may need to

122 The safety of routine iron supplementation in settings where infectious diseas

123 eliminate the inhibition of growth caused by iron supplementation in the absence of Mn2+.

124 the superiority of parenteral iron over oral iron supplementation in the treatment of chemotherapy-in

125 the hemoglobin S phenotype of the effects of iron supplementation in the treatment of mild anemia.

126 omized controlled trials of preventive, oral iron supplementation in young children (aged 0-59 mo) li

127 Iron supplementation increased ferritin from 11 +/- 14 m

128 Iron supplementation increased hepatic iron and serum he

129 Iron supplementation increased morbidity (mostly respira

130 Iron supplementation increased the activities of several

131 Thus iron supplementation increased the aerobic growth rate,

132 Iron supplementation increased the number of days with i

133 Iron supplementation increased the serum ferritin concen

134 While iron supplementation increases maternal iron status and

135 In cultured macrophages, iron supplementation induced reactive oxygen species and

136 ciency is a common cause of maternal anemia, iron supplementation is a common practice to reduce the

137 Dietary iron supplementation is associated with increased appeti

138 Where both conditions are prevalent, iron supplementation is complicated by observations that

139 women with depleted iron reserves, prenatal iron supplementation is important for meeting iron requi

140 e, but the risk of infection associated with iron supplementation is not well defined.

141 Oral iron supplementation is often associated with rapid onse

142 ntries, incomplete resolution of anemia with iron supplementation is often attributed to poor complia

143 Iron supplementation is recommended in populations in wh

144 g cause of anemia in sub-Saharan Africa, and iron supplementation is the standard of care during preg

145 C-reactive protein level and with oral or IV iron supplementation; it also reduced serum hepcidin lev

146 d-release preparations and intermittent oral iron supplementation lead to better overall compliance a

147 Iron supplementation led to increased birth weight.

148 Additional screening and iron supplementation may be warranted in this high-risk

149 ed by physiologic iron status, and therefore iron supplementation may have adverse effects even among

150 Consequently, while iron supplementation may improve pregnancy outcome when

151 Iron supplementation may increase malaria morbidity and

152 Previous evidence has shown that iron supplementation may increase malaria risk.

153 Furthermore, iron supplementation may prove to be beneficial for mood

154 These results suggest that maintenance iron supplementation may result in fewer infections than

155 Maternal hemoglobin (n = 1255), iron supplementation (n = 7484), food-based iron intake

156 Based on these data, routine IV iron supplementation of anemic, critically ill trauma pa

157 The study assessed the effect of early iron supplementation of breastfed infants and tested the

158 Early iron supplementation of breastfed infants is feasible an

159 merozoite preference for young erythrocytes, iron supplementation of iron-deficient individuals rever

160 e suggesting adverse effects associated with iron supplementation of iron-replete individuals.

161 s, and 4) the balance of benefit and harm of iron supplementation of iron-replete pregnant women and

162 rkshop related to iron screening and routine iron supplementation of largely iron-replete pregnant wo

163 Iron supplementation of the diet resulted in lower level

164 vary with the life stage and especially with iron supplementation of the diet.

165 Iron supplementation of the medium promoted translation

166 the absence of iron and grew poorly without iron supplementation of the medium, phenotypes consisten

167 Iron supplementation of these iron-depleted, nonanemic w

168 We investigated the effects of iron supplementation on adaptation to aerobic training a

169 We also reviewed effects of iron supplementation on birth outcomes among women at lo

170 olled clinical trial assessed the effects of iron supplementation on cognitive function in adolescent

171 On day 10, no significant effect of iron supplementation on DMT1 and FPN1 gene expression or

172 rial was conducted to examine the effects of iron supplementation on hemoglobin, HIV disease progress

173 is study was to assess the effect of dietary iron supplementation on insulin resistance and the role

174 Effects of prenatal iron supplementation on maternal postpartum iron status

175 to be learned about the benefits of maternal iron supplementation on the health and iron status of th

176 Variable effects of iron supplementation on the susceptibility to mycobacter

177 pression of DMT1 and FPN1 and the effects of iron supplementation on their expression and on iron abs

178 he underlying disease and not related to the iron supplementation only.

179 xchangeable iron stores in living cells upon iron supplementation or depletion, including labile iron

180 randomly assigned at age 6 mo to high or low iron supplementation or no added iron.

181 mpact of the current North American prenatal iron supplementation policy, this review highlights the

182 s also unknown, but it could be relevant for iron supplementation programs aimed at combating anemia.

183 cost point-of-care hepcidin assays would aid iron supplementation programs in the developing world.

184 y affects mother-child interactions and that iron supplementation protects against these negative eff

185 A therapeutic approach to iron supplementation, rather than a public health-based

186 of BCG-infected mice revealed that moderate iron supplementation reduced inflammation, as measured b

187 cental malaria was not increased by maternal iron supplementation (relative risk [RR], 1.03; 95% CI,

188 ate aerobic exercise on the effectiveness of iron supplementation remains unclear.This study aimed to

189 uld not grow in tissue culture media without iron supplementation replicated more rapidly within epit

190 Iron supplementation resulted in improved (P < 0.05) vig

191 cellular level of mitochondrial aconitase by iron supplementation resulted in increased RNA-binding a

192 Iron supplementation reversed this growth defect and was

193 our findings imply that in malarious regions iron supplementation should be accompanied by effective

194 Iron supplementation should be administered intravenousl

195 Iron supplementation significantly decreased P(FIT2)-GFP

196 Iron supplementation significantly decreased the risk of

197 Random-effects meta-analyses showed that iron supplementation significantly improved iron status

198 Six weeks of iron supplementation significantly improved serum ferrit

199 Compared with placebo, iron supplementation significantly improved the mean inc

200 We previously showed that iron supplementation significantly improves iron status

201 Iron supplementation significantly reduced the risk of m

202 Iron supplementation starting at an early age may preven

203 Iron supplementation strategies in the developing world

204 eans, is a novel and natural alternative for iron supplementation strategies where effectiveness is l

205 Data from pregnant women enrolled in the Iron Supplementation Study (Raleigh, North Carolina, 199

206 iency or iron deficiency anemia according to iron supplementation suggest that direct comparisons acr

207 was slightly but significantly higher after iron supplementation than after placebo (difference = 13

208 l adaptation, iron stores were greater after iron supplementation than after placebo and this differe

209 later in life.We investigated the effect of iron supplementation that was given to LBW infants on mi

210 uring the past year covers three main areas: iron supplementation, the regulation of iron absorption,

211 mpared with participants who did not receive iron supplementation, those who received iron supplement

212 dy, we used a rat model of long term dietary iron supplementation to identify stellate cell genes tha

213 are indicated to determine the potential of iron supplementation to modulate the clinical severity o

214 Iron supplementation was associated with greater increas

215 The combination of IPT and iron supplementation was most effective in the treatment

216 Iron supplementation was not associated with offspring B

217 With iron supplementation, weight gains were adversely affect

218 ansporters or iron absorption in response to iron supplementation, whereas down-regulation occurs dur

219 serious adverse events were associated with iron supplementation, whereas, in Nepal, no effects on m

220 Evidence is convincing that maternal iron supplementation will improve birth weight and perha

221 If true, iron supplementation will not be an effective anemia red

222 High-dose iron supplementation with iron dextran after the onset o

223 terminants compared to their expression with iron supplementation, yet the quantity of biofilm was no

224 IDA patients can be treated with iron supplementation, yet TT patients have diminished ca