戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 a significant risk factor for postinfectious irritable bowel and chronic fatigue syndromes.
2 ructive pathologies of the two main forms of irritable bowel disease (IBD), ulcerative colitis (UC),
3 individuals, and those with colon cancer and irritable bowel disease (IBD), we demonstrated that CD4C
4 rylation was observed in colonic mucosa from irritable bowel disease patients.
5 tive protease pathway in the pathogenesis of irritable bowel disease.
6 centage of individuals with gastrointestinal/irritable bowel disease.
7 d may offer a therapeutic avenue in treating irritable bowel disease.
8 n who do not fit a specific disorder such as irritable bowel, functional dyspepsia, or abdominal migr
9                           The prevalences of irritable bowel syndrome (39.4%) by Rome III criteria an
10 ), 6.78% in Crohn's disease (4/59), 5.82% in irritable bowel syndrome (51/877), and 4.9% in the remai
11 valences (per 100 residents) were stable for irritable bowel syndrome (8.3% and 11.4%, respectively)
12 subjects (aged 24-61 y, 6 men) with NCGS and irritable bowel syndrome (based on Rome III criteria), b
13 ota of patients with constipated-predominant irritable bowel syndrome (C-IBS) displays chronic dysbio
14 (FC), Gastrointestinal Symptoms Rating Scale-Irritable Bowel Syndrome (GSRS-IBS) and Hospital Anxiety
15 he Gastrointestinal Symptom Rating Scale for Irritable Bowel Syndrome (GSRS-IBS) into German and to e
16 red by Gastrointestinal Symptom Rating Scale Irritable Bowel Syndrome (GSRS-IBS) version.
17  also collected from 151 patients with CD or irritable bowel syndrome (IBS) (controls).
18 ed by the intestinal mucosa of patients with irritable bowel syndrome (IBS) affect the function of en
19                                              Irritable bowel syndrome (IBS) affects 7% to 21% of the
20 tudies have shown an increased prevalence of irritable bowel syndrome (IBS) after acute gastroenterit
21 patients (63.8%) had a previous diagnosis of irritable bowel syndrome (IBS) and 23 (28.8%) had one of
22 es from patients of colorectal cancer (CRC), irritable bowel syndrome (IBS) and controls to be run th
23         We investigated the role of TRPV1 in irritable bowel syndrome (IBS) and evaluated if an antag
24  symptoms, which occasionally mimic those of Irritable Bowel Syndrome (IBS) and Fibromyalgia Syndrome
25  hypnotherapy (HT) is effective in pediatric irritable bowel syndrome (IBS) and functional abdominal
26 tious gastroenteritis increases the risk for irritable bowel syndrome (IBS) and functional dyspepsia
27 ists are effective in treating patients with irritable bowel syndrome (IBS) and have anxiolytic effec
28                                              Irritable bowel syndrome (IBS) and inflammatory bowel di
29                   Abuse history is common in irritable bowel syndrome (IBS) and is associated with gr
30 Very few studies report on the prevalence of irritable bowel syndrome (IBS) and its correlates in the
31            Women have a higher prevalence of irritable bowel syndrome (IBS) and possible differences
32 nning to shape a pathophysiological model of irritable bowel syndrome (IBS) and related functional ga
33 cally divergent, ulcerative colitis (UC) and irritable bowel syndrome (IBS) are both associated with
34 isease (GERD), functional dyspepsia (FD) and irritable bowel syndrome (IBS) are common functional gas
35 healthy children and pediatric patients with irritable bowel syndrome (IBS) are not well defined.
36         Approximately 1 in ten patients with irritable bowel syndrome (IBS) believe their IBS began w
37 cture is used by patients as a treatment for irritable bowel syndrome (IBS) but the evidence on effec
38 pathophysiology, diagnosis, and treatment of irritable bowel syndrome (IBS) convened to audit the cur
39 elationship between giardiasis diagnosis and irritable bowel syndrome (IBS) diagnosis.
