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1 eople worldwide whose inadequate diet causes iron deficiency anemia.
2 orms causing gastrointestinal blood loss and iron deficiency anemia.
3 sociated with a 40% reduction in the risk of iron deficiency anemia.
4 firm the benefits of successful treatment of iron deficiency anemia.
5 ing cause of gastrointestinal hemorrhage and iron deficiency anemia.
6 tissues, while they continue to have severe iron deficiency anemia.
7 atodes cause gastrointestinal hemorrhage and iron deficiency anemia.
8 an females and significantly associated with iron deficiency anemia.
9 <20 y and education were not associated with iron deficiency anemia.
10 for HFE mutations had a lower prevalence of iron deficiency anemia.
11 (20.5%) were iron deficient; 24 of these had iron deficiency anemia.
12 240,000 toddlers and 3.3 million women have iron deficiency anemia.
13 e to be a safe and efficacious treatment for iron deficiency anemia.
14 PRSS6, whose mutations cause iron refractory iron deficiency anemia.
15 t challenge the entrenched oral treatment of iron deficiency anemia.
16 he United States who do not have symptoms of iron deficiency anemia.
17 l disorders had been treated for concomitant iron deficiency anemia.
18 nmol/L; P<0.05) and low in one patient with iron deficiency anemia.
19 n and appeared to be effective in correcting iron deficiency anemia.
20 s and underlies erythropoietic repression in iron deficiency anemia.
21 His blood count shows signs of mild iron deficiency anemia.
22 12 months of age in infants at high risk for iron deficiency anemia.
23 Hfe Tg mice exhibited hepcidin excess and iron deficiency anemia.
24 d to elevated hepcidin levels and consequent iron deficiency anemia.
25 in training regimens with iron deficiency or iron deficiency anemia.
26 addition of abnormal hemoglobin to classify iron deficiency anemia.
27 y in children, including iron deficiency and iron deficiency anemia.
28 < 0.05) in volunteers reporting to BCT with iron deficiency anemia.
29 ted patients with unexplained, mild, chronic iron-deficiency anemia.
30 following: osteoporosis or low bone mass or iron-deficiency anemia.
31 be considered for children with nutritional iron-deficiency anemia.
32 either patients with rheumatoid arthritis or iron-deficiency anemia.
33 reditary hemochromatosis and iron-refractory iron-deficiency anemia.
34 ally important biomarker in diseases such as iron-deficiency anemia.
35 distinguishes thalassemia-trait anemia from iron-deficiency anemia.
36 mia, 6 with secondary erythrocytosis, 2 with iron-deficiency anemia, 4 with hemochromatosis, or 5 nor
37 practical improvements in the management of iron deficiency anemia, a disorder that may affect as ma
38 riations in predictors of iron deficiency or iron deficiency anemia according to iron supplementation
39 gh-intensity infection had a greater risk of iron deficiency anemia (adjusted prevalence odds ratio:
42 need for successful interventions to reduce iron deficiency anemia among food-insecure children and
43 ecause of the tendency of infants to develop iron deficiency anemia and because of the documented adv
44 objective was to estimate the prevalence of iron deficiency anemia and examine potential reasons for
45 n models were used to identify predictors of iron deficiency anemia and iron deficiency without anemi
46 estigation of the potential contributions of iron deficiency anemia and iron treatment to this differ
50 nical study compared 96 female patients with iron-deficiency anemia and 60 healthy female control sub
51 or further evaluation, from which those with iron-deficiency anemia and active bleeding had been excl
52 gastrointestinal blood loss, and unexplained iron-deficiency anemia), and the performance of selected
54 Twenty-five percent of all anemia, 35% of iron deficiency anemia, and 73% of severe anemia were at
55 ity of iron fortification of food to improve iron deficiency, anemia, and biological outcomes is not
57 ong-standing type II diabetes, hypertension, iron deficiency anemia, aortic stenosis, and prior baria
62 a defect in iron absorption that results in iron-deficiency anemia, as revealed by an N-ethyl-N-nitr
63 nt deficiency among children worldwide, with iron-deficiency anemia associated with long-term adverse
66 h outcomes that are associated with maternal iron deficiency anemia, both Canada and the United State
