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1 droxyurea, aphidicolin, or etoposide induced irreversible cell cycle arrest after several population
2 e interferon (IFN)-mediated and p53-mediated irreversible cell cycle arrest and apoptosis as part of
3                                          The irreversible cell cycle arrest and apoptosis induced by
4 nce mechanisms, which ultimately induces the irreversible cell cycle arrest and apoptosis.
5 n of S- or G2-phase checkpoints resulting in irreversible cell cycle arrest and cell death.
6 hanism of cell death seemed to be due to the irreversible cell cycle arrest at the G2-M checkpoint, r
7           p21Waf1/Cip1/Sdi1 induction caused irreversible cell cycle arrest in both G1 and G2/M, and
8 nsformation, it can paradoxically promote an irreversible cell cycle arrest known as oncogene-induced
9 fe span, at the end of which they undergo an irreversible cell cycle arrest known as replicative sene
10  that observed for p53 alone and produced an irreversible cell cycle arrest that displayed features o
11            Cellular senescence is a state of irreversible cell cycle arrest that has been documented
12            Cellular senescence is a state of irreversible cell cycle arrest that has been involved in
13        Cellular senescence is a programme of irreversible cell cycle arrest that normal cells undergo
14 xpression of VentX in cancer cells caused an irreversible cell cycle arrest with a typical senescence
15 tion is the initiation and maintenance of an irreversible cell cycle arrest with the complex involvem
16 re of centriole duplication that produced an irreversible cell cycle arrest within a few divisions.
17              Cellular senescence, a state of irreversible cell cycle arrest, is thought to help prote
18 ve senescence, an end stage characterized by irreversible cell cycle arrest, multiple genetic and fun
19 wed these cells to evade differentiation and irreversible cell cycle arrest.
20  is a signal transduction program leading to irreversible cell cycle arrest.
21 tal muscle, Rb has been reported to regulate irreversible cell cycle exit and muscle-specific transcr
22  Differentiation is a coordinated process of irreversible cell cycle exit and tissue-specific gene ex
23     We conclude that HES1 safeguards against irreversible cell cycle exit both during normal cellular
24 mal cells in culture leads to senescence, an irreversible cell cycle exit characterized by biochemica
25 oci (SAHF), are thought to contribute to the irreversible cell cycle exit in many senescent cells by
26 umor-suppressive process characterized by an irreversible cell cycle exit, a unique morphology, and e
27 ordered proteolytic events are essential for irreversible cell cycle progression and the robustness o
28                                              Irreversible cell cycle withdrawal occurs as normal kera
29 sults in terminal cell divisions followed by irreversible cell cycle withdrawal of hemoglobinized cel
30 cle regeneration after injury is limited by "irreversible" cell cycle exit.
31     Cellular senescence refers to a state of irreversible cell-cycle arrest combined with the secreti
32 enescence has historically been viewed as an irreversible cell-cycle arrest mechanism that acts to pr
33                             Senescence is an irreversible cell-cycle arrest that has a crucial role b
34 c changes in gene expression that trigger an irreversible cell-cycle arrest.
35 teins encompasses key regulators proximal to irreversible cell damage.
36 ) is a critical determinant of the extent of irreversible cell damage.
37 e cells did not co-localise with a marker of irreversible cell death (TUNEL).
38 production of PAP I led to slower growth and irreversible cell death.
39 non-selective cation channel, which leads to irreversible cell depolarization and unregulated Ca2+ en
40 (the reversibly immortalized IDH4 cells), or irreversible cell differentiation (HL60 promyelocytic le
41 ukaryotes can thus control low rates of near irreversible cell fate decisions through a balancing act
42                                              Irreversible cell growth arrest, a process termed cellul
43 in PrECs contribute to senescence-associated irreversible cell growth arrest.
44 t, indicating that inhibition was not due to irreversible cell injury and suggesting that other signa
45 ory response that can result in necrosis and irreversible cell injury to both local vascular endothel
46 the MPT is causally linked to the genesis of irreversible cell injury with TNF.
47                   Electric fields can induce irreversible cell membrane pores in direct relation to c
48                                  As death is irreversible, cells must continually integrate developme
49 armacological effects of a single dose of an irreversible cell permeant general (nonselective) caspas
50            Senescence is characterized by an irreversible cell proliferation arrest.
51 n has ceased, indicating that this is a true irreversible cell reprogramming event.
52 influences the transition from reversible to irreversible cell shape change, which defines the onset

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