40  suggestive of functional dyspepsia (FD) and irritable bowel syndrome (IBS) frequently overlap with t
41 unctional gastrointestinal disorders such as irritable bowel syndrome (IBS) has shifted fundamentally
42                                Patients with irritable bowel syndrome (IBS) have high surgical rates.
43             Only a fraction of patients with irritable bowel syndrome (IBS) have increased perceptual
44                                Patients with irritable bowel syndrome (IBS) have increased postprandi
45 odify pain end points in clinical trials for irritable bowel syndrome (IBS) highlights the knowledge
46 care consumption for patients diagnosed with Irritable Bowel Syndrome (IBS) in primary and secondary
47                                              Irritable bowel syndrome (IBS) is a chronic functional g
48                                              Irritable Bowel Syndrome (IBS) is a chronic/common condi
49                                              Irritable Bowel Syndrome (IBS) is a common condition cha
50                                              Irritable bowel syndrome (IBS) is a common functional ga
51                                              Irritable bowel syndrome (IBS) is a common gastrointesti
52                                              Irritable bowel syndrome (IBS) is a common gastrointesti
53                                              Irritable Bowel Syndrome (IBS) is a functional somatic s
54                                              Irritable bowel syndrome (IBS) is a gut-brain disorder i
55                                              Irritable bowel syndrome (IBS) is among the most common
56                           BACKGROUND & AIMS: Irritable bowel syndrome (IBS) is associated with intest
57                                              Irritable bowel syndrome (IBS) is characterized by alter
58                                              Irritable bowel syndrome (IBS) is more common in patient
59                                              Irritable bowel syndrome (IBS) is one of the most common
60                                              Irritable bowel syndrome (IBS) is subtyped as IBS with d
61 f psychosocial disturbances with more severe irritable bowel syndrome (IBS) is well recognized.
62                                Patients with irritable bowel syndrome (IBS) often relate symptoms to
63                                Patients with Irritable Bowel Syndrome (IBS) often relate their sympto
64 role of the microbiota in the development of irritable bowel syndrome (IBS) only recently has been co
65 ) signaling pathways have been implicated in irritable bowel syndrome (IBS) pathophysiology.
66 lay a role in central pain amplification and irritable bowel syndrome (IBS) pathophysiology.
67  HPA axis response to a visceral stressor in irritable bowel syndrome (IBS) patients and healthy cont
68 efore aversive pelvic visceral distention in irritable bowel syndrome (IBS) patients and their possib
69                 Visceral hypersensitivity in irritable bowel syndrome (IBS) patients has been documen
70  Syndrome (FMS) is a frequent comorbidity in Irritable Bowel Syndrome (IBS) patients with a higher fu
71                      This study investigated Irritable Bowel Syndrome (IBS) prevalence in a sample of
72                             The diagnosis of irritable bowel syndrome (IBS) relies on symptom-based c
73                                              Irritable bowel syndrome (IBS) remains an incompletely u
74                       The pathophysiology of irritable bowel syndrome (IBS) remains enigmatic; abnorm
75 ll but significant subgroup of patients with irritable bowel syndrome (IBS) report a sudden onset of
76                                Patients with irritable bowel syndrome (IBS) seen by a gastroenterolog
77 sorbed fermentable carbohydrates can provoke irritable bowel syndrome (IBS) symptoms by escaping abso
78 acy of psychological treatments for reducing irritable bowel syndrome (IBS) symptoms, the mechanisms
79 suspected food intolerances in patients with irritable bowel syndrome (IBS) using confocal laser endo
80 entable carbohydrates may induce symptoms of irritable bowel syndrome (IBS) via unclear mechanisms.
81 entable carbohydrates may induce symptoms of irritable bowel syndrome (IBS) via unclear mechanisms.
82 ept study for the treatment of patients with irritable bowel syndrome (IBS) with constipation (IBS-C)
83 st, which has been approved for treatment of irritable bowel syndrome (IBS) with constipation in wome
84 ciated with colonic transit in patients with irritable bowel syndrome (IBS) with diarrhea.
