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1 e, an understanding of fear-extinction learning in children is essential for (1) detecting the source of developmental su
2 We show here that while the central DNA binding domain is essential for anchoring at parS, this interaction is not r
4 Rather, our results demonstrate that the PVT is essential for balancing conflicting behavioral tendencies
5 ar understanding of the clinical benefits relative to costs is essential for both appropriate clinical use and rational p
6 ssociated noncoding intergenic RNA)-Nkx2.1 gene duplex that is essential for buffering Nkx2.1 expression, lung epithelial
7 Collectively, these results support that Usp36 is essential for cell and organism viability because of its r
10 a replicates intracellularly in macrophages, a process that is essential for disease progression.
13 f the compounds as the T3SS needle pore protein EspD, which is essential for effector protein translocation into host cel
14 Mitophagy is a cellular quality-control pathway, which is essential for elimination of unhealthy mitochondria.
15 tr1 functions as a Cu(+) transporter for Cu acquisition and is essential for embryonic development, a homologous protein,
16 s, our results suggest that coordinated firing during sleep is essential for establishing sparse activation patterns that
17 ing agents, and characterizing nanoparticle biodistribution is essential for evaluating their efficacy and safety.
18 involved in updating the memory about the perturbation that is essential for forelimb motor adaptation.
19 tability has been studied, recent findings indicate that it is essential for functions apart from mitochondrial bioenerge
21 Furthermore, the enzymatic activity of LSD1 is essential for H3K9me2 demethylation at cell cycle gene loc
22 Med14 facilitate a large-scale Mediator rearrangement that is essential for holoenzyme formation.
23 resents the principal PAT for Glut4 and that this mechanism is essential for insulin-regulated glucose homeostasis.
26 wann cell (SC) myelination in the peripheral nervous system is essential for motor function, and uncontrolled SC prolifer
27 d a polyketide synthase (PKS) gene PfmaE and showed that it is essential for multicellular conidial pigmentation and deve
28 for patients with relapsed/refractory Hodgkin lymphoma (HL) is essential for optimizing therapy with risk-adapted approac
29 A region encompassing KOW4 and KOW5 of Spt5 is essential for pausing, and mutations in KOW5 specifically
30 ell death (RCD) plays critical roles during development and is essential for plant-specific responses to abiotic and biot
31 t that maintaining physiological levels of mTORC1 signaling is essential for postnatal tendon development and maturation.
37 So far, it has been assumed that the pilin domain is essential for the allosteric propagation within the lectin
39 lated events in the periphery can influence brain processes is essential for the development of therapies to prevent or t
40 in the developmental disorders Joubert and MORM syndromes, is essential for the function of the primary cilium and maint
41 domain at the C-terminus of Swc5, called Bucentaur (BCNT), is essential for the histone exchange activity of SWR, wherea
42 This strongly suggests that glycylation is essential for the homeostasis of primary cilia, which has
43 ort that alpha-SYN present in dopaminergic nigral afferents is essential for the normal cycling and maintenance of neural
44 oles in assembling the functional polymerase complex, which is essential for the replication and transcription of influen
46 nine rings confirm that the precise geometry of these rings is essential for this synergistic activity.
47 NT5A signaling mechanism comprising NF-kappaB and MMP7 that is essential for TNBC cell invasiveness, thereby providing im
49 ism using imaging presence of right ventricular dysfunction is essential for triage; however, comprehensive transthoracic
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