68 RR, 0.48; 95% CI, 0.32-0.70) and the risk of iron deficiency anemia by 66% (RR, 0.34; 95% CI, 0.19-0.
69 e anemia of chronic disease, iron refractory iron deficiency anemia, cancer, hereditary hemochromatos
71 R: 0.6; CI: 0.4, 0.7), but the prevalence of iron deficiency anemia did not change significantly in c
72 (mk) mice and Belgrade (b) rats have severe iron deficiency anemia due to defects in intestinal iron
75 ns were undetectable or low in patients with iron deficiency anemia (ferritin < 10 ng/mL), iron-deple
76 hildren aged 9 to 48 months with nutritional iron-deficiency anemia, ferrous sulfate compared with ir
77 uction and its great value in distinguishing iron deficiency anemia from the anemia of chronic diseas
78 lavin deficiency (from 60% to 6%; P<0.0001), iron deficiency anemia (from 35% to 15%; P<0.007), and a
81 n Southeast Asia, Thalassemia trait (TT) and iron deficiency anemia (IDA) are the two most common ane
84 tinal workup fails to establish the cause of iron deficiency anemia (IDA) in a substantial proportion
85 Routine screening and supplementation for iron deficiency anemia (IDA) in asymptomatic, nonanemic
86 The actual prevalence of anemia, ID, and iron deficiency anemia (IDA) in California remains uncle
95 The prevalence of iron deficiency (ID) and iron deficiency anemia (IDA) was 10-32% and 2-5%, respec
96 uate the prevalence of iron deficiency (ID), iron deficiency anemia (IDA), and other measures of iron
97 based on iron biomarker concentrations were iron deficiency anemia (IDA), anemia of inflammation (AI
100 deficiency (ID; diminished iron stores) and iron deficiency anemia (IDA; poor iron stores and dimini
103 ts and young children in Sub-Saharan Africa, iron-deficiency anemia (IDA) is common, and many complem
104 ferritin, which was suitable for identifying iron-deficiency anemia (IDA), but less reliable than ser
105 of severe complications that may arise from iron deficiency anemia if it is not recognized and treat
106 eraction at the locus, impacting the risk of iron deficiency anemia in African Americans with specifi
111 preventing and treating iron deficiency and iron deficiency anemia in infants and young children.
114 appropriate for estimating the prevalence of iron deficiency anemia in poor Jamaican girls 13-14 y of
116 for pregnant women or routine screening for iron deficiency anemia in pregnant women or young childr
117 lance of benefits and harms of screening for iron deficiency anemia in pregnant women to prevent adve
119 o estimate prevalence of iron deficiency and iron deficiency anemia in the Jamaican girls: 7.6% and 4
122 e serine protease mutated in iron-refractory iron deficiency anemia, inhibits hepcidin expression by
133 eed on blood in the small intestine, causing iron-deficiency anemia, malnutrition, growth and develop
135 oglobin level, iron-deficiency (IDA) and non-iron-deficiency anemia (NIDA), and inflammatory markers.
138 women who are malnourished, have symptoms of iron deficiency anemia, or have special hematologic cond
139 The other major modes of presentation are iron-deficiency anemia, osteoporosis, screening of famil
141 during early development in iron-refractory iron deficiency anemia patients, who present with microc
143 Although much of the ethnic disparity in iron deficiency anemia remains unexplained, factors asso
144 black woman has heavy menstrual bleeding and iron-deficiency anemia.She reports nocturia and urinary
145 ntually the microcytic hypochromic anemia or iron deficiency anemia that is the clinical hallmark of
146 thickness (CT) in the eyes of patients with iron-deficiency anemia, the most common form of the anem
147 nd clinical trial in adults with NDD-CKD and iron deficiency anemia to compare the safety and efficac
148 pcidin levels for diagnosing iron-refractory iron deficiency anemia to global health applications suc
149 f iron status across the full continuum from iron deficiency anemia to iron deficiency to iron replet
150 common symptoms of irritable bowel syndrome, iron deficiency anemia, unexplained arthritis, and even
151 IDA) is a familial disorder characterized by iron deficiency anemia unresponsive to oral iron treatme
152 , and iron supplement use, the prevalence of iron deficiency anemia was 2.3 times higher in Mexican A
153 old income) >3.0, however, the prevalence of iron deficiency anemia was 2.6 +/- 0.9% in Mexican Ameri
158 ldbearing age were iron deficient; of these, iron deficiency anemia was found in 3% and 2% to 5%, res
161 Post administration of FCM, correction of iron deficiency anemia was observed with improved mean h
163 alence rates of anemia, iron deficiency, and iron deficiency anemia were significantly lower after 24
164 1%, 32.7%) to 6.8% (95% CI: 4.2%, 9.3%), and iron deficiency anemia, which was 6.2% (95% CI: 3.0%, 9.
165 uch that newborn homozygous Pcm mice exhibit iron deficiency anemia with increased duodenal Fpn1 expr
167 ted with reduced risk of iron deficiency and iron deficiency anemia without a concomitant increase in
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