85  treatments are needed for patients who have irritable bowel syndrome (IBS) with diarrhea.
86 showed the benefit of a 5HT(3) antagonist in irritable bowel syndrome (IBS) with diarrhoea (IBS-D) an
87 of PI sequelae among exposed was as follows: irritable bowel syndrome (IBS), 3.0; dyspepsia, 1.8; con
88  and male healthy subjects and patients with irritable bowel syndrome (IBS), a common chronic abdomin
89 ve been implicated in the pathophysiology of irritable bowel syndrome (IBS), a visceral pain syndrome
90 the most bothersome symptom by patients with irritable bowel syndrome (IBS), and actual distention ma
91  including inflammatory bowel disease (IBD), irritable bowel syndrome (IBS), and chronic constipation
92 s are abnormal in the ileum of patients with irritable bowel syndrome (IBS), and whether any abnormal
93 d genetic factors contribute to variation in irritable bowel syndrome (IBS), anxiety and depression.
94 ND & AIMS: Probiotics can reduce symptoms of irritable bowel syndrome (IBS), but little is known abou
95 ssants are effective in patients with severe irritable bowel syndrome (IBS), but the cost-effectivene
96 f these genetic variations with subgroups of irritable bowel syndrome (IBS), functional abdominal pai
97                                              Irritable bowel syndrome (IBS), functional bloating, fun
98 iated with three other functional disorders; irritable bowel syndrome (IBS), functional dyspepsia (FD
99  the area of functional GI syndromes such as irritable bowel syndrome (IBS), functional dyspepsia, an
100 ose was to evaluate the overlap frequency of irritable bowel syndrome (IBS), gastroesophageal reflux
101                             In patients with irritable bowel syndrome (IBS), pain amplification and h
102                                              Irritable bowel syndrome (IBS), which affects 11% to 14%
103                                           In irritable bowel syndrome (IBS), which is the most common
104 l gastrointestinal symptoms in patients with irritable bowel syndrome (IBS), yet there is limited evi
105 testinal microbiota and clinical features of irritable bowel syndrome (IBS).
106 pulation each year, and is a risk factor for irritable bowel syndrome (IBS).
107  have important roles in the pathogenesis of irritable bowel syndrome (IBS).
108 the pathophysiology of anxiety disorders and irritable bowel syndrome (IBS).
109  studies of existing diagnostic criteria for irritable bowel syndrome (IBS).
110 microbiota might also play a similar role in irritable bowel syndrome (IBS).
111 relieve symptoms for patients suffering from irritable bowel syndrome (IBS).
112 versus usual care alone for the treatment of Irritable Bowel Syndrome (IBS).
113 important role in the pathophysiology of the irritable bowel syndrome (IBS).
114  used to treat patients with nonconstipating irritable bowel syndrome (IBS).
115 l nervous system is altered in patients with irritable bowel syndrome (IBS).
116  measure patient-reported outcomes (PROs) in irritable bowel syndrome (IBS).
117 the progression and evaluate the severity of irritable bowel syndrome (IBS).
118 he development of drugs for the treatment of irritable bowel syndrome (IBS).
119 nities to develop new therapeutic agents for irritable bowel syndrome (IBS).
120 y be important in the pathophysiology of the irritable bowel syndrome (IBS).
121  Visceral hypersensitivity is one feature of irritable bowel syndrome (IBS).
122 n of 5-HT is involved in the pathogenesis of irritable bowel syndrome (IBS).
123 er-related differences in FGID, particularly irritable bowel syndrome (IBS).
124 ted to reduce symptoms in some patients with irritable bowel syndrome (IBS).
125 ociated with abdominal pain in patients with irritable bowel syndrome (IBS).
126 testinal microbiota and clinical features of irritable bowel syndrome (IBS).
127 sing diet in the management of patients with irritable bowel syndrome (IBS).
128  effective in the treatment of patients with irritable bowel syndrome (IBS).
129 agonists might be involved in development of irritable bowel syndrome (IBS).
130  by mutations in SCN5A also have symptoms of irritable bowel syndrome (IBS).
131 in, constipation, and bloating; diagnoses of irritable bowel syndrome (IBS); and tegaserod prescripti
132 alence of celiac disease among patients with irritable bowel syndrome (IBS); few data are available w
133 fies the FBDs into five distinct categories: irritable bowel syndrome (IBS); functional constipation
134 and polyols (FODMAPs) exacerbate symptoms of irritable bowel syndrome (IBS); however, their mechanism
135 red an important pathophysiologic symptom in irritable bowel syndrome (IBS); previous gastrointestina
136  compared to a milk product in subjects with irritable bowel syndrome (IBS, n = 28).
137 in the treatment of constipation predominant irritable bowel syndrome (IBS-C), a highly prevalent dis
138 ogenetics of CDC in constipation-predominant irritable bowel syndrome (IBS-C).
139           Patients with diarrhea-predominant irritable bowel syndrome (IBS-D) could benefit from a gl
140      Some patients with diarrhea-predominant irritable bowel syndrome (IBS-D) may have undiagnosed ce
141 ned diarrhea, including diarrhea-predominant irritable bowel syndrome (IBS-D).
142 efficacy and safety for diarrhea-predominant irritable bowel syndrome (IBS-D).
143 ouse model that reproduces major features of irritable bowel syndrome (long-lasting colon hypersensit
144 hat patients with symptoms of nonconstipated irritable bowel syndrome (NC-IBS) undergo testing for ce
145 04-1.1), diarrhea (OR, 53; 95% CI, 6.1-471), irritable bowel syndrome (OR, 4.8; 95% CI, 1.6-14), chol
146                              Post-infectious irritable bowel syndrome (PI-IBS) is a common gastrointe
147 he role of gut microbiota in post-infectious irritable bowel syndrome (PI-IBS) is convincing.
148 ith increased rate ratios (RRs) for incident irritable bowel syndrome (RR, 6.1; 95% confidence interv
149 o an online database called Transcriptome of Irritable Bowel Syndrome (TIBS).
150             Of them, 305 (16.5%) had AP-FGD [irritable bowel syndrome = 91(4.9%), functional dyspepsi
151 who are in remission and those who developed irritable bowel syndrome after enteric infection continu
152 in the treatment of constipation-predominant irritable bowel syndrome and chronic constipation.
153 ate the persistence, prevalence, and risk of irritable bowel syndrome and chronic fatigue 6 years aft
154 ing is associated with an increased risk for irritable bowel syndrome and chronic fatigue 6 years lat
155     Clinical studies show that patients with irritable bowel syndrome and colonic diseases frequently
156                         Although symptomatic irritable bowel syndrome and Crohn's disease patients ha
157 nic gastrointestinal diseases-liver disease, irritable bowel syndrome and dyspepsia, and inflammatory
158 associated with diarrheal conditions such as irritable bowel syndrome and enteric infections.
159  gastrointestinal tract disorders, including irritable bowel syndrome and gastroesophageal reflux dis
160 with postinfectious complications, including irritable bowel syndrome and Guillain-Barre syndrome.
161 ter, are at increased risk of postinfectious irritable bowel syndrome and inflammatory bowel disease
162                   Colon disorders, including irritable bowel syndrome and inflammatory bowel disease,
163 sit and gut microbial communities, including irritable bowel syndrome and inflammatory bowel disease.
164  long-term effects, including postinfectious irritable bowel syndrome and inflammatory bowel disease.
165 ensal gut flora in such common conditions as irritable bowel syndrome and inflammatory bowel disease.
166                                         Both irritable bowel syndrome and inflammatory bowel diseases
167 , appeared to be most genetically similar to irritable bowel syndrome and most environmentally simila
168 stion that SIBO may be a causative factor in irritable bowel syndrome and of its constituent symptoms
169  successfully treat constipation-predominant irritable bowel syndrome and recent studies show that ex
170 (EPI) confound interpretation of findings in irritable bowel syndrome and severe renal insufficiency.
171 al gastrointestinal disorders, most commonly irritable bowel syndrome but also other functional and o
172 nalities with inflammatory bowel disease and irritable bowel syndrome but were focused on associative
173 ely prescribing antibiotics to patients with irritable bowel syndrome can be endorsed.
174        This activation may have relevance to irritable bowel syndrome characterized by lower pain thr
175  in small intestinal bacterial overgrowth in irritable bowel syndrome continues, the utility and spec
176    While interest in bacterial overgrowth in irritable bowel syndrome continues, the utility and spec
177      In the exposed group, the prevalence of irritable bowel syndrome decreased by 6.7% (RR, 0.85 [95
178     The peripheral component is prominent in irritable bowel syndrome developing after infection (pos
179 ease in IBD risk for persons with a previous irritable bowel syndrome diagnosis.
180 opsies were also taken from 16 patients with irritable bowel syndrome diarrhea who comprised the cont
181 antibiotics and concurrently, postinfectious irritable bowel syndrome has been associated with a long
182                               Traditionally, irritable bowel syndrome has been considered to be a dis
183                                              Irritable bowel syndrome has been referred to as a funct
184                 The study of post-infectious irritable bowel syndrome has revealed the importance of
185  treatment options for diarrhoea-predominant irritable bowel syndrome have had not very promising res
186 valuate the effectiveness of acupuncture for irritable bowel syndrome in primary care when provided a
187 ment of the diarrhea-predominant form of the irritable bowel syndrome in women suggests the importanc
188     Underlying mechanisms that could lead to irritable bowel syndrome include genetic factors (most n
189         It has suggested novel approaches to irritable bowel syndrome including studies of serotonin
190 w we challenge the widely accepted view that irritable bowel syndrome is an unexplained brain-gut dis
191                                              Irritable bowel syndrome is characterized by altered sen
192 ecent studies have overthrown the dogma that irritable bowel syndrome is characterized by no abnormal
193                                              Irritable bowel syndrome is classified as a functional g
194 toms and the strong overlap between GERD and irritable bowel syndrome is due to the influence of NERD
195  noxious stimulation in the context of human irritable bowel syndrome is questionable.
196  interventions are enjoying a renaissance in irritable bowel syndrome management.
197                               Postinfectious irritable bowel syndrome may occur in 3% to 17% of patie
198 orphic nature of some pelvic diseases, e.g., irritable bowel syndrome or cystitis.
199                                              Irritable bowel syndrome patients form a heterogeneous g
200 een the control group and Crohn's disease or irritable bowel syndrome patients in terms Blastocystis
201 ersensitivity of primary afferent neurons in irritable bowel syndrome patients.
202                              Acupuncture for irritable bowel syndrome provided an additional benefit
203                                              Irritable bowel syndrome refers to abdominal discomfort
204                                              Irritable bowel syndrome remains an incompletely underst
205          The role of bacterial overgrowth in irritable bowel syndrome requires further study.
206                                    Identical irritable bowel syndrome symptoms are probably due to di
207 ortance of low-grade inflammation in causing irritable bowel syndrome symptoms.
208                              Noninflammatory irritable bowel syndrome was induced by intracolonic ins
209                            233 patients with irritable bowel syndrome were randomly allocated to eith
210 n important clinical feature associated with irritable bowel syndrome which in some patients has been
211 ion, 15 with functional bloating, and 3 with irritable bowel syndrome with alternating bowel habits)
212                                              Irritable bowel syndrome with constipation (IBS-C) affec
213                                Patients with irritable bowel syndrome with constipation (IBS-C) and p
214  GC-C) that reduces symptoms associated with irritable bowel syndrome with constipation (IBS-C).
215 C) includes functional constipation (FC) and irritable bowel syndrome with constipation (IBS-C).
216 ith functional intestinal disorders (27 with irritable bowel syndrome with constipation, 15 with func
217 ed by >2 symptoms of chronic constipation or irritable bowel syndrome with constipation, and with >2
218 tion subtypes of functional constipation and irritable bowel syndrome with constipation.
219                           Some patients with irritable bowel syndrome with diarrhea (IBS-D) have inte
220 abdominal pain and diarrhea in patients with irritable bowel syndrome with diarrhea (IBS-D) without c
221     Chronic idiopathic constipation (CC) and irritable bowel syndrome with predominant constipation (
222                                    The term "irritable bowel syndrome" was used to search Clinical Ev
223  chronic fatigue syndrome' and 'Dopamine and irritable bowel syndrome' was carried out until April 20
224 tic syndromes', 'Chronic fatigue syndrome', 'Irritable bowel syndrome', 'Fibromyalgia', 'Dopamine and
225  developing after infection (post-infectious irritable bowel syndrome) and this has proved a profitab
226 rome, reactive arthritis, and postinfectious irritable bowel syndrome) contribute considerably to the
227 scribed (e.g. inflammatory bowel disease and irritable bowel syndrome).
228 e into the development of new approaches for irritable bowel syndrome, a multifactorial disorder for
229 ysiology, etiology, pathogenesis, diagnosis, irritable bowel syndrome, and IBS.
230 broid food poisoning, histamine intolerance, irritable bowel syndrome, and inflammatory bowel disease
231   These include inflammatory bowel diseases, irritable bowel syndrome, and metabolic (i.e. obesity, n
232 robiota, such as inflammatory bowel disease, irritable bowel syndrome, and metabolic syndrome, to nam
233 owel diseases, celiac disease, food allergy, irritable bowel syndrome, and--more recently recognized-
234 festations of celiac disease that include an irritable bowel syndrome, anemia, osteoporosis, neurolog
235 ders, such as inflammatory bowel disease and irritable bowel syndrome, are associated with exaggerate
236 disorders (cardiovascular disease, diabetes, irritable bowel syndrome, asthma, and others).
237  to human physiology and to diseases such as irritable bowel syndrome, autism, anxiety, depression, a
238 EC has been implicated in the development of irritable bowel syndrome, but this remains to be confirm
239 terpersonal therapy, and antidepressants for irritable bowel syndrome, chronic fatigue syndrome, and
240 , management, and role in conditions such as irritable bowel syndrome, chronic fatigue, and autoimmun
241                       Chronic pain syndromes irritable bowel syndrome, chronic pelvic pain, and fibro
242 ur findings link duplications in TPSAB1 with irritable bowel syndrome, cutaneous complaints, connecti
243 expanded from 1946 to December 2014 for IBS, irritable bowel syndrome, diet, treatment, and therapy.
244 ted with chronic prostatitis/CPPS, including irritable bowel syndrome, fibromyalgia, and chronic fati
245 r directly co-morbid somatic disorders, e.g. irritable bowel syndrome, fibromyalgia, or migraine.
246   This issue provides a clinical overview of irritable bowel syndrome, focusing on diagnosis, treatme
247 d in the setting of differentiating IBD from irritable bowel syndrome, for grading inflammation, to d
248 nter placebo-controlled trial, children with irritable bowel syndrome, functional abdominal pain, or
249 female patients with moderate to severe FBD (irritable bowel syndrome, functional abdominal pain, pai
250  mechanisms may predispose the individual to irritable bowel syndrome, gastroesophageal reflux diseas
251 orders including inflammatory bowel disease, irritable bowel syndrome, infectious and antibiotic-asso
252 ith active UC, inactive UC, Crohn's disease, irritable bowel syndrome, infectious colitis, and micros
253 ute pancreatitis, bacterial gastroenteritis, irritable bowel syndrome, inflammatory bowel disease, he
254 nfection, small bowel intestinal overgrowth, irritable bowel syndrome, inflammatory bowel disease, po
255 , including Clostridium difficile infection, irritable bowel syndrome, inflammatory bowel diseases, i
256                                              Irritable bowel syndrome, interstitial cystitis, and oth
257  and of patients with the common symptoms of irritable bowel syndrome, iron deficiency anemia, unexpl
258       In women with constipation-predominant irritable bowel syndrome, linaclotide 1000 microg once d
259  fibromyalgia, generalized anxiety disorder, irritable bowel syndrome, migraine, obsessive-compulsive
260 hea and colitis, inflammatory bowel disease, irritable bowel syndrome, necrotizing enterocolitis, and
261 vergrowth is one of the causes suggested for irritable bowel syndrome, particularly for the diarrhoea
262 mens from patients with UC, Crohn's disease, irritable bowel syndrome, sporadic colorectal cancer, or
263 ents meeting current diagnostic criteria for irritable bowel syndrome, therapeutic approaches shown t
264 liaison psychiatric setting in patients with irritable bowel syndrome, where positive benefits have b
265                                              Irritable bowel syndrome, which affects 5-10% of the pop
266 ctional bowel disorders (FBDs), particularly irritable bowel syndrome, with the objective of elucidat
267  abdominal pain experienced by patients with irritable bowel syndrome, yet the molecules that confer
268 est drug to be approved for the treatment of irritable bowel syndrome-diarrhoea is rifaximin, which w
269  findings, are nonspecific, and can simulate irritable bowel syndrome.
270 es such as fibromyalgia, chronic fatigue and irritable bowel syndrome.
271 cupuncture is a cost-effective treatment for irritable bowel syndrome.
272 osed inflammatory bowel disease and 877 with irritable bowel syndrome.
273 be cost-effective for those with more severe irritable bowel syndrome.
274  other involving children suffering from the irritable bowel syndrome.
275 tion between infection with Blastocystis and irritable bowel syndrome.
276 rted polymorphisms in the pathophysiology of irritable bowel syndrome.
277 e clearly established a genetic component in irritable bowel syndrome.
278 abdominal pain and cramping in patients with irritable bowel syndrome.
279 tem, such as chemotherapy-induced emesis and irritable bowel syndrome.
280 ntirely functional, such as in some cases of irritable bowel syndrome.
281 s may have a role in nondiarrhea predominant irritable bowel syndrome.
282  - have developing roles in the treatment of irritable bowel syndrome.
283 overgrowth may be important for treatment of irritable bowel syndrome.
284 in is effective in preventing postinfectious irritable bowel syndrome.
285 ntary and alternative medicine therapies for irritable bowel syndrome.
286 eral hypersensitivity is a characteristic of irritable bowel syndrome.
287 possible therapies for certain patients with irritable bowel syndrome.
288 h mucosa and blood have been demonstrated in irritable bowel syndrome.
289 ses substantial morbidity and postinfectious irritable bowel syndrome.
290  Patients often have a previous diagnosis of irritable bowel syndrome.
291 ations in sensory processing associated with irritable bowel syndrome.
292 disorder, multiple chemical sensitivity, and irritable bowel syndrome.
293  ultimately provide a cure for patients with irritable bowel syndrome.
294 NR1) have been associated with some forms of irritable bowel syndrome.
295 ergrowth participates in the pathogenesis of irritable bowel syndrome.
296 hanced toxin sensitivity in a mouse model of irritable bowel syndrome.
297 psy, ataxia, pain, arrhythmia, myotonia, and irritable bowel syndrome.
298 y, in 36 women with constipation-predominant irritable bowel syndrome; colonic transit was normal in
299 ed to control (P<.001) and disease controls (irritable bowel syndrome; P<.001; rheumatoid arthritis;
300 searched by using medical subject headings ("irritable bowel syndrome;" "colonic diseases, functional

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top